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BPK 241 > Week 1 [not including lab] > Flashcards

Week 1 [not including lab] Flashcards

1
Q

Three components of evidence-based medicine

A

Best evidence, clinical expertise, patient values and preferences

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2
Q

3 components of injury prevention

A
  • Protection that reduces forces: protective equipment, technique development, balanced opponents (ex: weight classes), preventative taping, facilities, rules and enforcement
  • Strength of body parts: when, what - strength vs endurance, how
  • Screening of participants: Why? - determine if fit, establish athlete’s health; Team medicals? Who, what, when
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3
Q

What are forms of sport preparation?

A
  1. general conditioning
  2. specific training
  3. protective measures
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4
Q

Why is sport preparation important?

A
  • to decrease incidence of injuries and decrease severity of injuries
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5
Q

describe 4 subcategories of general conditioning

A
  • endurance
  • strength
  • power
  • flexibility
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6
Q

describe the equation for cardiac output

A

cardiac output = heart rate x stroke volume

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7
Q

fill in the blank: your fitness is proportional to your __________

A

resting heart rate over 1 min

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8
Q

How should you build your endurance?

A

20-60min exercise, HR > 150 bpm at least 3 times per week

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9
Q

Define: PRE

A

progressive resistance exercises

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10
Q

What are 2 examples of protective measures for sport preparation?

A
  • equipment: absorbs energy, disperses energy, deflects a blow, limits excess movement
  • nutrition: role of nutrients: growth repair and maintenance of all tissues, regulate body processes, provide energy
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11
Q

Describe the % of caloric intake that the following should make up:

  • proteins
  • carbohydrates
  • fats
A
  • Proteins (20-25% of caloric intake)
  • Carbohydrates (55-60% of caloric intake)
  • Fats (15-20% of caloric intake)
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12
Q

What happens to extra proteins that are consumed?

A
  • burned as energy or stored as fat
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13
Q

what is the most efficiently broken down and metabolized form of energy for the body?

A
  • carbohydrates
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14
Q

Where are carbohydrates stored and in what form?

A
  • liver and muscles

- glycogen

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15
Q

Fill in the blank: the average 150lb athletes carries _______ calories in the form of carbs

A

1500 - 2000

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16
Q

What is the primary fuel for light to moderate intensity exercise?

A

fats

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17
Q

Why are more efficient athletes more likely to burn fats than carbohydrates?

A

they can maintain the same effort level but at a lower intensity

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18
Q

Fill in the blank: the average 150lb athlete carries up to _____ in fat

A

80 000 calories

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19
Q

Fill in the blank: Fat to carb utilization switch typically occurs at ____ VO2 max but in athletes can happen at _____ VO2 max (marathon runners, endurance athletes)

A
  • 65%

- 70-80%

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20
Q

What is the role of vitamins for sport training?

A
  • Not metabolized for energy but play key role in metabolism of carbs and fats for energy during exercise
  • Involved in the repair and building of muscle protein in response to training
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21
Q

fat soluble vitamins vs water soluble vitamins

A
  • fat soluble vitamins (ADEK): can be stored in body fat and have an increased risk of toxicity
  • Water soluble vitamins (BC): excess is excreted in urine, needs to be replenished
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22
Q

What is the role of minerals for sport training?

A
  • electrolytes for proper hydration

- calcium for strong bones

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23
Q

Define: major minerals

A

100mg+/day (sodium, potassium, chloride, calcium, phosphorus, magnesium, sulfur)

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24
Q

Define: trace minerals

A

<20mg/day (iron, zinc, iodine, copper, manganese, fluoride, selenium, chromium, molybdenum)

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25
Q

Define: anemia

A

iron deficiency

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26
Q

4 roles of water for exercise

A

digestion, temperature control, eliminating waste products, prevents dehydration

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27
Q

Sodium ingestion during exercise helps with what?

A

maintaining or restoring plasma volume during exercise and recovery
- example: Sports drinks help retain water in the body and aid in hydration by increasing the absorption of fluid from intestines → muscles

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28
Q

Why should you be careful in your choice with sports drinks?

