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Periodontal Instrumentation Final > Week 3 > Flashcards

Week 3 Flashcards

1
Q

what is the origination and development of a disease

A

pathogenesiis

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2
Q

___ is a complex interplay between subgingival biofilm and host immune inflammatory events that develop in the gingival and periodontal tissues in response to bacterial challenge

A

periodontitis

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3
Q

what results in the breakdown of fibers of the PDL, clinical attachment loss and resorption of alveolar bone

A

pathogenesiis

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4
Q

T/F: perioidontal pocket is a favorable environment for bacteria to survive

A

TRUE

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5
Q

what is part of epithelial portion of dentogingival junction

A

gingival, sulcular, and junctional epithelium

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6
Q

what are the histologic stages of gingivitis and periodontitis

A
  • know this
  1. initial lesion
  2. early lesion
  3. established lesion
  4. advanced lesion
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7
Q

what develops within 2-4 days of plaque acclumination

A

initial lesion

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8
Q

what lesion corresponds to clinically healthy gingival tisssues

A

initial lesion

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9
Q

what develops after 1 week of continued plaque accumulation

A

early lesion

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10
Q

what corresponds to early gingiviitis (clinically eviident)

A

early lesion

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11
Q

what corresponds to established, chronic ginigiivitis

A

established lesion

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12
Q

what marks the transition from gingivitis to periodontitiis

A

advanced lesion

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13
Q

what are clinical signs of disease

A

pocket formation and bone resorptiion

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14
Q

types of immunity

A

innate and adaptiive

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15
Q

what is part of innate immunity that prevents pathogens from invading cells and tissues of body

A
  1. saliva
  2. epithelial tissues
  3. gingival crrevicular fluid
  4. neutrophils
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16
Q

what provide a focused and intense defenses against infections that overwhelm the innate immune respsonsess

A

adaptive immunity

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17
Q

what is part of adaptive immunity

A
  1. antigen presenting cells
  2. t-cells
  3. antibodies
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18
Q

what is the primary cause of gingival inflammation

A

plaque!

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19
Q

can you find calculus supragingivally or subgingival?

A

BOTH

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20
Q

inorganic composition of calculus

A

70-90% inorganic

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21
Q

4 modes of attachment for calculus

A
  1. organic pellicle on cementum
  2. attachment on enamel
  3. mechanical locking into surface irregularitie
  4. close adaptation of calculus to depressions on unaltered cementum and then penetration into cementum
22
Q

does calculus contribute directly to gingival inflammation

23
Q

what is the plaque formation process

A
  1. formation of pellicle
  2. initial adhesion/attachment of bacteria
  3. colonization/plaque maturation
24
Q

what is colonization/plaque maturation

A

shift from supragingival plaque with high gram+ organisms to subgingival plaque with high gram- organisms

25
what are the 5 microbial shifts as health progresses to periodontitis
1. gram + to - 2. cocci to rods 3. nonmotile to motile 4. faculative anaerobes to obligate anaerobes 5. fementing to proteolytic
26
"The host response to the bacterial challenge originating from the dental biofilm has been considered to a play a major role in initiation and tissue destruction of ___."
periodontal diseases
27
what is an adherent mass of salivary proteins, organized bacterial colonies and their associated cytotoxins
plaque
28
what is the primary causes of all iinfectious dental diseases (caries, ginigivitis, and periodontitis)
plaque
29
what is hard, tenacious mineralized deposits on enamel, dentin, and cementum
calculus
30
what is calculus made up of
Ca2+, PO4, salivary proteins, cellular debris and bacteria and their cytotoxins
31
What becomes embedded into cementum and becomes increasingly difficult to remove
Calculus
32
Where does most plaque accumulate
Below height of contour
33
What does normal gingiva look like
Pink, firm, knife-edged, stippled, no BOP
34
What are early sides of gingivitis than the visual sides of inflammation
Redness, swelling
35
2017 classifications of gingivitis
Localized gingivitis: less than 30% of teeth affected by BOP Generalized gingivitis: 30% or more of teeth affected by BOP
36
1999 classifications of gingivitis
1. Papillary gingivitis 2. Marginal gingivitis 3. Diffuse gingivitis
37
Severity of gingivitis per 2017 workshop
1. Mild gingival inflammation: area w minor change in color and little change in texture of tissues 2. Moderate gingival inflammation: area with glazing, redness, edema, enlargement and BOP 3. Severe gingival inflammation: area of overt and edema with tendency toward bleeding when touched rather than probed
38
Drug induced gingival enlargement is associated with what medications
1. Anti epileptic drugs 2. Calcium channel blocking drugs 3. Immunoregulsting drugs 4. High dose oral contraceptives
39
What is the increase in volume of an organ or tissue due to enlargement of its component cells
Hypertrophy
40
What is is when the cells remains approximately the same size but there is an increase in number of cells
Hyperplasia
41
What explorer is double ended with pointed, curved, flexible ends
11/12 lab 3
42
Can 11/12 be used in all areas of mouth? Can it be used to detect Supra and subgingival calculus
Yes, yes
43
You should use the tip of the 11/12 explorer
No
44
What do we use the 11/12 explorer for
Calculus detection
45
What type of strokes to use for 11/12 explorer
Up and down Oblique Horizontal
46
Where does calculus initially form
CEJ
47
Most calculus missed is where
CEJ
48
Various forms of subgingival calculus
1. Nodular - small to large bumps that are usually easy to detect 2. Veneer - a flat sheet of calculus that is usually difficult or impossible to detect
49
Correct positioning of 11/12 explorer
Terminal shank parallel to long axis of tooth and tip curved into interproximal area
50
When using 11/12 on posterior teeth, you should check the terminal shank where
Distal of premolar
51
Does the terminal shank of 11/12 cross over the midline and working end curves around anterior tooth?
Yes, it looks like a seatbelt

Periodontal Instrumentation Final (5 decks)

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