week 9: pain assessment Flashcards

1
Q

pain

A

An unpleasant sensory and emotional experience

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2
Q

purpose of pain

A

Warns us of tissue damage, potential tissue damage
Elicits a reflex to keep injury damage at a minimum
Becomes bad if people continue to injure themselves; they become ill

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3
Q

multimodal analgesia

A

Preferred; several classes of medication + non-pharmacological treatments
Uses different treatments with different mode of actions (opioids, non-opioids, analgesics, anticonvulsants, hot-cold therapy, massages, stretching - medications not always the answer)

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4
Q

pain management strategies

A

Reduce pain to acceptable level as defined by the patient; though not always 100% possible
Involve patient, family, HCP’s in goal setting, strategies
Make sure you thoroughly assess what type of pain it is for appropriate treatment, don’t delay it

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5
Q

nociceptors

A

Pain-sensing nerve cells in the peripheral systems; mechanical (pressure/touch), thermal (hot/cold), and chemical types
They are activated by noxious (hurtful) stimuli, generates nerve impulses
Releases substances like substances P, prostaglandins, fast impulse

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6
Q

pain transmission process**

A
  • Transduction: Finger prick, stimuli activates nociceptors; releases substance P, prostaglandin, nerve impulse is generated
  • Transmission: Goes to dorsal nerve of spinal cord, then up to the brain
  • Perception: Brain interprets “Oh, there’s pain” - this is all very fast
  • Modulation: Brain tells you to do something about it, neurotransmitters to block impulse so you stop feeling pain
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7
Q

pain meds

A

Anti-inflammatory drugs: Decreases prostaglandin synthesis; can’t transmit
Local anaesthetic: Blocks nerve ability to generate an action potential, send messages
Opioids/Anticonvulsants/Antidepressants: Acts on the CNS

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8
Q

when pain isn’t properly relieved

A

Sympathetic NS, Fight or Flight activates:
Heart rate increases, BP, respiration is abnormal, GI slows, immune dysfunction
MSK tension, Nervous dysfunction
Psychological distress

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9
Q

how to address pain

A

At least once per shift for inpatients/once per visit for outpatients/home care
Before, During, After procedures
Following treatment to try to reduce pain (did your intervention do anything?)

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10
Q

OPQRSTUV

A
  • Onset:
    When did your pain start?
    Palliative/Provocative:
    What makes your pain better or worse?
  • Quality:
    How would you describe your pain? (Dull, Sharp, Throbbing, Burning, Aching, Electric)
    Region/Radiating:
    Where is the pain and does it travel anywhere?
  • Severity:
    How bad is your pain? (None,Mild,Moderate,Severe), How would you rate it from 0-10?
    (Using other inventories, pain assessment tools)
  • Timing:
    Is there a time of day when the pain is worse? Morning? Night? Activities? Lying still?
  • Understanding:
    What do you think is causing the pain?
  • Values:
    Any cultural, religious, personal/family beliefs about how you want to manage your pain?
    Any medications? (“Afraid of addiction”, “Afraid of cancer”)
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11
Q

if the patient is verbally/cognitively intact

A

Ask them if they’re in any pain
Family/caregiver report if they’re children or unable to report - may be unreliable

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12
Q

pain assessment tools

A

Brief Pain Inventory: Questionnaire about pain, location, type, effects
Numerical Rating Scale (0-10, 0 = no pain, 10 = worst pain imaginable)
Visual Analogue Scale (Mark along this line from No pain —————– Worst pain)
Descriptor Scale: No pain / Mild pain / Moderate pain / Severe pain / Excrutiating pain
Faces Scale: Wong Baker or Revised Faces scale - Point to the face representing your pain
There are other, more comprehensive scales as well

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13
Q

cognitively impaired individuals

A

They also feel pain - just have difficulty communicating pain in ways we can understand
Comprehensively assess physical, environmental factors
Get as much info from as many sources; non-verbal behaviours and vocalizations
Feldt Tool to assess pain in non-verbal

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14
Q

pain behaviours in nonverbal individuals: absence indicators

A

Flat affect
Decreased interactions
Decreased intake (eat/drink)
Altered sleep
Could mean anything; process of elimination

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15
Q

pain behaviours in nonverbal individuals: active indicators

A

Rocking
Negative vocalizations (moan/groan)
Frown/grimace
Noisy breathing
Irritable/Agitated

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16
Q

functional assessment/future goals

A

Walk through a day in their life; things they’re able to do/want to do
How does the pain interfere with lifestyle/daily living? Work? Physical limitations?
Fill out a pain diary - tends to not be reliable
Assess their sleep quality
Goals for present and future

17
Q

psychological impact

A

PHQ-9: Depression Screening
Resources: Psychology, Social Work, Counselling, Mental Health Services

18
Q

signs of neuropathic pain vs nociceptive pain

A

Hyperalgesia: Exaggerated pain response (ex., crying out after a light pinch)
Allodynia: Something that shouldn’t feel painful feels painful
Paraesthesia: Numbness/tingling

19
Q

acute pain

A

Physical sensations caused by injury - nociceptive pain
Usually lasts seconds to weeks and goes away after healing - transient
Abrupt onset and short duration

20
Q

chronic pain

A

Pain lasting longer than 3-6 months, or longer than typical healing time
Has no biological advantage like pain reflex normally does
Can result from diseases; diabetes, shingles, trauma, surgery, amputation
Causes disabilities; arthritis, avascular necrosis
Can even occur without known injury/disease; fibromyalgia without injury
HUGE psychological/depressive/poor sleep part with it - affects Quality of Life
Patient pulls away from activities

21
Q

nociceptive pain

A

Caused by the direct stimulation of peripheral nociceptors
Caused by tissue damage, inflammatory processes
Characteristics:
Localized (damage location = pain location)
Might diffuse if deeper structures/viscera are involved
“Aching, Sharp, Dull” - descriptors
Further Classifications:
Somatic:
Superficial Somatic: Skin invasion, ulceration, skin conditions
Deep Somatic: Bone, muscle, soft tissue
Visceral: Related to organ damage
Usually radiates somewhere else - ex., pyelonephritis (kidneys) = CVA tenderness

22
Q

neuropathic pain

A

Distinctly different; caused by lesion/dysfunction in the nervous system
Spinal cord/brain/peripheral nerve injuries
CNS: Meningitis, MS, Strokes, Spinal Cord Injuries, HIV
PNS: Amputations (phantom limb), Stenosis, Shingles (dorsal root), Diabetes, Cellulitis
Sustained by abnormal processing of sensory input by CNS/PNS
Occurs more often than you think; 2-3% of developed world
“Burning, Tingling, Numbness, Electric Shocks” - descriptors
Assess:
Medical History, past/current pain history, use pain assessment tools
Allodynia, Hyperalgesia, Paraesthesia

23
Q

DN4 - Douleur neuropathique 4

A

10 items; sensory + physical exam, 1 point each, >4 = neuropathic pain
1. Characteristics of the Pain:
Burning
Painful Sensation of Cold
Electric Shocks
2. Symptoms of the Pain:
Tingling
Pins and Needles
Numbness
Itching
3. Physical Exam:
Hypoesthesia to touch (they don’t feel as much)
Hypoesthesia to prick
4. Pain provoked/increased by:
Brushing or cotton swab