Visceral Artery Imaging/Hemodialysis Access Evaluation Flashcards
how to tell if mesenteric ischemia is acute or chronic
patients with acute mesenteric ischemia, often from an embolus, will present with severe abdominal pain - possible medical emergency due to. possibility of intestinal necrosis
describe normal celiac artery velocity, >70% stenosis and occlusion
normal <200 cm/sec
> 70% = >200 cm/sec with post stenotic turbulence
occlusion = no detectable signal, retrograde common hepatic artery flow
describe normal SMA velocity, >70% stenosis and occlusion
normal <275 cm/sec
> 70% = >275 cm/sec with post stenotic turbulence
occlusion = no detectable signal, often reconstitutes distally via collaterals
describe normal IMA velocity, >70% stenosis and occlusion
normal/>70% stenosis = no established criteria
occlusion = no detectable signal
if celiac and SMA are normal, isolated IMA stenosis is:
unlikely to be symptomatic
often small - if noted to be large, may suggest collateralization
what are two possible connections between SMA and IMA
marginal artery of the colon (aka marginal artery of Drummond)
arc of riolan
IMA may also serve as collateral to the iliac arteries via
branches of the internal iliac artery
what changes after eating? Celiac artery or SMA
SMA
pre prandial - high resistance flow pattern, flow reversal often present
post prandial - converts to Low resistance
median arcuate ligament syndrome (MALS)
(also known asa arcuate ligament compression syndrome or celiac axis compression syndrome)
compression of the celiac artery origin by the median acute ligament of the diaphragm
stenosis may occur during normal breathing or expiration as the arcuate ligament compresses the celiac artery
what are the clinical signs of MALS
abdominal bruit which disappears with deep inspiration
on expiration - the median arcuate ligament compresses the ventral aspect of the celiac artery creating an “S” shaped vessel course and a stenosis
with deep inspiration - the ligament releases the artery, it straightens, and the stenosis resolves with PS <200 cm/s
what is the difference between essential hypertension and secondary hypertension
essential hypertension - no direct identifable cause
secondary hypertension - hypertension that is the result of some other disease, commonly of the kidney
renovascular hypertension
caused by renal artery stenosis. the release of renin, promoting conversion of angiotensinogen to angiotensin causing vasoconstriction and subsequent high blood pressure
this eventually results in renal failure
what is the landmark for identifying the renal arteries
the left renal vein as it crosses over the aorta just below the origin of the SMA
what is the normal PSV for kidneys
<180cm/sec
what is considered 60% or greater stenosis for kidneys
> 180-200 cm/sec