Test 2 Flashcards

1
Q

what are the 2 components of mood disorders?

A
  • depression
  • mania
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2
Q

what is a major depressive episode (mde)?

not the symptoms, but the brief outline of the disorder

A
  • lowest end of the spectrum
  • 2 week period, 5 or more of the symptoms
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3
Q

what are the 9 symptoms of a major depressive episode?

A
  1. depressed mood most of the day, nearly everday
  2. dimished interest/pleasure
  3. changes in apetite/weight
  4. changes in sleep patterns
  5. pychomotor retardation/agitation
  6. fatigue/loss of energy
  7. feelings of worthlessness or inappropriate guilt
  8. dimished concentration/ability to make decisions
  9. recurrent thoughts of death or suicide
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4
Q

if you have no history of mania or hypomania, and you have only 1 MDE, what does this result in?

A

major depressive disorder single episode

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5
Q

if you have no history of mania or hypomania, and you have more than 1 MDE, what does this result in?

A

major depressive disorder recurrent

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6
Q

the likelihood of having another mde ____ the more you experience them

A

increases

1e - 50% | 2e - 70% | 3e - 90%

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7
Q

what is dysthymic disorder? what is another name for it?

not the symptoms, but the brief outline of the disorder

A
  • depressed mood most of the day, more tha not, for at least 2 years
  • during a 2 year period, person has never been without symptoms for more than 2 months at a time
  • aka persistent depressive disorder in the dsm-v
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8
Q

what are the 6 symptoms of dysthymic disorder?

A
  1. poor appetite or overeating
  2. insomnia or hypersomnia
  3. low energy or fatigue
  4. low self-esteem
  5. poor concentration or difficulty making decisions
  6. feelings of hopelessness

presence, while depressed, of 2 or more ^^^

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9
Q

what is double depression?

A

persistent depressive disorder + major depressive episode = double depression

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10
Q

what is mania/a manic episode?

A
  • distinct period of abnormally elevated, expansive, or irritable mood
  • abnormally and persistently increased goal-directed activity/energy
  • must occur for more than one week OR have been hospitalized
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11
Q

what are the 7 symptoms of a manic episode?

A
  1. inflated self-esteem or grandiosity
  2. decreased need for sleep
  3. more talkative than usual/pressure to keep talking
  4. racing thoughts (“flight of ideas”)
  5. distractability
  6. increase in goal-directed activity
  7. excessive involvement in pleasurable activities with a potential for negative consequences

markes impairment in social and occupational functioning

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12
Q

what is hypomania/a hypomanic episode?

A
  • one step down from mania
  • same critieria BUT…
  • must persist for 4+ days and have NOT been hospitalized
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13
Q

what are the symptoms for hypomania?

A
  • same as mania
  • does NOT cause marked impairment
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14
Q

what are the 2 types of bipolar I disorder?

A
  • one or more manic episode(s) only
  • mde and manic episodes
  • stretches across the spectrum
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15
Q

what is bipolar II disorder?

A

presence of history of mde and hypomania (not mania)

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16
Q

what is cyclothymic disorder?

A
  • hypomanic + depressive symptoms
  • pattern lasts at least 2 years (1 year for children)
  • one step down from bipolar I (does not include the extreme ends)
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17
Q

what are the biological factors that cause mood disorders?

A
  • genetic predisposition
  • nt imbalances: serotonin, norepinephrine, domapnie (too much = mania, too little = depression)
  • endocrine system: cortisol
  • sleep disruption: destablizes mood
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18
Q

what are the psychological factors that cause mood disorders?

A
  • stressful life events
  • learned helplessness
  • pessimism
  • neuroticism
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19
Q

which medication is used to treat depressive disorders?

A
  • those that inhibit reuptake of nts
  • tycyclic antidepressants
  • monoamine oxidase inhibitors
  • SSRIs
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20
Q

which medication is used to treat bipolar disorders?

A
  • lithium: regulates rate of neurotransmission
  • valproate: increases GABA
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21
Q

what are psychological treatments for mood disorders? specially those that target behaviour.

A
  • behaviour activation: building mastery, pleasure, self-efficacy
  • sleep
  • routine
  • medication adherence
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22
Q

what are psychological treatments for mood disorders? specially those that target cognitive.

