Ventricular Arrhythmias Flashcards

1
Q

What is indicative of ventricular ectopy on ECGs?

A

wide and bizarre QRS

  • wide because it takes longer to depolarize
  • premature, late
  • NOT initiated by P waves
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

How does ventricular ectopy compare to BBB?

A

BBBs are preceded by P waves

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What primary cardiac diseases and metabolic/endocrine diseases can cause centricular arrhythmias?

A

CARDIAC = cardiomyopathies, infectious, inflammatory (myositis)

ENDOCRINE = K, Mg, Ca, hyperthyroidism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What drugs and toxins can cause ventricular arrhythmias?

A
  • Digoxin
  • Amphetamine
  • Theobromine
  • Albuterol
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What conditions commonly cause ventricular arrhythmia?

A
  • ischemia
  • GDV
  • sepsis
  • splenic disease
  • hypoxia
  • hypovolemia
  • trauma
  • pain
  • decreased sympathetic innervation - inherited arrhythmias in GSDs
  • increased sympathetic innervation - pheochromocytoma
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the difference between ventricular premature contraction and escape beats?

A

VPCs = early, randomly placed ventricular beat interrupting normal rhythm

ESCAPE BEAT = late, preceded by a pause where the heart is trying to compensate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is indicative of a singlet ventricular premature complex (VPC) on ECG?

A

wide and bizarre QRS complex not initiated by a P wave

  • come early and followed by a compensatory pause
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the most common causes of VPCs in dogs and cats? What treatment is recommended?

A
  • DOGS = large breed cardiomyopathy (Boxers, Dobermans)
  • CATS = cardiomyopathy, hyperthyroidism
  • congenital disease, CVD, GDV, myocarditis, anesthesia, infiltrative neoplasia, traumatic myocarditis, digitalis toxicity

a few VPCs once in a while can be acceptable in some breeds, but cause should really be investigated and treated (look at ECG > 30s)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is occurring in this ECG?

A

VPCs

  • wide and bizarre QRS complex not initiated by a P wave
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is occurring in this ECG?

A

sinus rhythm interrupted by 3 single VPCs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

How can the ventricle of origin be differentiated with VPCs on ECG?

A

RIGHT VENTRICLE = mostly positive QRS

LEFT VENTRICLE = mostly negative QRS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are some causes of right and left sided disease that can cause VPCs? What most commonly causes disease on both sides?

A

RIGHT VENTRICLE = arrhythmogenic right ventricular cardiomyopathy (ARVC) in Boxers and Bulldogs, less common (positive QRS)

LEFT VENTRICLE = DCM (Dobermans, Irish Wolfhound, Great Danes), HCM (cats), more common (negative QRS)

EITHER = diffuse myocarditis or myocardial infiltration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

VPCs, complexity:

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is occurring in this ECG?

A

couplet VPCs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is occurring in this ECG?

A

one VPC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is the R on T phenomenon? What 2 things does it indicate?

A

QRS fires very closely to the preceding T wave

  1. high risk for development of ventricular fibrillation
  2. anti-arrhythmic therapy warranted
17
Q

What is accelerated idioventricular rhythm? How does it appear on ECGs?

A

runs of VPCs at a rate less than 160 bpm and greated than 40 bpm

  • 4 or more VPCs
  • looks like VTACH, but much slower
18
Q

What is occurring in this ECG?

A

idioventricular rhythm

  • 4 VPCs
  • HR = 40-160 bpm
19
Q

What are 5 possible causes of idioventricular rhythm?

A
  1. splenectomy
  2. systemic disease
  3. myocardial depression
  4. hypoxia
  5. myocardial infarction
20
Q

What is an accelerated idioventricular rhythm (AIVR)? Is treatment necessary?

A

slow ventricular tachycardia —> alternation between VPCs and normal sinus rhythm

benign arrhythmia - does not perturb hemodynamics, tx not necessary (without ECF, likely nothing was noted wrong with the heart)

21
Q

What causes the development of accelerated idioventricular rhythm?

A

non-cardiac disease —> thoracic trauma, GI disease (GDV), splenic disease, pancreatitis, post-splenectomy

22
Q

What is occurring in this ECG?

A

accelerated idioventricular rhythm

  • VPCs alternate with normal sinus rhythm
23
Q

What are some clinical signs of single VPCs/AIVR? What therapy is recommended?

A
  • rarely any signs seen
  • dropped heart sounds
  • irregular/dropped pulses
  • weakness, syncope, sudden death

address underlying disease —> pain, stress, hypoxia, hypovolemia, GDV

  • specific tx only needed if the patient is hemodynamically affected by the underlying rhythm or it is multifocal
24
Q

What causes ventricular tachycardia? What 2 things are seen on ECGs?

A

stimulation of an ectopic ventricular focus

  1. runs of > VPCs @ >160-180 bpm sustained for > 30s
  2. QRS complexes with no relationship to P waves (hidden)
25
Q

When does ventricular tachycardia affect heart function? Why are they especially serious? What conditions are associated?

A

300 bpm >6-8 bpm

can be pre-fibrillatory and has direct effects on the heart and secondary organs due to poor perfusion

  • large dogs with DCM
  • uncommon in cats
26
Q

What is occurring in these ECGs?

A

sustained ventricular tachycardia

27
Q

What is the most common cause of sustained ventricular tachycardia? What acute and chronic therapies are recommended?

A

primary myocardial disease (DCM, ARVC, myocarditis, HCM) —> hemodynamically devastating, potential for rapid decompensation and development of ventricular fibrillation

ACUTE = Lidocaine, Procainamide

CHRONIC = Sotalol, Mexiletine

28
Q

What is ventricular fibrillation? How do patients present?

A

non-perfusing rhythm where cells of the myocardium depolarize chaotically

no pulse is felt and cardiac output approaches 0

29
Q

What are 2 signs of ventricular fibrillation on ECGs?

A
  1. rapid, chaotic, irregular rhythm with bizarre waves or oscillations
  2. no QRS or P waves (large = coarse, small = fine)
30
Q

What is occurring in this ECG?

A

ventricular fibrillation

31
Q

What conditions are associated with ventricular fibrillation?

A

severe systemic illness or cardiac diseas

  • shock, anoxia
  • trauma
  • myocardial infarction, myocarditis
  • electrolyte/acid-base imbalance
  • anesthetic reaction
  • digoxin toxicity
  • cardiac sx
  • electrical shock
  • hypothermia
32
Q

What treatment is recommended for ventricular fibrillation?

A

quick treatment with Epinephrine +/- electrical defibrillation (transthoracic vs. internal) +/- magnesium to stabilize resting membrane potential

  • no treatment = likely death
33
Q

What is ventricular asystole? What is indicative on ECG?

A

terminal rhythm due to no pacemaker impulse = no contraction, no pulses, no cardiac output with severe sinoatrial block or arrest

no QRS complexes

34
Q

What is occurring in this ECG?

A

ventricular asystole

35
Q

What are 5 parts of treating ventricular asystole?

A
  1. start CPR
  2. epinephrine, atropine
  3. treat underlying disease
  4. defibrillation
  5. precordial thump

rapidly fatal arrhythmia

36
Q

Ventricular arrhythmias:

A