Week 8 - Basic EKG Review Flashcards

1
Q

What is included in the AANA & ASA standards of basic monitoring?

A

Continuous ECG, Pulse ox, Temp, Ventilation, HR & BP every 5 min at least

*basic monitoring is meant to insure patient safety, cover ABCs

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2
Q

What is the purpose of the ECG monitor?

A
  • Gives us a picture of the heart
  • Lets us know how the patient is doing
  • Always relate the rhythm seen to other data to get the whole clinical picture
  • Base treatment decisions on the whole picture
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3
Q

What two leads are typically monitored for ECG? Why?

A

Usually lead II and V5

Looks at both sides of the heart – RCA and the left side

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4
Q

What are the steps in rhythm identification of an ECG?

A
  • Is it regular?
  • Is there P waves, are they regular in size and shape?
  • Does the QRS follow every P wave?
  • Are they regular in size and shape?
  • What are all the intervals?
  • What does the ST segment look like?
  • What does the QT look like?
  • Are there any aberrant beats?
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5
Q

What steps should you go through if there is a rhythm change?

A
  • Look to see how the pt is tolerating the change
  • Think of what may have caused the change
  • Base treatment on the answers from above
  • Think of the risks of your treatment, and the risk of doing nothing
  • Know how long you have to act – its a guess
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6
Q

What are the causes and treatment of Sinus Tachycardia?

A

HR > 100 (relative)

Causes: sympathetic stimulation, pain, hypovolemia, ischemia, impending CHF

Treatment: control pain, increase anesthetic depth, volume, beta blocker
*only use BB after doing the previous treatments – it is a last resort treatment

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7
Q

What are the causes and treatment of Sinus Bradycardia?

A

Sinus with HR < 60 (relative)

Causes: reflexes, volatile agents (Iso - too deep), loss of sympathetic tone, digoxin toxicity

Treatment:

  • removal of offending agent (tugging on the eye, insufflation)
  • atropine or glycopyrrolate
  • inotropic agents (beta agonists)
  • sympathomimetics (role of Phenylephrine and Ephedrine)
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8
Q

What are the causes and treatment of atrial fibrillation?

A

Irregular R-R, absence of P waves — Controlled if rate <100, likely chronic

Causes: mitral valve disease, CHF, hypoxia, hypovolemia, ischemia, electrolytes, cardiac surgery

Treatment: assess tolerance of loss of atrial kick, continuous fluids, reverse hypoxia, beta blockers, amiodarone, cardioversion
-less likely = diltiazem gtt (myocardial depressant on top of volatile agent), digoxin, procainamide

  • *Caution - Ca+ blockers and anesthetics do not mix
  • *Look for anticoagulant while doing preop
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9
Q

What are the causes and treatment of Atrial Flutter?

A

Sawtooth P wave pattern – Atrial Rate >150 and Ventricular Rate is variable

Causes: HTN, COPD, CAD, Dilated cardiomyopathy, ETOH intoxication, thyrotoxicosis

Treatment: if compromised, cardiovert (be ready to pace)
-beta blockers, calcium channel blockers, amiodarone

  • May diagnose with carotid massage or adenosine
  • Usually paroxysmal, may convert to NSR or A-Fib
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10
Q

What is a AV Junctional Rhythm? How do you treat it?

A
  • Narrow QRS, retrograde, inverted or absent P wave
  • Rate usually <60 (if >60 then it is accelerated junctional)
  • common during GA, resolves on its own
  • if accelerated, consider digoxin toxicity, MI or stress of recent cardiac surgery, also hx of rheumatic fever
  • May not respond to atropine, ephedrine may work
  • Consider beta blocker to relieve ischemia (common reperfusion rhythm)
  • Consider temporary pacing if hemodynamic compromise
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11
Q

What is Accelerated Idioventricular Rhythm? Causes?

A
  • Uniform widened QRS rate <60
  • Considered “slow VT” if 60-100

Causes: MI, reperfusion of cardiac surgery, digoxin toxicity, rheumatic fever, cardiomyopathy

  • sign of a sick heart, rarely responds to atropine
  • avoid the urge to use lidocaine (will worsen the “escape rhythm”) – instead try to relieve ischemia
  • be ready to pace
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12
Q

What are the causes and treatment of Ventricular Tachycardia?

