Voice Flashcards

1
Q

Multidisciplinary roles in voice evaluation

A
  • ENT evaluation completed first: provides laryngeal diagnosis and medical management plan
  • SLP provides voice therapy plan
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2
Q

What are the components of a voice evaluation?

A
  • Medical exam
  • Patient interview
  • Perceptual evaluation
  • Patient self-evaluation
  • Acoustic/aerodynamic measurements
  • Vocal fold movement/function
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3
Q

What are the goals of a voice assessment?

A
  • Determine etiology, physiology, or behavioral factors that created the disorder
  • Describe the voice symptoms
  • Evaluate how the disorder impacts each subsystem
  • Develop a management plan
  • Educate and motivate
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4
Q

What information does an SLP collect during the case history?

A
  • Description of problem/cause (associated factors, time of onset, duration/consistency of problem)
  • Medical history
  • History of previous voice disorders
  • Occupational/social voice use
  • Phonotrauma checklist
  • Vocal use habits
  • Family support
  • General health habits
  • Emotional state/stress level
  • Hearing
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5
Q

What “checklist” or “index” should complete during a voice eval? Provide rationales for selecting each one.

A

Reflux Symptom Index
- This helps identify whether reflux may be contributing to voice problems.

Vocal Behavior Checklist
- Evaluates vocal behaviors and habits that could negatively impact the voice, such as excessive talking, loud talking, throat clearing, or smoking. It provides insight into factors that may be contributing to vocal strain or injury, guiding recommendations for voice therapy or lifestyle changes.

Vocal Handicap Index
- The VHI measures the perceived impact of voice problems on a person’s daily life, including their social, emotional, and physical well-being.

Vocal Fatigue Index-2
- The VFI-2 evaluates the level of vocal fatigue or strain a person experiences, including symptoms like vocal tiredness, discomfort, or reduced endurance during speech. It assists in identifying individuals who may be overusing their voice or experiencing signs of vocal overexertion, which can inform treatment plans for preventing further vocal damage.

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6
Q

What should SLP observe throughout case history?

A
  • Anxiety/tension
  • Neck or laryngeal tension
  • Signs of hand, limb, or head tremor
  • Respiratory behavior during speech
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7
Q

What is the CAPE-V? Rationale for administration

A
  • Standardized tool used to assess auditory-perceptual characteristics - overall severity, pitch, loudness, roughness, breathiness, strain.
  • SLP rates these qualities based on spoken samples (vowel, sentences, conversational sample)
  • Establishes a baseline assessment of voice & allows for monitoring changes over time
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8
Q

What does each CAPE-V attribute indicate? What additional features should an SLP listen form?

A

Roughness: irregularity in the voicing source
Breathiness: audible air escape
Strain: hyperfunction

Additional –> diplophonia, vocal fry, asthenia, aphonia, pitch instability, tremor, wet/gurgly

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9
Q

What are two measures to collect during eval that assess laryngeal function?

A

S/Z ratio: estimate of laryngeal efficiency
- Sustain /s/ as long as possible; /z/ as long as possible
- Divide longest /s/ by longest /z/
- Indicated of efficient airflow during phonation

Maximum phonation time: respiratory capacity during speech
- Longest sustaining of /a/ at comfortable pitch & loudness

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10
Q

What additional parameters should be measured during a voice eval? (4)

A

Pitch
- Pitch glides
- Compare to average range

Volume
- Use sound level meter (compare to conversational level 60-65 dB)

Rate
- May contribute to laryngeal pathology

Resonance
- Assess presence of hyper/hyponasality
- Nonasal/nasal phrases

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11
Q

What is the ADSV? What values does it provide?

A

Analysis of Dysphonia in Speech and Voice: program that performs cepstral and spectral analyses

  • Noise-to-harmonics ratio: identifies dysphonic voice if harmonic energy doesn’t stand out from noise elements
  • Spectral slope: amplitudes of high frequency energy in a signal
  • Cepstral peak prominence: amplitude of peak relative to amplitude of overall signal energy
  • Cepstral-spectral Index of Dysphonia (CSID): represents acoustic dysphonia severity (higher score = dysphonic)
  • Fo: rate at which VFs vibrates during voiced speech sounds
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12
Q

Endoscopy

A

Direct visualizzino of vocal folds and laryngeal area

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13
Q

Flexible vs rigid endoscopy

A

Flexible (nasolaryngoscopy):
- Visualization of nasal cavity, soft palate, pharyngeal walls
- VFs during connected speech

