vascular surgery Flashcards

1
Q

what is carotid artery stenosis and how does it occur?

A

it is narrowing of the carotid arteries secondary to atherosclerosis
the plaque can break away and cause embolic strokes - high risk of CAD and MI

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2
Q

what are the risk factors and classification of carotid artery stenosis?

A

RFs: male, age, smoking, HTN, poor diet, reduced physical activity, hypercholesterolaemia
classification: reduction in diameter: <50% is mild, 50-69% is moderate and >70% is severe

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3
Q

how does carotid artery stenosis present?

A

asymptomatic until stroke or TIA, carotid artery bruit during systole

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4
Q

how is carotid artery stenosis investigated and managed?

A

ix: carotid USS, CT, MRI
mx: diet, exercise, smoking, comorbidities, anti platelets, lipid lowering meds, carotid endarterectomy, angioplasty and stenting

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5
Q

what are the complications of carotid endarterectomy?

A

facial, glossopharyngeal nerve, hypoglossal and recurrent laryngeal nerve injury

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6
Q

what is buergers disease?

A

it is thromboangiitis obliterans - inflammatory condition causing thrombus formation in small and medium vessels resulting in painful, blue discolouration of the finger tips and toes, worse at night, ulcers, gangrene, amputation and corkscrew collaterals

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7
Q

who does buergers disease affect? what is the treatment?

A

young males, smokers
mx - stop smoking, IV iloprost

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8
Q

what is chronic venous insufficiency?

A

it is when blood does not drain from the legs to the heart due to damage in the veins due to age, obesity, immobility, prolonged standing
the blood pools: venous HTN, usually in gaiter area - skin changes (haemosiderin staining, venous eczema, lipodermatosclerosis, atrophie blanche)

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9
Q

what are complications of chronic venous insufficiency?

A

cellulitis, poor healing after injury, skin ulcers, pain

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10
Q

how is chronic venous insufficiency managed?

A

monitor skin, avoid damage, emollients, topical steroids, potent if lipodermatosclerosis, loose weight, stay active, elevate legs, compression stockings, ABx, analgesia, wound care for ulceration

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11
Q

what is lymphoedema?

A

impaired lymphatic drainage - swollen with protein rich fluid - prone to infection - can be primary which is rare and genetic under 30y/o or secondary due to mastectomy and ALN removal

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12
Q

how is lymphoedema investigated?

A

stemmers test for tenting (none in lymphoedema), limb volume using circumferential measurements, water displacement and perometry, bioimpendance spectrometry, lymphoscintigraphy

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13
Q

how is lymphoedema managed?

A

massage, compression bandage, improve lymph drainage with exercises, loose weight, good skin care, lymphaticovenular anastamoses, ABx, CBT, antidepressants

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14
Q

what is lymphatic fildriasis?

A

infectious disease caused by parasitic worms - live in lymphatic system resulting in lymphoedema and skin thickening and fibrosis - elephanitis
found in asia and africa

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15
Q

what is the presentation of arterial leg ulcers?

A

distal - toes and dorsum of foot, PAD (6Ps), smaller, deeper, well defined, punched out, pale, less likely to bleed, painful at night / lying

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16
Q

what is the presentation of venous ulcers?

A

gaiter area, chronic venous changes, minor injury to leg, superficial, larger, irregular sloping border, more likely to bleed, less pain, worse on lying, better with elevation

17
Q

how are leg ulcers investigated and managed?

A

ix: ABPI, bloods, charcoal swabs, skin biopsy
arterial mx: refer - surgical revascularisation
venous mx: r/o arterial, vascular surgery, tissue viability, derm, pain and diabetes input, clean, debridement, dressing and compression, pentoxyfylline, ABx, analgesia

18
Q

what is peripheral arterial disease?

A

it is made of intermittent claudication, critical and acute limb ischaemia

19
Q

what is IC?

A

it is aching or burning in the legs following exertion and relieved within minutes of stopping
investigated with pulses and ABPI

20
Q

what is acute limb ischaemia?

A

it is due to thrombus or embolus cutting off blood supply - pale, perishingly cold, pulseless, pain, paraesthesia, paralysis
ix: USS and ABPI

21
Q

what is the management of peripheral arterial disease?

A

acute: A-E, IV opioids, IV unfractionated heparin, vascular r/v - intraarterial thrombolysis, surgical embolectomy, angioplasty, bypass surgery, amputation
PAD: quit smoking, control BP, DM, obesity, statins, exercise, clopidogrel, endovascular/surgical revascularisation using percutaneous transluminal angioplasty and stenting or surgical bypass or endarterectomy

22
Q

how is ABPI classified?

A

1: normal
0.6-0.9: claudication
0.3-0.6: rest pain
<0.3: severe