Vascular Emergencies Flashcards

1
Q

Causes for acute arterial occlusion

A

Embolus

Thrombosis

Direct trauma to arteries

will lead to distal ischemia to the occlusion if left untreated

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2
Q

Embolic occlusion

A

Caused by dislodgment of intravascular thrombus That travels dismally and occludes a small artery
- usually originates in the left ventricle of the heart but can come from anywhere

history of arrhythmias, MI or valvular heart disease are often suggestive of acute peripheral ischemia

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3
Q

Types of emboli

A

Cardiac: originate in the left atrium often in patients with:

1) current atrial fib/mitral valve disease
2) history of recent MI or ventricular aneurysm

Vascular: originate on luminal surfaces in vessels that are atherosclerotic. Often in patients with

1) atherosclerosis
2) high cholesterol patients

Tumor: rare but most commonly caused by atrial myxomas

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4
Q

Thrombosis occlusion

A

Caused by plaque disruption and resultant clot formation from an atherosclerotic vessel. Often found in patients with history of:

1) peripheral vascular disease
2) claudication
3) progressive pain at rest
4) non healing wounds of the distal extremities

Takes time to present and shows intermittent claudication with it

  • results in ischemia, irreversible anesthesia, paralysis and tissue infarction
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5
Q

Clinical findings of thrombi/emboli occlusion

A

Symptoms and signs:

  • extremity pain
  • paresthesia
  • paralysis of limbs (if prolonged)
  • pale/cool/CYANOTIC
  • lower or absent pulses

Imaging studies:

  • angiography will confirm affected limb
  • can also use CTA, MRAs and arterial duplex ultrasonography
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6
Q

Treatment for embolism/thrombi occlusion

A

1) Receive a general/vascular surgery consultation immediately
- if surgery required, its usually fogarty embolectomy

2) same time or immediately after,Insert a IV catheter and begin IV heparin as soon as possible unless contraindicated
3) Obtain labs of CBC, PT and PTT times as well as blood chemistries specifically blood types.

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7
Q

Acute peripheral ischemia “blue toe syndrome”

A

Symptoms

  • abrupt onset of small painful area on affected digit
  • affected area is tender cool and CYANOTIC
  • will be ASYMMETRIC w/ possible livedo reticularis (swollen mottled reticulated vascular pattern w/ purplish discoloration of the skin)
  • is an emergency

Treatment:
- treat proximal source of emboli with direct IV heparin or vascular surgery

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8
Q

Acute peripheral ischemia due to venous occlusion in whole extremity “phlegmasia cerulea dolens”

A

Symptoms:

  • massive acute swelling of affected leg
  • leg is edematous and doughy consistency
  • cyanosis and gangrene
  • color flow Doppler ultrasound or venography confirms diagnosis

Treatment:

  • immediate IV heparin at full dose
  • immediate general or vascular surgery consultations (thrombectomy) if Heparin Tx doesnt work or is contraindicated
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9
Q

Acute visceral intestinal ischemia

A

Symptoms

  • severe non-localized abdominal pain w/ hypotension
  • history of intestinal angina
  • pain out of proportion to physical findings
  • grooms intestinal bleeding
  • abdominal distention

Imaging/labs to confirm

  • mesenteric arteriography or CT
  • elevated leukocytes, metabolic acidosis and elevated lactate

Treatment:

  • IV with saline and blood to combat hypotension
  • stop bleeding with vascular surgery
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10
Q

Ruptured AAA

A

Symptoms

  • sudden onset of abdominal/flank pain
  • pulsation abdominal mass
  • hypotension

Screening and labs to confirm

  • bedside ultrasound
  • CT scan
  • low hematocrit w/ ECG signs of STEMI

Treatment:
1) consult surgery since this is needed (100% mortality if no surgery, 40% if surgery)

2) very rapid treatment of hypotension and shock before surgery if present( use oxygen and saline and blood perfusion)
3) if HTN is present DONT treat it

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11
Q

TAAs and thoracic dissection

A

Symptoms:

  • abrupt 10/10 chest w/ radiation to the back or upper abdominal
  • aortic murmur from aortic regurgitation may be heard
  • may hear muffled heart sounds (cardiac tamponade)
  • weak or pulselessness pulses
  • syncope
  • paralysis (only if carotid has been dissected as well)
  • hypertensive

Imaging and tests to confirm

  • chest CT or MRI
  • trans esophageal echo (TEE)
  • ECG often shows anterolateral STEMI characteristics
Treatment:
*GET SURGERY CONSULTATION*
- immediate treatment of hypertension 
Done via:
1) IV esmolol (to test BB use NOT to treat) 
2) IV labetalol 
3) nicardipine (if BBs are contraindicated or not tolerated) 
4) nitroprusside (if all else fails) 
  • also use fentanyl/morphine to control pain*
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12
Q

Common risk factors for TAA and AAA

A

HTN

Trauma

Pregnancy

Marfan syndrome

Ehlers-Danlos syndrome

Turner syndrome

Cocaine/elicit drug abuse

Coarctation of the aorta

Valve stenosis/replacements (especially aortic)

