Unit 4- GI/GU Flashcards
Ask about during medical HX with GI
Hx problems- IBD, IBS, GERD, constipation, change in bowels
ABD surgery
Gyn hx in women- LMP, method of contraception, STD risk
Medications
Order of abd assessment
Look- distension, surgical scars, peristalsis, pulsations, veins, tugor, hernias
listen- bowels, renal or aortic bruits
feel- painful area last, spleen/liver, rigidity, masses, pulsations, rebound tenderness
percuss- ascites, CVA tenderness, hepatosplenomegaly
Murphys sign
RUQ pain on deep innspiration, inflamed gallbladder
Rovsing sign
palpation of LLQ and pain in the RLQ = appendicitis
Obturator test
pain in RLQ on internal rotation of right hip= appendicitis
psoas sign
extension of right hip, and pain in the RLQ appendicitis
Causes of diffuse abd pain
IBD, IBS, gasatroenteritis, AAA, bowel obstruction, ischemic bowel
Causes of epigastric pain
MI, PUD, biliary disease, pancreatitis
Causes of LUQ pain
spleen, renal disease
Causes of periumbilical pain
early appendicitis, small bowel disease
Causes of LLQ pain
diverticulitis, PID, ovarian cyst, ectopic prego
Causes of RLQ pain
appendicitis, PID, ovarian cyst, ectopic prego
voluntary guarding
usually symmetric, muscles
more tense on inspiration,
usually doesn’t hurt to rise from supine to sitting position (using abdominal muscles),
lessens with distraction.
involuntary guarding
asymmetrical,
rigidity present on inspiration and expiration,
rising to sitting position greatly increases pain, doesn’t change with distraction
Appendicitis sx
anorexia
periumbilical pain that migrates to RLQ
N/V
+mcburneys point, rebound tenderness
+obturator, Rovsing and psoas sign
Cholelithiasis sx
colicky pain, located in epigastrium or RUQ and flank, occasionally R shoulder
Pain occurs within 1 hr after eating large meal, last several hours, residual aching can last for days
anorexia N/V Fever \+murphys sign guarding and rebound increase WBC, total bili, ALT, Alk phosp, and amylase
Pancreatitis sx
abrupt onset of severe epigastric pain that radiates to the back
pain increase with movement or lying supine (pt prefer to sit up and lean forward)
N/V sweating anxiety abd tenderness without guarding rigidity or rebound distension Absent bowel sounds fever tachycardic pallor hypotension
Increase amylase and lipase, WBC, ALT
Tx for appy
CT scan
surgery
Tx for choliliathisis
bowel rest (NPO)
pain management
IV abx
lap choly
Tx for pancreatitis
refer
KUB, CT
What is gastroenteritis
acute infectious diarrhea
usually self limiting
very young or elderly more at risk
causes of gastroenteritis
virus (70-80%)- rotavirus, adenovirus, water or person-to-person
Bacterial (10-20%)- s. aureus, c. diff
Parasites (<10%)- giardia
sx of gastroenteritis
viral- large volume watery stool
last 1-2 days
N/V, crampy, fever, malaise, dehydration
bacterial- may have bloody diarrhea, c. diff can occur 8 weeks after abx,
parasitic- watery diarrhea prolonger, cramps
get stool culture in gastroenteritis if
more than 3 days, <3mo old
>70 years
at risk (food service, day care worker)
tx for gasteroenteritis
supportive, assess for dehydration,
adults should avoid dairy, caffeien and alcohol
Eat rice, potatoes, wheat, banana, yogurt, soup, crackers
Imodium, Kaopectate
DO NOT use in severe or bloody diarrhea, high fever or systemic toxicity
diverticulitis define
inflammation of a diverticulum that ranges from icro perforation with localized inflammation, macro perforation with abscess or peritonitis
diverticulitis sx
aching abd pain usually LLQ, constipation or loose stools
N/V, low-grade fever, LLQ tenderness, palpable mass
+occult blood in stool, mild-moderate increase in WBC
pt dont seek attention until several days after onset
pt with perforation will have more severe sx
Tx of diverticulitis with mild sx
empiric abx without imaging, colonoscopy, CT colo, or barium enema
Clear liquid diet
usually improve in 3 days
high fiber diet
Tx of diverticulitis with severe sx
may need CT of abd in acute stage to eval severity
REFER for inpt, IV abx, possible surgery
GERD sx
heartburn usually 30-60 miin after meal with reclining, burning CP and regurgitation
Non-gi- asthma, chronic cough, laryngitis, sore throat, sleep disturbances
Alarm sx- >55yo, anemia, melena, hematemsis, dysphagia, weight loss, difficult/painful swallowing
tx for mild gerd
smaller meals, eliminate spicy or acidic foods, elevate HOB, dont lay down 3 hr post eat, weight loss
