Vascular Flashcards

1
Q

What is this?
How can it be confirmed and rationale for this
What are the management priorities

A

**Phlegmasia cerulea dolens **

Confirm via:
* US lower limb - non compressable deep veins, no variance tp doppler pulse vein
* CT Venogram - assess clot burden and degree of clot. Map for intervention

Management
* Analgesia - dose and route
* Anti cogulation heparin infusion with loading
* Vascular surgery for embolisation

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2
Q

describe image and most likelt diagnosis

A

AAA approx 8cm
likely a bleed in the wall as fresh blood is white

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3
Q

what is the role of a CT in a ruptured AAA

A

if in a centre where endovascular is used CT is essential for planning and stent sizing
if laparatomy then it just delays time to theatre

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4
Q

Ruptured AAA
what are the steps in management whilst awaiting to go to theatre

A
  1. Urgent cross match/FFP/platelets
  2. permissive hypotension about 90mmhg
  3. analgesia eg fent or morphine
  4. explain plan to patient/family
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5
Q

collapse and abdo pain
?diagnosis

A

AAA

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6
Q

with an US confirmed AAA what would influence decision to do a CT

A
  • are they shocked - if so then no
  • proximitiy to CT
  • pre surgical planning
  • co-morbid status of patient
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7
Q

What are the definitive treatment options for AAA

A

Open repair
endovasculat stent
palliation

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8
Q

chest pain
what are the important features

A

inferior stemi
STE 2,3,avf (3>2)
reciprical changes
hyperacute t waves

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9
Q

what are the CXR features of aortic dissection?

A
  • Widening of the superior mediastinum
  • Dilatation of the aortic arch
  • Change in the configuration of the aorta on successive CXR
  • Obliteration of aortic knob
  • Double density of aorta (suggesting true and false lumen)
  • Localised prominence along aortic contour
  • Displacement of trachea to right
  • Distortion of left main stem bronchus
  • Pleural effusion
  • Cardiomegaly
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10
Q

what featurs of bedside echo would differentiate acute MI from aortic dissection?

A

MI
* Regional wall motion abnormality
* hypokenesis
**
Aortic dissection**
* pericardial effusion
* dilated IVC (tampanade)
* Intimal flap
* AV incompetence

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11
Q

what are the classifications of aortic dissections?

A

Stanford
o A: Ascending aorta with or without descending
o B: Descending aorta only (distal to origin of left subclavian)

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12
Q

What is the management of acute aortic dissection

A
  • Resus bay
  • 2 large cannulas and send bloods and cross match
  • IV analgesia
  • IV rate control to 60 eg metoprolol
  • IV BP control under 140 eg GTN
  • Art line
  • urgent transfer to centre which can have capacity for surgical fixing
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13
Q

major abnormality and diagnosis in chest pain

A

Large pericardial effusion

Thoracic aortic dissection with pericardial effusion, impending tamponade

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14
Q

what are the PRN end of life meds

A
  • Analgesia: morphine 1-2 mg subcut Q3H or hydromorphone or fentanyl
  • Sedative: midazolam 2.5 mg Q3H
  • **Anti-secretions: **hyoscine 400mcg Q3h or glycopyrrolate
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15
Q

what are the life threatening complications of aortic dissection

A

pericardia tampanade
stroke
MI
Severe AR

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16
Q

what are the risk factors for aortic dissection

A
  • hypertension
  • bicuspid aortic valve
  • male
  • increasing age
  • CTD eg Ehlor danlos
  • coarctation of aorta
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17
Q

what does aortic dissection cause hypotension

A
  • pericardial tampanade
  • aortic incompetence from aortic root dissection
  • iatrogenic from drugs given
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18
Q

diagnosis

A

Type A aortic dissection

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19
Q

what are the clinical signs of aortic dissection

A
  • loss of pulses
  • neurology - from stroke or spinal ischaemia
  • AR murmur
  • becks triad - signs of tampadane
  • significant patient distress
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20
Q
A

type a thoracic dissection

21
Q

4 findings and diagnosis

A
  • necrotic 2nd toe
  • pus
  • cellulitis
  • erythema with blister

Diagnosis
Diabetic foot infection with necrotic toe

22
Q

diabetic foot:
Investigations and justifications

A
23
Q

what are the causes of limb ischaemia with examples?

