W2: ECG Interpretation Flashcards
Acute Coronary Syndrome Conditions
Unstable Angina
NSTEMI
STEMI
ACS MOA
coronary obstruction caused by rupture atehrosclerosis plaques. = clots
mild/brief ischemia leads to __
angina
prolonged ischemia leads to ___
MI
changes in ECG
release of cardiac enzymes
degree of ischemia is determined by
degree of occlusion
oxygen demand of myocardium
arterial occulusion signifance at ___ %
70
rupture of plaques –> thrombus =
dec lumen and dec BF
Unstable Angina
form of ACS (NSTEMI)
transient episodes of thrombotic vessel occlusion and vasoconstriction at site of plaque damage
sign of impending MI
UA: diagnostic results
ST depresison
T wave inversion
Types of MI’s
subdenocardial infarction (NSTEMI)
transmural infarction (STEMI)
Subdenocardial Infarction
thrombus disintegrates before full distal tissue necrosis
involves only the myocardium directly below the endocardiu
- partial wall thickness damage
ST depression
T wave inversion (w/o st elevation)
Transmural Infarction
thrombus lodges permanently in vessel
infarction extends from myocardium to endocardium to epicardium (full wall thickness damage)
need intervention!!!!
A Fib: Patho
rapidly firing action potentials in the atrial myocardium d/t myocardial remodelling (HTN, valvular/ischemic disease, genetics)
irregular rapid HR - poor blood flow
AFib: ECG
absent p waves
irregular R-R intervals
fibrillatory waves
high ventricle rate (400-600bpm)
AFib: Hemodynamics
inc risk for stroke
HF
Atrial Flutter: Patho
atrias contract rapidly (not all conduct to ventricles)
ventricle response varies
rapid regular rhythm
should suspect Atrial flutter in anyone with a resting HR >150
Atrial Flutter: ECG
sawtooth flutter at regular rate
Atrial Flutter: Hemodynamics
inc HR = cardiac insuffiency
Premature Arterial Cx (PACs): Patho
depolarization insitgated outside the SA node (early heartbeat initiated by atria)
PACs: ECG
premature + abnormal p wave
normal QRS complex
compensatory pause (artirial bigeminy)
PACs: Hemodynamics
benign but disrupted cardiac rhythm
PVCs: patho
venticles generate action potential before SA node ( early heartbeat initated by the ventricles)
PVCs: ECG
no p wave
wide/abnormal QRS
irregular R-R Interval
compensatory pause (skipping a beat)
PVC: Hemodynamics
benign, palpitations
VTach: Patho
rapid heartbeats from ventricles
ventricle rate >100bpm
common in people with structural heart disease
VTach: ECG
regular rhythm
no p wave
wide and abnormal QRS
VTach: Hmeodynamics
hemodynamic instability - hypotension- life threatning (emergency!!!!!!)
VTach: monomorphic
same shape/symmetry of QRS b/c start in same place in myocardium
VTach: polymorphic
variable QRS b/c depolarization at multiple points
Vfib: Patho
disorganzied contraction of ventricle muscle fibres
d/t CAD, MI (ischemia, scarring). tissue damage creates reentry patterns –> chaotic depolarizations
VFib: ECG
no identifiable waves or complexes
VFib: hemodynamics
extremely compromised CO, low chance of survival outside of hospital
First Degree Block: overview
results from slow action potential conduction through AV node d/t damage diease causing changes in structure/vagal tone.
takes a min, but all impulses get through
asymptomatic, no tx
First Degree Block: ECG
prolonged PR interval (>0.2s)
Second Degree Block: Overview
changes in PR interval, >0.2s
some impulses don’t reach ventriclesS
Second Degree Block: ECG
some p waves not followed by QRS complex
SDB: Mobits I
PR interval increases until no QRS complex
pause
cycle repeats
benign, low vagal tone
SBD: Mobitz II
blocked P wave
PR Interval constant
issues with bundle brances
tx: pacemaker
Third Degree (Complete Block)
complete block, no atrial impulses to ventricle. must generate on impulses to survive.
AV disassociation. No P wave have QRS complexes
atrial rate > ventricular rate
d/t ischemia, disease
CM: LOC, dizziness
tx: pacemaker
Amiodarone: MOA
blocks potassium + sodium channels
and beta-adrenergic + calcium channels –> dec HR and resistance
Amiodarone: Indication
VTach
Atrial dysrythmmias
PVCs
AFib/Flutter
Amiodarone: DE
cardiac stabiliy
normal heart rhyth,
increased VF threshold
Amiodarone: AE
thyroid d/o
liver toxicity
drug interaction
bradycardia
pulmonary fibrosis
hypotension
QT prolongation
cyanosis
photosensitivity
rashes
fatigue