PRN antacids or H2 receptor antagonist- pepcid, zantac, tagament
tx of mod-severe gerd
empiric therapy PPI
PPI- once daily for 4-8 weeks
if no response=refer
+ response= keep taking PPI for 8-12 weeks
Causes of PUD
NSAIDS, h. pylori
sx of PUD
Dyspepsia
hunger-like burning pain in epigastrcic area, pain may awaken, sx wax and wane
up to 60% NSAID induced have no sx
normal or sightly tender epigastric, new onset guaiac stools
dx of PUD
upper endoscopy with biopsy
h pylori- upper breath test or fecal antigen
medications for PUD
acid antisecretory agents: PPI have replaced H2
abx-eradicate h pylori
What is colic
healthy, well fed infant that cries for more than 3 hours/day, more than 3 days a week and more than 3 weeks
severe and paroxysmal crying that occurs mainly in late afternoon
knees drawn up and fists clenched- begins at 2-3 months
intussusception
most common in first 2 years of life
thriving infant 3-12 mo with paroxysmal, colicky pain, draws up knees and screams
use barium or air enema
intussusception s/sx
vomiting, diarrhea, 90% bloody BM with mucus within 12 hours
prostration and fever, tender distended abd, sausage shaped mass may be palpated in upper mid abd
umbilical hernias affect what population
full-term african american within 1st year of life
T or F: umbilical hernias need repaired in adults due to high risk of incarceration and strangulation
True
Inguinal hernias
75% of abd hernias
congenital in children but can be d/t obesity, chronic cough, ascites, chronic constipations with straining and lifting heavy objects
Management of hernias
DO NOT reduce strangulated- can cause gangrenous bowel to enter peritoneal cavity
REFER
alarm markers for referral of hernais
acute onset of colicky abd pain, N/V, edema
discoloration at the site
intestinal malroation
healthy infant suddenly refusing to eat, vomits bile, becomes inconsalable- usually develops distention, occurs during first 3 weeks of life
use upper GI to and barium enema to confirm
surgery to fix
T or F: Pyloric stenosis is most common in females
FALSE, males
signs of pyloric stenosis
usually begins at 2-4 weeks old, projectile vomiting, rapidly becomes projectile after every feeding
appears hungry, eats frequently, constipation, dehydration, weight loss and fretful, olive size mass can be felt on deep palpation in RUQ
what does pyloric stenosis show on US?
hypoechoic ring with thickness >4mm
Tx of pyloric stenosis
hydration, correct electrolyte abnormality prior to surgical repair
Lactose intolerance
sx usually 4-6yo, intestinal dilation, bloating, increase flatulence, pain and diarrhea
onset of sx 2hr after ingestion of milk
tx- lactase supplement to dairy products
cows milk intolerance
occurs in infance, blood in stool, often manifestation of allergies
ectopic pregnancy
unilateral lower quad pain, continuous and crampy, vag bleeding and low-grade fever
EMERGENT referal to OB
Mittelschmerz
spillage of fluid from ruptured follicular cystic-irritates peritoneum-mid cycle pain
sx-sudden onset localized lower quad pain-persist for few minutes to as long as 8 hr
anovulatory cycles
lead to normal follicular cyst growing large over several cycles- considerable pain on rupture reveal intrauterine rego
EMERGENT referral
Tx of acute cystitis in women
cephalexin, nitrofurantoin, bactrim
Fluoroquinolone for uncomplicated UTI
most common cause of acute cystitis
e. coli
Labs for acute pyelo
leukocytosis and a left shift
UA-pyuria, bacteriuria, hematuria, white cell casts
tx for acute pyelo
empiric therapy-ampicillin, cipro, levofloxacin, bactrim,
when to refer for acute pyelo
complications
urolithiasis
obstruction
5 types of stones
calcium oxalate calcium phosphate struvite uric acid cystine
contributing to the stones
high humidity and elevated temps
sedentary lifestyle: HTN, carotid calcification, cardiovascular disease, high protein and salt intake, inadequate hydration
*keep sodium <150/day
Urge incontinence is?
a. most common cause of persistent
(INVOLUNTARY) incontinence in the elderly
b. present when involuntary leakage
occurs from effort or exertion or from sneezing or coughing
c.incontinence less than 6 weeks
spontaneously resolves with tx of underlying condition
d.prevalence of prostate disorders,
incontinence in older men due to obstruction of urinary
outflow; symptoms = dribbling
e.inability or unwillingness to
toilet because of physical, cognitive, psychological, or
environmental factors. Common in hospital and nursing
home patients
a.
