Week 3 Lecture Flashcards

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1
Q

• Describe the biomechanical forces that act upon the patellofemoral complex and its clinical significance on
forces ect

A

Considerable force transmitted cross PFJ – ½ body weight during walking up to 25X body weight on lifting a weight with knees flexed at 90.
Patella = largest seasmoid bone

Patellofemoral articulation
-displaces the fulcrum of motion of the extensor mechanism anterior to the femur,

-this produces a mechanical advantage increasing the force of the quadriceps muscles in extending the knee

Patella centralizes the divergent forces of the quadriceps muscle and transmits the tension around the femur to the patellar tendon

  • greatest compression occurs in loaded flexion
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2
Q

How does the patella congruency change throughout normal ROM of the knee?
Biomechanics

A

In full extension the patella lies superior to the trochlear cartilage

Flexion 0-30 the patella articular surface begins to engage with the trochlea, little to no articulation (0-45)

Between 30-90 flexion, first the inferior and then the superior patella cartilage articulates with the trochlea cartilage (45-90)

Beyond 120 knee flexion contact decreases between the patella and trochlea

  • at 130 flexion most contact with lateral facet and odd facet
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3
Q

Patella anatomy

post surface
sizes of patella
trochlea

A

Posterior surface of patella has:
large lateral facet
Medial facet
Odd facet (on medial side)

Patella alta- high riding patella
Patella baja- low riding patella
Patella piccolo- small patella
(articular stress)

Trochlea is the intercondylar groove of femur
Medial femoral condyle is typically larger
Lateral femoral condyle hypoplasia leads to the under constrained patella & lateral dislocation

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4
Q

List the patellofemoral joint stabilisers

A

Proximal structures
Rectus femoris
Vastus intermedius
Quadriceps tendon

Distal structures
Patellar tendon

Medial structures
VMO
Medial patellofemoral ligament
Medial retinaculum

Lateral structures
ITB
Lateral patello femoral lig
Lateral retinaculum

can be grouped into passive and active stabilisers

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5
Q

Describe the Q angle

A

The effective line of pull of the quadriceps

Formed between two lines joining:
ASIS, center of patella
center of patella and the tibial tuberosity.

Normal Q-angle is 10-12 M and 15-18 F
The larger the angle = greater the lateral patella ‘pull’

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6
Q

List the most commonly diagnosed and least commonly diagnosed causes of
ANTERIOR KNEE PAIN

A

Most commonly diagnosed
Patellofemoral pain syndrome (PFPS)
Patella tendinopathy
Fat pad impingement

Less commonly diagnosed
Bursitis
Osgood Schlatters
Sinding larsen Johansson
Referred pain from the hip
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7
Q

Static influences on Q angle

A

Static (structural)
-Genu valgum

-Excessive femoral anteversion ( internal femoral torsion)
(Patella is induced laterally into trochlea as distal femur rotates under it.)

-Excessive tibial external torsion
Tibial tubercle is positioned laterally > lat patellar displacement

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8
Q

Dynamic influences on Q angle

A
Dynamic(Functional)
Non local issues
Prox:
Poor frontal & transverse plane control at the hip
Distal
Prolonged and/or excessive foot eversion
Loss of ankle dorsiflexion
Local Issues:
Lateral retinaculum contracture
Shortened TFL/ITB
Vastus lateralis contracture/tightness
Hamstring tightness (BF inserts partiallyinto lateral retinaculum
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9
Q

Poor motor control
• Discuss the significance of sub-optimal motor control around the hip and pelvis in relation to patellofemoral joint pain

A

DYNAMIC VALGUS
Adduction
Internal rotation
Contralateral pelvis drop of stance hip

Increases Q angle which lateralises patella
If hip control is poor distal femur is allowed to move medially under neath the patella and the patella is effectively lateralised

results in patella mal tracking during funcion > PFJP

Weaknes of certain muscles that oppose this:
Adductors- superficial gluteals
External rotators- Deep gluteal group
- Glut max & post fibres glut med

