Test 3 - Respiratory Dysfunction Flashcards

1
Q

General Aspects of Respiratory Infections

Described according to the anatomical area involved:

A
Upper respiratory tract
Nose, pharynx, larynx, 
upper trachea
Lower respiratory tract
Lower trachea, bronchi and
 bronchioles, alveoli
Croup syndromes
Infections of the epiglottis or larynx
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2
Q

In infants younger than __ months, maternal antibodies offer some protection.

A

In infants younger than 3 months, maternal antibodies offer some protection.
Espeically if the mom is breast feeding

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3
Q

In infants age __ to ___ months, the infection rate increases.

A

In infants age 3 to 6 months, the infection rate increases.

They don’t have immune system, exposure to more things, and losing maternal antibodies, mother may go back to work - baby go to daycare
In toddlers and preschoolers, there is a high rate of viral infections
Preschool, touching things,eating things, picking their nose, poor hygene and spread germs

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4
Q

In children older than __ years, there is an increase in GABHS and Mycoplasma pneumoniae infections.

A

In children older than 5 years, there is an increase in GABHS and Mycoplasma pneumoniae infections.
In school, it gets passed around
Increased immunity develops with age.

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5
Q

Size to children’s respiratory tract

A

Diameter of airways is smaller
Distance between structures is shorter, allowing organisms to rapidly move between
Short and open eustachian tubes

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6
Q

How do children get respiratory diseases

A
Immune system deficiencies
Allergies, asthma
Cardiac anomalies
Cystic fibrosis
Exposure to infections in daycare
Exposure to second-hand smoke
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7
Q

Seasonal Variation - respirtaory infection are most common during which seasons?

A

Seasonal Variation

Most common during winter and spring

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8
Q

Mycoplasma infections more common in which seasons?

A

Mycoplasma infections more common in fall and winter

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9
Q

Asthmatic bronchitis more frequent in which in of weather?

A

Asthmatic bronchitis more frequent in cold weather

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10
Q

RSV season typically which season?

A

RSV season typically winter and early spring

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11
Q

Generalized signs and symptoms and local manifestations differ in young children are…?

A

Apnea
Fever - you see it in younger children due to initial immune response (in neonate you see drop in temperature)
Anorexia, vomiting, diarrhea, abdominal pain
Cough, sore throat, nasal blockage or discharge
Respiratory sounds

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12
Q

Respiratory assessment should include:

A

RR, depth, rhythm,and effort
HR, O2 sat, hydration status,
Body temperature
Activity level and comfort

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13
Q

Nursing Interventions for Respiratory Infections

A

Ease the respiratory effort
Positioning (lay them up), suction, oxygenation (decrease effort), having them cough, trying to calm the toddler down, turn the lights down and put on a movie (distraction), managing fever

Manage fever
Tylenol, uncover them, put fan in the room, wet/cool towel on forehead and axella
Promote rest and comfort
Decrease metabolic demand - turn off the light, give transitional object, help them feel secure, distractions (movies, music)

Control infection
Make sure the visitor wash hands, make sure the visitors aren’t sick, keep kids out of the room, clamp down on visitors during flu seasons

Promote hydration and nutrition
Offer snacks (pt often don’t have appetite), if hard to breath usually don’t offer water but rather ice chips so they don’t aspirate.
Hydration over nutrition - push them to drink not to eat

Provide family support and teaching

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14
Q

Home management for Upper Respiratory Tract Infections (URIs)

A

Clear secretions -parents make sure knows how to use ball syringes (suction the kid’s nose)
Humidification - Cool mist only (warm causes bacteria and mold growth, and dangerous bc mist can burn them)
Small, frequent feedings
Fever management
Avoid OTC “cold” medicines

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15
Q

Pharyngitis - what is causes and risks

what is the clinical manifestations?

A

Causes and risks
Often viral
Gr A β hemolytic strep(strep throat)
Risk for Rheumatic fever, Acute Glomerulonephritis

Clinical manifestations
Abrupt onset, fever, HA, sore throat
Tonsils, pharynx inflamed, covered with exudate

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16
Q

What is diagnosis of pharyngitis

A

Diagnostics

Although 80-90% are viral, rapid strep test should be done

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17
Q

Therapeutic management for pharyngitis is…?

A

Oral PCN if strong suspicion of bacterial infection

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18
Q

Nursing considerations for pharyngitis is…?

