topic 1B: caridac Flashcards
role of tendons in heart valves
prevent vales from turnign inside out when the ventricles contact under high pressure
how do elastic fibres in artery walls contirbute to maintaing blood pressure
they stretch, then recoil. the recoil squeezes blood, mainting pressure and ensuring continous flow
4 structures of the ehart to support its funciton as a pump
chmabers- strong walls to pump blood
valves- ensure one way flow
septum- seperates oxygenated and deoxygenated blood
caridac muslce- contracts withuot fatigue
why do capillaries hve thin walls and how does this facilate diffusion
they hve one cell thick walls for diffusion.
how does an aneurysm form and wt r the consequences
it forms when an artery walls weaknes and swells due to plaque buildup. consequences r like breakage, internal bleeding, stroke, death
wt is atherosclerisis and how does it develop over time
atherosclerosis is the hardening of arteries. plaques build up, triggered by damage to the artery lining and can resitirct blood flow
wt is the relationship between high blood pressure and the development of cardiovascular diseases
high blood pressure can damage artery linings, leading to plaque foemation and increased risk of CVDs
2 sympotoms of angina
chest pain
breathlessness
wt causes myocardial infarction and how does it relate to atherosclerosis
myocaridal infarction (heart attack) is often caused by a blood clot due to atherosclerosis. plaques form atherosclerosis and damage arteris, leading to clots
how does cholesterol relate to heart disease and wt types of lipoproteins r invloved
high chloestoel specially LDLs can lead to heart disease. LDLs deposit cholesterol while HDLs help remove it
wt r stratins and how do they fucniton in reducing cholesterol levels
they lower cholesterol by blocking an enzyme in the liver, reudicng LDL productions imporving the LDL and HDL balance
5 treatments for managing atherosclerosis
lifestyle change
antihypertensives
statins
anticoagulants
platele inhibitors
diff between ischemic and hemorrhagic strokes
ischemic - blockage cuts off blood to the brain
hemorrhagic - bleeding from damaged brain capilaries
how does low vitamin C intake on cardiovascular health
low vitamin C may lead to scurvy and damaged blood vessels, leading to CVD risk
wt role do platelets play in the formation of blood clots related to atherosclerosis
plateletes stick to damaged plaque in arteries, trigering the blood clotting process and forming clot. this can block the artery causing a heart attack
how do LDLs and HDLs differ in their roles related to cholesterol and heart disease
LDLs carry cholesterol to cells and high levels can lead to plaque buildup
HDLs remove cholesterol from tissues and artiers reducing plaque
3 funcitons of peripheral arteries in teh criculatory system
carry blood away from the heart
control blood flow to organs by contristincg lumen size
regulate organ activity
how might individual genetic differences affect cholesterol metabolism and cardiovascular risk
genes affect fat metabolism, LDL and HDL balance and cholesterol levels.
why do large multicellular organisms needs internal transport systems
they need them to trnasport oxygen,nutrients, hormones to cells and remove waste efficeintly
how do thromboplastin and calcium ions contribute to blood clotting
thromboplastin starts clotting by converting prothrombin to thrombin
calcium ions r needed for this conversion to happen
wt adaptation allow mammals to sustain a higher metabollic rate compared to other animals
mammals hve double circulation
they also hve efficient oxygen transport to cells
how does the criculatory system differ fish and mammals
fish hve a single-loop system , where blood passes thru the heart one per circuit.
mammals hve a double-loop system, with seperate pulmoary and systemic cirucits
how does the biconcave shape of red blood cells aid their function
their biconcave shape increases the surface area to volme ratio, which helps oxygen to diffuse in and out of the cell quicker
wt role does dietary fats play in the risk of developing CVD
high saturated fat intake correlates with high blood cholesterol and LDLS, increasing CVD risk