The Eyes Flashcards
What is accommodation?
Eye convergence and pupillary constriction that enables us to look at and focus upon objects close to the eye (requires midbrain to be functional)
Describe the accommodation response.
- Light hits retina and info goes to the 1o visual cortex and then to the frontal eye field
- From CNIII n. CNIII goes to medial rectus causing vergence
- From EWN (PS), CNIII goes into ciliary ganglion where sphincter pupillae causes pupil constriction whilst ciliary body causes lens fattening
How and why does the ciliary body cause lens fattening?
Contraction of the ciliary body relaxes the suspensory ligaments enabling the lens to recoil thus making it fatter so the lens can better bend and refract light allowing you to focus on items close to face (elasticity of lens decreases with age decreasing this - why older people need reading glasses)
What is Argyll-Robertson pupil?
No pupillary light reflex BUT accommodation response is functioning as the PTN is knocked out bilaterally - commonly seen in tertiary neurological syphilis (hence the name ‘Prostitutes pupil’) and diabetic neuropathy
How could the Edinger-Westphal nucleus (EWN) become damaged? What symptoms would you see?
Vascular issues, tumours or brainstem problems could damage the EWN so there would be no direct or consensual reflex on damaged side and a dilated/unreactive pupil as there is no ciliary ganglion output
What symptoms would occur if there was a problem with CNIII?
No direct or consensual reflex on damaged side and pupil would be dilated/unreactive
Compression = loss of all CNIII functions causing full ptosis
Vascular lesion = sparing of pupillary functions with partial ptosis (vascular lesions affect motor fibres but not PS fibres)
What is a Marcus-Gunn pupil? How is it tested?
Medical sign observed during the swinging flashlight test where the patients pupils constrict less (so appear to dilate) when a bright light is swung from the unaffected to affected eye where affected eye senses light and produces pupillary sphincter constriction but to a lesser degree
What does Marcus-Gunn pupil indicate?
Optic nerve (CNII) lesion between retina and optic chiasm
Severe retinal disease
Contralateral optic tract (due to the different contributions of the intact nasal and temporal hemifields)
What are the 3 different muscles acting on the eyelid? What do they do and what is there innervation?
- Levator palpebrae superiosis (CNIII) - opens eye
- Orbicularis oculi (CNVII) - closes eye
- Superior tarsal (sympathetic) - opens eye
What would be the difference in symptoms if you lost the levator palpebrae superioris or the superior tarsal muscle?
CNIII lesion causing loss of levator palpebrae superioris = full ptosis
Loss of superior tarsal sympathetic supply = partial ptosis
How does the orbicularis oculi contribute to keeping the eye free of matter?
Forms a sphincter around the eye closing eyelids laterally to medially pushing tear fluid across the eye so grit and dust end up in the inner corners and can be wiped out
What is the golden rule regarding extraocular muscles?
ALL are innervated by CNIII (inc. sphincter pupillae, cillary body + levator palpebrae superioris) EXCEPT superior oblique (CNIV) and lateral rectus (CNVI) - their positions tell you what they do to the eye (think about what contraction/shortening would cause the muscle to do)
What are the extraocular muscles?
Superior rectus (SR) Inferior rectus (IR) Medial rectus (MR) Lateral rectus (LR) Superior oblique (SO) Inferior oblique (IO)
Where does the rectus muscles originate from?
Common tendinous ring at posterior of orbit
Where does the inferior oblique arise from?
Antero-medial floor of orbit (like a hammock sitting under the eye and cupping it)
If the eye muscles were to act on their own, what movements would they cause?
LR = abduction (lateral) MR = adduction (medial) SR = superior (elevation) + lateral IR = inferior (depression) + lateral IO = superior (elevation) + medial SO = inferior (depression) + medial