Rigid:
- Light source brighter than flexible
- Better visualization of VF tissue (lesions)
- CANNOT visualize VFs during connected speech
- Often used with stroboscopy

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14
Q

Stroboscopy

A

Vocal fold movement patterns & mucosal wave

Samples different parts of the vibratory cycle (slow motion movement of VFs)

Assesses vibratory function:
- Regularity, amplitude, mucosal wave, symmetry, vertical level, glottal closure, supra glottal activity, VF edge, VF mobility

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15
Q

List/explain vibratory function parameters
(Hint –> F, S, M, R, A, M, S, V, GC, PC)

Pneumonic –> Friendly Superheroes Make Really Amazing Moves, Saving Villages, Giving Courage, Protecting Cities

A
  1. Free edge contour: smoothness of free edge (**non-linear edge contour can interfere with glottal closure)
  2. Supra-glottic activity: constriction of supraglottic structures
  3. VF mobility: movement of each VF toward and away from midline (normal, reduced, absent)
  4. Regularity: consistency of cycles
  5. Amplitude: magnitude of lateral movement of VFs (**reduced amplitude due to INCREASED mass/stiffness, incomplete glottal closure)
  6. Mucosal wave motion: magnitude of movement of the mucosal membrane
  7. Phase symmetry: symmetry of motion during phonation
  8. Vertical level: whether VFs meet on same vertical plane during closed phase
  9. Glottal closure: appearance of glottis during closed phase of vibration
  10. Phase closure: duration of different phases during glottal cycle
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16
Q

What does Color High-Speed Video Endoscopy record? List advantages/disadvantages.

A

Records every vibratory cycle (accurately represents aperiodic vibration) and provides highly detailed views of VF vibration

Advantages:
- Valid for ALL voices
- Detailed assessment of symmetry/periodicity
- Detailed view of lesions

Disadvantages:
- Uses memory/storage
- Limited recording time
- Costly
- Requires use of rigid endoscope

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17
Q

What does Videokymography record?

A
  • 2-dimensional display of HSV data
  • Tracks changes in 2D image over time
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18
Q

Explain Electroglottography - What does it measure

A
  • Small, high frequency electrical signal passed between 2 electrodes
  • Measures varying VF contact patterns
  • Extracts fundamental frequency
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19
Q

What does Electromyography measure?

A
  • Functioning of laryngeal muscles
    **Analysis of VF paralysis
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20
Q

What instrument can be used to assess aerodynamic measures?

A

Pneumotachograph: airflow mask w/built in pressure sensors; determines change in pressure across a known resistance

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21
Q

Define the following aerodynamic measures - airflow, pressure, laryngeal resistance

A

Airflow: at midpoint of vowel, flow across glottis

Pressure: measure at lips, estimate subtotal pressure

Laryngeal resistance: pressure/airflow

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22
Q

What tool is used to assess nasality?

A

Nasometer:
- Measures amount of air escaping through nasal passages while a person speaks
- Compares acoustic energy coming from nose to energy coming from mouth
- Ratio provides insight into how much nasal resonance is present

**hypernasal: too much air escapes through noise
**hyponasal: too little air passes through nose

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23
Q

What tool can be used to measure lung volume?

A

Plethysmography: estimates lung volume levels during speech from chest wall excursion

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24
Q

List 4 common pediatric voice etiologies

A
  1. Laryngomalacia: immature laryngeal cartilage (most common cause of infant inspiratory stridor)
  2. Laryngeal webbing: membranous tissue connecting VFs
  3. Laryngeal cleft: opening between larynx and esophagus
  4. Puberphonia: high-pitched voice during puberty
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25
Q

Define functional voice disorder

A

Voice disorder in the absence of neurologic or structural pathology of larynx

OR

Laryngeal pathology develops secondary to vocal misuse

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26
Q

What is phonotrauma? Provide 3-5 examples

A

Using phonatory and other systems in an ineffective manner

Examples:
- Hard glottal onsets
- Habitual use of glottal fry
- Excess tension in laryngeal muscles
- Loud talking
- Talking in too high/low pitch

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27
Q

Vocal Nodules - Etiology

A

Etiology:
- Phonotrauma
- Elevated anxiety OR extraversion
- Caffeine intake
- Allergies
- GERD

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28
Q

Vocal Nodules - Tissue features

bilateral or unilateral?
visual appearance (early vs later)
location
BMZ affected?

