Week 2 (exam 2) Flashcards

Cardiac

1
Q

Heart failure

A
  • The inability of the heart to pump sufficient blood to meet the needs of the tissues for oxygen and nutrients
  • A syndrome characterized by fluid overload or inadequate tissue perfusion
  • The term heart failure indicates myocardial disease, in which there is a problem
    with the contraction of the heart (systolic failure) or filling of the heart (diastolic
    failure).
  • Some cases are reversible.
  • Most HF is a progressive, lifelong disorder managed with lifestyle changes and
    medications.
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2
Q

Clinical manifestations of RIGHT sided heart failure

A

Viscera and peripheral
congestion
 Jugular venous distention
(JVD)
 Dependent edema
 Hepatomegaly
 Ascites
 Weight gain

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3
Q

Clinical manifestations of LEFT sided heart failure

A

 Pulmonary congestion,
crackles
 S3 or “ventricular gallop”
 Dyspnea on exertion (DOE)
 Orthopnea
 Dry, nonproductive cough
initially (possibly coughing up. blood)
 Oliguria
 Cyanosis
 Increased RR
 Elevated pulmonary capillary wedge pressure

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4
Q

Medical Management of HF: Medication

A

 Angiotensin-converting enzyme inhibitors
 Angiotensin II receptor blockers
 Beta-blockers
 Diuretics
 Digitalis
 Intravenous infusions
-Dobutamine
-Milrinone
 Other medications
- Hydralazine
- Isosorbide

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5
Q

medical management of HF includes

A

Nutritional therapy
- Following a low sodium (no more than 2 g/day) diet and avoiding excessive fluid intake
(p.803)
Supplemental O2
ICD
Cardiac resynchronization therapy
Elevate HOB

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6
Q

Acronym for congestive HF

A

Up-right position
Nitrates
Lasix
Oxygen
Ace Inhibitors
Digoxin

Fluids
Afterload decreases
Sodium restriction
Test (dig levels, ABGs, and Potassium levels)

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7
Q

HF complications

A

Pulmonary edema
Cariogenic shock
Thromboembolism
Pericardial Effusion and Cardiac Tamponade

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8
Q

Pulmonary edema manifestations

A
  • Pink frothy sputum
  • SOB / difficulty breathing
  • crackly lungs
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9
Q

Pulmonary edema assessments and diagnostics

A
  • listen to lungs
  • are they having difficulty maintaining O2 status
  • chest x-ray
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10
Q

Pulmonary edema prevention

A
  • Diuretics (in-between giving blood)
  • sit them up in bed
  • have them dangle their feet
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11
Q

Pulmonary edema medical management

A

O2 therapy (non-rebreather, oxy mask, BiPap)
Diuretics
Vasodilators

both diuretic and vasodilator will impact blood pressure

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12
Q

Patho of cariogenic shock

A

Cardiac output is compromised → BP falls and tissue profusion is decreased → Blood supply for tissues and organs and for the heart muscle itself is inadequate → this impaired tissue profusion weakens the heart and impairs its ability to pump → the ventricle does not fully eject its volume of blood during systole → fluid accumulates in the lungs

this can occur suddenly or over a period of days
(p. 287)

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13
Q

Intra-Aortic Ballon Pump

A

If cardiac output does not improve, the IABP is a catheter with an inflatable balloon at the end. The catheter is usually inserted through the femoral artery and threaded toward the heart, and the balloon is positioned in the descending thoracic aorta. It inflates during diastole, and deflates just before systole.
(p. 289)

Takes workload off the heart

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14
Q

Cardiac Tamponade

A

Compression of the heart resulting from fluid or blood within the pericardial sac
- looks like RHF
- muffles heart sounds
- Hypotensive
- Diastolic and systolic BP get closer together

DX: echo
Tx: pericardialcentesis or pericardial window

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15
Q

Medications Used in
Cardiopulmonary
Resuscitation

A

 Epinephrine
 Vasopressin
 Norepinephrine
 Dopamine
 Atropine
 Amiodarone
 Sodium bicarbonate
 Magnesium

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16
Q

Acute coronary artery syndrome

A
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17
Q

Myocardial Infarction

A
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18
Q

Factors affecting heart rate

A

Autonomic intervention (medications, lifestyle changes, and wearing compression garments)
Hormones
Fitness level
Age

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19
Q

Factors affecting stroke volume

A

Heart size
Fitness level
Gender
Contractility
Duration of contractility
Preload
Afterload

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20
Q

Stroke volume = ___________ - _____________

A

End Diastolic Volume - End Systolic Volume

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21
Q

Invasive modalities of Hemodynamics monitoring ANDDDD possible complications

A

CVP
Pulmonary Artery Pressure (Swan Ganz catheter)
Intra-arterial pressure

Infection
pneumothorax
Ari embolism

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22
Q

Minimally invasive hemodynamics monitoring devices

A

Pulse Pressure Analysis
Esophageal Doppler
Probes
Fick Principle

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23
Q

Central venous pressure shows the…

normal pressure is…

A

pressure of the right atrium, normal pressure is 2-6
if it is above 6 they have fluid volume overload
if it is below 2 they have fluid volume deficit

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24
Q

Pulmonary Artery Catheter

A

Sits in the pulmonary artery and provides pulmonary artery pressure and wedge pressure.
Shows Left Ventricle function
Normal range is 15-25/8-15

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25
Q

Pulmonary Artery Occlusion pressure normal is (PAOP)

26
Q

An increase in PAP (pulmonary arterial pressure) indicates what?

