Exam 3 - Burns Flashcards

1
Q

Functions of the Epidermis, Dermis, and Subcutaneous tissue

A

Epidermis – repels pathogens prevents fluid loss
•Dermis- largest portion of skin – gets structure and strength- contains a lot of nerve endings and sweat glands
•Subcutaneous tissue- cushion between skin and muscle and bone- helps regulate body temp

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2
Q

Functions of the skin:

A

• Protection – invasion of bacteria
• Sensation- temp, pain, pressure, light touch
• Fluid balance – has the ability to absorb water, insensible perspiration
• Temperature regulation
• Vitamin Production – synthesize vitamin D 5-30 min sun exposure twice a week
• Immune response

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3
Q

Effects on skin while we age

A

• Dryness
• Thinning of skin
• Loss of sub q tissue- loss of temp regulation – loss of protection of muscles and bones
• Sweat and sebaceous glans decrease

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4
Q

Factors the determine burn severity are

A

age, depth of burn, how much body surface area was involved, was it and inhalation injury, where is the burn at, as well as previous medical hx

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5
Q

Burn depth

A
  1. First degree – superficial, outermost layer of skin, epidermis is still intact (sun burn, touching stove)
  2. Second decree- Involved entire epidermis and some of the dermis – blisters – scalds, direct contact injury, flash flame
  3. Third degree- total destruction of the epidermis and dermis- damage of nerve fibers, leathery skin, hair follicles and sweat glands are destroyed – full thickness
  4. Fourth degree- deep burn necrosis, injury extends into deep tissue and bone – leads to amputations
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6
Q

Once burn goes past _____ of total surface area it will produce both local and systemic effects.

A

30%

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7
Q

Burns cause

A

-wound edema
–generalized edema
–increased O2 and glucose consumption

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8
Q

Zone of Burn Injury – (burns can evolve over time)

A
  • Zone of Coagulation – right in the middle – cell death – necrotic tissue
  • Zone of Stasis – right outside the middle of the burn- injured cells that may be viable if they get persistent profusion, if not it will die off in 24-48 hours
  • Zone of Hyperemia – outside zone – minimal injury- likely to have full recovery of this skin tissue
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9
Q

What are the most common sites of injury

A

Skin and mucosa of upper airway are most common sites of injury

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10
Q

Electrical burns

A

Most complex of all the burn injuries because you cannot tell the extent of damage just by looking at the burn like you would be able to with other burn injuries- some of the burn could be occurring interiorly

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11
Q

Types of electrical burns

A

Flash injury
Conductive injury
Lightning injury

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12
Q

Flash injury

A
  • Has to do with light and heat without current, ball of fire, quick and hot
  • Fewer complications
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13
Q

Conductive injury

A
  • Electrical current that travels through the body (could go into the hand, through the body and out the foot)
  • Skeletal muscle injury
  • Compartment syndrome
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14
Q

Lightning injury

A
  • Direct strike of lighting or a side flash of lightning
  • Messes with electrical system of the heart, may cause pts to cardiac arrest
  • Can cause respiratory arrest
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15
Q

Compartment syndrome

A

the development of fluid/swelling around a bone blocking blood flow from any distal limbs. Their distal extremities will not have capillary refill, or their extremities will be cool to the touch. To treat this Drs may do an escharotomy (opens up skin and relieves pressure) – or a fasciotomy (cuts through fascia to relieve that pressure).

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16
Q

Labs for electrical burns

A
  • CK levels- tells us muscle damage – myoglobin gets released which plugs up the kidney and caused burgundy colored urine. Increase fluids to flush out myoglobin
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17
Q

Systemic complications from burns: cardiovascular

A

decrease in CO, decreased plasma volume (leads to hypovolemia and hypotension) increased workload on the heart because of increased O2 demand, capillary leaking

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18
Q

Systemic complications from burns: fluid and electrolytes

A

edema forming, inflammatory mediators are getting released causing fluid and electrolytes to move into the interstitial fluid. These fluid shifts lead to compartment syndrome. Increased potassium levels, as time goes on fluid will shift again and you will see low potassium levels

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19
Q

Systemic complications from burns: Pulmonary Alterations

A

involve upper or lower airway. Upper airway involved when there is direct thermal injury to face or neck. Lower airway is when there is and inhalation injury and happens below the glottis.

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20
Q

Systemic complications: Upper airway assessments/signs

A
  • Assess trachea
  • Strider
  • Drooling
  • Difficulty swallowing

Tina Doesn’t Stop Drooling

21
Q

Systemic complications: Lower airway assessments/signs

A
  • Impaired gas exchanges
  • Collapsed alveoli
  • Crackles due to increased mucous production
  • Bronchi may constrict causing wheezes
  • Ineffective airway clearance
  • Carbon monoxide poisoning- give 100% O2 to replace carbon monoxide molecule
22
Q

Systemic complications: Kidney Alterations:

A

acute kidney injury (due to hypovolemia, not enough blood to the kidneys). Acute tubular necrosis (intra renal kidney injury because myoglobin in the urine is plugging up the kidneys). Observe for signs and symptoms of acute kidney injury.

