Unit 2 Lecture Flashcards
integumentary system
- skin, hair, nails
- changes may be the first clue to other health problems
- functioning skin is essential for health/life
- reflects pt hydration, nutrition, emotional status
skin fun facts
- ) largest/heaviest organ
- ) waterproof
- ) insulating shield
- ) 16% body weight
epidermis
- outermost layer
- first line of defense against pathogens/irritants/moister loss
- thickness remains constant
- melanocytes
- cell that perceive pain, light touch, vibration, temp
melanocytes
- 2 types
- determine skin color
- in epidermis
dermis
- connective tissue- elastin and collagen
- contains bld vessels, nerves, sebaceous/sweat glands, lymph vessels, hair follicles
- thickness varies throughout life and between genders
collagen and elastin
- found in dermis
* provide resiliency, distensibility, elasticity, and turgor of skin
dermal thickness
- thinest at birth
- thickens until 4th decade and thins again
- thicker in men b/c more androgens
subcutaneous layer
- fat, loose connective tissue
- insulation
- storage of caloric reserves
- cushioning
- lose w/ aging
- provides skin recoil
pigments that determine skin color
- melanin
- carotene
- oxyhemoglobin
- deoxxygemoglobin
melanin
- amt genetically determined
- Amt increases by exposure to sunlight
- protects skin agains UV
carotene
- golden yellow
* in SQ fat, palms, soles
hemoglobin
- carries most of O2
- bright red when carrying O2 (oxyhemoglobin)
- blue when loses O2 (deoxyhemoglobin)
jaundice
•yellowish color
•deposition of bilirubin
•observed in sclera, nails, palms, soles
*hard to see in artificial light
cyanosis
- bluish color
- lack of O2 (central)
- lack of blood flow (peripheral)
albinism
- pale, milky skin, flaxen hari, and light irises
- 1/17,000 Americans
- lack melanin
- defenseless against sunlight and more prone to skin cancer
hair
- protects body from debris/invasion
* insulation
vellus
- short, fine, inconspicuous, unpigmented hair
* all over body
terminal
- course, thick, conspicuous, pigmented hair
* scalp, brows, eyelids, axillae, perineum, legs, chest
sebaceous glands
- support each hair follicle
- secrete sebum for moister/conditioning
- reduce H2O loss
sweat glands
•fxn in thermoregulation by controlling evaporation and resorption
eccrine sweat glands
- widely distributed
- open directly onto surface
- secrete sweat in response to environmental and psychological changes
apocrine sweat glands
- found in axillae, perineal areas
- stimulated by emotional stress
- responsible for BO
nails
- matrix in epidermal layer at distal fingers/toes
- nail plated (hardened keratine) grows at varying rates
- lunula- half moon just above cuticle
- growth/thickness can be affected by DZ
fxn skin
- barrier
- thermoregulation
- synth vit D
- sensory
- nonverbal comm.