A

A decrease in left sided heart function

27
Q

Treatment for Increased PAP

A

Dolbutomine and diuretics

28
Q

Tx for decreased PAP

A

Fluids, vasopressors

29
Q

Dythrhythmias

A

disorders or the formation, conduction, or both of the electrical impulses in the heart
they can cause disturbances of rate rhythm or both
can alter blood flow and cause hemodynamics changes
diagnosed by EKG

30
Q

Potential complications/collaborative problems of dysrhythmias

A

Cardiac arrest
Heart failure
Thromboembolic
event, especially
with atrial
fibrillation

31
Q

Cardioversion and defibrillation

A

Treat tachydysrhythmias by delivering an
electrical current that depolarizes a critical
mass of myocardial cells. When cells
repolarize, the sinus node is usually able
to recapture its role as heart pacemaker.
In cardioversion, the current delivery is
synchronized with the patient’s ECG.
 In defibrillation, the current delivery is
unsynchronized.

32
Q

Possible complication of pacemaker use

A

 Infection
 Bleeding or hematoma formation
 Dislocation of the lead
 Skeletal muscle or phrenic nerve stimulation
 Cardiac tamponade
 Pacemaker malfunction

get chest x-ray to make sure leads are placed in the correct spot

33
Q

Invasive methods to diagnose and treat recurrent dysrhythmias

A

Electrophysiological
studies
Cardiac conduction
surgery
Maze procedure
Catheter ablation
therapy

34
Q

Electrophysiological
studies

A

cardiac catheter that assesses the function or dysfunction of the SA and AV nodal areas and identifies the location of the conduction issue

35
Q

Maze procedure

A

open heart surgical procedure for refractory atrial fibrillation.
Because the procedure requires significant time and cardiopulmonary bypass, its use is reserved only for those patients undergoing cardiac surgery for another reason

36
Q

Catheter ablation
therapy

A

Catheter ablation destroys specific cells that are the cause of a tachyarrhythmia

37
Q

Cariogenic shock

A

occurs when the heart’s ability to contract and to pump blood is impaired and the supply of oxygen is inadequate for the heart and the tissues. fluid accumulated in the lungs

38
Q

Cariogenic shock symptoms

A

angina pain
arrhythmias
fatigue
feelings of doom
signs of hemodynamics instability
(P.287)

39
Q

Cardiac Tamponade

A

compression of the heart resulting from fluid or blood within the pericardial sac
pericardiocentesis can be done to treat

40
Q

Causes of cardiac arrest can be

A

MI
Arrythmias
Progressive bradycardia
Hypothermia
Medication overdose
Pulmonary Embolism
Hyperkalemia
Sever Hypoxia

41
Q

Signs of cardiac arrest

A

Unresponsive, no pulse, no breathing

42
Q

Agents for cardiac arrest

A

Epinephrine
Vasopressin
Norepinephrine
Dopamine
Atropine
Amiodarone
Sodium bicarbonate
Magnesium Sulfate

43
Q

Treatment of Acute Coronary Syndrome(MI)

A

Nitro
Aspirin/Plavix (if you can’t give nitro due to low BP)
Morphine/Fentanyl
Oxygen (only if O2 sat below 94%)
Anticoagulants
Beta Blockers (within 24 hours)

PCI (60 min door to balloon)

Thrombolytic agents (30 min door to needle)

44
Q

Three zones of damage with Acute Coronary Syndrome(MI)

A

Infarction, injury, ischemia

45
Q

aortic valve is best heard…

A

at the second intercostal space to the right of the sternal boarder

46
Q

Pulmonic valve is best heard…

A

over the second intercostal space to the left of the sternal border

47
Q

Tricuspid valve is heard over…

A

the 5th intercostal space to the lower left sternal border

48
Q

Mitral valve is heard over…

A

Apex, 5th intercostal space

49
Q

Infarction

A

MI, cells are dead
beyond hope of recovering those cells but you can attempt to stop the spread of necrosis

50
Q

Injury

A

Some recovery is possible
can still perfuse and restore it to become viable

51
Q

Ischemia

A

Full recovery is possible

52
Q

Valvular Stenosis
Valvular Regurgitation
Valvular Prolapse

A

Stenosis - narrowing of the vessel blood will become backed up (LHF)
Regurgitation- backwards flow of blood (can be result of a prolapse)
Prolapse - Leaflets do not close properly allowing blood to flow backwards

53
Q

Dilated cardiomyopathy

A

most common – ventricles enlarge and weaken and it primarily effect systolic function

54
Q

Hypertrophic cardiomyopathy

A

ventricles and septum enlarge and thicken which effects diastolic function and also restricts blood outflow

55
Q

Restrictive cardiomyopathy

A

ventricles become stiff and rigid which restricts filling during diastole

56
Q

S&S of cardiomyopathy

A

SOB
Fatigue
Dizziness
Arrthmias
Murmurs

57
Q

Dx for cardiomyopathy

A

Echo
EKG
Chest MRI
Chest X-Ray

58
Q

Medications given for cardiomyopathy

A

Diuretics
Digoxin
Antidysrhythmic agents
Antihypertensive agents

59
Q

Procedures done for cardiomyopathy

A

Procedures
Septal myectoy –part of heart muscle is removed
Septal ablation
Pacemake or LVAD
Heart transplant

60
Q

Abdominal aortic aneurysm S&S

A

Abdominal or back flank pain
Pulsating abdominal mass (bruit over the area as well)
Do not palpate the area

61
Q

Thoracic aortic aneurysm S&S

A

Severe back or chest pain
SOB
Difficulty swallowing and a cough