A

Too much sugar slows the absorption of water (careful of sports drinks with sugar)

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29
Q

How is fluid lost?

A

through skin (sweat), lungs, urination

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30
Q

Dehydration can lead to decreased blood volume. Why can this be problematic?

A
  • Stroke volume decreases
  • Exercising muscles receive not enough oxygen
  • By-products of exercise are not flushed out as regularly as they should be
  • Exhaustion sets in and performance suffers
  • Losing as little as 2% of total body weight in water can negatively affect athletic performance
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31
Q

Define: hyponatremia

A

too much water

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32
Q

Dehydration

  • early signs
  • late symptoms
  • fill in the blank: waterloss of _____ of body weight may = death
A
  • early: thirst, tiredness/weakness, headache, loss of appetite, dry mouth. dark urine, muscle cramps
  • late: nausea, hot to touch, dizziness, lack of coordination, confusion, fainting
  • > 10-20%
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33
Q

Define: cramping

A

excessive sweating with sodium/mineral loss, muscle twitching and cramps

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34
Q

Define: heat exhaustion

A

prolonged period of fluid loss via activity in high temperatures

35
Q

Define: heat stroke

A

failure of thermoregulatory system
- Sudden collapse, loss of consciousness, flushed hot skin, shallow breathing, rapid pulse, core temp of 106 degc or higher

36
Q

Heat disorder prevention

A
  • Fluid and electrolyte replacement
  • Gradual acclimatization (1 week: 80% acclimatization, 2 weeks = 100%)
  • Monitor temperature humidity
  • Identify susceptible individuals
  • Weight records
  • Uniforms
37
Q

What is the guiding principle of medicine?

A

Primum non nocere

- first do no harm

38
Q

emergency care: ABCD

A

Airway, breathing, circulation, disability

39
Q

What are some general points on emergency care? (5)

A
  • Keep head and neck stable
  • Keep warm
  • NPO: nil per os (nothing by mouth)
  • Get help
  • Serial repetition of ABCD’s record status
40
Q

What is the most common cause of asphyxiation of an unconscious patient?

A

obstruction of the oropharynx by the tongue

41
Q

Define: shock

A

state of insufficient blood flow to the tissues of the body as a result of problems with the circulatory system. Common problems are low blood volume, reduced bp, excessive dilation of blood vessels, low heart rate

42
Q

Signs and symptoms of shock

A
  • Decreased B.P.; weak pulse
  • Hypoxia
  • Reflexive H.R. increase (tachycardia)
  • Cool and clammy skin (warm and dry if its septic shock)
  • Anxiety, thirst, impaired consciousness
43
Q

Treatment of shock

A
  • N.P.O
  • ABCD
  • Maintain body temp, elevate the legs
  • Transport to medical centre ASAP
44
Q

Describe these common medical abbreviations:

  • SSx
  • Hx
  • PHx
  • Ax
  • Dx
  • DDx
  • Tx
A
  • signs and symptoms
  • history of current injury
  • past history
  • assessment
  • diagnosis
  • differential diagnosis
  • treatment
45
Q

Types of joints (6)

A 
Hinge: ankle
Saddle: carpometacarpal joint
Facet: spine
Pivot: vertebrae of the neck
Gliding: tarsal bones of the foot
Ball and socket: shoulder
46
Q

Explain the difference between a open vs closed fracture

A

open: compound
closed: simple

47
Q

Explain the difference between acute vs stress fractures

A
  • traumatic fractures (also called acute fractures) are caused by a direct blow or impact
  • Stress fractures are tiny cracks in a bone. They’re caused by repetitive force, often from overuse
48
Q

Response of bone and joints to stress depends on what? (4)

A
  • Structure (strength and stiffness)
  • Load (magnitude, duration, direction, repetition)
  • Support (muscle activation, equipment)
  • Past history
49
Q

Stress response of tissue depends on what? (4)

A
  • Load: external and internal forces acting on body
  • Mechanical stress: tissue response to forces
  • Viscoelastic properties: amount of resistance to stress
  • Yield point: elasticity of tissue can no longer hold back stress = mechanical failure (strain or sprain)
50
Q

