Test 3 Flashcards

1
Q

describe insecticide resistance

A

a heritable change in the sensitivity of an insect pop to insecticides reflected in the failure of that insecticide to achieve the expected level or control

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2
Q

insecticide resistance is _______ driven by _______

A

population evolution driven by natural selection

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3
Q

can resistance evolve to any management tactic that causes high mortality and/or reduced natality

A

yes

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4
Q

describe cross-resistance

A

development of resistance to one insecticide confers resistance to another insecticide

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5
Q

what does the rate of resistance depends on

A

-frequency of application
-level of insecticide-induced mortality
-genetics (monogenic vs. polygenic)
-reproductive and development rate

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6
Q

does increase pop isolation lead to higher or lower insecticide resistance

A

increased rate of insecticide resistance

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7
Q

does insecticide resistance evolve faster in monogenic or polygenic genes

A

polygenic will take longer to evolve because resistance involves multiple genes

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8
Q

describe metabolic resistance

A

resistant insects can destroy or excrete the toxic compound faster than susceptible ones

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9
Q

describe target-site resistance

A

a change in structure of the target protein that decreases herbicide binding to its usual site of action

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10
Q

describe penetration resistance

A

the insect will absorb insecticide slower and therefore will receive a smaller dose

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11
Q

describe behavioural resistance

A

insect will stop feeding or leave an area when it comes across an insecticide
-behavior needs to have a genetic basis

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12
Q

Name 4 resistance mechanisms

A
  1. metabolic resistance
  2. target-site resistance
  3. penetration resistance
  4. behavioral resistance
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13
Q

why is the colorade potato beetle so bad?

A

its resistant to at least 56 insecticides and has every type of resistance mechanism

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14
Q

why is the Colorado potato beetle so insecticide resistance

A

it feeds on solanaceous plants which have compounds that are like insecticides so its adapted to deal with insecticides

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15
Q

name 6 ways we can manage resistance

A
  1. practice IPM
  2. follow economic thresholds
  3. target most susceptible life stage
  4. apply according to the label
  5. rotate different MOAs
  6. preserve susceptible genotypes (field refuges)
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16
Q

describe toxicology

A

study of adverse effect associated with exposures to substances

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17
Q

describe toxicity

A

the inherent poisonous potency of a substance (relative to other stuff)

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18
Q

what is a fundamental concept of toxicology (paracelsus)

A

the dose makes the poison

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19
Q

what’s the difference between acute, subchronic, and chronic toxicity

A

acute: very short term exposure (24-48h)
subchronic: short term exposure (3-6m)
chronic: long-term exposure (2-4 yrs or more)

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20
Q

what is an LD50

A

does that will kill 50% of the pop you are testing on

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21
Q

does a lower LD50 mean the substance is more or less toxic

A

lower number means it takes less of it to kill you or cause an effect

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22
Q

what is ED50

A

dose that would produce the effect of interest in 50%

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23
Q

what is EC50

A

concentration that would produce the effect of interest in 50%

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24
Q

what is LC50

A

concentration that would kill 50%

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25
Q

what is LT50

A

time required to kill 50%

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26
Q

name 6 things that influence toxicity

A
  1. exposure route (touch, inhale)
  2. metabolism
  3. excretion
  4. species
  5. age
  6. nutrition/ state of health/ genetics
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27
Q

what makes up the exposure of a substance

A

how you come in contact and the duration

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28
Q

describe concentration vs. dose

A

conc: amount of substance per unit of solution
dose: amount of substance received by the organism

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29
Q

describe the meaning of dose

A

amount that gets in via exposure
= conc. of substance x amount of substance received

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30
Q

describe effect/response

A

physiological or behavioral change resulting from exposure to a substance (change in structure, function, or mortality)

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31
Q

name 4 sub-lethal effects

A

-growth and development (slowed growth)
-sexual characteristics (fertility)
-physiology (respiration)
-behavior (feeding, learning)

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32
Q

describe hormesis

A

where a low dose of a potentially harmful stressor can actually stimulate beneficial adaptive responses in an organism

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33
Q

what are some sub-lethal effect on pops.

A

-decreased size
-demographic changes
-pop genetic changes

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34
Q

what determines if the exposure will cause an effect

A

the dose

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35
Q

describe a hazard

A

anything that has the potential to cause harm
-related to the toxicity of a substance and exposure

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36
Q

describe risk

A

potential/probability that a hazard will cause harm or have adverse effects

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37
Q

describe risk assesment

A

process of quantifying risk based on info about a hazard and the amount of exposure to that hazard

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38
Q

how to you calculate risk

A

risk = hazard X probability of exposure

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39
Q

describe pest resurgence

A

insecticide kills pest and natural enemies but the pest rebounds to a higher density than before insecticide application

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40
Q

describe a secondary pest outbreak after applying insecticides

A

insecticide used to kill pest A also kills natural enemies of pest B and then the density of pest B increases
-you reduce the target pest but now have a new problem

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41
Q

when designing a non-target effects study what is important to consider

A

the experimental design should reflect the biology of the test insect and the properties and intended use of the insecticide
-exposure route
-exposure duration
- test concs
-life stage
-residual life

