cardiothoracic surgery Flashcards

1
Q

why is CABG needed?

A

usually due to atherosclerosis leading to MI, angina, stroke, TIA, PAD, mesenteric ischaemia

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2
Q

how is CABG done?

A

a graft vessel is taken from usually saphenous vein to bypass a blockage. The machine takes blood from RA or VC, pumps through it and adds O2 and removes CO2 and puts it back into AA. Heparin added to prevent clotting. Cardioplegia is achieved through high concentration potassium and cardioversion or adrenaline used to restore heart beat. Recovery around 3m.

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3
Q

what is intimal hyperplasia?

A

when a vein used in CABG becomes stenosed

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4
Q

what are complications of CABG?

A

death, stroke, infection, AKI, CI, MI, AF

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5
Q

what is pericardial effusion?

A

it is fluid in the pericardial sac - transudate, exudate (inflammation), blood, pus, gas
cardiac tamponade when the intracardial pressure is raised
reduces expansion - reduced CO

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6
Q

what are the causes for pericardial effusion?

A

decreased drainage - transudative - CHF, pulmonary HTN
inflammatory - exudative - infection, AI, injury, uraemia, cancer, medications
acute tamponade - MI, aortic dissection A, trauma

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7
Q

how does pericardial effusion present and how is it investigated?

A

presentation - SOB, CP, fullness, orthopnoea, hiccups, dysphagia, hoarseness, quiet HS, pulsus paradoxus, hypotension, increased JVP, fever, pericardial rub
ix: echo, fluid analysis, bacterial culture, viral PCR, cytology, tumour markers

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8
Q

how is pericardial effusion managed?

A

NSAID, aspirin, colchicine, needle pericardiocentesis, or surgical drainage, pericardial window or pericardiectomy is rare

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9
Q

summarise cyanotic heart disease?

A

right to left shunt - ASD, VSD, PDA, TGA
left>right pressure - Eisenmenger syndrome
complications: arrhythmias, HF, endocarditis, stroke, pulmonary HTN, Eisenmengers

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10
Q

summarise ASD?

A

usually PFO, ostium secundum or primum, dyspnoea, stroke, AF or flutter, mid systolic crescendo decrescendo murmur, fixed split second heart sound
mx: transvenous catheter closure or open heart sx, anticoagulants

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11
Q

summarise VSD?

A

pansystolic murmur over LLSE in 3rd ICS
systolic thrill on palpation
mx: transvenous catheter closure, open heart sx, ABx prophylaxis

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12
Q

summarise coarctation of the aorta?

A

usually around DA, HTN, systolic murmur below left clavicle, BP difference, LV heave, underdeveloped left arm and leg
ix: CT angiography,
mx: emergency surgery at birth if severe, percutaneous balloon angioplasty and stenting, open repair, manage HTN

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13
Q

summarise prosthetic heart valves?

A

usually a midline sternotomy scar
bioprosthetic - porcine - 10y lifespan
mechanical - 20y lifespan - need lifelong warfarin (can be starr edwards, tilting disc or st jude) - clicks replace S1 and S2
can get thrombus, IE and anaemia

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14
Q

summarise aortic stenosis?

A

ejection systolic murmur high pitched, crescendo decrescendo in systole, radiate to carotids, slow rising pulse, exertional syncope, narrow pulse pressure
causes: RHD, idiopathic
mx if severe: transcatheter aortic valve through femoral artery under XR guidance - bioprosthetic

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15
Q

summarise mitral regurgitation?

A

2nd most common
reduced EF, CCF, pansystolic high pitched whistling murmur, 3rd HS
causes: RHD, idiopathic, IE, IHD, CTD

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16
Q

what is the main risk of valve replacement?

A

IE: 2.5% of surgical valve replacements or 1.5% of TAVI
15% mortality
gram +ve cocci such as staph, strep or entero

17
Q

summarise heart and lung transplant?

A

they mostly come from deceased donors - indications are IHD, cardiomyopathy, CHD, COPD, PF, CF, pulm HTN
variations - single or double lung, heart and lung, heart
lateral thoracotomy scars for lung and midline sternotomy for heart
time between the death and donation is cold ischaemic time - cooled down and operation started immediately as soon as transplant arrives
cardioplegia, bypass, cardioversion
post transplant steroids and immunosuppression
complications - cardiac allograft vasculopathy, primary graft dysfunction, bronchiolitis obliterans syndrome, dehiscence of bronchial anastomases

18
Q

what are the most common types of lung cancer?

A

non small cell - 80% and small cell - 20%

19
Q

which lung cancer is most likely to have paraneoplastic syndrome?

A

SCLC - neurosecretory granules

20
Q

how does mesothelioma present?

A

asbestos link, SOB, cough, reduced weight, clubbing, haemoptysis, pneumonia, paraneoplastic syndromes

21
Q

what is the 2ww criteria for lung cancer?

A

CXR if >40 with clubbing, lymphadenopathy, recurrent chest infections, increased platelets, lung cancer chest signs
consider if >40 with 2MUS and never smoked or >40 with 1 MUS and smoker - MUS: cough, SOB, fatigue, weight loss, reduced appetite

22
Q

how is lung cancer investigated?

A

CXR (hilar enlargement, peripheral opacities, pleural effusion, collapse, staging CT, PET CT, bronchoscopy with endobronchial US, histology and biopsy)

23
Q

how is lung cancer managed?

A

NSCLC: segmentectomy, lobectomy, pneumonectomy, RT
SCLC: chemo and RT
endobronchial treatment with stenting and debulking

24
Q

where does thoracic aortic aneurysm commonly occur?

A

in the ascending aorta

25
Q

what are the normal values for thoracic aorta?

A

<4.5cm ascending and <3.5cm descending

26
Q

what is the difference between false and true thoracic AA?

A

false: two inner layers rupture - blood in outer layer due to trauma
true: all 3 layers intact but dilated

27
Q

what are complications of thoracic aortic aneurysm?

A

dissection, rupture, aortic regurgitation

28
Q

what is the presentation of rupture?

A

haematemesis, haemoptysis, cardiac tamponade due to bleeding into mediastinum, back pain, chest pain, collapse, haemodynamic instability, death

29
Q

what are the risk factors and presentation of thoracic aortic aneurysm?

A

RFs: men, age, smoking, HTN, FHx, marfan/CTD
presentation: chest or back pain, cough, SOB, stridor, hiccups, dysphagia, hoarseness

30
Q

how is thoracic aortic aneurysm investigated/managed?

A

ix: echo, CT, MRA
mx: stop smoking, diet, exercise, optimise mx of comorbidities, surveillance, thoracic endovascular aortic repair, open surgery

31
Q

what is pneumothorax and what causes it?

A

it is air in the pleural space which separates the lung from the chest wall
causes: spontaneous, lung pathology, trauma, iatrogenic

32
Q

how is pneumothorax investigated and managed?

A

ix: CXR, CT thorax
mx: <2cm and asymptomatic: conservative - f/u in 2-4w
>2cm - aspiration and reassessment
if fails twice - chest drain in triangle of safety
pleurodesis or pleurectomy if required
tension - large bore cannula in 2nd ics MCL then chest drain