Venous Flashcards
Define Phlegmasia alba/cerulea dolens
Extreme form of iliofemoral DVT where swelling is so extensive the arterial circulation is compromised. Can lead to gangrene and limb loss.
What is the most dangerous complication of DVT?
PE
What is the most common complication of DVT?
venous insufficiency
Give five ways venous anatomy differs from arterial anatomy.
thinner walls flow direction shape changes (circular to dumbbell) based on pressure presence of valves perforators
Name the leg perforators.
Perforators of the Femoral Canal
Peritibial Perforators
Posterior Tibial Perforators
What are the five Doppler characteristics of normal venous flow?
Spontaneous: flow exists in medium to large veins
Phasicity: velocity varies with respiration
Augmentation: confirms patency
Valsalva response: shows if valves are competent
Competency: unidirectional flow toward heart through valves, if not, reflux
What two methods are used in duplex examination of veins?
Compression/coaptation on short axis
Doppler evaluation on long axis
What is effort thrombosis? What is its other name?
Compression of the subclavian vein causing thrombosis.
Paget-Schroetter Syndrome
Thrombosis in the vein causes what sort of response?
Inflammatory - pain in first two weeks
What skin changes are noted with venous disease?
Edema Rubor from inflammatory process Brawney color Pallor - phegmasia alba dolens Cyanosis - plegmasia cerulea dolens
What will lyse the thrombus? In what % of cases?
Plasminogen, in 20% of cases.
Describe the progression of DVT, in three stages.
Acute: first 2 weeks; hypoechoic, spongey, dilated, may have free floating tail, collaterals, active inflammation & pain
Subacute: weeks to 6 months: more hyperechoic, well adhered, may be dilated, collaterals
Chronic: bright, calcific, fibrous cords, retracted, wall thickening, sequelae occur
What are the risk factors for DVT?
Virchow’s Triad:
- Stasis: ICU/CCU, bedrest, long ride, stroke
- Trauma: surgery, accident
- Hypercoaguable: cancer, pregnancy, dehydration, Protein C, Protein S, Factor V Leiden, Antithrombin 3 deficiency
Describe the difference between primary and secondary varicose veins.
Primary: limited to superficial system
Secondary: underlying deep venous incompetence and/or obstruction
Describe venous insufficiency.
Valvular incompetence (deep, superficial, or perforating) permits flow reversal (reflux), leading to venous HTN, venous claudication, ulcers, stasis dermatitis, etc.
What is VI caused by? (3)
Recanalized segments of thrombosed veins
Dilation of veins
Congenital absence of competent valves
List six sequellae of chronic VI.
Edema Fibrous deposits Ulceration Hemociterin - brawny appearance Pain
What are the three goals of a venous exam for VI?
Determine the venous systems involved.
Determine the level.
Determine if VI is due to incompetence or obstruction or both.
What are the invasive tests for VI?
Venography, both ascending and descending. Ascending looks for DVT, descending looks for VI.
Ambulatory venous pressures: uses needle in foot for pressure readings.
What are the non-invasive tests for VI?
CW Doppler
Photoplethysmography (PPG) - infrared sensor, dorsiflexion. Refill time > 20 sec. is normal.
Air Plethysmography (APG)
Duplex: reversal .5–1.0 mild, > 1 significant
What does APG measure:
Is there VI?
Is there venous HTN distally?
Does the calf muscle work?
What is the duplex protocol for checking for VI?
• Evaluate for DVT
• Sample sites for VI/reflux: superficial and deep, B-Mode & Doppler
• Check perforators
Determines presence and location of VI and outflow obstruction.