Traumatic Brain Injury Flashcards
Intracranial Fluid
10% CSF, 12% blood and 78% brain tissue that makes up 1.9L and does not alter the volume
Cerebral Blood Flow
requires 750-1000mL of blood due to the inability to store glucose and O2 but high demands for them.
Deficits in Cerebral Blood FLow Markers
<50 cerebral ischemia and >1500 is max constriction and intracranial hypertension occurs
Mean Arterial Pressure MAP
systolic BP + 2(diastolic BP)/3
ABG Levels in the Brain
High CO2 (>45mmHG) or low O2 (<50mmHG) relaxes smooth muscles, dilated cerebral arteries, and decreased cerebral vascular resistance Low O2 severe (<35mmHG) leads to anaerobic metabolism, lactic acid accumulation, high H+ concentration, low pH, increased cerebral vascular resistance, and vasodilation when autoregulation is lost
Cerebral Perfusion Pressure
the pressure needed to ensure brain functioning, MAP + ICP
Intracranial Pressure ICP
balance of components in the brain, one goes screwy, the rest compensate. typical levels are 5-15mmHG, >20mmHG is brain damage
Factors that Influence ICP
cardiovascular diseases BP intra-ab and intrathoracic pressure body position temp ABGs, mostly CO2 intrinsic mechanisms that control ICP
Monro-Kellie Doctrine
state of dynamic equilibrium where the one component goes screwy, the rest compensate and fix their mess
Pressure-Volume Curve/Relationship
Stage I is intracranial compartments increase in volume, throwing off the others, autoregulation is intact
Stage II is compliance beginning to be exhausted, increased of high ICP if there are no changes
Stage III comp mechanisms are exhausted and start to exhibit the mild S&S
Stage IV we be fucked, at risk for hypoperfusion, herniation, and death, where there is a stroke occurring
Herniation
brain tissue is forcibly shifted from the compartment of greater pressure to = the pressure gradient
Brain Injury
any trauma to the skull, scalp and brain that includes an alteration in LOC, no matter how brief
Primary Brain Injury
the initial time of injury that results in displacement, bruising and damage of the three components of the skull
Secondary Brain Injury
hypoxia, ischemia, hypotension, hypertension, edema or increased OCP that occurs from a primary injury later on
Scalp Lacerations
minor brain injury. Don’t be alarmed if it bleeds a lot, that is just how it be