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42
Q

what are the types of studies for a non-target effects study

A

-lab
-semi-field
-field/greenhouse

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43
Q

is insecticide exposure in lab studies lower or higher than what would be in real life

A

higher

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44
Q

what does it mean if the insecticide is toxic or not toxic in lab

A

not toxic in lab = prob not toxic in field
toxic in lab = may or may not be toxic in the field

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45
Q

why can semi-field studies be unrealistic

A

insects are confined to a treated plot

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46
Q

what field study is non-target effects most realistic

A

field studies

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47
Q

what are pros and cons of doing a study in a lab

A

pros: low expense. low difficulty, high control
cons: over exposure

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48
Q

what are pros and cons of doing a study in a field

A

pros: realistic
cons: high expense, high difficulty and low control

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49
Q

what is the goal of biological and chemical control

A

to reduce or minimize toxic effects and/or insecticide exposure to natural enemies

50
Q

what are 4 ways to minimize toxic effects and/or insecticide exposure to natural enemies

A
  1. choose less toxic/ more selective products
  2. target pest hot spots
  3. time insecticide apps differently
  4. provide untreated refugia (reservoir to keep natural enemies)
51
Q

what is an ecologically significant trait or Spotted wing drosophila

A

they have a serrated ovipositor that penetrates the surface barrier of soft-skinned fruit to lay eggs

52
Q

why do the Spotted wing drosophila have rapid dispersal

A

-cryptic nature
-global fresh fruit trade
-lack or regulation
-rapid development
-multiple hosts facilitate establishment

53
Q

what fruit is at most risk from Spotted wing drosophila

A

strawberries (but has many hosts)

54
Q

whats the threshold for fruit infested by Spotted wing drosophila

A

0 tolerance threshold for invested fruit
-ripening fruit + SWD activity = spray

55
Q

why is the Spotted wing drosophila such a bad pest

A

-highly fecund
-large host range
-multiple generation
-able to disperse
-no native natural enemies

56
Q

how can you monitor the larvae activity of SWD

A

-examine fruit weekly for eggs and larvae
-place fruits in a salt solution
-place fruit in a bag and leave in a sunny location

57
Q

does susceptibility to SWD decrease or increase as fruit ripens

58
Q

how can you predict if it will be a bad year for SWD

A

if there is a warm winter and warm spring there is a high prob or early emergence

59
Q

what temps do SWD prefer

A

moderate temps and dont like it when its dry

60
Q

where do female SWD prefer to lay their eggs in the canopy

A

where there are lower temps and higher humidity so in shaded fruits and more dense areas of the canopy

61
Q

name 4 factors that affect SWD environment suitability

A
  1. irrigation type (want drip system)
  2. presence of weed mat (good)
  3. pruning intensity (more=better)
  4. surrounding veg (don’t want?)
62
Q

what are ways to manipulation SWD habitat in field

A

-pruning to open the canopy
-sanitation (remove infested fruit)
-ground cover management (want bare ground)

63
Q

what life cycle f SWD is most important to target

A

adults before the eggs are laid because all other life stages are protected

64
Q

what are 3 postharvest practices for reducing SWD

A
  1. refrigeration
  2. chemical treatments
  3. irradiation
65
Q

how are growers coping with SWD

A

-better traps
-exclusion netting
-biocontrol
-sterile insect release
-collaboration
-modifying behavior (gum)

66
Q

what are semiochemicals

A

chemicals that mediate interactions between organisms
-pheromones (intraspecific communication) –allelochemicals (interspecific communication)

67
Q

describe a sex pheromone

A

produced by one sex and attracts the opposite sex (usually produced by females)

68
Q

describe an aggregation pheromone

A

attracts both sexes and usually is produced by males

69
Q

what do kairomones do

A

favour the recipient (attract insect to a plant they want to eat)

70
Q

what do allomones do (produced by plant to repel herbivore

A

favour the emitter

71
Q

what do synomones do

A

favour the recipient and emitter

72
Q

how are semiochemicals used in IPM

A

-trapping (monitoring, mass trapping)
-mating disruption
-push/pull

73
Q

how is mass trapping used on SWD

A

they are attracted to VOCs released by gum-like substance which disrupts their behaviour

74
Q

describe how mating disruption works

A

synthetic sex pheromone is released into crop to prevent mating

75
Q

describe push-pull

A

pest is simultaneously pulled from the crop and pushed towards a trap or trap crop

76
Q

what are the problems with ECB being in corn

A

-yield loss
-stalk and ear rots
-mycotoxins in grain
-harvestability

77
Q

what was the response to Cry1Fa resistance from ECB

A

-removal of single toxin Bt hybrids and Cry1F hybrids replaced with Cry1F + Cry1Ab
-increased resistance monitoring
-increased research on ECB phenology

78
Q

what is the life cycle of ECB

A

overwinter in corn stalks, then emerge and mate outside corn fields, then lay eggs on corn so larvae can feed on plant

79
Q

what is the best option for managing for ECB

A

transgenic corn

80
Q

describe mating disruption

A

the release of synthetic pheromone into a crop system to disrupt mating activity thus reducing the pest pop

