UW Peds Cards Flashcards

1
Q

EKG showing left-axis deviation; tall, peaked P waves (d/t RAH) from ASD; small or absent R waves in precordial leads; plus CXR showing normal-sized heart

A

Tricuspid atresia

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2
Q

CXR of tricuspid atresia

A

decreased pulmonary vascular markings and a normal sized heart

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3
Q

Murmur of tricuspid atresia

A

Holosystolic murmur loudest at LLSB (VSD)

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4
Q

increased pulmonary markings and cardiomegaly from excessive pulmonary blood flow and biventricular volume overload

A

CXR of CAVCD

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5
Q

displacement of a malformed tricuspid valve into the RV –> severe TR and RAH

A

Ebstein’s anomaly

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6
Q

Tall P waves and RAD on EKG plus extreme cardiomegaly from heart failure

A

Ebstein’s anomaly

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7
Q

Increased or decreased pulmonary vascular markings on CXR with TOF?

A

Decreased

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8
Q

Defect in which all 4 pulmonary veins fail to make their normal connection to the LA, so the RA receives blood from pulmonary and systemic circumlation

A

TAPVR

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9
Q

EKG of TAPVR

A

RVH and RAD

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10
Q

CXR of truncus arteriosus

A

cardiomegaly and increased pulmonary vascular markings

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11
Q

truncus arteriosus is strongly a/w

A

DiGeorge

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12
Q

benign murmur increases or decreases with standing

A

Decreases

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13
Q

pathologic murmur increases with

A

standing or valsalva

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14
Q

Pathologic murmurs may be a/w loud, fixed split or single S2 or decreases or absent

A

femoral pulses

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15
Q

HOCM murmur increases with standing because

A

standing decreases venous return and preload, which increases the obstruction

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16
Q

3 cardiac anomalies a/w Turner

A

bicuspid aortic valve, coarctation, aortic root dilation (w/ inc risk of aortic dissection)

17
Q

MVP is a/w

A

Marfan, Ehlers-Danlos, OI_

18
Q

PDA is a/w

A

congenital rubella, Char syndrome

19
Q

TOF is a/w

A

Down, DiGeorge

20
Q

VSD is occ a/w

A

trisomies 13, 18, 21

21
Q

carotid pulse has a dual upstroke, + strong apical impulse, + systolic ejection murmur along LSB

22
Q

Maneuvers that increase either preload or after load, such as squatting, leg raise, or sustained hand grip, increase LV cavity size in HOCM and thereby

A

decrease outflow obstruction –> decreased intensity of murmur

23
Q

Maneuvers that decrease preload, such as Valsalva, abrupt standing, or amyl nitrate administration, decrease LV size in HOCM and thereby

A

increase intensity of murmur

24
Q

loud S2 due to pulmonary HTN, systolic ejection murmur from increase flow across P from L to R shunt across AD, holosystolic murmur of VSD

A

findings on auscultation in CAVSD

25
Q

pansystolic murmur that is loudest at LLSB plus diastolic rumble at apex due to increase flow across the mitral valve

26
Q

stridor at age 6 mos - 6 yrs with barky cough

27
Q

strido most severe at age 4-8 months that worsens in supine position and improves in prone position

A

Laryngomalacia

28
Q

stridor that presents before age 1 year, improves with neck extension, and is a/w cardiac abnormalities; does NOT improve with steroids, rac epi, or albuterol

A

vascular ring

29
Q

w/u harsh holosystolic murmur best heard at LLSB

A

echo to determine location and size of VSD and r/o other defects

30
Q

accentuated peripheral pulses with continuous flow murmur last LSB

31
Q

mid systolic click with late systolic murmur

32
Q

Syndrome a/w several cardiac abnormalities including AS, PS, or septal defects

A

Williams syndrome

33
Q

tx PDA

A

indomethacin to close; prostaglandin E1 to keep open