Traumatic Occlusal Forces Flashcards

1
Q

Other conditions affecting the
periodontium
(5)

A

Systemic diseases
Periodontal abscess or
periodontal/ endodontic lesions
Mucogingival deformities and
conditions
Traumatic occlusal forces
Tooth and prosthesis related
factors

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2
Q

Occlusal Trauma (OT) Diagnosis

A

Injury resulting in tissue changes within the attachment
apparatus (periodontal ligament, cementum and supporting
bone) as a result of occlusal forces (etiology)

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3
Q

Occlusal Trauma (OT) Diagnosis
Occlusal forces = !

A

Teeth

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4
Q

Occlusal Trauma (OT) Diagnosis
May occur in an

A

intact periodontium or in a reduced
periodontium affected by periodontal disease

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5
Q

What is a ‘reduced’ periodontium?
Based on an in vitro study, reduced is loss of —%
of bone support

A

> 60

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6
Q

AAP World Workshop 2017
Occlusal trauma and excessive*
occlusal forces
Questions asked:
(2)

A
  • Does occlusal trauma (OT) initiate periodontal disease?
  • Does occlusal trauma lead to progression of existing
    periodontal disease?
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7
Q

AAP World Workshop 2017
Occlusal trauma and excessive*
occlusal forces
Questions asked:
* Does occlusal trauma (OT) initiate periodontal disease?
* Does occlusal trauma lead to progression of existing
periodontal disease?

*Point of interest-title of workshop includes —, but terminology is
changed to —

A

excessive
traumatic

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8
Q

Why the Change?

A

Research performed for over 100 years but used different
animal models and experimental design (magnitude,
direction and location of forces)
* Sheep
* Human necropsy
* Beagles
* Squirrel Monkeys
Overall, past studies showed lack of ‘cause and effect’.
i.e. Occlusal Trauma (OT) did not cause pocket formation or
lead to loss of connective tissue.

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9
Q

Parts of the Periodontium
Affected by Occlusal Forces

A
  1. Cementum
  2. PDL
  3. Alveolar Bone Proper
    The gingiva and junctional epithelium are not
    affected by occlusal forces.
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10
Q

Classification of Traumatic Occlusal Forces
on the Periodontium (2017)
1. Occlusal Trauma
(3)

A

A. Primary occlusal trauma
B. Secondary occlusal trauma
C. Orthodontic forces

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11
Q

Occlusal Trauma
Variables:
(4)

A
  1. Direction of force.
  2. Magnitude of force.
  3. Duration of force.
  4. Frequency of occurrence
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12
Q

Trauma From Occlusion
1. Considered to be —.
2. Forces of occlusion — the adaptive capacity
of the periodontium

A

pathologic
exceed

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13
Q

Primary Occlusal Trauma 2017

A

Traumatic occlusal forces applied to a tooth or teeth
with normal periodontal support

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14
Q

With Primary occlusal trauma, clinically may see

A

adaptive mobility (does not progress)

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15
Q

Primary Occlusal Trauma 2017
Example is

A

‘high’ restoration with mobility resolving
following reduction.

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16
Q

Secondary Occlusal Trauma 2017

A

Injury resulting in tissue changes from normal or
traumatic occlusal forces applied to a tooth or teeth
with reduced periodontal support
* May be seen as progressive mobility &/or pain

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17
Q

Trauma from Occlusion
A. Compression side
(4)

A
  • PDL space is reduced as fibers are compressed
  • Loss of fiber orientation
  • Increased capillary permeability, rupture of blood
    vessels and hemorrhage into PDL perivascular
    spaces (edema)
  • Resorption of alveolar bone proper (root resorption
    if severe) then widening of PDL space
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18
Q

Minor Trauma (from occlusion)
(5)

A
  • Increased capillary permeability, dilation
  • Edema, disturbed fluid exchange
  • Vascular damage with stasis, clotting, thrombosis
  • Lowered periodontal resistance?
  • Accompanying tissue effects, usually minor
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19
Q

Trauma from Occlusion
B. Tension side
(4)

A
  • Increase in PDL space
  • Rupture of PDL fiber bundles
  • Compression of PDL blood vessels and
    hemorrhage into perivascular spaces
  • Deposition of new alveolar bone and decrease in
    PDL space (If severe, cemental tears)
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20
Q

Severe Trauma (from occlusion)
(5)

A
  • Crushing (pressure) injury - necrosis at furca, alveolar crest
  • Extravasated RBCs, hematoma, necrosis, vascular damage
  • Well-defined necrosis, including PDL, cementum, bone
  • Degenerative changes (hyaline, mucoid, liquefaction)
  • Repair from PDL, endosteal cells, bone marrow, Haversian
    systems (rear resorption)
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21
Q

Term: Primary Occlusal Trauma
Definition:
Manifestation:

A

Traumatic occlusal
forces applied to tooth
or teeth with NORMAL
periodontal support

Adaptive mobility (not
progressive or
pathologic)

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22
Q

Term: Secondary Occlusal Trauma
Definition:
Manifestation:

A

Normal or traumatic
occlusal forces applied
to a tooth or teeth with
reduced periodontal
support

Progressive mobility
(may exhibit mobility
and/or pain on
function)
Consider splinting?