23
Q

Systemic complications: Immunologic Alterations:

A

pt looses their first couple layers of skin, puts them at a higher risk of infection

24
Q

Systemic complications: Thermoregulatory Alterations

A

loss of skin pt looses ability to regulate body temp, may become hypothermic

25
Systemic complications: Gastrointestinal Alterations
1. Paralytic ileus 2. Curlings Ulcer 3. Translocation of bacteria 4. Abdominal compartment syndrome
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1. Paralytic ileus
- related to decreased GI motility – gut isn’t getting perfused – decreased nerve impulses that leads to decreased bowel sounds or no bowel sounds. Fix by putting NG tube in with Low intermittent suction
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2. Curlings Ulcer
- erosion of GI tract from ischemia Tx with H2 blocker or PPI
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3. Translocation of bacteria
- bowel becomes more permeable so stool will shift into peritoneal space - may cause peritonitis, leads to severe sepsis
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4. Abdominal compartment syndrome
- fluid shifts into intertidal space of abdomen causing a lot of pressure within the abdominal cavity which can lead to abdominal organ ischemia - Abdominal distention - Oliguria - Difficulty ventilating these pts because the abdomen is push up on the lungs
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Phases of burn care
1. Emergency or resuscitative phase 2. Acute or intermediate phase 3. Rehabilitation phase
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1. Emergency or resuscitative phase
On the scene care: remove pt from source of injury, remove restrictive objects, establish an airway, supply O2, start IV, cover wound (clean dry cloth or gauze, irrigate wound if it’s a chemical burn) 62.4 chart
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1. Emergency or resuscitative phase: Medical Management
- Focus on ABCs, encourage pt to cough, monitor their airway - Get baseline weight, labs, and vitals - Fluid resuscitation – give LR
34
ABA Formula:
Thermal burn: 2ml of LR * pt weight in Kg *total body surface area of burn Electrical burn: 4ml of LR * pt weight in Kg* total body surface area of burn
35
Parkland Formula:
applied only in 2nd and 3rd degree burns 4ml of LR * body weigh in Kg *total body surface area of burn / 2. First half give in the first 8hrs and second half give in the second 16 hours.
36
2. Acute or intermediate phase
Begins 48-72 hours after the burn injury
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2. Acute or intermediate phase: Medical Management
- Restoring fluid balance- LR – monitoring for fluid excess – monitor electrolytes - Infection prevention – neutropenic precautions - Wound cleaning – debridement of nonviable skin tissue and hair. Use warm water and mild soap to clean. - Topical antibacterial therapy – look up what meds these are - Wound dressing - Wound debridement - Wound grafting - must have viable tissue to put skin graft on - Pain management - Hyper metabolism
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2. Acute or intermediate phase: collaborative problems
- Acute Respiratory Failure - Distributive shock - Acute kidney injury - Compartment syndrome - Paralytic ileus - Curling's ulcers
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1. Natural debridement
When the nonviable tissue separated from viable tissue naturally
40
2. Mechanical debridement
Surgical tools separate and remove escar, dressing changes aid in this
41
3. Surgical debridement
Excision of full thickness of the skin, down to where the surgeon sees viable tissue (if the tissue starts to bleed) they will get rid of necrotic tissue, down to wear they see viable tissue and then they will put a skin graft on
42
4. Chemical debridement
Topical antimicrobial agents, used in conjunction with antibacterial. They have silver in them and can cause debridement on viable tissue if not careful.
43
Cultured epithelial autograft (CEA):
used in burns that cover more than 90% TBSA - Care of graft site: needs to be occlusive dressing – first dressing change is 2-5 days after the graft placement – keep site elevated (reduces edema) - Care of donor site : clean dry dressing, throbostatic agent applied to relieve bleeding – can take 7-14 days for it to heal.
44
Homografts and Xenografts
- Biological dressing: biobrane, nylon and silicone – helps prevent infection - Temporary wound coverage. - Homografts are taken off another pt and Xenografts are taken off animals
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Biosynthetic and synthetic dressings:
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Types of pain
- Background pain: always there, take long-acting analgesics - Breakthrough pain: related to activity and movement, acute and severe pain. Take short acting analgesics (IV morphine, fentanyl, dilaudid) - Procedural pain: due to daily dressing changes, OT/PT, plan to medicate these pts 30-40 min before they are going to have a dressing change
47
Modulation of hyper-metabolism
exaggerated stress response, early nutrition is necessary for these pts, they will have tube feeds high in carbs and protein
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3. Rehabilitation phase
Psychosocial support
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Abnormal wound healing
- Hypertrophic or keloid scars - Prevention and treatment of scars: Compression sleeves, scar massage, reconstructive surgery