Unit 1: Stroke: Hemorrhagic Flashcards

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1
Q

Hemorrhagic Stroke

A

bleeding into the brain

-blood is a irritant to the brain

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2
Q

3 Main subtypes of Hemorrhagic Stroke

A
  • Nontraumatic Subarachnoid Hemorrhage (SAH)
  • Intracerebral Hemorrhage (ICH)
  • Intraventricular Hemorrhage (IVH)
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3
Q

Subarachnoid Hemorrhage

A

caused by a ruptured aneurysm (weakened blood vessel) and less commonly caused by an arteriovenous malformation (AVM; mass of arteries and veins that is not connected by a capillary network)

  • sudden severe headache; “thunderclap” headache b/c intensity of the pain at onset
  • neck stiffness and pain occur b/c of the irritation of the meninges, at base of skull, where pooling of blood occurs
  • photosensitivity w/ meningeal irritation or inflammation
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4
Q

Aneurysm

A

weak, dilated vessel

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5
Q

Arteriovenous Malformation (AVM)

A

mass of arteries and veins that is not connected by a capillary network

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6
Q

Risk Factors for Subarachnoid Hemorrhage (SAH)

A
  • hypertension
  • smoking
  • heavy alcohol use
  • use of sympathetic nervous stimulants (cocaine)
  • female gender
  • hx of cerebrovascular disease
  • postmenopausal state
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7
Q

How does a Cerebral Aneurysm Occur?

A
  • as a result of an inherent weakness or gradually acquired weakness of the medial layer in a segment of a blood vessel; muscular layer that adds shape and tone to the vessel; a weakness in a segment of this layer causes an outpouching of the blood vessel through the outermost adventitial layer
  • aneurysm ruptures in the thinnest walled portion of the aneurysm (dome)
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8
Q

Clinical Manifestations of Hemorrhagic Stroke

A
  • similar to ischemic; motor deficit, visual field cuts
  • severe headache
  • early and sudden changes in LOC
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9
Q

Medical and Surgical Management

A

prevent and minimize complications such as aneurysm rebleeding and cerebral vasospasm (causes narrowing of blood vessel segments)

  • Aneurysm clipping: aneurysms are secured by applying a titanium clip to the neck of the aneurysm or
  • Aneurysm coiling: deploying platinum coils into the aneurysm during angiography; reduces blood flow to the aneurysm
  • surgical evacuation of a hematoma below the tentorium b/c risk of brainstem compression
  • intraventricular hemorrhage managed with CSF drainage
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10
Q

Complications of Hemorrhagic Stroke

A
  • Subarachnoid Hemorrhage causes neurological and systemic complications; ischemic stroke, cerebral edema, pulmonary edema, myocardial ischemia/infarction, acute respiratory distress syndrome (ARDs)
  • Vasospasm (causes change in consciousness, new focal (localized) motor weakness
  • Hyponatremia
  • Seizures (blood is irritant to brain)
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11
Q

Complications: Hyponatremia

A

may be caused by SIADH or by cerebral salt wasting syndrome (renal salt wasting syndrome)

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12
Q

Nursing Assessments

A
  • Know the patients baseline for neurological status
  • Serial neurological assessments; q 1-2 hours
  • Vital Signs; q 1-2 hours or more when administering meds that alter BP
  • ECG
  • Serum Electrolytes
  • Intake and Output
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13
Q

Nursing Assessments: Vital Signs

A

after hemorrhagic stroke, close monitoring of vital signs (BP) is necessary to prevent rebleeding or expansion of hematoma

  • If BP lower than prescribed targets, there is risk of hypoperfusion
  • If BP higher than prescribed targets, there is risk of increased ICP and rebleeding
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14
Q

Nursing Assessments: Electrocardiogram (ECG)

A

identification of rhythm disturbances (A-fib, ST segment, or T wave changes associated with MI) is essential in determining potential causes of stroke and intervening in complications of stroke

  • after SAH, may experience myocardial stunning
  • trending of 12-lead ECGs and cardiac enzymes allows identification of potential myocardial infarction and/or reversible myocardial stunning
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15
Q

