Tiffany's Spine Cards Flashcards

1
Q

What are kids predisposed to upper cervical spine #s? (3)

A
  1. Horizontal cervical facets
  2. Ligamentous laxity
  3. Large head to body ratio
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

In pediatric spine trauma, where are the majority of injuries?

A

80% are in c-spine. Younger kids (<8yo) are mostly upper cspine. Older kids are throughout cspine.
In adults, 30% spine trauma involve cspine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How do you properly immobilize pediatric c-spines in trauma? (2)

A

2 options given large head to body ratio
1. Backboard w recess for occiput
2. Standard backboard w pad under torso
Normal spine board causes forced cspine flexion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is pseudosubluxation of the c-spine? What locations are the most common?

A

Anatomic variant in kids <8yo due to more horizontal facet joints. Most common C2 on C3. 2nd most common is C3 on C4

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How do you differentiate cervical pseudosubluxation from true injury? (4)
What fracture may be a cause of true C2 on C3 subluxation?

A
  1. Pseudosubluxation reduces on extension lateral views
  2. No hx of trauma
  3. No anterior soft tissue swelling
  4. Swischuk’s line: within 1.5mm of C2
    True injury: hangman’s #
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is Swischuk’s line?

A

Differentiate cervical pseudosubluxation in kids from true traumatic injury
Swischuk’s line on lateral XR. AKA spinolaminar line
Draw along anterior cortex of posterior arch from C1 to C3
If physiologic variant, anterior cortex of C2 posterior arch will be within 1mm of line
True injury if >1.5mm from line

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

True or False: cervical pseudosubluxation in kids is associated w increased morbidity and mortality

A

False. No increase in either. Treatment is observation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Describe how pediatric C-spine lordosis changes w positioning

A

With neutral position, loss of lordosis. Lordosis restored w extension. This is physiologic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is normal ADI for kids and adults?

A

Kids <8yo : <5mm
Kids >8yo and adults : <3mm
This excludes conditions such as RA or syndromes where there is ligamentous laxity. In these conditions, ADI is less useful. Better to look at SAC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

True or False: in pediatric trauma, CT is an appropriate screening tool for c-spine trauma?

A

False. Inappropriate radiation dose and most injuries seen on XR
Can consider CTA to R/O vertebral artery injury in c-spine #s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

In pediatric trauma, when is MRI an appropriate screening tool for s-spine trauma? (2)

A
  1. Obtunded
  2. Neuro deficit
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

In traumatic pediatric atlantoaxial instability due to ligamentous injury, what are the treatment options for younger and older kids?

A
  • Younger: Brooks fixation + halo
  • Older: C1 lateral mass to C2 pedicle screw fusion. May not need halo
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

RC: Which of the following is associated w an increased risk of complications w halo treatment in kids?
A. Re-tightening the screws at an appropriate interval
B. Placing the ring 2cm above the pinna
C. Placing the ring closer to the skull
D. using 6 pins instead of 4

A

Answer: B
This was a tough question and still not sure what is the best answer. Maybe some misremembering
Halo pin should be tightened. Ring should be 1cm superior to pinna. Any higher may risk ring migration and dislodgement.
Ring should have 1-2cm clearance. Not sure what “closer to skull” means
Kids should have more than 4 pins. Typically 8

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is SCIWORA?

A

Spinal cord injury without radiographic abnormality. Only in kids
Diagnosis of exclusion
Occurs because spine is more elastic than spinal cord. Spine may have stretched without #, injury cord
May not have XR or MRI findings

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Why may patients w SCIWORA present w delayed neuro deficits?

A

2 theories
1. Poor initial exam
2. Hypotension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Describe the differences in SCIWORA injuries in younger kids (<10yo) and older kids

A
  • Younger: more cervicothoracic junction injuries. Complete injuries
  • Older kids: more mid-thoracic injuries. Incomplete injuries. More likely to have recovery
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What is the treatment for SCIWORA?

A

No anatomic injury to fix. External immobilization x12ks then activity modification x12wks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

RC: In pediatric patients w SCIWORA, all are true except:
A. Thoracic spine is most common
B. Anatomic and physiologic injuries don’t match
C. Cervical neuro injury has worse prognosis than lumbar neuro injury
D. 25% can present w delayed neuro changes

A

Answer: A (thoracic is most common. Not true)
Cervical SCIWORA is most common
Anatomic and physiologic injuries don’t match: neuro manifestations don’t match anatomic injuries seen on imaging
They can present w delayed neuro changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What is an apophyseal ring?

A

Secondary ossification center of vertebral body. Connects to disc annulus fibrosus via Sharpey’s fibers. Completely ossifies at 18yo. Weak point that may fracture

20
Q

What are apophyseal ring #s?

A

AKA limbus #. Unique to kids. More common in lumbar spine. Present w radicular pain
Treatment: excise fragment. Unlikely to improve nonop

21
Q

What are the causes of atlantoaxial rotatory subluxation in kids? (5)

A
  1. Infection: most common. Grisel’s disease
  2. Trauma: 2nd most common
  3. Recent head or neck surgery
  4. Congenital
  5. Associated conditions: Down’s, RA, tumor
22
Q

What is Grisel’s disease?

A

Atlantoaxial rotatory subluxation due to infection. After upper respiratory or retropharyngeal infection, lymphatic edema may cause subluxation. Usually lasts 1wk

23
Q

What is the Fielding classification?