- ID
- wound repair
- excretion metabolic waste
skin as barrier
- protects against injury (physical, chemical, thermal, UV)
- prevents penetration of organisms
- prevents loss of H2O and elecrolytes
skin and perception
•touch, pain, temp, pressure
skin and thermoregulation
- sweat glands- cool
* subcutaneous tissue- insulates
skin and expression
•huge role as canvas for the artistic/spiritual
pediatric skin
- apocrine immature fxn until puberty
- eccrine produced in lower amounts
- more terminal hair in axillae/perineum at puberty
comedones
- blackheads and acne
- form during puberty when apocrine glands mature and large ants sebum is builds up on hair follicles of face, neck, chest, back
Linea nigra
•hormonal changes in preggo cause darkened line from umbilicus to pubic area
older adult skin
- atrophy
- loss of elastin/collagen
- sub fat -> decreased resilience, sagging, wrinkling
- increased visibility of superficial vascular structures
- increased shearing/bruising
- rougher- less epi repain
- slower wound heal
health history subjective data of integument
•ID of
- ) disease
- ) abuse
- ) risk for pressure ulcer/cancer
concerning integument symptoms
- rash
- pruritis- itching
- non healing
- moles/lesions
- ecchymosis
- dry/too moist
- hair loss
melanoma
- first degree relatives w/ increase pt risk
- 10% of pt have relative
- key is to teach early signs and use of sunscreen
- results from exposure to UV-B
- increased risk if dysplastic nevi or > 50 moles
- found in face, shoulder, upper arms, back (men), legs (women)
integument past history
- skin diseases
- diabetes/peripheral vascular disease
- allergies/sensitivities
- (sun)burns
inspecting skin
- start at head/scalp
- use good lighting/draping
- note areas of brittle/broken/absent hair
- note pigmented areas- freckles/moles/birthmarks
- look for widespread color change
- progress down body to feet
- palpate for moisture, temp, texture, mobility, and turgor
- inspect skin folds
benign lesions
•freckles, moles, birth marks, skin tags, cherry angiomas
turgor
- measure of skin elasticity
- speed at which skin returns to normal after lifted
- decreases w/ thinning of dermis and reduced elastin production (aging)
- affected by hydration status
tenting
•skin with decreased turgor remains elevated after being pulled and released
•loss of elasticity due to dehydration/aging
* test at sight w/ less sun exposure
edema
- excess fluid in interstitial spaces
- skin puffy/tight
- localized indicates injury
- systemic- fluid goes to dependent part of body
- pitting/nonpitting
1+ pitting
- 2 mm depression
- barely detectable
- immediate rebound
2+ pitting
- 4 mm deep pit
* few sec rebound
3+ pitting
- 6 mm deep pit
* 10-12 sec rebound
4+ pitting
- 8 mm deep pit
- very deep
- > 20 sec rebound
physical exam lesions
•locations/distribution •pattern/shape/color •type •elevation •exudate *must describe exactly what is seen
primary lesion
•lesion develops on skin that was normal
secondary lesion
- changes in lesion over time
* seen in over treatment, scratching, infection of primary
vascular lesion
- develop b/c of blood supply problem to skin
- purpura
- port wine stain
- telangiectasias
- venous lake- varicose veins
pt w/ decreased mobility
- susceptible to skin damage/ulceration/microorganisms
- sustained compression obliterates blood flow
- distortion of soft tissue
- moisture promotes maceration of tissue and skin breakdown
pressure ulcers
- caused by sustained compression, friction, shearing forces, moisture
- easier to prevent than heal
- use Braden scale- low score= high risk
- common at sacrum, butt, greater trochanter, knees, heels, elbows, skin folds, back of head, under tubing
Braden Scale
•an assessment tool for predicting the risk of pressure ulcers •score of less than 18 is risk •based on the total of scores in 1.) sensory perception 2.) moisture 3.) activity 4.) mobility 5.) nutrition 6.) friction/shear
stages of pressure ulcers (I-IV)
I- alteration of intact skin; changes in temp, consistency, sensation, color
II- partial thickness skin loss of eli/dermis
III- full thickness skin loss w/ damage to subcutaneous tissue and maybe muscle; necrosis
IV- full thickness skin loss w/ damage to underlying muscle/bone/etc; necrosis
assessing for vascular blood supply
- direct pressure of finger or on skin surface of nail beds
