W2: ECG Interpretation Flashcards
Acute Coronary Syndrome Conditions
Unstable Angina
NSTEMI
STEMI
ACS MOA
coronary obstruction caused by rupture atehrosclerosis plaques. = clots
mild/brief ischemia leads to __
angina
prolonged ischemia leads to ___
MI
changes in ECG
release of cardiac enzymes
degree of ischemia is determined by
degree of occlusion
oxygen demand of myocardium
arterial occulusion signifance at ___ %
70
rupture of plaques –> thrombus =
dec lumen and dec BF
Unstable Angina
form of ACS (NSTEMI)
transient episodes of thrombotic vessel occlusion and vasoconstriction at site of plaque damage
sign of impending MI
UA: diagnostic results
ST depresison
T wave inversion
Types of MI’s
subdenocardial infarction (NSTEMI)
transmural infarction (STEMI)
Subdenocardial Infarction
thrombus disintegrates before full distal tissue necrosis
involves only the myocardium directly below the endocardiu
- partial wall thickness damage
ST depression
T wave inversion (w/o st elevation)
Transmural Infarction
thrombus lodges permanently in vessel
infarction extends from myocardium to endocardium to epicardium (full wall thickness damage)
need intervention!!!!
A Fib: Patho
rapidly firing action potentials in the atrial myocardium d/t myocardial remodelling (HTN, valvular/ischemic disease, genetics)
irregular rapid HR - poor blood flow
AFib: ECG
absent p waves
irregular R-R intervals
fibrillatory waves
high ventricle rate (400-600bpm)
AFib: Hemodynamics
inc risk for stroke
HF
Atrial Flutter: Patho
atrias contract rapidly (not all conduct to ventricles)
ventricle response varies
rapid regular rhythm
should suspect Atrial flutter in anyone with a resting HR >150
Atrial Flutter: ECG
sawtooth flutter at regular rate