VT Flashcards

1
Q

Definition?

A

Constant rapid ventricular contraction rates, can be sustained or non-sustained. Sustained VT is a ventricular rhythm faster than 100 bpm lasting 30 secs or needing to be terminated earlier due to haemodynamic instability.

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2
Q

RF?

A
  • CAD
  • AMI
  • LV systolic dysfunction
  • Hypertropic cardiomyopathy
  • Long/short QT syndrome
  • Brugada syndrome
  • Ventricular pre-excitation
  • Arrhythmogenic right ventricular cardiomyopathy
  • Electrolyte imbalance
  • Drug toxicity
  • Chagas disease/cardiomyopathies
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3
Q

ddx?

A
  • SVT aberrancy-doesn’t meet criteria of VT
  • SVT with pre-excitation-“
  • Electrical artefact-motion or tremor
  • Sepsis-sinus rhythm and cultures
  • Panic-sinus rhythm and A/D scores high
  • Hyperthyroidism-snus rhythm or AF-raised TSH and low TSH
  • Acute haemorrhage-sinus, bleeding
  • Pheochromocytoma-high adrenaline, sinus tachy, evidence of tumour or high steroid hormones
  • Pericarditis-concave up ST elevation and PR depression and CXR/echo findings
  • Caffeine-sinus tachy
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4
Q

EPIDEMIOLOGY?

A

Age: Elderly
Sex: Male
Ethnicity:

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5
Q

Aetiology?

A
  • Cardiac scars
  • Conduction disorders
  • Drugs
  • Long QT syndrome
  • Congenital long-QT syndrome
  • Acquired long QT syndrome
  • Drugs
  • Antiarrhythmics
  • Electrolyte imbalances
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6
Q

CP?

A
  • Asymptomatic if non-sustained
  • Palpitations
  • Hypotension
  • Syncope
  • Chest pain/pressure
  • Cardiogenic shock
  • Loss of consciousness
  • Progression to VF
  • Sudden cardiac failure
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7
Q

Pathophysiology?

A
  • Monomorphic VT
  • Re-entry circuit
  • Polymorphic VT-abnormal ventricular repolarisation
  • Decreased cardiac output-non-synchronised rhythm leads to less blood flow into ventricles hence low cardiac outputs causing haemodynamic compromise and so signs of this show
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8
Q

Investigations?

A
  • ECG-
    • wide complex tachycardia (QRS 120 milliseconds or greater) at a rate of 100 bpm or greater; may show presence of atrioventricular dissociation,
    • previous myocardial infarction;
    • QRS duration: >140 milliseconds with right bundle branch block morphology, or
    • QRS duration >160 milliseconds with left bundle branch block morphology (this does not apply to patients on anti-arrhythmic drugs);
    • right superior axis or left bundle branch block morphology and any right axis;
    • baseline ECG may demonstrate QT interval prolongation or evidence of Brugada syndrome or arrhythmogenic right ventricular cardiomyopathy
  • Electrolytes-hypokalaemia and hypomagnesaemia
  • Troponin I
  • CK-MB
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9
Q

Management all?

A
  • Cardioversion

* Anti-arrhythmics-amiodarone

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10
Q

management torades de pointes?

A
  • IV mg sulphate and correct electrolyte imbalances
  • Isoprenaline infusion
  • Temp/permanent pacing
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11
Q

management catecholamines?

A

• Beta-blockers
• Implantable cardioverter defib
Catheter ablation

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12
Q

Non-sustained?

A

• Electrolyte imbalances correction
• Catheter ablation
ICD

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13
Q

Prognosis?

A

• Idiopathic-good-should be evaluated carefully or can be ablated to be cured
• Non-idiopathic-high risk of death if left untreated
Heart disease adversely affects prognosis

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14
Q

Complications?

A
• Malfunction of implantable cardioverter defibrillator system malfunction
• V fib
• Sudden cardiac death
• Infection
• Cardiomyopathy
Amiodarone-thyroid dysfunction
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