A

Tissue features:
- Bilateral
- White, callous-like swellings on vibratory margin
- Increased mass & stiffness on VF cover
- Located –> juncture of anterior 1/3 and posterior 2/3 of VF margin (point of greatest impact)
- Early = soft/reddish…..Later = white/hard
- BMZ = 3x normal thickness

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29
Q

Vocal Nodules - Vibratory features/Aerodynamics

A

Vibratory:
- Incomplete GC
- Aperiodic vibration
- (+/-) Decreased amplitude or symmetry

Aerodynamics
- Increased airflow
- Decreased pressure
- (+/-) Changes in laryngeal resistance

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30
Q

Vocal Nodules - Perceptual/Acoustic features

A
  • Breathy, rough
  • (+/-) Strained, pressed
  • Reduced pitch
  • Reduced pitch range & control
  • Reduced loudness levels & range
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31
Q

Vocal Polyps - Etiology

A

Phonotrauma
- Single event of excessive use
- HIGH association w/smoking

OR

Unknown

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32
Q

Vocal Polyps - Tissue features

2 types
unilateral vs bilateral
visual appearance
location
BMZ affected?

A

Sessile: closely adhering to mucosa, broad based
Pedunculated: attached by a slim stalk

  • Pliable fluid-filled sacks (vascular, edematous)
  • Unilateral
  • Location –> anterior 1/3 of VF
  • NO affect on BMZ
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33
Q

Vocal Polyps - Vibratory/Aerodynamic features

A

Vibratory:
- Incomplete GC
- Aperiodic vibration
- (+/-) Decreased amplitude or symmetry

Aerodynamics:
- Increased airflow
- Decreased pressure
- (+/-) Changes in laryngeal resistance

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34
Q

Vocal Polyps - Perceptual/Acoustic features

A
  • Breathy, rough
  • (+/-) Strained/pressed
  • Overall pitch & pitch range decreased
  • Decreased loudness level & range
  • Diplophonia
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35
Q

Reinke’s Space Edema

What is it?
Etiology

A

Generalized edema along the entire length of the VFs

Etiology:
- Most often associated with smoking (chronic mucosal irritation)

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36
Q

Reinke’s Space Edema - Tissue features

A
  • Generalized edema in the superficial layer of lamina proprietary (Reinke’s space)
  • Increase in tissue mass
  • DECREASED stiffness
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37
Q

Reinke’s Space Edema - Vibratory/Aerodynamic features

A

Vibratory:
- Incomplete VF closure
- Asymmetry/aperiodicity

Aerodynamics:
(+/-) Changes in airflow
- May see increased laryngeal resistance

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38
Q

Reinke’s Space Edema - Voice features

A
  • Significantly rough, breathy, strained
  • LOW pitch
  • Decreased pitch & loudness range
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39
Q

Traumatic Laryngitis

What is it?
Etiology

A

Inflammation of VFs and larynx

Etiology:
- Phonotrauma (screaming, excessive smoking/drinking)
- Inhaled chemical exposure
- Chronic allergies
- GERD

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40
Q

Traumatic Laryngitis - Tissue features

A
  • VFs are edematous and reddish
  • Chronic = VF thickening
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41
Q

Traumatic Laryngitis - Vibratory/Aerodynamic features

A

Vibratory:
- Incomplete VF closure
- Asymmetry and aperiodicity

Aerodynamics
- Increased laryngeal resistance

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42
Q

Traumatic laryngitis - Voice features

A
  • Rough, breathy, strained
  • Reduced pitch/pitch range
  • Reduced loudness level/range
  • Voice WORSENS w/prolonged speaking
  • Chronic THROAT clearing
43
Q

Vocal Fold Hemorrhage

What is it?
Etiology

A

Bleeding into VF due to burst blood vessel

Etiology:
- Often from single episode of traumatic use
- Intubation, laryngeal trauma
- Hormonal predisposition
- Overuse of aspirin prior to heavy voice use

44
Q

Vocal Fold Hemorrhage - Tissue features

A

Hemmorhagic area - reddish in color & edematous

  • Usually unilateral
45
Q

Vocal Fold Hemorrhage - Vibratory/Aerodynamic features

A

Vibratory:
- Reduced amplitude
- Increased stiffness
- Incomplete VF closure
- Asymmetry/aperiodicity of vibration

Aerodynamics:
- Variable effects on pressure, airflow, resistance

46
Q

Muscle Tension Dysphonia

What is it?
Etiologies

A

Excessive laryngeal musculoskeletal tension

Etiology:
- Anxiety, GERD, infection, edema, posture

47
Q

MTD - Tissue features (primary vs secondary)

