Topic 3 - Dyspnoea Flashcards

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1
Q

5 main causes of dyspnoea

A
  • Neurological
  • Airway obstruction
  • Respiratory compromise
  • Cardiovascular compromise
  • Thoracic muskuloskeletal compromise
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2
Q

Mild (partial) airway obstriction management

A
  • Reassurance
  • Encourage spontaneous efforts to clear airway
  • Inappropriate or premature intervention can result in severe or complete obstruction
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3
Q

Severe airway obstruction management (concious)

A
  • Up to five sharp back blows
  • Up to five chest thrusts
  • Repeat if required
  • Ensure ongoing assessment of airway and concious state
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4
Q

Severe airway obstraction (unconcious)

A
  • Remove foreign body under direct visualisation

If not:

  • O2
  • Gentle IPPV
  • LMA/ETT
  • Appropriate resuscitation
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5
Q

Croup and clinical manifestations

A

Croup is a viral ilness that causes inflammatory oedema of the subglottis

  • Coroyzal prodrome (URTI), hoarse voice, inspiratory stridor, harsh ‘barking’ cough, widespread wheeze, increased WOB and fever
  • Affects children 6mths to 3 years generally
  • 2-5 day symptoms, worse at night
  • NOTE - nebulised adrenaline is a temporising measure only
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6
Q

Croup - differential diagnosis

A
  • Inhaled foreign body
  • Epiglottitis
  • Bacterial tracheitis
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7
Q

4 components of bronchospasm in asthma

A
  • Bronchospasm
  • Inflammation and oedema of airways
  • Mucous plugging
  • Airway smooth muscle hyperplasia and hypertrophy
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8
Q

Asthma classifications

A
  • Mild, moderate, severe, life-threatening
  • Gradual onset
    • Over days or weeks - higher mortality - responds less well to treatment
  • Rapid onset
    • Often has a precipitating trigger and responds quickly to treatment
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9
Q

Electrolyte complications of asthma

A
  • Hypokalemia
  • Hypocalcemia
  • Lactic acidosis
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10
Q

Risk factors for life-threatening exacerbations in astma

A
  • Prior ICU admission and prior intubation
  • Three or more admissions to hospital over the last 12/12
  • Currently taking steroids for asthma
  • Poor compliance with medications
  • NOTE - SpO2 is a POOR indicator of severity - will not decrease until later stages
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11
Q

Important patient history factors in asthma

A
  • Previous asthma history (including ICU admissions and intubations, number of admissions in the last 12 months etc.)
  • Co-existing medical conditions
  • Asthma triggers if known and cause of current episode
  • Duration of symptoms (? physical exhaustion)
  • Medication use and compliance
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12
Q

IPPV in asthma

A
  • Prolonged expiratory phase
  • ADULT: 4-6 b/m
  • LARGE CHILD: 8-10 b/m
  • SMALL CHILD: 10-15
  • INFANT:15-20
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13
Q

Asthma Managment

A
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14
Q

Main causes of cardiogenic APO

A
  • Left ventricular failure (LVF)
  • Increased intravscular volume
  • Pulmonary venous outflow ovstruction (eg. mitral valve stenosis)
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15
Q

Main causes of non-cardiogenic APO

A
  • High output states such as:
    • Septicemia
    • Anaemia
    • Thyrotoxicosis
  • Systemic increased vascular permiability
    • Pancreatitis
    • Eclampsia
    • DIC
    • Burns
  • Toxins/environmental
    • Submersion
    • Toxic inhlation
    • HAPE
  • Other
    • Head injury
    • PE
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16
Q

Clinical featured of APO

A
  • Crackles at bases, progressing to higher fields as progresses
  • Pink, frothy sputum is severe cases
  • Respiratory distress
  • JVD
17
Q

APO management

A
18
Q

COPD and exacerbation management

A
19
Q

Anaphylaxis definition

A

Any acute onset illness with typical skin features plus involvement of respiratory and/or cardiovascular and/or persistent severe gastrointestinal symptoms OR any acute onset of hypotension or bronchospasm or upper airway obstruction where anaphylaxis is considered possible

20
Q

Resolved anaphylaxis and transportation

A

Due to possibility of byphasic response, all patients should be transported to a medical facility

21
Q

Anaphylaxis management

A
22
Q

Salbutamol (Pharmacology)

A
  • B2 agonist
  • Lowers serum potassium levels via direct stimulation of the sodium/potassium ATPase pump
23
Q

Salbutamol (Indications)

A
  • Bronchospasm
  • Suspected hyperkalemia (with QRS widening AND/OR AV dissasociation)
24
Q

Salbutamol (Contraindications)

A
  • KSAR or hypersensitivity to salbutamol
  • Patients <2 years old
25
Q

Salbutamol (Precautions)

A
  • APO
  • IHD
26
Q

Salbutamol (Side Effects)

A
  • Anxiety
  • Tachyarrhytmias
  • Tremors
  • Hypokalemia and metabolic acidosis
27
Q

Salbutamol half-life

A

1.6 hours

28
Q

Ipratropium Bromide (Pharmacology)

A
  • Inhibits cholinergic vasomotor tone by decreasing intracellular intracellular Ca2+ (blocks acetylcholine)
29
Q

Ipratropium Bromide (Indications)

A
  • Moderate OR severe bronchospasm
30
Q

Ipratropium Bromide (Contraindications)

A
  • KSAR or hypersensitivity to anticholinergics
  • Patients <2 years
31
Q

Ipratropium Bromide (Precautions)

A
  • Glaucoma
32
Q

Ipratropium Bromide (Side Effects)

A
  • Dilated pupils
  • Dry mouth
  • Palpitations
33
Q

Laryngoscopy contraindications

A
  • Suspected or known epiglottitis
34
Q

Laryngoscopy complications

A
  • Laryngospasm
  • Hypoxia due to ventilation delay
  • Upper airway and mouth trauma
  • Exacerbation of underlying c-spine injury
  • Vomiting
35
Q

CPAP - mechanism

A

Increases intrathoracic pressure to decrease venous return and decrease preload, afterload and improve cardiac function

36
Q

CPAP contraindications

A
  • Patients <16
  • GCS <9
  • Inadequate ventilatory drive
  • Hypotension
  • Pneumothorax
  • Facial trauma
  • Epistaxis
37
Q

CPAP complications

A
  • Aspiration
  • Gastric distension
  • Hypotension
  • Corneal driving
  • Barotrauma