Define: collagen

A

strong structural protein with little elasticity

- Stress is linear in relation to elongation

51
Q

Define: elastin

A

elastic structural protein

- Stress takes longer than collagen to increase in relation to elongation

52
Q

Define: strain

A

stretching and tearing of contractile tissue

53
Q

Define: sprain

A

stretching and tearing of non-contractile tissue

54
Q

Phases of soft tissue injury repair

A
  • Acute inflammatory phase (0-72 hours)
  • Proliferation/repair phase (2 days - 6 weeks)
  • Remodeling/ maturation phase (4 weeks - 6 months, longer?)
55
Q

Define: inflammation

A

protective tissue reaction

56
Q

When is an inflammation considered clinical?

A

if it causes pain or disability

57
Q

Define: rubour inflammation

A

redness caused by increased blood flow

58
Q

Define: calor inflammation

A

heat caused by increased blood flow

59
Q

Define: tumour inflammation

A

swelling caused by fluid accumulation

60
Q

Define: dolor inflammation

A

pain caused by local tissue pressure

61
Q

Define: functio laesa inflammation

A

loss of function due to swelling and tenderness

62
Q

How long does acute inflammation last for?

A

0-72 hours

63
Q

What are the 2 components of acute inflammation?

A
  • vascular events and cellular events
64
Q

Fill in the blank: acute inflammation events are mediated by _______ derived from plasma proteins or cells (_______) and are produced in response to or activated by stimuli

A
  • chemical mediators

- cytokines

65
Q

Describe the vascular events that occur during inflammation (5)
- VSCVI

A
  • Vasoconstriction: blood vessels constrict for a brief period
  • Stasis: increased blood viscosity
  • Coagulation cascade
    (Thromboplastin → prothrombin → thrombin → fibrinogen → insulin fibrin clot)
  • Vasodilation quickly follows (increased blood flow and increased vascular permeability)
  • Increased vascular permeability; hallmark of acute inflammation (protein and fluid leakage from artery)
66
Q

Describe the cellular events that occur during inflammation (4; MRAM)

A
  • Margination: blood vessel dilation; slows blood where vessel has dilated; allows white blood cells to migrate closer to edge of blood vessel
  • Rolling: blood cells start to roll as they get closer to edge of blood vessel and pick up endothelial receptors with their own receptors
  • Adhesion: blood cells stick to endothelial cell wall
  • Migration (diapedesis/extravasation) → chemotaxis: endothelial cell walls expand which allows leukocytes to squeeze through; Chemotaxis: Leukocytes follow chemical signals being released by foreign agents or damaged cells (injury) to site of injury
67
Q

What are the benefits to inflammation?

A

supplies things to injury site:

  • clotting factors
  • anti-infection material and processes (leukocytes, phagocytes (macrophages), antibodies, stick microorganisms to phagocytes, attack foreign cells (put holes in their membranes)
  • nutrients (O2, amino acids, glucose, etc.)

Stimulates proliferation of endothelial cells (neovascularization) and fibroblasts (cells that synthesize collagen and extracellular matrix)

68
Q

cons of inflammation

A

Excessive or chronic inflammation may lead to tissue destruction, fibrosis, and necrosis (death)

edema: swelling caused by extra fluid trapped in body’s tissue
- Restricts blood flow if no room for swelling
- Produce pain, which can limit movement

69
Q

Describe the difference between onset, duration, and outcomes for acute and chronic inflammation

A

acute:

  • onset: immediate
  • duration: few days
  • outcomes: resolution, abscess formation (pus), chronic inflammation

chronic:

  • onset: delayed
  • duration: up to many months/ years
  • outcomes: tissue destruction, fibrosis, necrosis
70
Q

Explain 3 methods of treatment to inflammation

A
  • Remove underlying cause
  • Treat local effects
  • Treat systemic effects
71
Q

What are some ways to remove the underlying causes of inflammation

A
  • incise and drain abscess
  • Antibiotics
  • Remove foregin body
  • Remove mechanical stress
72
Q

