Test three b Flashcards

1
Q

Two types of HF based on cause

A

Primary causes = underlying cardiac props (CAD, cardiomyopahy)

Precipitating causes = anemia, pulmonary disease, hypervolemia

Others: diabetes, tobacco, obesity, lipids, age
NOT GENETICS

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2
Q

Systolic HF

A

Pathophysiology: heart has thin walls, so LV can’t pump well

Etiology: impaired contractile function (MI); increased afterload (HTN), cardiomyopathy, or mechanical abnormalities

Diagnosis: decrease in LV EF

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3
Q

Diastolic HF

A

Pathophysiology: heart has thick walls, so ventricals can’t relax and fill –> decreases SV and CO –> venous engorgement in pulmonary and systemic systems

Etiology: LV hypertrophy from chronic htn, aortic stenosis, or hypertrophic cardiomyopathy

Diagnosis: presence of HF symptoms with normal EF

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4
Q

Mixed HF

A

Pathophysiology: Poor systolic function is further compromised by dilated LV walls that are unable to relax

Etiology: seen in disease states such as dilated cardiomyopathy

Symptoms: Low systemic BP, low CO, and poor renal perfusion

Compensation: Ventricular dilation, ventricular hypertrophy, activation of SNS and RAAS

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5
Q

Left sided HF

A

most common
blood backs up into LA and pulmonary veins
pulmonary congestion and edema

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6
Q

Right sided HF

A

usually caused by LSHF, but not always
blood backs up into RA and venous circulation
peripheral edema, hepatomegaly, JVD
**cor pulmonale can also cause RSHF

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7
Q

Manifestations of HF

A

Fatigue and anemia
edema
skin changes
behavioral changes
angina

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8
Q

LSHF signs and symptoms

A

Signs
-LV heaves
-pulsus alternans
-elevated HR
-PMI displaced inferiorly and posteriorly
-crackles
-S3 and S4
-pleural effusion
-mental status changes
-restlessness/confusion
-shallow respirations, dry cough, pink sputum

Symptoms
-dyspnea and orthopnea
-wekaness
-paradoxical nocturnal dyspnea
-nocturia

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9
Q

RSHF signs and symptoms

A

Signs
-RV heaves
-murmurs
-JVD
-edema
-weight gain
-elevated HR
-ascites
-anasarca

Symptoms
-fatigue
-anxiety/depression
-anorexia and GI bloating
-nausea
-dependent, bilateral edema
-RUQ pain
-hepatomegaly

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10
Q

Clinical manifestations of Acute Decompensated Heart Failure

A

CAD –> LV failure –> Pulmonary edema where alveoli fill with serosanguineous fluid

-anxiety, pallor, cyanosis, cold/clammy skin
-dyspnea, RR over 30, orthopnea
-coughing with blood tinged sputum
-lung crackles, wheezes, rhonchi
-tachycardia, htn, hpotn,

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11
Q

Etiology of mitral valve stenosis

A

rheumatic heart disease usually

also:
congenital mitral stenosis
rheumatoid arthritis
systemic lupus erythematosus

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12
Q

mitral valve regurgitation etiology

A

MI
rheumatic heart disease
mitral valve prolapse
ischemic papillary muscle dysfunction
infective endocarditis

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13
Q

aortic valve stenosis etiology

A

congenital aortic valve stenosis in kids

in older ppl, its bc of rheumatic fever or degeneration

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14
Q

aortic valve regurgitation etiology

A

Acute: IE, trauma, aortic dissection

chronic: htn, rheumatic heart disease, congenital bicuspid aortic valve, syphilis, chronic arthritic conditions

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15
Q

results of mitral and aortic stenoses

A

mitral
-higher pulmonary vascular pressure due to backup

aortic
-left ventricular hypertrophy and increased myocardial oxygen consumption from increased myocardial mass

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16
Q

results of mitral and aortic regurgitation

A

mitral (chronic)
-left atrial enlargement
-LV dilation, hypertrophy, and decreased CO

aortic
-volume overload
-declining myocardial contractility and increased volume in LA/lungs
-pulmonary htn
-RV faliure

17
Q

Mitral valve stenosis clinical manifestations

A

dyspnea on exertion
hemoptysis
fatigue
afib
palpitations
loud S1
low diastolic murmur

18
Q

mitral valve regurgitation manifestation

A

Acute
-new systolic murmur with rapid dvlpmt of pulmonary edema and cardiogenic shock

