The Endocrine Response to Training Flashcards

1
Q

The anabolic hormones of the endocrine (3)

A

Testastrone, IGF1, cortisol

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2
Q

Hormone-Receptor Complex

A

Testosterone and stress can alter the number of androgen

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3
Q

Muscle Remodeling

A

Muscle remodelling involves the disruption of muscle fibres in response to mechanical loading, resulting in the inflammatory process (immune cells and catabolic hormones) and subsequent release of anabolic hormones

increases receptor and membrane permeability to hormones and nutrients

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4
Q

What is Acute vs. Chronic Adaptations

A

Acute effects refer to those that happen during and just a9er training, chronic ones are long-term adaptation

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5
Q

Anabolic hormones are affected by the following strength training variables

A

exercise modality (involved musculature), •exercise sequence, •intensity (load), •sets and repe@@ons (volume),•rest period.

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6
Q

Acute Effects

A

acute effects, high-volume strength training seems to produce the greatest change in hormone levels

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7
Q

Chronic Effects

A

note that in as little as five weeks, exercise stops having a hormonal response

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8
Q

Training status and hormone response

A

Training background and strength levels are most important, as these are indicative of Type II fibre content and androgen receptor content

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9
Q

Testosterone

A

Testosterone is the primary anabolic hormone and has both direct and indirect effects on protein synthesis

promotes the growth hormone responses from the pituitary gland.

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10
Q

How does testosterone affect the nervous system

A

It acts on the nervous system through interacting with neuron receptors and increasing the amount of neurotransmiKers, both of which increase force production

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11
Q

Concurrent training: The interference effect

A

Endurance exercise interferes with resistance exercise sessions via residual fatigue and substrate depletion

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12
Q

Concurrent training: Neural Development

A

Increases in maximal strength during the initial weeks of strength training can be attributed largely to the increased motor unit activation of the trained agonist muscles

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13
Q

DOES CONCURRENT TRAINING INTERFERE WITH NEURAL DEVELOPMENT

A

large gains in maximal force there was an increase in the maximum integrated electromyograms (EMGs) in the leg extensor muscles during a concurrent training programme lasting 21 weeks

Increase in EMG
Increase in number motor units
Increase rate of coding

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14
Q

Concurrent training:Where an interference effect has been demonstrated, it is purported to manifest as:

A

1) alteration in the neural recruitment patterns of skeletal muscle
2) limitation in force generation
3) increased neuromuscular fatigue from increased training demands of high volume endurance training

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15
Q

Concurrent training: Muscular disruption

A

Likely impact of testosterone and cortisol interference due to mixed endocrinal responses to training

Endurance training may decrease muscle fibre size in order to accommodate increases in capillary and mitochondrial density

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16
Q

Concurrent training: Muscular disruption

A

A lack of development in muscle CSA during concurrent training could be attributed to chronic muscle glycogen depletion (down regulating the signaling cascade required for protein accretion, as well as reducing training performance) and an increase in catabolic hormones

17
Q

Concurrent training: Molecular singling

A

Excessive bouts of endurance exercise are known to reduce rates of protein synthesis for several hours following the cessation of training

18
Q

Concurrent training: Molecular singling

A

endurance training the metabolic signalling pathways that are linked to substrate depletion and calcium release and uptake into the sarcoplasmic reticulum are activated

19
Q

Concurrent training: what is molecular singling and what is its function

A

The secondary messenger adenosine monophosphate-activated kinase (AMPK) is activated, as its role is to increase mitochondrial function to enhance aerobic capacity

20
Q

Concurrent training: Cardiovascular development

A

In elite endurance athletes, strength training can lead to enhanced long- term (> 30 min) and short-term (< 15 min) endurance capacity

Impact on peripheral adaptations (e.g. capillary and mitochondria density)

21
Q

Relationship between lactate and testerone

A
3x10 short rest periods
the highest release of 
- growth hormone 
- high levels of lactate
- hydrogen ions