Week 1-Human Digestive & Absorption, Anatomical Structures and Physiological Structures Flashcards

1
Q

GIT

A

The gastrointestinal tract - mouth, esophagus, stomach, small intestine, large intestine, rectum anus
accessory organs - liver, pancreas and gall bladder

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2
Q

Oral Cavity (mouth)

A
  • chewing increases surface area
  • when mixed with saliva food becomes bolus
  • saliva
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3
Q

Saliva

A

– Lysozymes: Break down bacteria
– Mucus: Lubricates and holds bolus together
– Salivary amylase: starts breaking down starch
– Lingual lipase: starts breaking down triglycerides

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4
Q

Sphincters

A

– Lower esophageal sphincter
– Pyloric sphincter
– Sphincter of Oddi
– Ileocecal sphincter (or “valve”) – Anal sphincter

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5
Q

Lower esophageal sphincter

A

Prevent backflow (reflux) of stomach contents into esophagus

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6
Q

Pyloric sphincter

A

Control the flow of stomach contents into the small intestine

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7
Q

Sphincter of Oddi

A

Control the flow of bile from the common bile duct into the small intestine

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8
Q

Ileocecal sphincter

A

Prevent the contents of the large intestine from reentering the small intestine

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9
Q

Anal sphincters

A

Prevents defecation until person desires to do so

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10
Q

GIT Motility

A
  1. Peristalsis
  2. Segmentation
  3. Mass movements
  4. Elimination via sphincters
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11
Q

Peristalsis

A

Contractions of the GIT wall muscle for movement of bolus and chyme
• Peristalsis is triggered by the autonomous nervous
system. Hydration and bulk of feces play a role:
stretching of intestinal wall promotes peristalsis

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12
Q

Segmentation

A

Back and forth movement to allow breaking down

of bolus and chyme into smaller particles so enzymes can access

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13
Q

Mass Movement

A

Peristalsis over widespread area

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14
Q

Digestion

A

Process of breaking down foods into molecules that can be absorbed by erythrocytes and delivered into blood and lymphatic system

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15
Q

Absorption

A

uptake of monomers and micro-nutrients from the lumen of the GIT through absorption cells (enterocytes) into blood or lymph for transport to organs/cells
Ageing= decreased HCL, therefore decreased absorption and digestion

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16
Q

Pathway of food

A

• Food mixed in the bolus goes through lower
eosophageal sphincter into stomach
• Mixed with stomach secretions becomes chyme
• Pyloric sphincter allows chyme into the small intestine
• Gastric inhibitory peptide (GIP) slows the release of
the chyme from the stomach into the small intestine

17
Q

Gastrin:

A

Hormone that controls release of HCl and

pepsinogen

18
Q

Parietal cells

A

produce hydrochloric acid (HCl)

19
Q

HCL

A

• Denatures proteins
• Destroys bacteria and viruses
• Converts pepsinogen into the active form pepsin:
enzyme for protein digestion
• Aids in minerals absorption
• Chief cells: produce and release gastric lipase

20
Q

Mucus:

A

Protects the stomach from “being digested” by HCL

21
Q

Chief cells

A

produce and release gastric lipase

22
Q

Small intestine

A

• Site of most of the digestive and absorptive processes
• Three sections: duodenum, jejunum and ileum: a total
of 6m in length
• Villi and microvilli on the intestinal wall: increase the
surface area of the lumen by 600 times to optimise the
absorption capacity

23
Q

Goblet cells:

A

produce mucus

24
Q

Endocrine cells

A

produce hormones

25
Q

Enterocytes

A

absorb nutrients and produce

metabolism enzymes