What are some ways to treat local effects of inflammation

A
  • P.O.L.I.C. E. then heat
    (Protect, optimal loading, ice, compression, elevation)
  • N.S.A.I.D. (nonsteroidal anti-inflammatory medications)
  • Physiotherapy, chiropractic, RMT, acupuncture, naturopath
73
Q

What are some ways to treat systemic effects of inflammation

A
  • Antipyretics: medication to fight fever (ASA, acetaminophen)
  • Analgesics: pain medication (NSAID, narcotics)
  • Antibiotics: fights bacterial infection
74
Q 
Itis?
Myositis
Arthritis
Tendonitis
Bursitis 
Vasculitis
Dermatitis
A

itis = inflammation
Myositis: inflammation of the muscles that you use to move your body
Arthritis: joint inflammation
Tendonitis (proper term: tendinopathy, in most cases it is tendinosis): inflammation of a tendon
Bursitis: bursa inflammation
Vasculitis: blood vessel inflammation
Dermatitis: skin inflammation causing redness and itchyness

75
Q

Describe the proliferation/ repair phase

A
  • Characterized by proliferation of capillaries (neovascularization) and fibroblasts which synthesize granulation tissue aka scar tissue (a collagen and extracellular matrix with cross-linking)
  • As the quantity of collagen at the injury site increases, the number of fibroblasts decrease
  • Mobilization or stress applied to the healing tissue decreases the number of cross-links and increases the strength of the tissue
  • However, early mobilization may negatively impact the healing process by prematurely reducing the number of cross links
  • several months to years
76
Q

Describe early scar tissue structure

A
  • More type 3, less type 1
  • Weak (H-bonds, rather than covalent bonds, between collagen fibres) and susceptible to re-injury
  • Poorly vascularized
  • Disorganized
77
Q

Describe the maturation phase after a sprain

A
  • aka remodelling phase
  • occurs 1-2 weeks post injury up to a year+
  • Long term process that involves realignment and maturation of collagen fibres that make up scar tissue
  • Myofibroblasts re-orient collagen fibrils in the direction of loading; increased stress and strain causes collagen fibres to realign to position of maximum efficiency. Parallel to lines of tension; Gradually assumes more normal appearance and function
  • Lasts 10-12 months, total maturation phase may take years to be fully complete
78
Q

How does movement help the maturation phase? (3)

A
  • increases synthesis
  • increases lysis
  • reorganizes (decreases in weaker type 3 collagen and an increase in stronger type 1 collagen)
79
Q

What are the benefits of icing after exercise

A
  • Vasoconstriction (hunting response)
  • Decreased bleeding into injured tissue
  • Decrease in inflammatory response
  • Decreased swelling
  • Decreased pain - decreases excitability of free nerve endings (nociceptors)
  • Decreased muscle spasms
  • Decreased cellular damage - decreased secondary tissue hypoxia
  • Decrease or delay of cell death
80
Q

Implications of cold on performance

A
  • Decreased pain sensation
  • Golgi tendon organ less sensitive (numb pain)
  • Muscle spindles less sensitive
  • Increase stiffness at myotendinous junction
  • Muscle more susceptible to fatigue if cooled below 25degC
81
Q

What are the benefits of heat after exercise

A
  • Vasodilation: increased blood flow
  • Increase in inflammatory response
  • Increased swelling
  • Decreased pain
  • Decreased muscle spasm
  • Decreased stiffness of soft tissues
  • Increase metabolism within cells that are warmed
82
Q

Fill in the blank: Critical temperature threshold for beneficial effects of heat is ____; Can be achieved through 15 minutes of ________

A
  • 39degC

- general exercise (dynamic warm up)

83
Q

Describe gross vs fine motor skills

A

Gross motor skills pertain to skills involving large muscle movements, such as walking, or running. Fine motor skills involve use of smaller muscles, such as grasping, object manipulation, or writing

84
Q

Describe open vs closed motor skills

A

Closed: static environment (running on a treadmill in your home gym)
open: dynamic environment (running a race outdoors alongside many other runners)

BPK 241 (7 decks)

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