Ventricular Tachycardia Flashcards

1
Q

define ventriular tachycardia?

A

a regular broad complex tachycardia originating from the ventricles

rate usually more than 120bpm

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2
Q

define sustained ventricular tachycardia?

A

ventricular rhythm faster than 100bpm lasting at least 30 seconds or requiring termination early due to haemodynamic instability

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3
Q

define non- sustained ventricular tachycardia?

A

ectopic ventricular rhythm with wide QRS complex (120 milliseconds or greater), rate faster than 120 bpm, lasting for at least 3 beats that spontaneously resolves in less than 30 seconds.

MEDICAL EMERGENCY

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4
Q

explain the aetiology/ risk factors of VT?

A

Electrical impulses arise from a ventricular ectopic focus– an ectopic focus is an excitable group of cells within the atria/ventricles which cause a premature heart beat outside the normally functioning circulation i.e. abnormal pacemaker sites

Can impair cardiac output causing hypotension, collapse and acute cardiac failure.

Torsades de pointesis a rare form of ventricular tachycardia. It can self-correct but it can also both lower arterial blood pressure (leading to syncope) and is a precursor to ventricular fibrillation and therefore sudden death. It can be caused by drugs such as tricyclic antidepressants and amiodarone and is also associated with long QT syndrome (a congenital condition), malnutrition and alcohol abuse. It is treated with intravenous magnesium and anti-arrhythmics.

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5
Q

what can cause Torsades de pointes and how can it be treated?

A

tricyclic antidepressants and amiodarone and is also associated with long QT syndrome (a congenital condition), malnutrition and alcohol abuse. It is treated with intravenous magnesium and anti-arrhythmics.

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6
Q

what is torsades de pointes?

A

Rare form of VT

It can self correct- but can also both lower arterial blood pressure ( leading to syncope)-> it is therefore a precursor to ventricular fibrillation and sudden death

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7
Q

What are the risk factors for VT?

A

Coronary heart disease

Structural heart disease

Electrolyte deficiencies (e.g. hypokalaemia, hypocalcaemia, hypomagnesaemia)

Use of stimulant drugs (e.g. caffeine, cocaine)

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8
Q

what are the presenting symptoms of VT?

A

signs of ischaemic heart disease/ haemodrynamic compromise

Chest pain

palpitations

dyspnoea

dizziness/ syncope

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9
Q

what are the signs of VT?

A

dependent on degree of haemodynamic stability

Weak pulse

Respiratory distress

Bibasal crackles

Raised JVP

Hypotension

Anxiety

Agitation

Lethargy

Coma

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10
Q

what are the investigations for VT?

A

ECG

Electrolytes- (e.g. hypo/hyperkalaemia, hypomagnesaemia) derangement can cause arrhythmias

Drug levels- check for DIGOXIN TOXICITY

Cardiac enzymes- troponins and CK-MB to check for recent ischaemic event

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11
Q

why is it important to investigate electrolytes when investigating VT?

A

hypo/ hyperkalaemia, hypomagnesia derrangement cause arrythmias

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12
Q

what can be seen on the ECG for VT?

A

can be difficult to distinguish VT and SVT with abberant conduction-> if in doubt treat as VT

May want to do 24 hour ambulatory ECG

Rate= more than 100bpm

broad QRS complexes

AV dissociation

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13
Q

when is ICD considered in the management of VT?

A

sustained VT causing syncope

sustained VT with ejection fraction less than 35%
previous cardiac arrest due to VT or VF

MI complicated by non-sustained VT

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14
Q

outline the management for VT?

A
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15
Q

How is torsades de pointes treated?

A

polymorphic VT-> give magnesium sulphate

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16
Q

what are the complications of VT?

A

congestive cardiac failure

cardiogenic shock

VT may deteriorate into VF

17
Q

summarise the prognosis of VT?

A

good if treated rapidly

long term prognosis depends on underlying cause