Intra-aorta catheterization

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13
Q

peripheral arterial aneurysms

A

Symptoms

  • pulsation mass on physical examination
  • Pain out of proportion
  • intestinal angina
  • edema/swollen leg w/ cyanotic apperance
  • may have high femoral BP
  • most common areas for peripheral arterial aneurysms are femoral, popliteal, splenic arteries and the SMA

Imagining and tests to confirm:

  • ultrasound
  • increased lactate/metabolic acidosis and increased WBC counts
  • arteriography defines where the cutoff begins

Treatment:
- IV heparin and notify surgeon immediately for surgery (if heparin doesnt stop it)

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14
Q

Lower DVT

A

Symptoms:

  • unilateral swelling, warmth and redness of the affected limb
  • pain in the affected limb that worsens overtime and does not respond to meds
  • (+) homan sign (pain in posterior calf from dorsiflexion

Imaging and labs to confirm:

  • ultrasounds of the leg confirm however venography is the gold standard if possible
  • D-dimer elevation can help confirm but is not specific

Treatment:

  • start IV heparin (LMW) or factor 10 inhibitors if heparin contraindicated
  • use surgery if this cant be fixed
  • requires heparin or factor 10 treatment for life usually
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15
Q

Superficial thrombophlebitis

A

Symptoms:

  • pain/tenderness/induration and redness along the vein
  • no edema in extremities
  • may feel like a string of beads

Treatment:

  • only treat symptoms if no complication (will run its course)
  • surgery and parenteral antimicrobials if complications occur (becomes septic or moves to a bigger vein)
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16
Q

Difference between emboli and thrombi cases

A

Emboli:

  • most likely to be acute onset after an MI
  • typically are formed in the left ventricle (80%) or left atrium (10%)

Thrombi:

  • most likely to be gradual onset due to atherosclerosis
  • intermittent claudication occurs (burning sensation that occurs during activity but goes away at rest)
17
Q

6 Ps of acute ischemia

A

Parasthesia (early)

Pallor (early)

Pulselessness (most late)

Pain (early)

Paralysis (late)

Poikilothermia (early)

18
Q

Peripheral arterial aneurysms most common locations

A

MOST COMMON IN MEN AND PREGNANT WOMEN

  • femoral and popliteal are most common followed by:
    1) splenic artery
    2) hepatic artery
    3) SMA

cardinal sign is symptoms similar to thrombi/emboli events with pain out of proportion

19
Q

What is a Mycotic aneurysm?

A

Any aneurysm that becomes infected

20
Q

What is the most common origin of distal arterial emboli?

A

85% are found in the LEFT VENTRICLE

15% are found in PROXIMAL AORTA

These are really the only 2 options with all others occurring <2%

21
Q

AAA facts

A

50-50% male female ratio

Most are under 50 and non smokers

Women are more prone to ruptured AAAs

Family History of AAAs makes the patient more likely to have it as well as

1) CT genetic disorders
2) being old
3) HTN

22
Q

Most common post AAA repair complications

A

1) Aortoenteric fistula is the most common (w/ duodenum being the most common location)
- presenting w/ massive hemorrhage from the GI tract with hx of having an AAA assume AE fistula until proven else wise (must get immediate surgery)

2) graft infections (mycotic aneurysms)
3) pseudoaneuysms
4) endovascular repair leaks

23
Q

Ankle brachial index

A

Ratio of blood pressure at the ankle to the blood pressure in the upper arm

Normal ratio = .9 or greater

  1. 7-0.9 = mild obstruction/occlusion
  2. 4-0.7 = moderate obstruction/occlusion
  3. 0-0.4 = severe obstruction/occlusion

Steps to get it:

1) patient must be placed supine with everything on table
2) both arm sides BP are taken (usually the higher systolic is used)
3) take ankle/foot BP (posterior tibial artery or dorsalis pedis artery which ever is higher)
4) use which ever BP is higher with the same side as the arm and determine ratio.

24
Q

Why are thrombolytics controversial treatment for emboli/thrombi?

A

While it will treat and allow reperfusion to the area, there is a strong chance that busting these can result in smaller thrombi/emboli that will grow over time

25
Q

Pseudo aneurysms

A

Often caused by trauma and includes post operative angioplasty
- usually a hematoma that surrounds the vessel and appears like a aneurysm on imaging

26
Q

When does an AAA officially need to be treated aggressively (repair)?

A

Until size is >4.5cm or AAA dissection symptoms present

  • If aggressively treating an AAA, DONT lower HTN if it is present (this is opposite with TAA) since this will cause lower limb necrosis*
  • treatment includes massive transfusion protocol and vascular surgery immediately
27
Q

Why is esmolol used more than labetalol?

A

Esmolol:

  • slower half life (can determine if patient can handle it and if not get it out quicker)
  • only blocks B- receptors (labetalol blocks A-receptors also 7B:1A)
28
Q

Differences between viable, threatened and dead tissue

A

Viable:

  • sensation and motor is intact w/ good pulses
  • have hours to repair without losing

Threatened:

  • loss of sensation and pulses but motor is still intact
  • have minutes to repair without losing

Dead:
- loss of sensation, motor and pulses