Transient incontinence is?
a. most common cause of persistent
(INVOLUNTARY) incontinence in the elderly
b. present when involuntary leakage
occurs from effort or exertion or from sneezing or coughing
c.incontinence less than 6 weeks
spontaneously resolves with tx of underlying condition
d.prevalence of prostate disorders,
incontinence in older men due to obstruction of urinary
outflow; symptoms = dribbling
e.inability or unwillingness to
toilet because of physical, cognitive, psychological, or
environmental factors. Common in hospital and nursing
home patients
c.
Functional incontinence is?
a. most common cause of persistent
(INVOLUNTARY) incontinence in the elderly
b. present when involuntary leakage
occurs from effort or exertion or from sneezing or coughing
c.incontinence less than 6 weeks
spontaneously resolves with tx of underlying condition
d.prevalence of prostate disorders,
incontinence in older men due to obstruction of urinary
outflow; symptoms = dribbling
e.inability or unwillingness to
toilet because of physical, cognitive, psychological, or
environmental factors. Common in hospital and nursing
home patients
E.
Stress incontinence is?
a. most common cause of persistent
(INVOLUNTARY) incontinence in the elderly
b. present when involuntary leakage
occurs from effort or exertion or from sneezing or coughing
c.incontinence less than 6 weeks
spontaneously resolves with tx of underlying condition
d.prevalence of prostate disorders,
incontinence in older men due to obstruction of urinary
outflow; symptoms = dribbling
e.inability or unwillingness to
toilet because of physical, cognitive, psychological, or
environmental factors. Common in hospital and nursing
home patients
b.
Overflow incontinence is?
a. most common cause of persistent
(INVOLUNTARY) incontinence in the elderly
b. present when involuntary leakage
occurs from effort or exertion or from sneezing or coughing
c.incontinence less than 6 weeks
spontaneously resolves with tx of underlying condition
d.prevalence of prostate disorders,
incontinence in older men due to obstruction of urinary
outflow; symptoms = dribbling
e.inability or unwillingness to
toilet because of physical, cognitive, psychological, or
environmental factors. Common in hospital and nursing
home patients
d.
Dyspepsia is
predominant epigastric pain for at least 1 mo, epigastric fullness, N/V, heartburn,
everyone older than ___ get endoscopy with dyspepsia
60,
or selected younger with alarm features
In all others test for h. pylori
T or F: prescribe empiric PI in those with h. pylori who dont improve after eradication
True
pancreatic disease
pancreatic carcinoma and chronic pancreatitis=chronic epigastric pain
what is functional dyspepsia
most common cause of dyspepsia
increased visceral afferent sensitivity, gastric delated emptying or impaired accommodation to food or stressors
often younger, signs of anxiety or depression
What is biliary tract dysfunction
abrupt onset of epigastric or RUQ pain r/t cholelithiasis, elithiasis, or choledocholithiasis
tx of dyspepsia
initial empiric in all younger than 60 with no alarm feature (PPI)
all others get upper endoscopy with subsequent tx and cause
obtain gastric bisy- during endoscopy to test for h. pylori
If positive for h. pylori give abx
diagnostic studies for dyspepsia
upper endo- choice for GI ulcer, erosive esophagitis, increased GI malignancy
abd CT scan
sx of N/V w/out abd pain causes
food poisoning, infectious gastroenteritis, drugs, systemic illness
sx of N/V w/ severe pain and vomiting
peritoneal irritation, acute gastroparesis, intestinal dysmotility, psychogenic disorders, CNS or systemic disorder
Causes of vomiting in the morning before breakfast
prego, uremia, ETOH, increased ICP
Causes of vomiting immediately after meals
bulimia and psychogenic
causes of vomiting of undigested food one to several hr post meal
gastroparesis or gastric outlet syndrome
special exam for severe or protracted vomiting
electrolytes for assess hypokalemia, azotemia, or metabolic alkalosis (due to gastric contents)
special exam for flat and upright abd xray or CT
severe pain or suspicion of obstruction to look for intraperitoneal air or dilated loops of small bowel
What does upper endo determine?