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23
Q

Problem
The lesion of Occlusal Trauma can only be confirmed
—, so must use
other surrogate indicators
(2)

A

histologically by block section biopsy

  • Clinical
  • Radiographic
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24
Q

Proposed clinical and radiographic indicators of
occlusal trauma
(11)

A
  1. Fremitus (palpable or visible
    movement of a tooth when subject to
    occlusal forces)
  2. Thermal sensitivity
  3. Mobility
  4. Discomfort/pain on chewing
  5. Occlusal discrepancies (working
    &/or balancing interferences)
  6. Widened periodontal ligament space
  7. Wear facets
  8. Root resorption
  9. Tooth migration
  10. Cemental tear
  11. Fractured tooth
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25
Q

Fremitus

A

A palpable or visible movement of a tooth when
subjected to occlusal forces.

26
Q

Clinical Signs/Symptoms of
Occlusal Trauma
1. — of affected teeth.*
2. Radiographic evidence of —
3. Positive Hx of —
4. — teeth.
5. Evidence of working and/or balancing
side —

A

Mobility
thickened PDL.
clenching or bruxism.
Missing or tilted
occlusal interferences

27
Q

Mobility Assessment
1=
2 =
3 =

A

first distinguishable sign of
movement > than normal
(physiologic)

movement which allows crown to
move 1 mm from its normal position
in any direction

tooth may be rotated or depressed
in alveoli

28
Q

Mobility Assessment
Must use

A

2 rigid instruments, NOT fingers

29
Q

Occlusal Discrepancies
(2)

A
  • Working &/or balancing interferences
  • Wear facets (BUT may be normal part of aging!)
30
Q
  • Working &/or balancing interferences
    (3)
A
  • Evidence of occlusal slide in CR or CO
  • Evidence of occlusal interferences in protrusive
    mandibular movement
  • Extremely steep cuspal inclines
31
Q

Bruxism

A

Grinding, clenching or clamping of the teeth.
* The force may damage tooth or attachment
apparatus

32
Q

Signs & symptoms of bruxism:
(7)

A
  • Increased mobility
  • Pulpal sensitivity / bite tenderness
  • Non-masticatory / excessive occlusal
    wear
  • Dull percussion sound
  • Muscle tenderness / spasm /
    hypertrophy / tiredness (am)
  • TMJ pain / jawlock
  • Audible sounds
33
Q

Other Manifestations of Traumatic
Occlusal Force
(3)

A
  • Malocclusions
  • Tooth migration
  • Fractured teeth
34
Q

Radiographic Signs
(5)

A
  • Widened PDL space
  • Thickening of lamina dura
  • Vertical (angular) bone loss
  • Furcal bone loss
  • Alveolar radiolucency &/or
    condensation
35
Q

Periodontal Ligament Dimensions
* PDL thickest at —
Less at —
* Varies with —

A

apices & alveolar crest (0.20
mm);
mid-root (0.15 mm)
functional / force status of tooth

36
Q

Problems with surrogate indicators
* Existing — may contribute to
mobility
* — may be due to ‘normal’ function rather
than parafunctional habits (bruxism, clenching,
grinding)
* Altered — of teeth may be due to other factors

A

loss of attachment
Wear facets
vitality

37
Q

Indications for Occlusal Adjustment
(4)

A
  • Traumatic injuries / soft tissue injury; food
    impaction
  • Increasing mobility or fremitus
  • Parafunctional habits
  • In conjunction with orthodontic /
    orthognathic therapy
38
Q

Contraindications to Occlusal
Adjustment
(5)

A
  • Absence of a pre-treatment diagnosis
  • As prophylactic therapy or only treatment
    for periodontal disease
  • As primary therapy of bruxism
  • Severe extrusion or malpositioned teeth
  • When periodontal inflammation has not
    been controlled
39
Q

Effect of Periodontal Treatment on
Mobility
Tooth mobility negatively affects outcome of
Tooth mobility generally will decrease once

A

periodontal therapy and maintenance

inflammation is controlled

40
Q

‘Recommendations’ from Workshop
* If see signs and symptoms of occlusal trauma and patient’s comfort
and function are impacted then perform