A

Nursing considerations
Warm saline gargles, cool compresses, tylenol/motrin, encourage PO fluids, rest
Teach about administration of meds
Strep is contagious for 24 hours after antibiotics are started

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19
Q

Etiology and clinical manifestation for Tonsillitis?

A

Tonsillitis
Etiology: often occurs with pharyngitis, common in young children, viral or bacterial

Clinical Manifestations:
Enlarged tonsils (“kissing”)
Difficulty swallowing/breathing
Mouth breathing
Sleep apnea
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20
Q

Therapeutic management for Tonsillitis?

A

Therapeutic management - antibiotics
Tonsillectomy/Adenoidectomy
Nursing care: pain relief, minimize bleeding, close observation of breathing, cool clear fluids, observe for bleeding, avoid emesis and clearing of throat

avoid chips or things that can scratch the throat, or vomiting, causing the scab to come off.

Observe for frequent swallowing - that may be due to bleeding

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21
Q

Influenza Clinical manifestations

A

Clinical manifestations
Mild mod or severe, dry throat, cough, general myalgia and malaise, fever, and chills (every fiber of your body hurts)
Lasts 4-5 days minimum

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22
Q

Therapeutic management for influenza

A

Symptomatic treatment - fluids, tylenol

Antiviral medications lessen severity

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23
Q

Prevention for influenza

A

Yearly flu vaccines in children over 6 months of age
Family are the “vaccines for the young infant” - infant can’t get the flu vaccine, so people around the baby need to be vaccinated
Health care worker vaccines

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24
Q

Croup Syndromes

A

Croup Syndromes - fall and winter

Croup
croup won’t kill you, more towards infants
Croup gets worst for 3 nights and then gets better
Make sure to get a good history for pt w stridor - inspiratory sound (hear it on inspiration, sounds like a gasp)

Causes: foreign body - potato chips, lego, etc

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25
Q

Epiglottitis

A

Epiglottitis
Sicker, drooling, and can kill you, you want to leave them alone - never stick anything in mouth or cause pain, dont stick a tongue blade, it would reflexively swell
Get steroids

Characterized by hoarseness, barking cough (sounds like a seal), inspiratory stridor, and varying degrees of respiratory distress
Croup syndromes affect the larynx, trachea, and bronchi
Epiglottitis, laryngitis, laryngotracheobronchitis (LTB), tracheitis

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26
Q

Acute Epiglottitis-

A

Acute Epiglottitis- A Medical Emergency
Clinical manifestations
Rapid progression
Sore throat, pain, tripod positioning, retractions
Inspiratory stridor, mild hypoxia, distress
Watch for: absence of spontaneous cough, drooling, agitation/anxiety
Therapeutic management
Potential for respiratory obstruction
Nursing considerations – do not attempt to inspect the throat! Keep child as calm as possible.
Allow position of comfort
Prevention requires Haemophilus influenzae type b (Hib) vaccine, incidence greatly decreased since vaccine

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27
Q

Acute Laryngotracheobronchitis (LTB)

A

Acute Laryngotracheobronchitis (LTB)
Most common croup syndrome
Generally affects children younger than 5 years of age
Organisms responsible
RSV, parainfluenza virus, Mycoplasma pneumoniae, influenza A and B viruses
Manifestations of LTB
Inspiratory stridor
Suprasternal retractions
Barking or seal-like cough
Increasing respiratory distress and hypoxia
Can progress to respiratory acidosis, respiratory failure, and death
Therapeutic Management of LTB
Airway management
Maintain hydration (oral or IV)
High humidity with cool mist
Nebulizer treatments
Epinephrine (racemic) – rapid onset, peak effect at 2 hours
Steroids

28
Q

Infections of the Lower Airways

A

Considered the“reactive”portion of the lower respiratory tract
Includes bronchi and bronchioles
Cartilaginous support is not fully developed until adolescence
Constriction of airways

29
Q

Bronchitis

A
Also known as tracheobronchitis
Definitions – inflammation of the large airways
Causative agents – primarily viral
Clinical manifestations
Dry hacking cough
Worsens at night
Mild, self-limiting disease
30
Q

Bronchiolitis and RSV

A

Definitions: common acute viral illness, maximum effect and the bronchiolar level
Respiratory syncytial virus (RSV) :
occurs cyclically in winter and spring
Most common cause of hospitalization in <1 year of age
Severe RSV at <1 y is a significant risk of developing asthma