gastric outlet obstruction
What does an abd CT imaging determine?
cause of small intestinal obstruction
What does a nuclear scintigraph study or c-octanoic acid breath test determine?
gastroparesis, shows delayed emptying and no evidence of gastric outlet obstruction
example of a serotonin 5-ht-receptor antagonist
ondansetron- effective in preventing chemo and radiation induced emesis and pstop when given prior to treatment
efficacy is enhanced by combined therapy with a steroid
corticosteroids
enhance efficacy of serotonin antagonist for preventing acute and delayed N/V in pt receiving emetogenic chemo
dopamine antagonists
promethazine
work by blocking dopaminergic and have sedative effects
se:extrapyramidal reactions and depression
antihistamines and anticholinergics for n/V
meclizine, dimenhydrinate, scopolamine
work by stimulating labyrinth. give with oral vitamin B6 and doxylamine given FIRST LINE during PREGO
se: drowsiness
Cannabinoids for N/V
stimulate appetite and antiemetic
dronabinol- good for chemo and CNS pt
T or F: Hiccups can cause metabolic acidosis
FALSE- can cause respiratory alkalosis
causes of hiccups
gastric distention, sudden temp change, ETOH, heightened emotion
recurrent: GI, CNS, CV, and thoracic disorders
Assessment of hiccups include looking at what body systems?
Neuro
serum creatinine
liver chem test
CXR
Unclear cause= CT/MRI or head, chest, and abd, EKG, upper endo
Drug given for hiccups
Chlorpromazine
What is primary constipation?
unattributed to any structural abnormality or systemic disease - most common
What is normal colonic time?
approx. 35 hr, anything >72=ABNORMAL
What is secondary constipation?
cauesd by systemic disorders, medications, or obstructing colonic lesions
neoro gut dysfunction, myopathies, endocrine disorders, or electrolyte abnormalities
What is fecal impaction
sever impaction in the rectal vault that may lead to obstruction either partial or complete large bowel
causes-meds, pyshiatric disease, bed rest, neuro/spinal disorders
fiber will exacerbate sx in these disorders
colonic inertia
defecatory disorders
opioid-induced
IBS
When to give laxatives
in intermittent or chronic bases for constipation that does not respond to dietary or lifestyle changes
osmotic laxatives
daily
increase secretions of water into lumen
Miralax
Purgative laxative
rapid tx
mag citrate
may cause hypermagnesemia
stimulant laxatives
pt with no response to osmotic agents
stimulate fluid secretion and colonic contraction
bisacodyl, senna, cascara
Chronic excessive belching
supragastric belching or true air swallowing= behavioral seen in psychiatric pt
etiology of flatus
increased ingestion of lactose, polyols, and fructants or disorder of malabsorption
Avoid what in flatus
gum chewing or carbonated beverages, assess lactose intolerance
bloating cause
production of excess gas or impaired gas propulsion
treatment of bloating
reduce fermentable sugars with restricted diet, reduce intake of dietary fat
Rifaximin
Acute diarrhea
<2 weeks, most commonly caused by invasive or noninvasive pathogrens
what is acute inflammatory diarrhea?
fever and bloody diarrhea
colonic tissue damage r/t invasion or a toxin
acute inflammatory diarrhea s/sx
small volume diarrhea, LLQ cramps, urgency and tenesmus
fecal leukocytes or lactoferrin
causes of infectious dysentery
e coli
CMV
when should a stool sample be sent with diarrhea
7-14 days fr analysis for viral, protozoan, and bacterial pathogens
when to seek medical evaluation in diarrhea
6+ unformed stools in 24hr profuse watery diarrhea and dehydration frail older pt or nursing home immunocompromised exposure to abx hospital-acquired diarrhea systemic illness
tx for diarrhea
PO fluids
rest bowel
frequent feedings of tea, flat carbonated beverages and soft easily digested foods
antidiarrhea safe in pt with ild-mod diarrheal illness
loperaide
bismuth
abx- empiric tx, fluoroquinolones, bactrim
DONT GIVE macrolides
Chronic diarrhea last longer than
4 weeks
causes of chronic diarrhea
carbohydrate malabsorption*
laxative abuse
malabsorption syndromes
secretory conditions for chronic diarrhea
increased intestinal secretions or decreased absorption=high vlm watery diarrhea w/ normal osmotic gap
caused by endocrine tumors and micro colitis
inflammatory conditions for chronic diarrhea
IBS, UC, chrons
abd pain, fever, weight loss, hematochezia
malabsorptive conditions for chronic diarrhea
small mucosal intestinal disease, intestinal resections, lymphatic obstruction, small intestinal bacterial overgrowth, pancreatic insufficiency
weight loss, osmotic diarrhea, steatorrhea, and nutritional deficiencies
motility conditions for chronic diarrhea
IBS
lower abd pain+altered bowel habits w/out evidence of serious organic disease
chronic infections conditions for chronic diarrhea
protozoans, intestinal nematodes, c.diff
systemic conditions conditions for chronic diarrhea
thyroid disease, DM, collagen vascular disorders
Which produces greasy or malodorous diarrhea
a. inflammatory
b. secretory process
c. malabsorption
c.