Chronic
-weakness
-fatigue
-exertional dyspnea
-palpitations
-S3 gallop
-murmur throughout systole

19
Q

Aortic valve stenosis manifestations

A

angina
syncope
dyspnea on exertion
normal or soft S1
diminished or absent S2
systolic murmur
S4

20
Q

Aortic valve regurgitation manifestation

A

Acute:
-abrupt onset of dyspnea, chest pain
-LV failure and cardio shock

Chronic
-fatigue
-exertional dyspnea
-orthopnea
-parxysmal nocturnal dyspnea
-water hammer pulse
-heaving precordial impulse
-diminished or absent S1, S3, or S4
-Austin Flint murmur

21
Q

Drugs for atrial dysrhythmias

A

ccb
bb
digoxin
antidysrhythmic drugs
electrical cardioversion

22
Q

Non drug treatment for valve issues

A

percutaneous transluminal balloon valvuloplasty
-through femoral artery to stenotic valve
-inflate and separate leaflets
-good for old ppl who can’t handle surgery

23
Q

surgeries for valve issues

A

mitral commissurotomy (stenosis)
open surgical valvuloplasty (regurgitation)
annuloplasty

24
Q

what should INR be?

A

2.5 to 3.5

25
Q

PAD

A

progressive narrowing of the arteries from the thickening of arterial walls

26
Q

PAD risk factors

A

tobacco!!!!!
CKD
DM
HTN
high cholesterol
female
black
high CRP
fam history
hypertriglyceridemia
age
hcy
boesity
sedentary
stress

27
Q

leading cause of PAD

A

atherosclerosis
-in femoral popliteal area (in non diabetics)
-below knees in DM

28
Q

PAD clinical manifestations

A

intermittent claudication (from muscular anaerobic respiration)

Atypical leg symptoms (burning, tightness, weakness)

Neuropathy (tingling, numbness, shooting, burning)
-loss of bloodflow to neurons reduces pain sensation

Skin gets thin, shiny, taught, and hairless - pallor on elevation and vice versa

Rest pain often at night due to limb elevation

Critical limb ischemia = rest pain for more than 2 weeks, ulcers, gangrene –> more likely if HF, DM, or history of stroke

29
Q

Complication of peripheral artery disease

A

atrophy of skin and muscles
delayed healing, wound infection, and necrosis
-ulcers on bony prominences –> develop gangrene
-uncontrolled pain, sepsis, or osteomyelitis indicate amputation need

30
Q

Diagnostic studies for PAD

A

Doppler Ultrasound

Segmental blood pressure –> PAD if drops more than 30

Ankle brachial index -> divide ankle SBP by higher brachial SBP –> PAD is under 0.9 –>old ppl and those w/ DM have false high ABI

Angiography: contrast and fluroscopy

Angiography and magnetic resonance angiography determine location and extent of PAD

31
Q

Risk factor modification for PAD

A

Stop tobacco use
Manage DM (keep HbA1C below 7%)
Manage Lipids (statins)
Manage HTN (below 140/90 if normal ppl; below 130/80 if DM or kidney issues) –> ACE inhibitors

32
Q

Drug therapy for PAD

A

ANTIPLATLET AGENTS
-aspirin or clopidogrel

NO ANTICOAGULANTS

Cilostazol (don’t use if HF) and Pentoxifylline for claudication

33
Q

Priority when giving anticoagulation meds

A

draw baseline labs first
do bleeding time test –> will increase once meds taken –> platelets won’t decrease

34
Q

Exercise therapy for PAD

A

super important- esp for women
-30-45 mins at least 3x a week for 3 months

35
Q

Interventional radiology catheter based procedures for PAD

A

PTA –> balloon pumps up narrowed vessel
Stents –> placed after balloon pump
Atherectomy –> can use disk or laser
Cryoplasty –> PTA with coldness to limit restonosis

36
Q

Surgeries for PAD

A

Peripheral artery bypass surgery
Femoral popliteal bypass
endarterectomy and patch graft angioplasty
amputation

37
Q

Post op for PAD surgeries

A

check extremity every 15 mins and then hourly for color, temp, cap refill, peripheral pulses, sensation, and movemnt

bleeding, hematoma, thrombosis, emolization, compartment syndrome

ABI not recommended bc could cause thrombosis

38
Q

Nursing interventions for PAD

A

Pain management –> may be aggressive
Positioning –> get them walking; not too much time with dependent leg
Supportive care –> graduated compression stockings
Infection prevention