A

Primary: muscle misuse patterns WITHOUT observable secondary pathology

Secondary: occurs w/other pathology (e.g., nodules, polyps, chronic laryngitis, neurologic disorder)

48
Q

MTD - Vibratory/Aerodynamic features

A

Vibratory:
- Supraglottic hyperfunction

Aerodynamics:
- Increased laryngeal resistance
(+/-) Changes in pressure/airflow (dependent on patterns of constriction)

49
Q

MTD - Voice/Clinical features

A
  • Palpable tension in extrinsic muscles
  • Strained, tense, rough
  • Worsens w/prolonged speaking
  • HIGH pitch
  • Decreased loudness level/range
50
Q

What are two models of hyperfunction?

A

Phonotraumatic hyperfunction: likely to result in subsequent pathology (GREATER collision forces)

Nonphonotraumatic vocal hyperfunction: produces dysphonia, less likely to result in subsequent pathology

51
Q

Adducted vs Non-adducted Hyperfunction

A

Adducted: TVF tightly approximated
- Supraglottic compression

Non-adducted: TVF tight/tense but incompletely adducted
- With or without supraglottic compression

52
Q

Patterns of Hyperfunction - Medial/Lateral vs Anterior-Posterior

A

Medial/Lateral Compression:
- Hyper-adduction is side to side manner
- Medial compression occurs at FVF and/or TVF

Anterior-Posterior Contraction:
- Anterior to posterior contraction
- Reduced space between epiglottis and arytenoid prominences

53
Q

Psychologically-Related Voice Disorder

Define
Clinical Presentation
Associated w/

A

Voice symptoms in the absence of any physical, structural, or organic problems

Clinical Presentation:
- Dysphonic or aphonic
- Normal phonation or vegetative tasks

Associated With:
- Sudden onset
- Traumatic emotional event
- Upper respiratory infection

54
Q

Puberphonia

A

Adolescent raises pitch to or above prepuberty level

Clinical Presentation
- Excessively high pitch

55
Q

Define organic voice disorder

A

Voice disorder resulting from organic diease/trauma causing structural abnormalities of the vocal folds

56
Q

Infectious Laryngitis - Etiology/Tissue Features

A

Etiology: bacterial or viral infection

Tissue Features:
- Edema: swollen VFs (exacerbated by coughing)
- Erythmea: reddened VFs (irritation, dilated blood vessels)

57
Q

Infectious Laryngitis - Vibratory/Aerodynamic Features

A

Vibratory:
- Incomplete VF closure
- Asymmetry/aperiodicity

Aerodynamics:
- Increased laryngeal resistance

58
Q

Infectious Laryngitis - Voice Features

A

**Same as traumatic laryngitis:
- Rough, breathy, strained
- Reduced pitch/pitch range
- Reduced loudness level/range
- Voice worsens w/prolonged speaking
- Chronic throat clearing

59
Q

Infection Laryngitis - Recovery/Treatment

A

Recovery: swelling resolves within 7-14 days

Treatment:
- Vocal rest
- Modified speaking habits: soft voice, breath support
- Increased hydration (water intake, humidifier)

60
Q

Papilloma

What is it?
Etiology
Types

A

Wart-like growth

Etiology: viral (HPV)

Subglottal, glottal (impedes VF vibration/respiration), and respiration

61
Q

Papilloma - Tissue, Vibratory Features

A
  • Stiff tissue in affected region
  • Decreased VF amplitude, aperiodicity
62
Q

Papilloma - Voice Features

A
  • Breathy, harsh
  • Can be aphonic
    (+/-) Low pitch
    (+/-) Stridor during inspiration
63
Q

Papilloma - Treatment

A
  • Surgical & antiviral agents
  • Voice therapy to reduce hyperfunctional/compensatory behaviors
64
Q

Laryngo-pharyngeal reflux

Tissue features
Symptoms

A
  • Reddening of posterior 1/3 of VFs and arytenoids
  • Voice often worse in morning
65
Q

Granuloma

What is it?
Etiology

A

Vascularized growth in response to tissue damage

Etiology:
- Occurs after initial contact ulcer
- Tissue healing response

66
Q

Granuloma - Tissue, Vibratory, Aerodynamic Features

A
  • Unilateral mass on cartilaginous portion of VFs
  • May impeded VF closure
    (+/-) Decreased vibratory amplitude
  • Increased airflow, decreased pressure, DECREASED resistance
67
Q