- skin should blanch (pale) and promptly return to normal upon release
- delayed return of color indicates decreased circulation
arterial insufficiency
- vascular lesion
- poor arterial perfusion
- pale around b/c blood not getting to
- punched outlook
- well defined margins
- painful
venous stasis ulcer
- vascular lesion
- red around b/c blood not getting out
- venous valves don’t work
- blood pools/leaks, causing breakdown
- chronic pain
macule
•1° lesion •non palpable •change in color of skin •up to 1.0 cm *freckle
patch
- 1° lesion
- non palpable
- macule > 1.0 cm
vesicle
- 1° lesion
- palpable
- serous fluid filled
- up to 1.0 cm
- Ex: chix pox; blister; herpes
bulla
•1° lesion •palpable • > 1.0 cm vesicle •serous fluid *big vesicle
pustule
•1° lesion
•puss filled
*acne
burrow
- 1° lesion
- fluid filled
- slightly raised tunnel in epi
- mites/scabes
papule
•1° lesion •solid •distinct borders •up to 1.0 cm *mole
nodule/tumor
•1° lesion
•solid
• > 1 cm
*wart; cyst
plaque
- 1° lesion
- solid mass
- > 1.0 cm
- often coalescence of papules
- flat top (plateau)
wheal
•1° lesion
•solid mass
*hives/insect bites
crust
- 2° lesion
- dried residue of skin exudates
- dried serum, pus, blood
scale
- 2° lesion
- thin flak of dead
- psoriasis; dandruff
fissure
- 2° lesion
- linear crack in skin due to excessive dryness
- tinea pedis
erosion
- 2° lesion
- nonscarring loss of superficial epi
- moist
- doesn’t bleed
ulcer
- 2° lesion
- deeper loss of eli/dermis
- bleed and scar
excoriation
- 2° lesion
* linear or punctate erosions caused by scratching
lichenification
- 2° lesion
- visible/palpable thickening of epi w/ roughing of skin
- thick, leathery skin due to rubbing/scratching
- atopic dermatitis, eczema
scar
- 2° lesion
* extra connective tissue arises from injury/disease
keloid
•2° lesion •hypertrophic scarring that extends beyond borders of initial injury •overgrowth of granulation tissue •firm/rubbery *Mike's arm
purpura
- vascular lesion
- deep reddish purple
- flat
- > 3 mm
petechiae
- vascular lesion
* small purpura (1-3 mm)
port wine stain
- vascular lesion
- ruby red mark on skin
- due to abnormal aggregation of caps.
- vascular birth mark
telangiectasias
- vascular lesion
- dilated small vessels
- spider veins
venous lake
- vascular lesion
* varicose veins
mongolian spots
- common in dark-skinned native am. and hispanic babies
- flat gray-blue in color
- caused by pigment that didn’t make it to top layer of skin when formed
- harmless and fade with age
striae gravidarum
- atrophic, pinkish/purplish scar-like lesions that later become white
- due to weakening of elastic tissues during pregnancy
- on breasts, thighs, and butt
- also associated with overweight, rapid growth during puberty, Cushing’s syndrome, and corticosteroid use
ephelides freckles
•flat light brown spots that appear in sun and fad in winter
lentigines freckles
- small tan, brown, or black spots that do not fade in winter
- bigger than freckles
- in older people- sun exposure
dermatophyte
- mold-like fungi that thrives in warm, moist ares
- contagious
- tx w/ topical agents
tinea capitis/corporis
- ringworm
* capitis- of scalp
tinea pedis
•athletes foot
hirsutism
- female has male pattern hair distribution
* found often with polycystic ovarian syndrome
performing skin self examination (SSE)
•ABCDE method Asymmetry Border irregularity Color variation (esp. blue/black) Diameter 6 mm or greater Evolving shape/size/color
melanoma prevention
- reduce midday sun exposure- when UV-B rays most intense
- use at least 15 SPF
- reapply every 2 hrs
older adult nose/ears
- continue to grow
- skin sags w/ loss of subcutaneous fat
- lesions likely
silent thyroiditis
•slight enlargement of thyroid in some preggo
fontanel
- a space between the bones of the skull in an infant or fetus, where ossification is not complete and the sutures not fully formed
- main one is between the frontal and parietal bones
hypothyroidism
- cold intolerance
- coarse hair
- thinning brows
- fatigue
- anorexia
- dry skin
- irregular menstruation
- weight gain
hyperthyroidism
- heat intolerance
- thin hair
- weight loss
- anxiety
- increased BP, HR, diaphoresis
- muscle weakness/fatigue
- exophthalmia- bulging eyes
facial features not symmetrical
•indicates CN7 (facial) nerve damage