TMJ/Facial Pain Flashcards
What are extra-articular disorders?
-Makes up majority of TMJ complaints (95%)
-Hand to side of the face
-Can range from muscular pain to other conditions that present as TMJ pain
What are causes of muscle pain?
-Muscle fatigue
-Heavy exercise
-Dehydration
-Pregnancy
-Hypothyroidism
-Decreased Mg or Ca
-ETOH abuse
-Renal failure
What is Hilton’s orthopedic law?
The nerves that innervate a joint, also innervate the muscles that move the joint along with overlying skin
TMJ- CN V
What are conditions that can mimic extra-articular pain?
Pulpitis, pericoronitis, otitis, sinusitis, parotitis, trigeminal neuralgia, atypical facial pain, temporal arteritis, nasopharyngeal Ca, Eagle’ssyndrome/fractured styloid
What are extra-articular conditions that cause limited jaw function?
-Odontogenic/non-odontogenic infections
-myositis/myofacitis
-myositis ossifacans
-neoplasms
-scleroderma
-coronoid hyperplasia
-zygomatic arch fractures
-psychological (hysteria, extra pyramidal reactions)
-tetanus
Describe the facial pain cycle.
-Predisposed susceptible muscle group
-Precipitating traumatic event
-Perpetuating para-functional habit
What is the management for extra-articular disorders?
Non-invasive measures:
-Jaw rest
-Soft diet
-Stretch/PT
-Ice/moist heat
-Orthotic splint
-Amitriptyline10-30 mg at bedtime to prevent bruxism
-NSAIDs (mobic 7.5 mg per day)
-Behavioral counseling
What are intra-articular disorders?
Less common ~5%, finger pointing directly to joint
What are developmental causes of intra-articular disorders?
Condylar hyperplasia, condylar hypoplasia
-Tx with reestablishment of occlusion, reconstruction with orthognathic surgery, costochondral graft, custom alloplastic implant
What are neoplastic causes of intra-articular disorders?
Benign: Osteoma, synovial chrodomatosis, osteochondroma
Malignant: Fibrosarcoma, osteosarcoma, synovial sarcoma
What are traumatic causes of intra-articular disorders?
Subluxation, dislocation, fracture
Describe arthritic intra-articular disorder.
-Pathogenesis for degenerative changes of articular cartilage
-Stress from bruxism
-chronic microtrauma
-Compression and shearing
-Chondrocyte damage (collaganases)
-Proteoglycan chain splitting and water loss
-Loss of cartilage resilience
-Chondromalacia
Describe the imaging for TMJ evaluation.
Panorex: Initial screening
CT: Bony pathology
MRI: Soft tissue, disc position
Describe MRI TMJ imaging.
T1: Bright: Fat, bone marrow, better for anatomy
T2: Bright: Water/edema, better for anatomy
-Earliest findings of internal derangement is T2 hyper-intensity in the bilaminar zone
-Late stage: T2 disc deformity (biconvex, thickened, folded, perforation)
-Anterior or anteriolateral disc displacement is most common finding
-With progression of disease, disc does not reduce, becomes fixed anteriorly during opening/closing
-Fluid accumulates in joint
-Cortical erosion with condylar head flattening and anterior osteophytes
-Subchondral marrow edema followed by low signal sclerosis
Describe Wilkes Stage I
Early Stage
-Clinical: No significnt mechanical symptmos other than early opening reciprocal click, no pain or limited ROM
-Radiographic: Slight forward displacement, good contour of disc
-Anatomic: Excellent for slight anterior displacement
Describe Wilkes Stage II
Early/Intermediate Stage
-Clinical: One or more episodes of pain, beginning major mechanical problems, mid to late opening loud click, transient catching/locking
-Radiographic: Slight forward displacement, beginning disc deformity with slight thickening
-Anatomic: Anterior disc displacement, disc deformity with good central articulating area
Describe Wilkes Stage III
Intermediate Stage
-Clinical: Multiple episodes of pain with restriction of function, major mechanical symptoms with locking
-Radiographic: Anterior disc displacement with significant deformity/prolapse
-Anatomic: Marked anatomic disc deformity with anterior displacement, no hard tissue change
Describe Wilkes Stage IV
Intermediate/Late Stage
-Clinical: Slight increase in severity over intermediate
-Radiographic: Increase in severity showing early degenerative changes, deformed condylar head, sclerosis
-Anatomic: Hard tissue degenerative remodeling with osteophytes, adhesions in anterior and posterior recess, no perforation of disc
Describe Wilkes Stage V
Late stage
-Clinical: Crepitus, grating, grinding, continual pain, chronic restriction of motion, difficult function
-Radiographic: Disc perforation, gross anatomic deformity of disc and hard tissue with degenerative arthritic changes
-Anatomic: Gross degenerative changes of disc and hard tissue, disc perforation, multiple adhesions, osteophytes, flattening of condyle and eminence, subcortical cyst formation
What are the goals of surgical treatment of the TMJ
In patients that have failed non-surgical therapy
Goals: Reduce adverse joint loading, reduce inflammation and pain, establish normal painless ROM
Indications: Significant pain and/or dysfunction when non-surgical therapy has failed, evidence of disease on imaging, localized pain to the joint the better the prognosis (more diffuse pain=worse prognosis), failure to manage associated myofascial pain and dysfunction will worsen the surgical success rate