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10
Q

Discuss the role of sub-optimal quadriceps function in patellofemoral joint pain

A

Weak gluteals > increased compensatory quads activation
T/f increassed load at the knee
VMO has no critical role providing medial support ofPFJ

IN PFP group vastus lateralis was activated for much longer than VMO even though they switch on at the same time

  • Quadriceps activity may be better observed in 45-90 degrees of flexion and for CKC 0-45 degrees may reduce compression
  • Assessment of IRQ in OKC pain position is useful differential diagnosis for PFPS
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11
Q

Describe the clinical features

and the factors which may lead to the development of patellofemoral joint pain

A

Body chart
Pain anterior
Crepitus

Typicaly secondary to repetitive/cyclic loading with gradual onset. Primary onset due to trauma

          	Aggravating factors:   Activities with repetitive loaded knee flexion-extension
          	Provoked by prolonged knee flexion Extrinsic factors:
          	Training type and loads
          	Occupation
            Changes in footwear, intensity, surface
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12
Q

• Describe the key objectives of treatment and management and methods to achieve those objectives in patients with patellofemoral joint pain

A

Motor control impairment
AIM: normalise suboptimal postures loading strategies, movement control &/or muscle imbalances

* more in guide p 294
Quad training
Hip strength training
Patellar taping
Combined interventions
Strengthening and flexibility excercises
Stretch lateral constraints
Strengthen medial constraint
Straighten top to bottom

In which ranges is it least painful for strengthening and flexibility excercises?

CKC-0-45 (low)
OKC 45-90 (mod)

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13
Q

Which patients respond best to patellar taping?

A

Those with
Higher levels of pretaping pain
Less lateral patellofemoral tilt
Larger Q angle

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14
Q

In what ranges is it least painful for strenthening and flexibiliity excercises around the knee?

A

Ranges Contact area CKC pain OKC pain

0-45 Low LOW max
45-90 high max MOD

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15
Q

• Describe the clinical features and
key management strategies
of other non-traumatic causes of anterior knee pain including patella tendinopathy,
and bursitis (including Breaststrokers knee/pes anserine bursitis)

A

edit this ??

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16
Q

OSD
&
SLJD

Describe &
Management

A
A type of Adolescent traction apophysitis
Osgood schlatters(OSD)(osteochondrosis)

Involves tibial tubercle and the immature ossification centre beneath
Derangement of normal processes of bone
Localised tissue necrosis followed by full regeneration of healthy bone tissue

Sinding larsen Johansson
Affects distal pole of patella

Mx of these
Symptoms resolve when skeletally mature
Sport participation guided by symptoms
Address any coexisting factors or muscle imbalances

17
Q

Fat Pad irritation syndrome

Decribe
Causes

MOI
Aggravating factors
Managemet

A

(Hoffas Syndrome)

Causes
Local trauma, post surgery, genu recurvatum ( knee hyperextension)
Inferior pole tilt ( inf pole buried deep in FP)

Pain ant and inf , swollen & puffy

MOI-Typically repetative loading (2) w gradual onset
-Trauma (1) acute injury

Aggravating factors
Loaded WB activities
Prolonged standing, stairs
End of range extension activities

Biomechanics
Common posture hyperextension sway back ( post capsule overstretching)

Solution: bracing/ taping
motor relearning/posture correction
Post pelvic tilt
Neutral knee
Increase dorsiflexion range if limited
18
Q

Iliotibial band friction syndrome

Pathology
Causes

ITB function
Dynamic changes

A

ITB function-
controlled adduction
Knee joint stabiliser
Assists knee joint extension

@30 F to E moves and to lat fem epicondyle
@>30 F moves posteriorly

Source of symptoms
Compression of highly innervated layer of fat and connnective tissue that separates ITB from epicondyle

Causes
Dynamic valgus- weak hip adductors
Tight TFL/ ITB
Increasd hip adduction & IR
Increased max rear foot inversion