A

target the negative and biased thinking

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23
Q

risk of suicide is ____ among those with mood disorders

which disorder, particularly?

A

significantly increased, particularly in bipolar

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24
Q

what are the risk factors associated with suicide and mood disorders?

A
  • male gender
  • history of previous suicide attempts
  • comorbid substance use
  • periods of severe depression or mania
  • feelings of hopelessness or worthlessness
  • lack of social support
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25
Q

what is the difference between pessimism and neuroticism?

A
  • P is a way of thinking, N is a personality trait that influences emotional regulation
  • P fuels hopelessness and depression
  • N makes individuals emotionally reactive and prone to anxiety, depression, and mood swings
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26
Q

emotional disorders occur ____ after a traumatic inccident

A

one month

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27
Q

what are ‘criterion a’ stressors?

A

exposure to actual or threatened death, serious injury, or sexual violence through direct exposure, witnessing, learning that it happened to a close relative/friend or indirect exposure to aversive details of the trauma

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28
Q

who are more likely to experience indirect exposure of a traumatic experience?

A
  • therapists
  • first responders
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29
Q

what are the 4 PTSD symptom categories?

A
  • re-experiencing
  • avoidance
  • negative changes to mood and cognition
  • reactivity
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30
Q

explain the ‘re-experience’ component/symptom of ptsd

A
  • nightmares, intrusive memories, flashbacks
  • e.g., a car accident survivor hears a car horn and suddenly feels as though they are back in the crash
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31
Q

explain the ‘avoidance’ component/symptom of ptsd

A
  • avoidance of the feelings or reminders of the event through emotional numbing, avoiding people/places/circumstances
  • e.g., a first responder who witnessed a tragic event at work starts avoiding certain parts of the town where it occured
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32
Q

explain the ‘negative changed to mood and cognition’ component/symptom of ptsd

A
  • affects thoughts, emotins, and beliefs about oneself, others, and the world
  • e.g., a police officer feels emotionally detached and unable to feel joy, even in positive situations
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33
Q

explain the ‘reactivity’ component/symptom of ptsd

A
  • disturbed sleep, hypervigilance, increased startle response
  • e.g., a shooting survivor umps or panics when hearing fireworks or loud noices
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34
Q

what are the 2 ptsd specifiers?

A
  • with delayed expression
  • with dissociative symptoms
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35
Q

what is delayed expression in ptsd?

A
  • symptoms do not fully appear until at least 6 months after the traumatic event
  • some surpress or disconnect from the trauma until a trigger brings them to the surface
  • trigger for onset may be a lack of distraction (e.g., retirement)
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36
Q

what are dissociative symptoms of ptsd?

A

applies when an individual with ptsd experiences dissociation in response to trauma

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37
Q

what is dissociation?

A
  • a disconnection between thoughts, memories, identity, emotions, and perception of reality
  • depersonalization/derealization
  • coping mechanism to help manage extreme distress or overwhelming emotions
  • normative to disordered

not a single disorder, but a symptom that appears in various disorders

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38
Q

what is depersonalization?

A
  • feelinng detached from oneself
  • e.g., i feel like i’m watching myself from the outside
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39
Q

what is derealization?

A
  • feeling like the world isn’t real
  • e.g., everything looks foggy or dreamlike
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40
Q

what are causes for ptsd?

A
  • anxious predisposition
  • intensity and severity of trauma
  • lack of social support
  • brain changes explain why ptsd patients stuck in their trauma and react strongly to reminders
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41
Q

how does an overactive amygdala change/affect ptsd?

A
  • excessive fear responses
  • hypervigilance
  • emotional deregulation
42
Q

how does a shrinking in hippocampus change/affect ptsd?

A
  • flashbacks
  • memory gaps
  • difficult separating past/present
43
Q

what is neuroplasticity and how is it a ptsd maintenance factor?

A
  • brain adpats to trauma by reinforcing fear pathways in the amygdala and weakening memory regulation in the hippocampus
  • repeated stress exposure keeps these circuits active
  • avoidance behaviour reinforce these changes by preventing reprocessing of traumatic memories
44
Q

what are some ptsd psychological treaments?

