Week 5- Heart Failure Flashcards

1
Q

What is congestive heart failure?

A
  • Occurs when the heart is unable to pump suffcient blood to meet the metabolic needs of the body
  • As a result, blood backs up into either the pulmonary circuit, systemic circuit, or both
  • Usually occurs secondary to another condition
  • May present as an acute episode, but is chronic
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2
Q

What causes left sided heart failure?

A
  • Left ventricle is most commonly damaged during an MI
  • Chronic HTN- long term effects of having to pump against the increased after load
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3
Q

Left Sided Heart Failure

A
  • In both cases, the right side of the heart continues to pump normally and deliver normal amounts of blood to the pulmonary circulation
  • The left side of the heart is unable to pump the blood from the pulmonary vessels’
  • As a result, blood backs up behind the left ventricle and the pressure in the left atrium and pulmonary veins increases
  • As the pulmonary veins become engorged with blood, serum is forced out of the pulmonary capillaries and into the alveoli
  • Acute cardiogenic pulmonary edema
  • Resp symptom but is a cardiac issue
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4
Q

What will the pt present with if they have Left sided failure?

A
  • Pt. present with SOB, especially when lying supine
  • Never lie pt supine (can make a stable patient go into cardiac arrest, can only breathe sitting up) NEVER LAY SUPINE
  • Increased fluid in the vessels leaks into the interstitial spaces causing narrowing of the bronchioles= wheezing
  • Air passing through the fluid filled alveoli= crackles
  • Pt will present with tachypnea, cheyene- stokes respirations
  • Pt may cough up the edema fluid in the form of foamy, blood tinged sputum
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5
Q

S/S of the LHF

A
  • Restlessness (impending doom)
  • Agitation
  • Confusion
  • Severe SOB
  • Tachypnea
  • Abnormality high or low BP
  • Crackles and possibly wheezes
  • Frothy, pink sputum
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6
Q

What is the management for LHF?

A

Aimed at:
- improving oxygenation
- Decreasing workload on the heart
- Reducing the preload so that the LV is less overburdened

  • Vitals, cardiac monitor, SPO2, ETCO2 (35-45 mmHg)
  • O2
  • IV
    12 Lead ECG
  • Position pt. upright unless BP or LOA precludes it (legs dangling if possible b/c keeps the fluid down and decreases the workload on the heart)
  • Nitro- SL. 0.4-0.8mg, depending on BP
  • Effects vasodilation
  • Reduction in cardiac workload and improved cardiac output (lowers BP, making heart not have to work as hard)
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7
Q

With wheezes, why should bronchodilators be avoided?

A
  • B/c they caused increased tachycardia and this will increase the workload and worsen CHF
  • If you give a pt with left side heart failure ventolin this is a bronchodilator and will open the bronchioles causing more fluid to move in causing them to drown
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8
Q

What is CPAP used for?

A
  • Lungs have a decreased ability to keep the alveoli open due to the fluid
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9
Q

How does CPAP work?

A
  • CPAP works to hold the alveoli open through the respiratory cycle so that O2 and CO2 can diffuse normality
  • Mask can be removed for medication administration
  • CPAP causes an increase in intrathoracic pressure- may cause a slight decrease in BP (need to have a slightly higher BP)
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10
Q

When should you stop CPAP?

A

If the respiratory status is very poor, oxygen saturations levels are dropping ETCO2 is increasing or LOA is decreasing, stop CPAP and begin to ventilate via BVM
- Consider advanced airways if needed

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11
Q

Paroxysmal Nocturnal Dyspnea

A
  • Severe attacks of SOB and coughing that typically happen at night
  • Usually wakes the person from sleep
  • Often occurs with LHF where the cause is chronic
  • Often occurs with LHF where the cause is chronic overload as opposed to MI
  • These pt’s will often complain of 1-2 weeks of SOB at night
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12
Q

Right Sided Heart Failure

A
  • Most commonly occurs as a result of LHF
  • As blood backs up from the left side of the heart and into the lungs, the right side has to work harder to pump blood into the engorged pulmonary arteries
  • Eventually, the right side is unable to keep up to the increased workload, and it too fails
  • Can result in pulmonary embolism or pulmonary HTN
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13
Q

Pulmonary HTN

A
  • Chronic increased resistance of blood through the lungs- backing blood into the right ventricles= increased workload= failure
  • Pulmonary embolism- forward blood flow through the pulmonary system is blocked by a clot= increased workload on RV= failure
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14
Q

S/S of right sided heart failure

A
  • Presence or absence or pulmonary edema will assist you in determining if the RHF is secondary to LHF
  • If pulmonary edema is present= secondary to LHF
  • If non= lung or right sided heart problems
  • When the right side of the heart fails- pressure backs up behind the right ventricle and increases pressure in the systemic veins causing them to become engorged
  • Distention to external jugular veins
  • Over time, serum is forced out of the veins into the surrounding tissues creating edma
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15
Q

What is the management of RHF?

A
  • RHF, by itself is seldom life threatening
  • Usually develops gradually, over weeks (likewise it requires days to reverse)
  • Prehospital treatment- make the pt. comfortable
  • Monitor
  • If pt. presents with signs of LHF as well, treat accordingly
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16
Q

Cardiogenic Shock

A
  • Occurs when the heart is so severely damaged that it can no longer pump sufficient blood to maintain tissue perfusion
  • MI nearly always produces some impairment of the left ventricle
  • Occurs when 40% or more of the left ventricle has been infarcted
  • Can occur after resuscitation, ROSC
17
Q

S/S of Cardiogenic Shock

A
  • Confusion/ comatose due to decreased cerebral perfusion (if awake, restless and anxious)
  • Pale, cold skin- from massive peripheral vasoconstriction
  • Poor SPO2 readings- fingers and hands are barely perfused
  • Rapid, shallow respirations- often crackles
  • Rapid, thread pulse
  • As these compensatory mechanisms fail, the BP will drop
  • Be aware of relative hypotension (happens inside the body)
  • Absolute hypotension (happens outside the body and can see where it’s coming from)
18
Q

What is the management for cardiogenic shock?

A
  • Focused on improving oxygenation and perfusion without increasing the workload on the heart
  • 100% supplemental O2
  • Place the pt. supine unless pulmonary edema is present
  • IV- TKVO
  • Cardiac monitor and 12 Lead
  • Transport quickly