Nursing Assessments: Electrolytes

A

-sodium: to identify disorders of salt and water imbalance resulting in hyponatremia, which places patients who have suffered a stroke at high risk for cerebral edema and neurological deterioration

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16
Q

Nursing Assessments: Intake and Output

A
  • evaluates potential sodium and water imbalance

- approximating volume status

17
Q

Nursing Interventions

A
  • Perform bedside swallow eval
  • Administer Meds as ordered
  • Elevate HOB to 30 degrees
  • Positioning of HOB if endovascular procedure (15 degrees then increasing gradually)
  • Place feeding tube
  • Aspiration precautions
  • Frequent Positioning; elevate weak limbs to minimize edema
  • Seizure precautions
  • Administer calcium channel blocker
  • often care is supportive
18
Q

Nursing Interventions Specific to Hemorrhagic Stroke

A
  • Seizure precautions (blood is irritant)
  • Administer Calcium Channel Blockers
  • Often care is supportive
19
Q

Interventions: Perform bedside swallow screening

A

after a stroke, may experience swallowing dysfunction, at risk for aspiration and subsequent pneumonia
>swallowing is complex and involves structures in the brainstem, thalamus, and cerebral cortex as well as tongue and facial movements, many of which can be compromised after stroke
>cranial nerves IX, X, XI, XII contribute to swallowing and innervate the palate and pharynx, assisting in airway protection
-when these cranial nerves have been damaged, aspiration of food or fluid into the lungs can occur

20
Q

Interventions: Nasogastric or Postpyloric feeding tube

A
  • for nutrition and medication administration
  • to facilitate enteral feeding to allow time for swallowing function to improve or for more formal swallowing evaluation to be completed
  • if swallowing dysfunction is thought to be a long-term problem or permanent, a percutaneous endoscopically placed gastrostomy tube may be placed by surgeon or gastroenterologist
21
Q

Methods to prevent aspiration

A
  • providing supervision of the patient while eating to observe for clinical manifestations of aspiration or choking
  • maintaining HOB at least 45 degrees or greater while eating or drinking
  • reducing distractions to assist a patient in concentrating on eating and drinking
  • advocating for evaluation of patient by SLP
  • tucking the chin when swallowing
  • thickening liquids with fiber additives
  • prescribed therapeutic food preparation (soft or pureed)
  • communicate appropriate precautions and diet to the other healthcare team members that may be involved w/ feeding the patients (PCT)
22
Q

Medications prescribed for secondary stroke prevention

A

-Antihypertensives: used to reduce BP to prevent long-term damage from excessive shear and reduce chance of intracerebral hemorrhage
>Beta blockers (metoprolol [Lopressor])
>Calcium channel blockers (prevent vasospasm, which vasospasm constricts vessels)
>Diuretics

23
Q

Metoprolol (Lopressor)

A

antihypertensive; beta blocker

  • used to reduce BP and HR
  • Side Effect: bradycardia, HF, pulmonary edema, fatigue, weakness, erectile dysfunction
  • Nursing: monitor BP, EKG, and pulse frequently during dose adjustment and during therapy; monitor vital signs and EKG q 5-15 min during and for several hours after parenteral administration; if HR <40 bpm (and if cardiac output is also decreased) administer atropine IV
24
Q

Calcium Channel Blockers

A
  • antihypertensive
  • dilates coronary arteries in both normal and ischemic myocardium and inhibit coronary artery spasm
  • prevents vasospasm, which vasospasm constricts vessels causing high BP)
  • Side Effects: constipation, dizziness, tachycardia, fatigue, flushing, nausea
25
Q

Diuretics

A

cause diuresis and subsequent mobilization of excess fluid

  • decreases BP; for hypertension
  • Side Effect: dehydration electrolyte imbalances, hypovolemia
26
Q

Nurse Teachings

A
  • stroke diagnosis
  • activation of EMS team
  • warning S/S of stroke
  • risk factors for stroke
  • smoking cessation
  • medications for secondary prevention of stroke