WK2-Wound Etiologies Part 1 Flashcards
What is the general pathophysiological mechanism of arterial insufficiency ?
AI wound is one that results from a lack of blood flow which deprives a certain area of oxygen
Can be due to: Trauma, acute embolisms, diabetes mellitus, rheumatoid arthritis, thromboangiitis ( Buerger’s disease), Arteriosclerosis and atherosclerosis.
What is the organization scheme of arteries from large arteries to vessels that actually permeate tissues ? and what are the 3 layers to the smallest organization of arteries ?
arteries > arterioles > capillaries
capillaries from outer most to innermost: tunica adventitia, tunica media, tunica intima
What is intermittent claudication ?
Intermittent claudication is pain due to poor tissue perfusion during activity, it typically stops within 1-5 minutes of ceasing activity, ~50% stenosis will result in claudication
the pain is usually distal to site of occlusion
What is ischemic rest pain ?
a step above intermittent claudication in where there is a burning pain at rest exacerbated with elevation.
What are the general characteristics of a wound caused by AI ?
severe pain unless masked by some comorbidity
found on the LE, distal toes, dorsal foot, areas of trauma
pale granulation tissue, shiny, loss of hair in area, anhydrous skin, yellow nails, no drainage, decreased pulse, cool temperature
What is the first line of testing for AI and VI ?
ABI, ankle brachial index
mmHg LE/mmHg UE
At what ABI should debridement begin to be clinically judged ?
.7
What is the general pathophysiological mechanism of venous insufficiency ?
condition where the veins, particularly in the LE have difficulties sending blood back to the heart,
due to sustained venous hypertension that can be due to incompetent veins, bicuspid valves failing to close, valve damage as in varicose veins
What are the two theories that describe how venous hypertension causes tissue damage
WBC trapping theory: WBCs gather in area and release things that damage walls due to high venous pressure
Fibrin cuff theory: distension makes veins leaky, leading to edema in intersitium
What are the general characteristics of VI ulcers ?
mild to moderate pain unless masked
found on medial malleolus, medial leg, or areas of trauma
irregular shape, glossy, coating, red ruddy wound bed, copious drainage
edema, hemosiderin staining
normal pulse, or decreased
normal to mild warmth
What is the general treatment for VI ulcers ?
Compression unless ABI is under .5
What is the average healing time of VI ulcers ?
8 weeks
How many weeks in should a referral for a VI ulcer be referred out ?
4 weeks
For VI ulcers, what ABI is a contraindication to compression ?
<0.5
How does a pressure injury occur ?
a pressure injury occurs due to excess pressure on the skin causing a breakage, typically over a bony prominence
can be common in those with spinal cord injuries, hospitalized patients, individuals in long term care facilities