31
Q

Pathophysiology of Bronchiolitis and RSV

A

Pathophysiology
Affects the epithelial cells which swell and protrude into the lumen
Lumina fill with mucus
Leads to hyperinflation (they can breath out but can’t get the air out), hypoxia (bronchial with mucous not exchanging), atelectasis

32
Q

Diagnostics of Bronchiolitis and RSV

A

Diagnostics
Nasopharyngeal swab - you don’t want the mucous, you want the nasopharyngeal cell
CXR: hyperinflation

33
Q

Therapeutic management of Bronchiolitis and RSV

A

Therapeutic management
Most managed at home, Humidified O2, Periodic suctionin- especially before feedings, Fluids PO or IV, Bronchodilators?
Prevention of RSV: prophylaxis (synagis), handwashing
Nursing considerations: isolation, close monitoring, teach mom to provide care at home, suctioning

34
Q

Pneumonia

A

Inflammation of the pulmonary parenchyma
Causative organism varies greatly by age category
The most useful classification is etiologic agent: Viral, Bacterial, Mycoplasma, Aspiration of foreign substances
Clinical Manifestations
Vary depending on Age, Etiology, Systemic reaction to infection, Extent of the lesions, Degree of obstruction

35
Q

Pertussis (Whooping Cough)

A

Pertussis (Whooping Cough)
Caused by Bordetella pertussis
Highly contagious
In the United States, it occurs most often in children who have not been immunized
Highest incidence is in spring and summer
Hallmark: severe, paroxysmal coughing, followed by characteristic “whoop”, Young infants may present with apnea -
Heart rate will drop, stats will drop
Post-tussive emesis common, often vomit afterwards
Three phases
Catarrhal - Increase secretion, running nose and eyes, look like any type of cold
Paroxysmal - last a long time
Convalescent -

36
Q

Asthma

A

Asthma
Chronic inflammatory disorder of the airways
Recurring episodic symptoms
Wheezing, Breathlessness, Chest tightness, Cough (especially at night)
Limited air flow or obstruction that reverses spontaneously or with treatment
Bronchial hyper responsiveness
Narrowed airways causes forced expiration.
Air trapping
Reduced alveolar air exchange
Inflammation now recognized as playing a key role in asthma

37
Q

Asthma Severity Classification in Children 5 Years and Older

A

Step I: Intermittent asthma
Sx <2 days/week, no limitations on ADL
Step II: Mild, persistent asthma
Sx > 2x/wk but not daily, night time 1-2x/mo, some limitations to ADLs
Step III: Moderate, persistent asthma
Daily symptoms, freq night sx, minor limitations to ADLs
Step IV: Severe, persistent asthma
Continued daytime sx, freq night sx, use of relievers several times per day, extreme limitations on ADL

38
Q

Asthma Diagnostic evaluation

A
Diagnostic evaluation
Dyspnea, wheeze, cough
PFTs, PEFR, allergy testing
Therapeutic management
Pharmacologic therapy
Nonpharmacologic therapy
Allergen/trigger control
39
Q

Asthma Nursing considerations

A

Nursing considerations
Importance of patient and family teaching
Need to control triggers in hospital
Smoking cessation

40
Q

Asthma Drug Therapy for Asthma

A

Drug Therapy for Asthma

Long-term control medications
Preventive (Controller) DAILY use, even when feeling well

Quick relief medications
Rescue (Reliever) - albuterol, xopenex

Metered-dose inhaler (MDI)
Always use with spacer/mask

Corticosteroids - First line treatment
Significant improvement of all asthma parameters

Albuterol, terbutaline
Β Adrenergic agonist, rapid onset treatment for acute bronchospasm

Dilates bronchioles 
prevent EIB (exercise-induced bronchospasm) - exercise induced 

Long-term bronchodilators
(salmeterol [Serevent])
Not used in children <12 years

Theophylline
(monitor serum levels) rarely used, causings significant agitation, and tachycardia, if serum too high pt vomits
only in ED when not responding to maximal therapy

Leukotriene modifiers (singulair)
Mediates inflammation, given in combination with β agonist, steroids for long term control
41
Q

Asthma Intervention

A

Asthma Intervention
Exercise/play
Evaluate for EIB (exercise induced bronchospasm)
Chest physical therapy (CPT)
Breathing exercises - encourage cough and deep breathing

