Trans - Pediatric Cardiac PE Flashcards

1
Q

Harrison’s groove

A

rib deformity and sign of chronic heart failure with diaphragm enlargement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

palpating for heaves - movement on which part of the hand indicates LV heave? RV heave?

A

[1] RV heave - palm/sternum

[2] LV heave - fingers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

cause of thrills

A

turbulence of underlying murmurs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

thrills can first be felt at what grade of murmurs

A

4/6

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

the stethoscope diaphragm is useful for picking up which sounds?

A

high pitched sounds - S1, S2, murmurs of aortic and mitral regurgitation, pericardial friction rubs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

the stethocscope bell is useful for picking up which sounds?

A

low pitched sounds - S3, S4, murmur of mitral stenosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

how is ascultation of the pediatric patient done? why?

A

C manner (infraclavicular –> parasternal –> apex –> axilla), because most pediatric heart conditions are congenital

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

where is the normal splitting of S1 detected?

A

lower left sternal border

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

physiological splitting of S2 occurs when?

A

during inspiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

why does the physiological splitting of S2 occur?

A

During inspiration, the increase in negative intrathoracic pressure causes increased central venous return. Because this increased volume passes through the pulmonary trunk, causing the pulmonic valve to close a bit later than normal.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

components of S2

A

A2, P2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

which is louder? A2 or P2?

A

A2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

[T/F] S3 or S4 in an athlete is an abnormal finding

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

how are murmurs differentiated from normal heart sounds?

A

murmurs have a longer duration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

things to evaluate in auscultation [6]

A
[1] rate
[2] rhythm
[3] quality of heart sound
[4] S1/S2
[5] extra heart sounds
[6] murmur
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

where is S1 louder?

A

apex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

where is S2 louder?

A

base

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

midsystolic murmur - description

A

begins after S1, stops before S2; gaps between murmur and heart sound

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

midsystolic murmur - lesions

A

deals with blood flow across semilunar valves

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

holosystolic murmur - description

A

starts with S1 and stops at S2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

holosystolic murmur - lesions

A

often due to regurgitant flow across AV valves

22
Q

late systolic murmur - description

A

in mid- late- systole persisting up to S2

23
Q

late systolic murmur - lesions

A

mitral valve prolapse (may or may not be preceeded by systolic click)

24
Q

early diastolic murmur - description

A

starts right after S2 without gap, fades into silence before next S1

25
early diastolic murmur - lesions
often accompany regurgitant flow across incompetent semilunar valves
26
middiastolic murmur - description
begins quickly after S2
27
middiastolic murmur - lesion
turbulent flow across AV valves
28
S3 rumbles - description
middiastolic murmur associated with S3, associated with torrential inflow
29
Austin Flint murmur - description
begin in middiastole, may be confined to middiastole or continue through presystole
30
S3 rumbles - lesion
AV regurgitation, ASD
31
Austin Flint murmur - lesion
aortic regurgitation
32
pericardial friction rub - causes
inflammation of pericardial sac
33
pericardial friction rub - components [3]
[1] atrial systole [2] ventricular systole [3] ventricular diastole
34
patent ductus arteriosus
open channel persists between aorta and pulmonary artery
35
patent ductus arteriosus - type of murmur
continuous murmur with silent interval late in diastole and loudest in late systole
36
[T/F] friction rubs are a type of heart murmur
F, not caused by flow
37
crescendo-decrescendo murmur - lesions
present in obstructive lesions
38
crescendo-decrescendo murmur - why?
crescendo - ventricles try to pump blood through obstruction | decrescendo - blood passes through, pressure is released
39
aortic regurgitation - type of murmur
diastolic
40
aortic stenosis - type of murmur
systolic
41
pulmonic regurgitation - type of murmur
diastolic
42
pulmonic stenosis - type of murmur
systolic
43
mitral stenosis - type of murmur
diastolic
44
mitral regurgitation - type of mumur
systolic
45
tricuspid regurgitation - type of murmur
systolic
46
tricuspid stenosis - type of murmur
diastolic
47
ventricular septal defect - type of murmur
uniform holosystolic murmur (due to pressure gradient present in systole)
48
patent ductus arteriosus - type of murmur
continuous murmur (due to pressure difference in both systole and diastole)
49
atrial septal defect - ascultatory findings [3]
[1] diastolic rumble in tricuspid (stenosis) [2] pulmonic murmur (due to increased blood flow from right ventricle to pulmonic valve) [3] widely fixed split S2 (due to increased blood flow through pulmonic valve)
50
atrial myxoma
mass in atrium causing mitral inflow obstruction
51
atrial myxoma - type of murmur
crescendo-decrescendo murmur in diastole