A
  • imaginal exposure with relaxation training
  • eye-movement desensitization and reprocessing (EMDR)
  • retrain brain
45
Q

what is imaginal exposure with relaxation training?

A
  • clients mentally revisit traumatic events in a safe setting while practicing relaxation techniques
  • helps reduce avoidance, desensitize emotional distress, and reprocess trauma
46
Q

what is eye movement desensitization and reprocessing?

A
  • clients recall traumatic memories while following bilateral stimultion (eye movements, tapping)
  • helps the brain reprocess trauma and reduce emotional intensity
47
Q

how can you retrain your brain as a ptsd treatment?

A
  • reducing amygdala hyperactivity (lowering fear response)
  • strengthening hippocampal function (improve memory processing)
  • rewiring trauma memories from survival mode (amygdala) to rational processing (pre-front cortex)
  • allowing new, adaptive beliefs to replaces fear-based thinking
48
Q

what are the biological treatments for ptsd?

A
  • SSRIs
  • Repetitive Transcranial Magnetic Stimulation (rTMS)
49
Q

how does ssri treat ptsd?

A

regulate brain’s overactive fear response and mood alternations

50
Q

what is a repetitive transcranial magnetic stimulation?

A
  • magnetic pulses stimulate underactive brain regions associated with ptsd symptoms
  • strengthens connections between the prefrontal cortex (thinking brain) and the amygdala (fear center) to improve emotional regulation and reduce hyperarousal
51
Q

what is an acute stress disorder?

A
  • occurs up to 1 month after a traumatic event
  • can be short-term ptsd precursor
  • may reslve and not lead to ptsd, but is a significant risk factor (40-80%)
52
Q

what is an adjustment disorder?

A
  • emotional and/or behvaioural reactions to life stress (divorce, job loss, move) that is out of proportion to the stressor
  • may include depression, anxiety, and connduct problems (e.g., aggression, rule breaking)
53
Q

what is complex-ptsd?

A
  • caused by prolonged exposure to traumatic events, often interpersonal in nature (long-term abuse)
  • includes symptoms of ptsd + emotional dysregulation, interpersonal problems, complex changes in self-concept (e.g., lacking identity)
54
Q

which type of ptsd is not currently recognized in the dsm-5-tr? where is it recognized?

A

complex-ptsd, and is recognized in the icd-11

55
Q

how does complex-ptsd impact relationships?

A

problems with trust, intimacy, and a tendency
to avoid relationships or to seek out unhealthy relationships

56
Q

what is a treatment for complex ptsd?

A

May require longer-term psychotherapy that focuses on coping with traumatic memories + improving emotional regulation, self-concept, and relationship skills (eg., DBT)

57
Q

what are the 3 criteria for anorexia nervosa (a.n.)?

A
  1. dietary restriction -> low body weight (guideliine is less than 18.5 BMI, severity level is based on weight)
  2. intense fear of weight gain or becoming fat OR behaviours that inerfere w/ weight gain
  3. disturbed perception of body OR undue influence of body on self-evaluation OR denial of seriousness of low weight
58
Q

what are the 2 sub-types of anorexia nervosa?

A
  • restrictive type (AN-R)
  • binge-purge type (AN-BP)
59
Q

what is the restrictive type of anorexia nervosa?

A
  • restrictive eating as hallmark
  • absence of regular bingeing of purging
  • compulsive exercise common
60
Q

what is the binge-purge type of anorexia nervosa?

A
  • recurrent binges AND/OR
  • recurrent purging
61
Q

what are the issues with the bmi criteria?

A
  • binge-purge frequency should be considered as electrolyte imbalances, psychological distress and behaviours
  • rapid weight loss can be unhealthy, even if BMI is still in the “normal” range
  • delay diagnosis, discourage treatment, overlook serious cases (they don’t look “sick/thin enough”)
62
Q

in bulimia nervosa, what is a binge?

A
  • time frame: unusually large amount of food in a 2-hr time span
  • quantities: unusually large amount of food, generally high-fat, high-calorie
  • loss of control: where the person cannt stop themselves from overeating
63
Q

what is bulimia nervosa (BN)?

