Visual System Flashcards

1
Q

Visual System

A
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2
Q

Vision

A
  • Very large area devoted to this sense in humans.
  • Light passes through cornea > pupil > lens forms inverted & reversed image on the retina.
  • Where the optic nerve enters the retina, no rods or cones, creates the optic disc which is the blind spot.
  • Not superimposed so no functional blind spot.
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3
Q

Vision

A
  • The fovea is the area of central fixation; region of highest visual acuity (highest amount of receptors).
  • The macula surrounds the fovea, also high visual acuity.
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4
Q

Optic Field of View

A
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5
Q

Photoreceptors

A
  • Rods.
  • More numerous; 20:1.
  • Do not detect colors & good for low light.
  • Cones.
  • Lots in the fovea.
  • Can see color.
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6
Q

Receptive Field

A
  • Receptive field of a neuron in visual pathway: portion of visual field where light causes excitation or inhibition on bipolar cells.
  • Bipolar cells synapse onto ganglion cells that send axons into the optic nerve which exits through optic canal of sphenoid.
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7
Q

Optic Diagram

A
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8
Q

Optic Nerve, Chiasm, Tract

A

•Some fibers from the optic nerve cross over at the optic chiasm.

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9
Q

Lateral Geniculate Nucleus (LGN) & Extrageniculate Pathways

A
  • Axons of retinal ganglion cells in optic tract synapse of the LGN of the thalamus.
  • Continue to the primary visual cortex via optic radiations.
  • Few fibers bypass LGN & enter brachium of superior colliculus (this is extrageniculate pathway).
  • Relay to pretectal & superior colliculus go to Pulvinar > temporo-Parieto-occipital association love.
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10
Q

Figure 11.6 Geniculate and Extrageniculate Visual Pathways

A
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11
Q

Optic Radiations to Primary Visual Cortex

A
  • Axons leave LGN (“C” shape around lateral ventricle) to go to primary visual cortex by Calcarine fissure.
  • Called optic radiations.
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12
Q

Primary Visual Cortex

A
  • Upper portions à superior bank of calcarine fissure.
  • Lower portions à inferior bank of calcarine fissure.
  • Upper bank lesions:
  • Contralateral inferior quadrant defects.
  • Lower bank lesions:
  • Contralateral superior quadrant defects.
  • Fovea: 50% of visual cortex.
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13
Q

Left visual hemifields of the left and right eyes mapped to the primary visual cortex of the RIGHT hemisphere.

A
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14
Q

Visual Processing in Neocortex

A
  • Most input arrives in layer 4.
  • Thick layer due to functional importance.
  • Subdivided into sub-laminae 4A, 4B, 4C α, and 4C β.
  • Layer 4B has a lot of myelinated axon collaterals forming pale looking stria of Gennari can be seen in the gray matter with naked eyeball.
  • Primary visual cortex (AREA 17) sometimes called striate cortex.
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15
Q

Primary Visual Cortex

A
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16
Q

Assessment of Visual Disturbances

A
  • Detailed description of nature of visual disturbance.
  • Such as time of course, if positive for phenomena (bright lights) or negative (regions of decreased vision.)
  • Description of region of visual field defect of each eye involved.
17
Q

Some Terms to Describe Visual Disturbances

A
18
Q

Evaluation of Visual Disturbance

A
  • Detailed history.
  • Ophthalmoscopic exam.
  • Visual acuity (Snellen chart).
  • Visual fields.
19
Q

Localization of Visual Field Defects

A

•Visual field testing

20
Q

Visual Field Deficits

A
  • Monocular scotoma.
  • Monocular visual loss.
  • Bitemporal hemianopia.
  • Homonymous visual field defects? (retrochiasmal lesions.)
  • Contralateral homonymous hemianopia.
  • Macular sparing.
21
Q

Figure 11.15 Effects of Lesions in the Primary Visual Pathways

A
22
Q

VISUAL PATHWAY

A

https://faculty.washington.edu/chudler/vispath.html

23
Q

Blood Supply & Ischemia In Visual Pathways

A
  • Retina receives blood from branches of ophthalmic artery.
  • Well visualized on ophthalmoscopic exam as emerge from optic disc.
  • Emboli.
  • Stenosis due to diabetes, HTN, or intracranial pressure.
  • Vasculitis.
  • Central artery occlusion or branch > infarct of all or part of retina.
24
Q
A
25
Q

Optic Neuritis

A
  • Inflammatory, demyelinating disorder of the optic nerve.
  • Pathophysiologically related to multiple sclerosis.
  • Mean onset 30 y.o., acute or slowly progressive; days>weeks. Recovery begins by 2 weeks, complete 6-8weeks.
  • 2:1, Female:Male.
  • 50% or more will develop M.S.
  • Eye pain especially with movement, monocular problems, often central scotoma.

Papillitis, sometimes pale disc appearance