81
Q

name 2 benefits of mating disruption

A
  1. few non-target effects
  2. hard to develop resistance
82
Q

what are lepidopteran pheromones made of

A

straight chain fatty acids derived chemicals, alcohols, aldehydes and acetates

83
Q

what is a plume

A

when females release pheromones in air

84
Q

what 3 factors are male and female mating behaviors dependent on

A
  1. age
  2. temp
  3. photoperiod
85
Q

what is the behaviour call when a female releases plume

86
Q

name 2 forms of competitive mating disruption

A
  1. competitive attraction (false trail following)
  2. induced allopatry (M drawn away from F)
87
Q

describe competitive disruption

A

males being drawn to pheromone dispensers rather than females

88
Q

describe competitive attraction

A

when dispensers are more attractive/numerous than females

89
Q

describe induced allopatry

A

males hang around dispensers instead of around females

90
Q

name 2 forms of non-competitive disruption

A
  1. desensitization (elevated response threshold, sensory blockage)
  2. suppressed calling/mating
91
Q

describe non-competitive disruption

A

males are prevented from responding to an authentic pheromone signal but don’t orient towards a dispenser

92
Q

describe desensitization

A

exposure to a high conc. of pheromones makes males unresponsive to pheromone for a period of time

93
Q

describe suppressed calling/mating

A

females detect high level of pheromone and stop calling

94
Q

what are the 2 theoretical approaches to mating disruption

A
  1. many dispensers but low conc per dispenser
  2. few dispensers but high conc per dispenser
95
Q

what is an implication of using mating disruption in IPM

A

pheromone lures can be placed in traps and used to monitor pests

96
Q

what happens to pheromone traps when implementing mating disruption

A

males cant find females or trap
-trap shutdown

97
Q

how do you monitor under trap shutdown

A

employ different lures (pheromone +synergist, semiochemicals, trap for F)
monitor for females

98
Q

describe acoustic mating disruption

A

using electronic devices that mimic calling males or mask the sound of males

99
Q

what are the issues with western bean cutworm

A

-feeds on field corn and dry edible beans
-results in yield dec. and inc. disease susceptibility
-gibberella and DON toxin

100
Q

what life stage of the western bean cutworm is best to target

A

egg masses bc they are hard to reach with insecticides once larvae burrow

101
Q

what regions is the western bean cutworm the largest problem

A

sandy regions

102
Q

what are the management strategies and what is effective and ineffective

A

-tillage (not E)
-biological (minor E)
-genetic, Bt (E)
-insecticides (E)

103
Q

what are the monitoring strategies for western bean cutworm

A

-pheromone traps
-scouting for egg masses
-insecticide

104
Q

what is the hypothesis for where western bean cutworm will be located

A

regions will lower plant density, convex, and sandy

105
Q

describe strong migratory connectivity

A

there is no gene flow between pops of the same species during migration

106
Q

describe weak migratory connectivity

A

pops may mix during the year during migration and may have gene flow
-events during one period of the annual cycle may impact the pop during subsequent periods

107
Q

why is understanding the migratory connectivity of a species crucial

A

for developing year-round predictive pop models

108
Q

what is the best way to track insect migrants

A

isotope analysis

109
Q

what are 3 applications of isotope analysis

A
  1. tracking migratory pests to estimate where a pop originated
  2. determining where invasive pests originated
  3. determining the degree of connectivity between portions of a migratory pests annual cycle
110
Q

what host does the spotted lanternfly prefer

A

grapevines

111
Q

why does the spotted lanternfly spread so easily

A

-females indiscriminately lay egg masses
-adults have sticky feet

112
Q

why is the invasion potential of SLF high in ON

A

-eggs are able to survive cold conditions
-many favourite hosts in ON

113
Q

is tree of heaven a required host for SLF

A

preferred but NOT required

114
Q

why are SLF a plant stressor

A

they are phloem feeders and produce lots of honeydew

115
Q

name 4 cultural management strats for SLF

A
  1. exclusion netting
  2. egg removal
  3. tree of heaven removal
  4. backpack vacuums
116
Q

name 5 reasons why SLF is a successful invader

A
  1. wide host range
  2. high fecundity
  3. cryptic life stages (eggs)
  4. introduced with no natural enemy
  5. human activities facilitate long-distance spread
117
Q

where is IPM increasing

A

-developed nations
-high-value crops (orchard)

118
Q

how many farmers are below the threshold of IPM

119
Q

vertical vs. horizontal integrations

A

vertical = one species
horizontal = management of several pest classes at once

120
Q

what are barriers to IPM adoption

A
  1. weak grower knowledge base
  2. user preferences and risk aversion
  3. vested interests and corporate responsibility
  4. traditional practices and emerging tech
  5. hard and soft policy levers
121
Q

what are 5 reasons IPM is losing popularity

A
  1. no universal definition
  2. barriers to adoption
  3. inconsistencies between concepts and practice
  4. continued reliance on insecticides
  5. inadequate research on IPM
122
Q

what are the 6 IPM tactics

A
  1. biocontrol
  2. chemical control
  3. behavioral control
  4. genetic control
  5. cultural control
  6. physical control