42
Q

Status Asthmaticus

A

Status Asthmaticus
Respiratory distress continues despite vigorous therapeutic measures
Sweating, remains sitting upright, refusal to lie down, agitation, absence of breath sounds, only speaking a few words at a time, level of conciousness is deminishing (meaning not enough air is getting in)= immediate intervention
Emergency treatment is epinephrine 0.01 mL/kg subcutaneously (maximum dose, 0.3 mL)
Magnesium sulfate - infuse over 20 minutes usually
Potent muscle relaxant, decreases inflammation
Monitor for side effects, can cause severe hypotension
Vital signs q10minutes for an hour
Concurrent infection in some cases - pt may also have pneumonia, thus start pt on antivirals

43
Q

Acute Asthma Care

A

Acute Asthma Care
Calm nursing presence - pt and family might get anxious seeing you etc.
Auscultation of breath sounds, air movement
Monitor with pulse oximetry
Titrate O2 - Choice of O2 delivery devices
Allow older children to sit up if they are more comfortable in that position
Allow parents to remain with children
Oral vs IV fluids

44
Q

Goals of Asthma Management

A

Goals of Asthma Management
Avoid exacerbation, Avoid allergens
Relieve asthmatic episodes promptly, Relieve bronchospasm
Monitor function with a peak flow meter
Self-management of inhalers, devices, and activity regulation
Support child, adolescent, and family
Identifying Asthma Triggers
Viral infection, Exercise, Perfumes, Smoke
One of the most important components of decreasing episodes of exacerbations
Differs in each person - was there a weather change, different setting, food, etc
Common triggers (not an exhaustive list)
Viral infections (most common trigger for infants) “he never gets these symptoms unless he has cold, Perfumes, strong odors
Smoke: tobacco, marijuana, wood, BBQ, fires, Exercise, Changes in temperature, Animal dander - cats.dog, Dust and mold, Pollen, Food allergies

45
Q

Cystic Fibrosis (CF)

A

Cystic Fibrosis (CF)
Exocrine gland dysfunction that produces multisystem involvement
Most common lethal genetic illness among Caucasian children
Autosomal recessive trait - need a defective gene from both parents
Child inherits a defective gene from both parents, with an overall incidence of 1:4
Approximately 3% of the U.S. Caucasian population are symptom-free carriers

46
Q

Pathophysiology of CF

A

Pathophysiology of CF
Predominantly affects the respiratory tract and the pancreas - produce cement like mucous
Increased viscosity of mucous gland secretions
Responsible for many of the clinical manifestations
Elevation of sweat electrolytes
Increase in several organic and enzymatic constituents of saliva
Increase in sodium and chloride in saliva and sweat
Exocrine Gland Dysfunction in CF

47
Q

Clinical Manifestations of CF

A

Clinical Manifestations of CF
Pancreatic enzyme deficiency - doesnt let them digest fat
Progressive chronic obstructive pulmonary disease (COPD) associated with infection
Sweat gland dysfunction
Failure to thrive - due to fighting all infection, they’re not absorbing food
Increased weight loss despite increased appetite
Gradual respiratory deterioration

48
Q

Presentation of CF

A

Presentation of CF
Wheezing respiration; dry, nonproductive cough, Generalized obstructive emphysema
Patchy atelectasis, Cyanosis
Clubbing of fingers and toes
Repeated bouts of bronchitis and pneumonia
Seem like “sickly baby” - aren’t gaining weight, are sick often
Meconium ileus - Earliest recognizable manifestation of CF
Prolapse of the rectum - due to the stool
Distal intestinal obstruction syndrome
Excretion of undigested (fat) food in stool - Stool is bulky, frothy, and foul smelling
Wasting of tissues (not absorbing fat, thus not absorning vitamin A,D,E,K (fat soluble))
Delayed puberty in females
Sterility in males
Parents report that children taste “salty” - when kissing their child
Dehydration, Hyponatremic or hypochloremic alkalosis, Hypoalbuminemia

49
Q

Diagnostic Evaluation of CF

A
Diagnostic Evaluation of CF
Quantitative sweat chloride test - make them sweat and test the sweat
Chest x-ray
Pulmonary function tests (PFTs)
Stool fat and/or enzyme analysis
Barium enema
50
Q

Treatment Goals for CF

A

Treatment Goals for CF
Prevent or minimize pulmonary complications, Adequate nutrition for growth, Assist the child in adapting to a chronic illness

