week 3 <3 Flashcards
amiodorone
administered when heart is in a-fib to help prevent clots
formula & amount of normal cardiac output
CO= HR x SV
*4-8 L per minute
formula of cardiac index
CI= cardiac output / body surface area
*normal = 2.8 to 4.2
formula for mean arterial pressure
MAP = SBP + 2DBP / 3
in patients with heart failure, their pulse pressure
decreases
in relation to the heart…chemicals associated with the sympathetic nervous system are
epinephrine & norepinephrine
- INCREASE contractility, HR, & vasoCONSTRICTION
In relation to the heart… (a) chemical associated with the parasympathetic nervous system are
acetylcholine
*DECREASES– contractility, HR, & vasoDILATION
baroreceptors
found in carotid sinus & aortic arch & function to MAINTAIN NORMAL BLOOD PRESSURE
chemoreceptors
MAINTAIN ABG’S - cardiorespiratory hemostasis
2 locations =
(1) BRAIN - stimulated by pH and PCO2 (arterial co2)
(2) peripheral in carotid & aortic bodies - stimulated by pH & PO2
** hyperventilation stimulates them
testing for orthostatic hypotension includes
- BP while lying down
- sit-up & wait 5 min then take BP
- stand and take BP
8 sites for palpating arteries
- carotid
- brachial
- radial
- ulnar
- femoral
- popliteal
- posterior tibial
- dorsalis pedis
3 types of drugs for a slow heart
***these drugs help speed it up
- Beta Blockers = -lol
- Calcium channel blockers = -zem
- digoxin = -digitalis
atropine
anticholinergic drug used to elevate heart rate bradycardia
used only when symptoms are present
atroPINE = PINE on top of the tree = ELEVATE
3 cardiac biomarkers
Troponin ( 7 days)
Creatinine kinase (up to 3 days)
Myoglobin (within 24 hrs)
normal cholesterol levels
< 200 mg/dL
c-reactive protein range
< 0.3 mg/dL
EKG 5 step interpretations
- # of R peaks x 10 = bpm
- rhythm = space between R peaks
- presence of P wave next to QRS
- PR interval less than 5 mini boxes
- QRS wave (present, upright, & tight)
V-fib on an EKG looks like & is treated by:
*flopping squiggly line
- use DEFIB
- admin drugs LAP
(L) lidocaine
(A) amiodorone
(P) praocainamide
**caused by v-tach. post MI, & electorlyte imbalances
V-tach on an EKG looks like
*TOMBSTONES
*no pulse = d-fib
pulse = cardioversion
*caused by LOW K+ & MG, & MI
a-fib on an EKG
no p wave (starting hill) fibrillation flopping
- TTE to rule out clots
- cardioversion
- digoxin
- anticoagulants
*caused by COPD, HTN
before administering digoxin check for
A.T.P.
**digoxin DIGS for deeper contraction
A = apical pulse is 60 +
T = toxicity (<2,0, n/v, green halos)
P = potassium less than 3.5
NO ANTACIDS
atrial flutter
*saw TOOTH w/ 2 T’s
treated same as a-fib
- TTE to rule out clots
- cardioversion
- digoxin
- anticoagulants
treatment for supraventricular tachycardia
** super fast — hr
synchronized cardioversion = priority if HR & RR is elevated & Bp is low
- pt to bear down or ice cold stimulation
- rapid push of adenosine
torsades de pointes on EKG
*wavy tornado
caused by low MG, MI, & hypoxia
treated w magnesium sulfate
magnesiums role on heart muscles
magnesium MELLOWS out the heart muscles
if low = overactive heart
p-wave depicts
atrial fibrillation
ORS Wave depicts
ventricular rhythm
“lack of QRS complexes”
patient is in asystole