A

Atlantoaxial rotatory subluxation
Type 1: unilateral facet subluxation w intact transverse ligament. Most common
Pivots on odontoid
Type 2: unilateral facet subluxation w 3-5mm anterior displacement
Other facet acts as pivot. Transverse ligament may rupture
Type 3: Bilateral anterior facet displacement, >5mm
Decreases SAC. Risk decrease neuro deficit
Type 4: posterior displacement of C1
Due to hypoplastic dens or odontoid #. Decreases SAC. Risk serious neuro deficit

24
Q

How do the presentations of traumatic vs congenital torticollis differ?

A

Traumatic: primary cause is facet subluxation. Head tilted towards subluxation. Chin and secondary sternocleidomastoid (SCM) spasticity on opposite side. SCM prevents further subluxation.
Congenital: primary cause is SCM spasticity. Head tilted towards SCM, chin on opposite side.
Summary: in traumatic, chin and SCM on same side. In congenital, SCM and chin on opposite sides

25
Q

What is the gold standard imaging in diagnosing atlantoaxial rotatory subluxation?

A

Dynamic CT. Take CT w head straight, then max rotation to right and left.
Will see fixed C1 on C2 rotation that doesn’t change w head movement

26
Q

What are the treatment options and indications for atlantoaxial rotatory subluxation? (3)

A
  1. Soft collar for subluxation <1wk duration
  2. Halo traction for subluxation 1wk to 1mo duration, then hard collar
  3. Posterior C1-C2 fusion for subluxation >1mo or recurrence
27
Q

What conditions are associated w congenital muscular torticollis? (3)

A
  1. Other packaging disorders
  2. Plagiocephaly: asymmetric skull flattening
  3. Congenital atlanto-occipital abnormalities
28
Q

What is the presentation of congenital muscular torticollis like?

A
  • Palpable mass from contracted SCM at birth
  • Not painful
29
Q

What is the differential diagnosis of pediatric torticollis? (3)

A
  1. Congenital muscular
  2. Atlantoaxial rotatory subluxation
  3. Klippel-Feil
30
Q

What is the management of congenital muscular torticollis? (2) What are the indications?

A
  1. Passive stretching: <1yo and mild deformity (<30deg limited rotation)
  2. SCM Z-plasty lengthening: failed stretching or significant deformity
31
Q

What are the complications associated w untreated congenital muscular torticollis? (3)

A
  1. Permanent rotatory deformity
  2. Positional plagiocephaly
  3. Facial asymmetry
32
Q

Define spondylolysis

A

Pars defect. Most common at L5

33
Q

In pediatric spondylolisthesis, what are risk factors for progression? (4)

A
  1. High grade slip
  2. Slip angle >55deg
  3. Younger age
  4. Dysplasia
34
Q

How do you measure slip angle in isthmic spondylolisthesis?

A

Angle between
- Line perpendicular to line along posterior edge of S1
- Line along superior L5 endplate

35
Q

How do you measure lumbosacral kyphosis? What does this measurement mean?

A

Measures rotational deformity in isthmic spondylolisthesis
Angle between:
- L5 superior endplate
- Canal: posterior edge of S1 body
LSK < 80deg means poor quality of life. Very kyphotic.

36
Q

How do you determine the severity of isthmic spondylolisthesis? (3)

A
  1. Translational deformity: slip percentage (Myerding)
  2. Slip angle
  3. Rotational deformity: lumbosacral kyphosis angle
37
Q

How does pelvic incidence change during isthmic spondylolisthesis?

A

Increases with worsening spondylolisthesis severity.
PI = PT + SS
Higher PT + SS means more shear force at lumbosacral junction

38
Q

How does sacral slope and pelvic tilt affect sacropelvic balance?

A

Based on a normogram
Balanced sacrum and pelvis: high SS, low PT
Unbalanced: low SS, high PT. Vertical sacrum and retroverted pelvis

39
Q

How do you determine spinopelvic balance?

A

C7 plumb line. In neural, should run from center of C7 to posterosuperior corner of S1
If negative balance: line is >2cm posterior to S1. Lumbar hyperlordosis
If positive balance: line is >2cm anterior to S1. Due to hip flexion contracture or flat back syndrome

40
Q

What are the principles of operative management of isthmic spondylolisthesis?

A
  • In kids: always circumferential fusion (can do all from posterior approach, however)
  • Low grade slips: in situ fusion
  • High grade slips: in situ fusion vs partial reduction
  • Need partial reduction if unbalanced sacropelvis (low SS, high PT) or spinopelvis (anterior C7 plumb line)
  • Partial reduction is preferred over full reduction to minimize neuro complications. Reduce to low grade slip
41
Q

Regarding spondylolisthesis, what is the definition a low grade slip vs high grade?

A

Low grade: slip <50% anterior translation (Myerding 1 +2). Includes spondylolysis without listhesis
High grade: slip >50% (Myerding 3+). Includes spondyloptosis

42
Q

What is the Wiltse classification?

A

Etiology based classification of spondylolisthesis
Congenital
Isthmic
Degenerative
Traumatic
Pathologic

43
Q

What are the most common locations of isthmic vs degenerative spondylolisthesis?

A

Isthmic: L5/S1
Degenerative: L4/L5

44
Q

How do you measure sacral slope, pelvic tilt, and pelvic incidence? What is the relationship between them?

A

PI = PT + SS
Pelvic tilt: angle between
- Line perpendicular to the floor
- Line from hip center to center of S1 superior endplate
Sacral slope: angle between
- Line parallel to floor
- Line along S1 superior endplate
Pelvic incidence: angle between
- Center of S1 superior endplate to hip center
- Line perpendicular to S1 superior endplate

45
Q

True or False: pelvic incidence changes w position?

A

False. SS and PT change w position.
Pelvic incidence remains constant w position

46
Q

True or False: pelvic incidence changes from childhood to adulthood?

A

True. Increases during childhood to adulthood due to bipedal walking