A

occlusal adjustment in
conjunction with periodontal therapy

41
Q
  • Evaluate and record — before, during and after treatment
A

occlusion

42
Q
  • Treatment of occlusal trauma ‘may
A

slow the progression of
periodontitis and improve the prognosis

43
Q

Orthodontic Forces
Animal studies- certain orthodontic forces can
adversely affect the periodontium and cause (4)

A

root
resorption, pulpal disorders, gingival recession
and alveolar bone loss

44
Q

Orthodontic Forces
Observational studies-

A

teeth with a reduced but
healthy periodontium (no inflammation) may
undergo successful tooth movement without
compromising periodontal support

45
Q

Occlusal Hyperfunction
1. — increase in occlusal force.
2. Considered to be a — adaptation
and not a — entity

A

Slight
physiologic
pathologic

46
Q

Clinical Symptoms of Occlusal
Hyperfunction
1. Increase in number and diameter of —
2. Increased —
3. Increased density and thickness of —
4. Radiographic evidence of —.
5. Slight or undetectable tooth —

A

collagen fiber bundles in PDL
width of PDL.
alveolar bone proper (lamina dura).
osteosclerosis
mobility

47
Q

Occlusal Hypofunction
(3)

A
  1. A mild weakening of the tooth supporting
    structures due to lack of physiologic
    stimulation.
  2. Considered to be a physiologic adaptation
    and not a pathologic entity.
  3. Can only be diagnosed by histology
48
Q

Occlusal Hypofunction
1. — in number of PDL fiber bundles
but normal orientation.
2. — physiologic turnover and
remodeling of alveolar bone.
3. — of PDL space.
4. — change in tooth mobility

A

Decrease
Decrease
Narrowing
No

49
Q

Disuse Atrophy

A

Total removal of occlusal forces resulting in
lack of the level of physiologic stimulation
required to maintain normal form and
function.
Physiologic adaptation and not considered
pathologic

50
Q

Clinical Symptoms of Disuse Atrophy
(3)

A
  1. Radiographic evidence of decreased width
    of PDL space.
  2. Increased tooth mobility is always present.
  3. Absence of occlusal antagonist.
51
Q

Disuse Atrophy
1. — of the principle fiber
bundles of the PDL.
2. — PDL width.
3. — in number of bony
trabeculae, i.e., localized osteoporosis

A

Loss of orientation
Narrowed
Significant decrease

52
Q

Trauma From Occlusion
Trauma from occlusion, in the absence of
inflammation, does not cause:
(3)

A
  • gingivitis
  • periodontitis
  • pocket formation
53
Q

The Role of occlusion in the Dental Implant and
Peri-implant condition: A Review. Graves CV,
Harrel SK et al.,
Open Dent J 2016, Nov 16;10:594-601
“Several articles demonstrated that occlusion and
occlusion overload could detrimentally affect the
— condition, while other articles did not
support these results.”

A

peri-implant

54
Q

The Role of Occlusion in Implant Therapy:
A Comprehensive Updated Review.
Sheridan RA, Decker AM et al.
Implant Dent 2016 Dec; 25(6):829-838
PubMed database review 1950 to September 2015
* Findings

A
  • Recommendations still lacking regarding implant
    occlusion but include
  • Mutually protected occlusion with
  • Anterior guidance
  • Wide freedom in centric relation (decrease cuspal
    inclines)
  • Reduce occlusal overload (more implants, less
    cantilevers)
  • Close monitoring for parafunctional habits
55
Q

Harrell communication on AAP Forum
2019
Performed comprehensive lit review for textbook chapter on occlusion in
the failure of implants most articles were case reports/opinion articles
& related to prosthetic failure.
Recommend
(2)

A
  • Occlusal adjustment (prior to implant restoration)
  • Hard acrylic bite guard in all cases (or where parafaunctional habits
    are suspected)
56
Q

Conclusions
Traumatic Occlusal Forces (TOF)
* No evidence that this causes —
* Limited evidence (animal and human) that it
causes —
* Observational studies that TOF may be
associated with severity of —
* Animal model-
* Human-

A

periodontal
attachment loss in humans
inflammation in the periodontal ligament
periodontitis

may increase alveolar bone loss
no evidence

57
Q

Traumatic Occlusal Force and
Relationship to
(2)

A
  1. Non carious cervical lesions
    (NCCL’s) /Abfraction
  2. Recession
58
Q

Traumatic Occlusal Force(s) and
Abfraction
NO EVIDENCE that TOF causes

A

non-carious cervical
lesions (NCCLs). Most studies used finite element
analysis (not clinical)
NCCLs may result from abrasion, erosion or corrosion

59
Q

Recession

A

EVIDENCE from observational studies that Traumatic
Occlusal Force does NOT cause gingival recession

60
Q

Abfraction

A

No credible clinical evidence to support existence of
abfraction
Therefore there can be no evidence implicating
abfraction as cause of recession!