Week 4 - Cardiovascular Flashcards
Cardiovascular examination
- Note general appearance and measure blood pressure and heart rate.
Color, respiratory rate, level of anxiety, BP, HR - Estimate the level of jugular venous pressure.
- Auscultate the carotids (bruit) one at a time.
- Palpate the carotid pulse including carotid upstroke (amplitude, contour, timing) and presence of a thrill.
- Inspect the anterior chest wall (apical impulse, precordial movements).
- Palpate the precordium for any heaves, thrills, or palpable heart sounds.
- Palpate and locate the PMI or apical impulse.
- Palpate for a systolic impulse of the right ventricle, pulmonary artery, and aortic outflow tract areas on the chest wall.
- Auscultate S1 and S2 in six positions from the base to the apex.
- Identify physiologic and paradoxical splitting of S2.
- Auscultate and recognize abnormal sounds in early diastole, including an S3 and OS of mitral stenosis and an S4 later in diastole.
- Distinguish systolic and diastolic murmurs, using maneuvers when needed. If present, identify their timing, shape, grade, location, radiation, pitch, and quality
Cardiac apex
Has the point of maximal impulse, locates the left lower border of the heart and found at 5th intercostal space
S1
closure of mitral and tricuspid valves
S2
closure of pulmonic and aortic valves
- Splits into
- A2 (aortic valve closure) and P2 (pulmonic valve closure)
- A2 louder, P2 softer
S3
abrupt deceleration of inflow across the mitral valve (systolic HF)
S4
increased left ventricular end diastolic stiffness with decreased compliance (diastolic HF)
Systole
ventricular contraction (aortic valve is open blood flow from LV into the aorta)
Diastole
ventricular relaxation (pulmonic valve open, blood flow from left atrium into relaxed LV)
Screening for individual CV risk factors
- Family history
- Smoking
- Unhealthy diet
- Obesity
- Physical activity
- Hypertension
- Dyslipidemias
- DM
- AF
When do you do a lipid screening?
Measure lipid levels every 5 years for people 40-75 without existing CVD
How do we treat CV disease?
Start low dose statins for patients 40-75 who have one or more risk factors (DM, smoking, HTN, HLD) and have a 10 year calculated risk of greater than 10%
Jugular Venous Pressure
- Provides an index of right heart pressures and cardiac function
- Accurately predicts elevations in fluid volume in HF
- JVP falls with loss of blood or decreased venous vascular tone and increases with right or left heart failure, pulmonary hypertension, tricuspid stenosis, AV dissociation, increased venous vascular tone, and pericardial compression or tamponade.
- JVP measured at >3cm above sternal angle considered elevated
Assessing the carotid artery
Auscultate for bruit first – narrowing of arteries due to atherosclerosis
- Have the patient hold their breath, listen with the diaphragm
Palpation – could dislodge a plaque resulting in stroke
- Feel for carotid upstroke, amplitude, contour, presence/absence of thrills
- Assessment characteristics of the carotid pulse
Amplitude – correlates with pulse pressure
* Contour of pulse wave – speed of upstroke, duration of summit, speed of downstroke
Thrills – vibrations like a purring cat
Pulsus Alternans
force alternates due to strong and weak ventricular contractions
- Indicative of left ventricular dysfunction
Paradoxical pulse
greater than normal drop in SBP during inspiration