51
Q

Respiratory Manifestations of CF

A

Respiratory Manifestations of CF
Present in almost all CF patients, but the onset and extent vary
Viscous secretions are difficult to expectorate and obstruct bronchi and bronchioles
Cause atelectasis and hyperinflation
Stagnant mucus leads to destruction of lung tissue
Stagnant mucus provides a favorable environment for bacteria growth

52
Q

What is respiratory progression

A

Respiratory Progression
Gradual progression follows chronic infection
Bronchial epithelium is destroyed
Infection spreads to peribronchial tissues, weakening the bronchial walls
Peribronchial fibrosis
Decreased exchange of O2 and CO2 - end up on oxygen all the time, kids become slim

53
Q

What is further respiratory progression

A
Further Respiratory Progression
Chronic hypoxemia causes contraction and hypertrophy of muscle fibers in pulmonary arteries and arterioles
Pulmonary hypertension
Cor pulmonale
Pneumothorax
Hemoptysis
54
Q

Respiratory Management of CF

A

Respiratory Management of CF
CPT (chest physiotherapy) and postural drainage - Therapy vests - vibrate to help move the mucous
Bronchodilator medication, Forced expiration
Aggressive treatment of pulmonary infections
Home IV antibiotic therapy, Aerosolized antibiotics
Steroids or non-steroidal anti-inflammatory drugs (NSAIDs)
Transplantation

55
Q

Gastrointestinal (GI) Tract from CF

A

Gastrointestinal (GI) Tract
Thick secretions block ducts, leading to cystic dilation, degeneration, and diffuse fibrosis
Prevents pancreatic enzymes from reaching the duodenum - have to take handful and pancreatic enzyme
Impaired digestion and absorption of fat, or steatorrhea, occurs
Impaired digestion and absorption of protein, or azotorrhea, develops
Earliest manifestation may be meconium ileus
Endocrine function of the pancreas is initially unchanged
Eventually, pancreatic fibrosis - could kill off the pancreas occurs; may result in diabetes mellitus
Focal biliary obstruction results in multilobular biliary cirrhosis - may cause heart, liver transplant
Impaired salivation

56
Q

GI Management in CF

A

GI Management in CF
Replacement of pancreatic enzymes
High-protein, high-calorie diet, as much as 150% of the recommended daily allowance (RDA)
Treat constipation
Reduction of rectal prolapse
Salt supplementation
Treat gastroesophageal reflux- Place the patient in the upright position after meals

57
Q

Endocrine Management of CF

A

Endocrine Management of CF

Monitor blood glucose levels, Diet, Exercise, Regular eye examinations

58
Q

Prognosis of CF

A

Prognosis of CF
Estimated life expectancy for a child born with CF in 2008 was 37.4 years
CF continues to be a progressive, incurable disease
Organ transplantation has increased the survival rate
Heart–lung and bilateral lung transplantation
Liver and pancreas transplantation
For lung transplants, the survival rate is 75% at 1 year and 55% at 3 years following transplantation.
Maximize health potential
Nutrition, Prevention and early aggressive treatment of infection, Pulmonary hygiene

59
Q

Family Support for the Child with CF

A

Family Support for the Child with CF
Coping with the emotional needs of the child and family
Developmental care
Child requires treatments multiple times each day
Frequent hospitalizations
Implications of genetic transmission of disease

60
Q

What is the difference between long term control medication, quick relief medications and metered dose inhaler?

A

Long-term control medications
Preventive (Controller) DAILY use, even when feeling well

Quick relief medications
Rescue (Reliever) - albuterol, xopenex

Metered-dose inhaler (MDI)
Always use with spacer/mask

61
Q

What is corticosteriods?

A

Corticosteroids - First line treatment

Significant improvement of all asthma parameters

62
Q

What is albuterol/terbutaline?

A

Albuterol, terbutaline
Β Adrenergic agonist, rapid onset treatment for acute bronchospasm

Dilates bronchioles 
prevent EIB  (exercise-induced bronchospasm) - exercise induced
63
Q

What is sameterol/servent?

A

Long-term bronchodilators
(salmeterol [Serevent])
Not used in children <12 years

64
Q

What is theophyline?

A

Theophylline
***(monitor serum levels) rarely used, causings significant agitation, and tachycardia, if serum too high pt vomits
only in ED when not responding to maximal therapy

65
Q

What is Leukotriene modifiers (singulair)?

A
Leukotriene modifiers (singulair)
Mediates inflammation, given in combination with β agonist, steroids for long term control