Granuloma - Voice Features

A

May NOT affect voice

  • Breathy, pressed, effortful
  • Intermittent dysphonia
  • Vocal fatigue
  • Decreased pitch/decreased dynamic range
    (+/-) Laryngeal apin
68
Q

Granuloma - Treatment

A
  • Medical management of reflux
    (+/-) Surgery
  • Voice therapy (reduced hard glottal attacks, train improved phonatory patterns)
69
Q

VF Cyst - Etiology/Types

A

Etiology:
- Congenital
- Blocked granular duct (retention of mucous)
- Traumatic contact

2 Types: mucous retention or epidermoid cysts

70
Q

VF Cyst - Tissue Features

Shape/color
Unilateral vs bilateral
Affected location

A
  • Opaque and spherical
  • Usually occur unilaterally
  • Affect glottal margin or superior/inferior surfaces of VFs
71
Q

VF Cyst - Vibratory/Aerodynamic Features

A
  • Rigidity of involved VF
  • (+/-) Increased airflow, decreased pressure
72
Q

VF Cyst - Voice Features/Treatment

A
  • Breathy, strained, raspy
  • Decreased pitch

Surgical management + voice therapy

73
Q

Laryngeal Web

What is it?
Two forms

A

A band of tissue connecting VFs

Congenital Form:
- Failure of normal tissue reabsorption in embryonic development

Acquired Form:
- Bilateral trauma of VFs (can occur after surgery)
- Prolonged trauma: tissue grows together

74
Q

Laryngeal Webbing - Tissue, Vibratory, Aerodynamic Features

A
  • Starts at anterior commissure
  • Inhibits normal vibration, reduced amplitude
  • Reduced airflow
    (+/-) Changes in pressure
75
Q

Laryngeal Webbing - Voice Features

A
  • High pitch
  • Stidor/respiratory distress
76
Q

Laryngeal Webbing - Treatment

A
  • Surgical management: cut webbing & place wedge to prevent fusion
  • Voice therapy to optimize voice given structurally altered system
77
Q

Laryngomalacia

A

Most frequent congenital condition –> immature cartilage development

78
Q

Sulcus Vocalis - Two Forms

A

Congenital (most common)
Acquired: phonotrauma

79
Q

Sulcus Vocalis - Tissue, Vibratory, Aerodynamic Features

A
  • Longitudinal groove parallel to glottis
  • Oval/spindle shaped closure

(+/-) Compensatory supraglottal hyperfunction

80
Q

Sulcus Vocalis - Voice Features

A
  • Breathy, hoarse
  • Decreased loudness level
  • Vocal fatigue
  • Increased pitch
81
Q

Sulcus Vocalis - Treatment

A
  • Voice therapy to improve function

If not sufficient: surgical management (medicalization injection OR excision of sulcus with laser)

82
Q

Define Presbyphonia

A

Dysphonia related to age-related structural changes to VFs and larynx

83
Q

Presbyphonia - Tissue, Vibratory and Aerodynamic Features

A
  • VF atrophy
  • Bowing of VFs
  • Reduced amplitude, reduced mucosal wave
  • Increased airflow, decreased pressure, decreased resistance
84
Q

Presbysphonia - Voice Features/Treatment

A
  • Breathy, hoarse, decreased loudness, unstable phonation (pitch breaks)

Treatment:
- Voice therapy to increase glottal closure (PhoRTE, RVT, VFEs)
- Surgery: medialization by injection

85
Q

Carcinoma - Etiology

A

Smoking, environmental irritants, metabolic disorders, idiopathic

86
Q

Explain Carcinoma Staging

A

Staged:lower number, better prognosis

Size and extent (T1-4)
- T1 = 1 site of the larynx
- T2 = 2 sites of the larynx
- T3 = Tumor limited to larynx, impaired mobility of one VF
- T4 = Tumor has extended beyond the larynx

Lymph node involvement (N0-3)
- N0 = No evidence of lymph nodes involved
- N1 = Tumor has spread to 1 or more regional lymph nodes
- N2 = Tumor has spread to extent between N1 and N3
- N3 = Tumor has spread to more distant or numerous regional
lymph nodes

Metastasis occurrence (M0/1)

87
Q

Carcinoma - Clinical Presentation

A
  • Hoarseness ( >2 weeks)
  • Lump or tenderness in neck
  • Dysphagia
  • Stridor or dyspnea
88
Q