Which produces pus or bloody diarrhea
a. inflammatory
b. secretory process
c. malabsorption
a.
Which produces watery diarrhea
a. inflammatory
b. secretory process
c. malabsorption
b.
T or F: presence of nocturnal diarrhea, weight loss, anemia, positive FOBT warrant further eval
True
labs for malabsoprtion
anemia
- folate
- iron deficiency
- vit b12
hypoalbuminemia
labs for and inflammatory conditions
anemia
- folate
- iron deficiency
- vit b12
hypoalbuminemia
increased ESR/ C-reactive protein
labs for secretory diarrhea
hyponatremia
nonanion gap metabolic acidosis
T or F: Hct is a good early indicator of blood loss
FALSE
causes of upper GI bleed
PUD portal HTN mallory-weiss tear vascular anomalies gastric neoplasms erosive gastritis erosive esophagitis trauma
T or F: All pt with upper GI bleed should undergo endo within 48 hr of ED arrival
FALSE
within 24 hr
Gerd is exacerbated by
meals
bending
recumbency
Dysfunction of the gastroesophageal sphincter
transient relaxations of the LES- triggered by gastric distention by a vasovagal reflex
T or F: treatment is NOT warranted for pt with typical GERD suggesting uncomplicated reflux
TRUE
Heartburn and regurgitation should be treated with?
daily H2-receptor antagonists or
PPI for 4-8 weeks
alarm features with GERD
troublesome dysphagia
odynophagia
weight loss
iron deficiency anemia
T or F: Barium esophagography should be done to diagnose gerd
FALSE
tx for mild GERD
lifestyle modificiations and medical interventions
eat smaller meals and eliminate acidic foods/fatty foods/chocolate/peppermint/alcohol/ cigarettes
weight loss
avoid laying down 3 hr post meal
antacids take before meals
Tx for GERD
once daily PPI (omeprazole, lansoprazole, pantoprazole)
gastritis is put into what 3 categories
erosive and hemorrhagic
nonerosive, nonspecific
specific types
erosive/hemorrhagic gastritis causes
alcoholics, critically ill, taking NSAIDS
often asymptomatic, may cause epigastric pain, N/V, anorexia , upper GI bleed
major risk factors for erosive/hemorrhagic gastritis
vents, coagulopathy, trauma, burns, shock, sepsis, CNS injury, liver/kidney disease, MODS
tests for erosive/hemorrhagic gastritis
upper endo, labs (low Hct)
stress gastritis
mucosal erosions and subepithelial hemorrhages may develop w/in 72hr critically ill pt.
tx-continuous PPI
NSAID gastritis
less incidence of endoscopically visible ulcers
increase risk of MI, CVA, death
upper endo if- severe pain, weight loss, vomiting, GI bleed, anemia
tx- po PPI
alcoholic gastritis
excessive consumption=dyspepsia, N/V, minor hematamesis
tx- H2 receptor, PPI
portal hypertensive gastropathy
gastric mucosal and submucosal congestion of capillaries and venules
asymptomatic, can cause chronic GI bleed
tx- propranolol
non-erosive types
h. pylori
pernicious anemia
eosinophilic gastritis
h. pylori gastritis
inflammation w PMNs and lymphocytes
higher in non-whites and immigrants
transmission- person to person
tx- abx
fecal antigen immunoassay and urea breath test
d/c PPI 7-14d and abx for at least 28 days
Pernicious anermia gastritis
rare autoimmune, involves fundic glands w/ resultant achlorhydria, decreased intrinsic factor secretion, vit B12 malabsorption
T or F: most NSAIDS induced ulcers are asymptomatic
True
What are the 2 causes of PUD
NSAIDs and chronic H pylori infection
Alcohol, dietary factors, stress DO NOT cause ulcer disease
Duodenal ulcers are caused by___
h pylori
Tx of Uncomplicated H pylori–associated
ulcers:
PPI x14d
T or F: must confirm h pylori eradication for all pt more than 4 week after completion of abx therapy and 2 weeks after d/s of PPI
True
Hallmark sign of PUD
epigastric pain- dyspepsia
dyspepsia is relieved with what
food or antacids
sign of gastric outlet syndrome
significant vomiting and weight loss
nonhealing ulcers are suspicious for what?