A
  • thrombotic - atheroscletoric plaque
  • embolic - AF, CCF, AAA
  • other - dissection. septic embolic
24
Q

Society for vascular surgery catergoes for limb ischaemia

A
25
Q

what are the medications for limb ischaemia

A
  • IV fentanyl aliquots and titrate to pain
  • heparin infusion titrate to APTT
26
Q

what are the interventional options for limb ischaemia

A
  • Catheter-directed thrombolysis
  • Embolectomy
  • Bypass
27
Q

features of limb ischaemia that suggests arterial embolism over thrombosis - on history

A
  • sudden symptoms
  • AF
  • no history of atherosclerotic disease
  • endocarditis
  • atrial myxoma
28
Q

features of limb ischaemia that suggests arterial embolism over thrombosis - on exam

A

presence of AF
murmurs
clinical signs of endocarditis
good proximal pulses
clear site of demarcation
more extensive gangrene or limb threatened

29
Q

with limb ischaemia what are the complications of reperfusion

A

hyperkalaemia
compartment syndrome
renal failiure
rhabdo
re-occlusion and ischaemia
hypotension

30
Q
A
  • Embolic – from AF
  • Thrombotic
  • Traumatic – penetrating injury
  • Iatrogenic – IVC or IV drug arterial injection
  • Aortic dissection extending to subclavian artery
  • Neuro – spinal SAH
  • Thoracic outlet syndrome
31
Q

two findings/signs

What is the most important test?

A
  1. Pembertons sign
  2. goitre

CT chest ?mass

32
Q

what is pembertons sign and what does it suggest

A

engorged neck veins when raising arms

SVC obstruction

33
Q

what are the causes of SVC obstrutcion

A
  • mediastinal mass - lymphoma, thymoma, lung Ca,
  • retrosternal thyroid
  • aortic aneurysm
  • indwelling venous catheter
  • TB
34
Q

key findings and diagnosis

A

widened mediastinum
loss of aortic knob contour
pleural effusion

Aortic dissection

35
Q

AAA
main abnormalities and cause

A
  • Relative hypoxia with A-a gradient of approx 130 – likely causes include aspiration pneumonia/pleural effusion
  • HAGMA with anion gap 19 – likely cause hypoperfusion of gut from aortic dissection (this
    supported by raised lactate)
    Raised creatinine and elevated potassium – likely involvement of renal A in dissection
36
Q

severe abdo pain
findings

A

AAA
* Significant intraluminal clot
* Suggestion of rupture

37
Q

what is a massive transfusion in adult patient

A

over one whole blood volume in 24 hours or half in 4 hours

38
Q

what are the target parameters with massive transfusion

A
39
Q

young with chest pain
what are the abnormal findings

A

sinus tachy
RBBB

40
Q
A
41
Q

finding

A

saddle embolus

42
Q
  • define
    massive
    submassive
    nonmassive PE
A

* Massive PE
Acute PE + haemodynamic compromise (no longer defined by size)
* BP < 90 for 15min
* Vasopressor requirement
* Pulseless

* Submassive PE
Acute PE, not haemodynamically compromised but:
* Right heart strain (ECG / Echo / BNP)
* Troponin Leak

  • Non-massive PE
    Acute PE + none of above
43
Q

what are the 8 featurs of the PERC

A
44
Q

treatment for PE peri arrest

A

Improve oxygenation: HFNO2 50L/min, titrate fio2 to target SaO2 92-96%

Vasopressors: Noradrenaline infusion target BP 90-100

Systemic thrombolysis: Alteplase IV
* Deteriorating or arrest = 50mg alteplase bolus

  • Stable 100mg (10mg bolus, 90mg over 90min)
45
Q

what are the indications for thrombolysis in PE

A

cardiac arrest
massive PE with hypotension
Right heart strain or troponin leak
extensive clot burden
hypoxia

46
Q

what are the causes of a false negative d dimer

A

small clot burden
mature thrombus
recent bleeding
cancer
collection error

47
Q

components of wells score

A
48
Q

What are the ECG findings in PE

A

Sinus tachy
PR depression
RAD
RBBB
dominant R wave V1
LAD
atrial arrhytmia

49
Q
A