General Surgery Flashcards

1
Q

what are stomas?

A

they are artificial openings of hollow organs - opened onto the surface of abdomen and contents are collected into bag

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2
Q

what is the difference between colostomy and ileostomy?

A

ileostomy has more irritant contents so has a spout, is more likely to be in RIF and has liquid contents, colostomy is in LIF, solid and flat to skin

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3
Q

when is an end stoma permanent?

A

after APR or panproctocolectomy

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4
Q

what are complications of stomas?

A

hernia, psychosocial, local skin irritation, loss of bowel length, dehydration, malnutrition, constipation, stenosis, obstruction, retraction, prolapse, bleeding, granulomas

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5
Q

list differentials for acute abdominal pain?

A

generalised: ischaemic colitis, ruptured AAA, peritonitis, intestinal obstruction
RUQ: biliary colic, cholecystitis, cholangitis
epigastric: gastritis, peptic ulcers, pancreatitis, ruptured AAA
loin to groin: ruptured AAA, renal colic, UUTI
testicular: torsion, epididymo-orchitis
central: ruptured AAA, ischaemic colitis, early appendicitis, intestinal obstruction
RIF: appendicitis, ectopic pregnancy, ruptured ovarian cyst, ovarian torsion, Meckel’s diverticulum
LIF: diverticulitis, ectopic, ruptured cysts, torsion
suprapubic: LUTI, retention, PID, prostatitis

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6
Q

what are the signs of peritonitis?

A

guarding, rigidity, rebound tenderness, coughing test, percussion test

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7
Q

how is the acute abdomen investigated?

A

A-E
FBC, U&Es, LFTs, CRP, amylase, INR, Ca, hcg, ABG, lactate, G&S, cultures
AXR, CXR, A USS, CT abdo

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8
Q

how is the acute abdomen managed?

A

A-E, escalate, NBM, NGT, IVf, IV ABx, analgesia, VTE risk assess, prescribe regular meds, XM

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9
Q

what are the risk factors for bowel cancer?

A

FHx, FAP, increasing age, HNPCC, IBD, diet, obesity, sedentary lifestyle, smoking, alcohol

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10
Q

how does bowel cancer present?

A

mass (abdo/rectal), weight loss, unexplained abdominal pain, rectal bleeding, iron deficiency anaemia, change in bowel habit

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11
Q

what is the 2 ww criteria for bowel cancer?

A

> 40y with unexplained abdominal pain and weight loss
50 with unexplained PR bleeding
60 with change in bowel habit or anaemia

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12
Q

how is bowel cancer investigated?

A

screening: FIT >60-60 as per 2ww or 60-74 every 2 years
colonoscopy and biopsy, sigmoidoscopy, CT colonography, regular screening if RFs, staging CTTAP, CEA for relapse

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13
Q

what classification is used for bowel cancer?

A

Duke’s and TNM

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14
Q

how is bowel cancer managed?

A

surgical resection - lap, open, robotic
hemi colectomy, APR, anterior resections, Hartmann’s
chemotherapy, radiotherapy, palliation

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15
Q

how is bowel cancer followed up?

A

CEA, CTTAP

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16
Q

what are some complications of surgery for bowel cancer?

A

pain, bleeding, infection, damage to structures, ileus, anaesthetic risks, risk of changing to open, leak or fail of anastamoses, VTE, change in bowel habit, failure to remove, adhesions, stoma, hernia, lower anterior resection syndrome

17
Q

what is volvulus?

A

it is when the bowel twists around itself and mesentery - closed loop bowel obstruction - ischaemia, necrosis and perforation
investigated with AXR and CT

18
Q

summarise sigmoid volvulus?

A

it is more common and in older patients
it is caused by chronic constipation and lengthening of mesentery attached to sigmoid - faecal overload - sinks down
associated with high fibre and laxatives
shows coffee bean sign on AXR

19
Q

summarise caecal volvulus?

A

less common, more in younger patients
foetal sign on AXR

20
Q

what are the risk factors for volvulus?

A

neuropsychiatric disorders, nursing home, chronic constipation, high fibre, pregnancy, adhesions

21
Q

how does volvulus present?

A

vomiting (green bilious), distension, diffuse pain, absolute, constipation, no flatulence

22
Q

how is volvulus managed?

A

NBM, NGT, conservative - endoscopic decompression for sigmoid
surgical - laparotomy, Hartmann’s (sigmoid), ileocaecal resection, right hemicolectomy (caecal)

23
Q

what is cholangitis?

A

it is infection and inflammation of bile ducts - emergency - sepsis

24
Q

what causes cholangitis and how does it present?

A

it is caused by obstruction, infection, ERCP
E coli, enterococci, Klebsiella
it presents with RUQ pain, fever and jaundice

25
Q

how is cholangitis investigated and managed?

A

Ix: abdo USS, CT, MRCP, endoscopic USS
Mx - acute: NBM, IVf, culture, IV ABx, HDU/ICU
definitive: ERCP for stones (and cholangiopancreatography, sphincterectomy, removal, dilatation and stenting, biopsy), percutaneous transhepatic cholangiogram for drainage to relieve obstruction if ERCP not suitable or failed

26
Q

what is cholangiocarcinoma?

A

it is bile duct cancer - adenocarcinomas - can be intra or extra hepatic

27
Q

what are the risk factors for cholangiocarcinoma?

A

liver flukes, PSC

28
Q

how does cholangiocarcinoma present?

A

pale stools, dark urine, itching, weight loss, RUQ pain, palpable gallbladder, hepatomegaly

29
Q

how is cholangiocarcinoma investigated?

A

CT/MRI, biopsy, CTTAP, Ca 19-9, MRCP, ERCP

30
Q

how is cholangiocarcinoma managed?

A

radio and chemo (only curative if early presentation)
stents, palliative surgery, chemo, radio, EoL control