Unit #1 Flashcards
Lab value: Sodium (Na+)
135-145
Lab value: Potassium (K+)
3.5-5.0
lab value: Calcium (Ca+)
8.5-10.5
lab value: Magnesium (Mg+)
1.3-2.1
lab value: Chloride (Cl-)
97-110
lab value: Phosphate (PO4)
2.5-4.5
lab value: Hydrogen ion concentrate or potential of Hydrogen (pH)
7.35-7.45
lab value: Partial pressure of Oxygen (PaO2)
80-100
lab value: Carbon dioxide (CO2)
35-45
lab value: Partial pressure of Carbon Dioxide (PaCO2)
35-45
lab value: Bicarbonate (HCO3)
24-30
lab value: Carbonic Acid (H2CO3)
1.2
lab value: BUN
8-25
lab value: Creatinine
0.6-1.5
lab value: Glucose
65-110
lab value: Serum Osmolality
280-300
lab value: Hematocrit percentage (Hct)
36%-52%
S/Sx of Hypernatremia
fluid overload- will see: lethargy, drowsiness, stupor, or comatose)
deep tendon reflexes decrease- will see muscle weakness, twitching, etc.
Disease process with Hypernatremia
Kidney failure- poor kidney excretion
Tx for Hypernatremia
Diuretics to promote Na loss
ie: Lasix or Bumex
S/Sx of Hyponatremia
Sudden onset acute confusion or increased confusion- can see seizures, coma, or death
General muscle weakness- will see decrease tendon reflexes
Disease process for Hyponatremia
Prolonged use of diuretics
tx for Hyponatremia
decreased use of diuretics, give IV fluids
S/Sx of Hyperkalemia
Bradycardia, hypotension, ECG changes (peak T waves)
tingling/burning sensations followed by numbness in hands and feet
Disease process for Hyperkalemia
hospitalized pts undergoing treatment, chronically ill, elderly pts, or kidney failure
Tx for Hyperkalemia
Give Potassium-excreting diuretics (loop diuretics- lasix)
Stop all potassium food/drug sources
S/Sx of Hypokalemia
Shallow respirations, check mucus membranes and nail beds
muscle weakness, orthostatic hypotension
Disease process for Hypokalemia
Cushing’s syndrome, diuretics, corticosteroids, insulin drip (regular insulin)
Tx for Hypokalemia
Give IV or PO potassium, change to potassium sparing diuretics
S/Sx of Hypercalcemia
Renal stones, bone pain, abdominal pain, N/V
Polyuria, insomnia
Disease process for Hypercalcemia
Hyperparathyroidism
Tx for Hypercalcemia
re-hydration, increase salt intake
S/Sx of Hypocalcemia
petechiae, Chvostek’s sign (facial twitch on one side)
Disease process for Hypocalcemia
Hypoparathyroidism, vitamin D deficiency
Tx for Hypocalcemia
IV calcium gluconate
S/Sx of Hypmagnesemia
Weakness, N/V, decreased Resp rate
Decreased BP, decreased blood Calcium
Disease process for Hypermagnesemia
Kidney failure, over dose of antacids and laxatives
Tx for Hypermagnesemia
IV calcium gluconate
S/Sx of Hypomagnesemia
muscle cramps, abnormal Heart rate or rhythms
tremors, parasthesia
Disease process for Hypomagnesemia
Chronic diarrhea, malabsorption, alcoholism. or diuretics
Tx for Hypomagnesemia
PO Magnesium or IV Magnesium Sulfate
To decrease pH level, give:
Bicarb
5% dextrose in water
hypotonic or isotonic
0.9% Sodium Chloride
isotonic
5% dextrose in 0.9% Sodium Chloride
Hypertonic
5% dextrose in 0.45% Sodium Chloride
Hypertonic
Lactated Ringers
isotonic
3% normal Saline
Hypertonic
5% dextrose in Lactated Ringers
hypertonic or isotonic
Isotonic solutions are similar to:
blood serum (stays in the vascular space and expands volume)
Hypertonic solutions:
pulls fluids out of cells, into the vascular space (given for fluid overload)
Hypotonic solutions:
pull fluids into the cells, out of the vascular space (given for dehydration)
3 advantages of IV therapy:
Therapeutic effect is quicker
Control over rate of administration
Great for pts who cannot tolerate otehr routes
3 disadvantages of IV therapy:
CANNOT reverse toxic dose as quickly or effectively
Increased drug to drug interference
Can be more expensive
3 common complications of IV therapy:
Phlebitis- inflammation of vein
Infiltration- fluids leak into tissues
Thrombosis- blood clot in vein caused by extremes in osmolarity or pH
Advantages and Disadvantages of PICC lines
Advantage: decreased r/f infection, short-term use
Disadvantage: easily damaged