malignancy
When should H2 receptor antagonist be given in uncomplicated peptic ulcers?
daily at bedtime
What is zollinger-ellison syndrome?
very rare
PUD severe and atypical gastric acid hypersecretion
gastrin-secreting gut tumors-excessive acid production by GI tract- mostly arise in gastrinoma triangle
what is the gastrinoma triangle?
portal hepatitis, neck of pancreas, 3rd portion of duodenum
T or F: ZES is very similar to PUD so will go undetected for many years
TRUE
ZES signs
diarrhea, GERD, steatorrhea, weight loss
NG aspirationof stomach acid stops diarrhea
ulcer patient with _____ or family hx of ulcers should be screened for ZES
hypercalcemia
What is the most sensitive and specific method for identifying ZES?
increased fasting gastric concentration >150
obtain when NOT taking H2 for 24hr or PPI for 6days
What is celiacs disease?
permanent dietary disorder to gluten due to immunologic
most present in childhood or adulthood
sign of celiacs
weight loss, chronic diarrhea, abd distention, growth retardation, dyspepsia, muscle wasting, hyperactive bowel sounds
dematitis, herpetiformis, IDA, osteoporosis
40% dont have symptoms
Dx celiac
abnormal serologic test+small bowel biopsy
IgA tissue transglutaminase antibody***
dietary supplements for celiacs disease
folate iron zinc calcium vit a, b6, b12, d, and e
sx of lactase deficiency
diarrhea
bloating
gas
abd pain post dairy
dx of lactase deficiency
hydrogen breath test
lactase deficiency
can arise from GI disorders that affect proximal small intestinal mucosa
chrons, celiac, viral gastroenteritis
What supplements do pt with lactase deficiency need?
calcium d/t risk for osteoporosis
sx of appendicitis
RLQ pain, +mcburneys point low grade fever leukocytosis colicky pain feel constipated
What can happen if appy left untreated?
gangrene
Gold standard screen for appy
CT scan
IBS
chronic functional disorder >6mo
characterized by abd pain w/ alterations in bowel habits
more common in women
T or F: IBS will interfere with sleep
FALSE
What to avoid in IBS
fatty foods, alcohol, caffeien, spicy foods, grains
T or F: drug therpay should be given to all IBS pt
FALSE
only given to mod-severe cases that dont respond to conservative measures
MEds for IBS
antispasmodic antidiarrheal anticonstipation psychotropic agents serotonin receptor antagonist
UC or Crohns has bloody diarrhea, fecal urgency, anemia, and low albumin?
UC
UC or Crohns is dx with sigmoidoscopy?
UC
UC or Crohns should have a colonoscopy?
not UC
UC or Crohns is tx w/ steroids
UC
UC or Crohns should not have antidiarrhealth agents
UC
UC or Crohns affects rectum to large bowel
UC
UC or Crohns affects anywhere from the mouth to anus?
crohns
UC or Crohns has continuous diffse inflammation
UC
UC or Crohns has patch inflammation
crohns
UC or Crohns has hematochezia
UC
UC or Crohns has mucus/pus
UC
UC or Crohns hassmall bowel disease
crohns
UC or Crohns has abd mass and where
chrons, RLQ
UC or Crohns has extra intestinal
BOTH
UC or Crohns has SBO
crohns
UC or Crohns has colonic obstruction
crohns
UC or Crohns has strictures and fistulas
crohns
UC or Crohns has thin and which one has thick bowel wall
thin=uc
thick=crohns
UC or Crohns has insidious onset with intermittent low grade fevers, RLQ pain
crohns
UC or Crohns is associated with smoking
crohns
UC or Crohns is acommon with chronic inflammatory disease
crohns
UC or Crohns has arthralgia, arthritis, iritis
crohns
UC or Crohns will have at least one surgery
crohns