Carcinoma - Stroboscopy

A
  • Small lesion visibly in early stages
  • Affect vibratory movement: increased stiffness, decreased amplitude, restricted/absent mucosal wave
  • Incomplete VF closure
89
Q

Carcinoma - Management Team/Treatment

A

Multi-disciplinary team: otolaryngologist, oncologist, SLP, social worker, psychologist, nutritionist, nurse, dentist

Radiation (side effects –> swelling of mucosa, fatigue, dryness, mouth sores, stiffness)

Chemotherapy (side effects –> skin problems, immunosuppression, nausea, hair loss, mouth sores)

Surgery:
- Lesion removal on true vocal fold only
- Hemilaryngectomy – partial laryngectomy, vertical direction
- Supraglottal laryngectomy
- Total laryngectomy
- Some oral structures may also be removed

90
Q

What is Laryngeal Dystonia? Types?

A

Syndrome of sustained, uncontrolled muscle contractions resulting in abnormal, unintended actions

2 subtypes:
- Adductor-type (ADLD)
- Abductor-type (ABLD)

90
Q

Define neurological voice disorder

A

Voice disorder associated w/damage to the nervous system

91
Q

Adductor Laryngeal Dystonia Characteristics

A
  • Effortful voice production (strained-strangled)
  • Voice stoppages during voiced sounds
  • TA involved
  • Vegetative voice function may be normal
92
Q

Abductor Laryngeal Dystonia Characteristics

A
  • Intermittent breathy breaks during production of voiceless consonants
  • Breathy breaks associated w/over-abduction of VFs
  • Pitch breaks
  • Normal vegetative voice function
  • (+/-) CT, PCA, TA involvement
93
Q

Onset of Laryngeal Dystonia

A

Occurs after:
- Upper respiratory infection
- Laryngeal injury
- Emotional stress

94
Q

Symptoms w/LD (both types)

A
  • Worse w/prolonged speaking
  • Worse when stressed/tired
  • Task-specific
  • Hyperfunctional voicing patterns
95
Q

Videoendoscopy/stroboscopy Signs of ADLD & ABLD

A

ADLD
- Hyperadduction at voice onset
- Intermittent hyper adduction
- Supraglottic compression

ABLD:
- Abduction at onset of words
- Supraglottic compressions

Larynx structure = normal

96
Q

Treatment of Laryngeal Dystonia

A

Botulinum toxin (BOTOX) injection
- Inhibits release of acetylcholine at neuromuscular junction
- Creates flaccid paralysis in injected muscle

Behavioral treatment after injection to achieve optimal voice (e.g., flow phonation)

97
Q

What is Voice Tremor? Origin regions? Characterized by…

A

A form of dystonia (cerebellum, connection pathways to brainstem nuclei) –> regular, rhythmic modulation of voice signal

Characterized by:
- Periodic modulation in frequency or intensity of voice
- Tremor isolated to larynx, or generalized to other speech structures
- Most noticeable during sustained voicing

98
Q

How do you differentially diagnose MTD, LD, tremor?

A

MTD: consistency
LD: inconsistency
Tremor: vowels worse

99
Q

Treatment of Voice Tremor

A

Behavior Tx:
- Increasing/changing pitch
- Shortening phrase length
- Shortening vowel length within words
- Using breathy/pressed voice quality

BOTOX
Pharmaceutical - beta blockers

100
Q

Parkinson’s Voice/Speech Characteristics

A
  • Monopitch, monoloudness
  • Reduced stress
  • Imprecise consonants
  • Breathy voice
101
Q

Treatment for PD

A

Lee Silverman Voice Treatment Program

  • Increase respiratory support
  • Increase phonatory effort
  • High intensity
  • Increase vocal fold adduction
  • Improve loudness level
102
Q

Clinical Presentation of Paradoxical Vocal Fold Motion Disorder (PVFM)

A
  • Choking sensation, throat tightness
  • Inspiratory stridor
  • Affects inspiratory phase of respiratory cycle

Symptoms occur with –> emotional stressors, allergens, GERD, physical exertion

Compensatory behaviors –> cough to open airway, throat clearing

103
Q

Etiology/Predisposing Conditions of PVFD

A

Etiologies:
- UMN/LMN
- Brainstem lesion
- LPR/GERD
- Psychological factors

Predisposing:
- Stress, Asthma, over-exposure to irritating fumes, childhood episodes of sexual abuse