Week 2 Flashcards

1
Q

• Discuss the indications for imaging in traumatic knee injury (Ottawa Knee Rules)

A
  • Age 55 or over
  • Isolated tenderness of the patella (no bone tenderness of the knee other than the patella)
  • Tenderness at the head of fibula
  • Inability to flex knee to 90 degrees
  • Inability to weight bear immediately and in the casualty department (4 steps= unable to transfer weight twice onto each lower limb regardless of limping)
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2
Q

• Describe the common mechanisms of injury of the menisci and ligaments of the knee

Two main sub-groups

A

Non-contact (80%)

  • Valgus stress with knee in ext rotation AND near EROM extension
  • Deceleration with an attempt to change direction
  • Forced hyperextension

Contact
-Mechanisms = valgus force to knee

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3
Q

Discuss the risk factors for ACL injury

There are 4 subheadings

A
Biomechanical:
              	Knee valgus
              	Anterior tibial shear
              	Tibial IR or ER
              	Lateral trunk motion
              	Dynamic foot pronation
              	GRF

Neuromuscular:
Relative hamstring recruitment
Hip abduction strength
Limb and trunk proprioception

Gender:
Female 2X8 more at risk

Anatomical:
High BMI
Femoral notch width
Generalised or specific joint laxity
Prior injury
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4
Q

• Discuss methods of prevention of non-contact ACL injuries

A
  • Stance foot kept closer to the body’s midline
  • Torso kept upright and rotated toward the desired direction of travel
  • Increased knee flexion angles
  • Control of lateral sway

-Hip neuromuscular control
(isometric hip abduction & ER independantly predict future ACL injury)

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5
Q

Outcomes post ACL Injury

Likely profile of an individual who will return to preinjury level of performance

A
  • Low knee effusion
  • Less episodes of knee instability(self reported)
  • High IKDC scores(self reported knee function)

Low pain
low TSK 11 score
Higher quadriceps peak torque
Less pre/post surgical change in Tegner score(self reported physical activity)

  • = biggest indicator

ACL injury has 3x greater risk of OA after AC treated with recon than contralateral healthy knee

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6
Q

Mechanisms of ACL injury
and the structures affected in order of damage
3-4 kinds

A

Hyperextension
1 posterior capule torn/stretched
2.ACL
3.PCL

O’donoghues /unhappy triad/Valgus knee force with knee in slight flexion

  • Classic presentation:
    1. Superficial MCL then Deep MCL
    2. Tear of medial meniscus- via pull of MCL on attachment to medial meniscus
    3. Rupture of ACL
  • can also result in # of lateral tibial condyle
  • Alternative veiw
    1. Superficial MCL then deep MCL
    2. Tear of lateral meniscus - compression shearing as lat compartment dislocates posteriorly
    3. Rupture of ACL

VARUS FORCE TO THE KNEE- uncommon contact MOI

  1. Lateral collateral ligament
  2. ACL tear
  3. Tear of fibres to popliteus
  4. Fracture of medial tibial condyle
  5. Lateral meniscus tear
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7
Q

ACL diagnosis

A
Patient heard crack/pop.snap 
Felt knee go in and out
Initial intense pain
Rapid hot effusion within 2 hours
Hemoarthrosis = 80% chance of ACL tear
PE
-Exclude PCL & #'s
-lachman’s, anterior draw, pivot shift
-X-ray for associated bony injury
 (tibial spine avulsion, postero-lateral tibial plateau injury)
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8
Q

PCL diagnosis

A
  • Posterior sag test

- Stability tests (posterior draw, reverse Lachman’s)

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9
Q

MCL diagnosis

A

Tender on palpation
Lack of knee extension
Nil or minimal swelling
Positive to valgus testing
(pain, gapping, altereded end feel)

Pain & stiffness with extension & twisting

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10
Q

LCL diagnosis

A
Nil-minimal swelling
Lateral joint line tender on palpation
loss of terminal extension
Positive varus stress test
(pain, gapping, altered end feel)
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11
Q

PCL mechanisms of injury

A

Falling on tibia/blow to tibia forcing it backwards
Football tackle
Dashboard injury

Forced knee hyperextension injury with foot plantarflexed

Hyperextension ( as complication of ACL rupture)

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12
Q

Role of superficial MCL

A

Superficial MCL
-Primary restraint to VALGUS loads in all degrees of knee flexion

  • primary restraint to ER of tibia
  • secondary restraint to Anterior tibial translation
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13
Q

Mechanism of injury of superficial MCL

A

Valgus force with knee slighly flexed(odonoghues triad)
(MCL only)

Valgus force with knee extended
(Posterio medial capsule also torn)
if more severe ACL may also be affected

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14
Q

Role of LCL

& mechanism of injury

A
  • primary restraint to Varus forces at knee
  • Restraint to IR of tibia

Very uncommon
Varus stress to the knee( direct blow to anteromedial tibia)

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15
Q

Kinematics of the menisci of the knee

A
  • With knee flexion the menisci elongate posteriorly
  • lateral moves more than medial
  • Medial more vulnerable to injury
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16
Q

Mechanism of Injury of menisci

A

1 low energy mechanism
twist on flexed knee ( tib ER + knee valgus)
in degenerative middle aged pop

2 High energy mechanism
Twisting involved in sport
Male>female , medial >lateral

3 associated with other injury
Lateral meniscus with ACL injury>
result of compression/sheering forces as lateral compartment dislocates posteriorly

Medial meniscus with MCL injury>
via pull of MCL on attachment to medial meniscus

17
Q

Menisci function

A

Load transmission & shock absorbtion
Joint congruity enhanced
Stability enhanced

Joint lubrication
Articular cartilage nutrition
Proprioception

18
Q

Diagnosis of meniscal tear

A
S/E
patient age
catching or locking
can usually weight bear through pain
Effusion around joint line( mild & slow)

P/E
Pain with loaded deep flexion

loss of end range extension with springy end feel
hamstring spasm and acute locking
joint line tenderness
Pain with varus/valgus stress tests
Pain & clicking w
- mcmurrarys test     flex ext w varus/valgus
-thelasy test
-eges test

MRI non invasive gold standard

19
Q

Define sensitivity and specificity and their role in diagnosis

A

SnOUT:
sensitivity high
a neg test willl rule the disorder out
(low risk of false negative)

SpIN
Specificity high
a pos test will rule the disorder in
(low risk of false positive)