A

Uniform, widened QRS, rate 100-250

Causes: CAD, ventricular dysfunction, recent MI (<96hr), cardiomyopathy, hypokalemia, low Mg, central line placement

Treatment: depends on rate & duration, tolerance, extent of known disease

  • amiodarone, lidocaine, possibly procainamide
  • ICD placement
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13
Q

What are the causes and treatment of Torsades de Pointes?

A

Continuously changing, widened QRS, looks like “twisting around a point”

Causes: R on T phenomena (reason for synchronized cardioversion), ischemia

Treatment: magnesium, cardioversion – placement of ICD

  • may terminate spontaneously or degenerate to VF
  • if associated with relative bradycardia, consider Isuprel, Epinephrine, overdrive pacing
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14
Q

What is the treatment of Ventricular Fibrillation? (ACLS algorithm)

A
  • High Quality CPR
  • Shock
  • CPR, Access
  • Shock
  • Epi, Shock (Vasopressin no longer in ACLS)
  • Amiodarone (or Lidocaine), -Shock
  • CPR, shock, repeat, think 5Hs and 5Ts
  • Where is AIRWAY (when it is convenient
  • CPR and SHOCKING are the most important components*
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15
Q

What are the 5 H’s and 5 T’s?

A
Hypovolemia
Hypoxia
Hydrogen ion
Hypo/hyperkalemia
Hypothermia
Tension Pneumothorax
Tamponade, Cardiac
Toxins
Thrombosis, Pulmonary
Thrombosis, Coronary
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16
Q

What are the different heart blocks?

A

First Degree Block – long PR

Second Degree Block (Mobitz 1) – prolonging PR until dropped QRS

Second Degree Block (Mobitz 2) – no pattern, dropped QRS

Third Degree Heart Block

17
Q

What leads look at the inferior side of the heart? What vessel correlates?

A

II, III, aVF

RCA

18
Q

What leads look at the lateral side of the heart? What vessel correlates?

A

I, aVL, V5-V6

Left Circumflex

19
Q

What leads look at the anterior side of the heart? What vessel correlates?

A

I, aVL, V1-V4

LAD and Left Circumflex

20
Q

What leads look at the anteroseptal side of the heart? What vessel correlates?

A

V1-V4

LAD

21
Q

What happens to a pacer when you place a magnet?

A

It will convert it to DOO/VOO, rate typically 85 (Medtronic)

-removing the magnet will make pacer resume original setting

  • ALWAYS have a magnet available during surgery
  • Encourage bipolar not monopolar cautery (ground away from pacer)
22
Q

What do the 1st, 2nd, and 3rd letters of pacer settings stand for?

A
1st = Chamber Paced (A,V,D,O)
2nd = Chamber Sensed (A,V,D,O)
3rd = Mode of Response (triggered, inhibited, dual)

Occasionally:
4th = programmability, communication, & rate modulation (P, M, C, R, O)
5th = antitachyarrhythmia/ defibrillation functions (P, S, D, O)

23
Q

What is an ICD?

A

Internal cardiac defibrillator

  • programmed to try to overdrive pace out of VT, will deliver shock if necessary
  • shocking capacity may be turned off for surgery, may sense cautery as VF and shock it if not disabled
  • must reprogram in PACU if deactivated

Use Magnet for ICDs (turns off shocking capacity but doesn’t affect pacer portion)

24
Q

What are the anesthetic considerations for placement of pacemakers?

A
  • May be done under local or MAC (we are rarely involved unless pt is really sick)
  • May be for lead changes in MOR
  • ICDs may occasionally be done under MAC, may need heavy MAC for programming
  • Fluoro may be used

*Communication w/ Cardiologist is essential

25
Q

What are the considerations for anesthesia in the EP lab?

A
  • May be MAC or GA or both
  • May be for device, lead removal or replacement, cardioversion or more commonly AF/VT ablation
  • If GA, need to go to recovery after procedure
  • Risks of procedures determine level of monitoring you will do, plan for the worst
  • Don’t rely on the cardiologist for arterial line, place your own if indicated
  • Requires fluoro and lead