A
  • recurrent binge eating
  • recurrent inappropriate compensation (e.g., laxative use, fasting, vomiting, excessive exercise)
  • over-evaluation of shape and weight
  • anorexia nervosa criteria not met (weight)
  • often restricted eating outside of binges
  • 1 time a week for 3 months
64
Q

what is binge eating disorder?

A
  • recurrent binge eating episodes
  • marked distress about binge eating
  • no recurrent inappropriate compensation
  • 1 time a week for 3 months
65
Q

what are some associated features with eating disorders?

A
  • mortality: highest of all psych disorders since suicide is most common cause of death followed by cardiac arrest
  • cardiac problems: electrolyte imbalances, prolapse, reduced muscle mass, slowed heart rate
  • dental problems: stomach acid coming up due to purge
  • anemia
  • comorbid psych disorders
66
Q

what is the prevalence in eating disorders/related disorders in men?

A
  • around 25% of individuals with eating disorders are men, though underdiagnosis is common
  • men are less likely to seek treatment due to stigma and misconceptions
67
Q

what is the differences in presentation in eating disorders/related disorders in men?

A
  • more likely to focus on muscle gain, leanness, and control over physique
  • especially true in gay communities and athletes
  • gay and bisexual men: 7x more likely to develop an eating disorder
  • may engage in excessive exercise, supplement use, and rigid dieting
68
Q

what type of eating disorder is most common in men?

A

body dysmorphic disorder with muscle dysmorphia

69
Q

how do men have barriers to treatment in eating disorders?

A
  • social stigma: eating disorders are seen as a “female issue”
  • medical professional may overlook symptoms in men
  • less research and targeted treatment approaches for men with EDs
70
Q

what are the main causes for the glorification of thinness?

A
  • media and advertising
  • health and fitness culture
  • cultural and social rewards

accompanied by the stigmatization and discrimination against larger body

71
Q

adolescent girls who diet have ____ times the changes of developing ____ within ____

A

18, an eating disorder, 6 months

72
Q

what can food restriction lead to?

A
  • food obsession
  • binge eating
  • rapid weight gain
  • impair cognitive functioning
73
Q

what are the biological factors associated with eating disorders?

what are the key findings in twin studies?

A
  • both AN and BN have more than 50% heritability
  • higher condorance in monozygotic twins than in dizygotic twins
  • genetic factors contribute to personality traits linked to EDs (e.g., perfectionism, impulsivity, anxiety)
74
Q

what are the psychological factors in eating disorders?

A
  • developmental experiences: high control or family relationships/attitudes, criticism or pressure
  • trauma and abuse: trauma survivors may develop eating disorders due to shame, negative body image and/or as a way to cope
  • peer pressure and comparison culture, especially during adolescence
75
Q

how can personality associate with eating disorders?

A
  • perfectionism: high self-expectations, fear of failure/criticism/rejection
  • anxiety sensitivity: avoiding risk, strong need for control, obsessiveness
  • impulsivity: more common in bulimia/binge-eating disorder
76
Q

how do cognitive factors affect eating disorders?

A
  • black-and-white thinking about food, weight, and self-worth
  • distorted perception of body image
  • low self-esteem and identity struggles
77
Q

how does emotional regulation difficulties affect eating disorders?

A
  • difficulty coping with distress
  • restriction = emotional numbing thru bingeing = emotional escape
78
Q

what are some barriers to seeking help in eating disorders?

A
  • functions being served by the eating disorder may deter help-seeking
  • e.g., emotion regulation, trauma avoidance, social reward
  • shame and denial
  • barriers related to access
79
Q

what are some cognitive-behavioural treatment to eating disorders?

A
  • exposure necessary
  • weight restoration
  • eating feared foods
  • regular eating schedule
  • moderate exercisses
  • mindfulness/dbt
80
Q

eating disorders are similar to what?

A
  • phobias
  • obsessive-compulsive problems
81
Q

what are the stages/cycles of sleep?

A
  • stages 1 to 4: non-REM sleep
  • stage 5: REM sleep, rapid-eye movement, muscle atonia, vivid dreaming
82
Q

how long does each sleep cycle last?

A

90 minutes, with multiple cycles per night

83
Q

REM sleep becomes ____ in later cycles

A

longer and more frequent

84
Q

what happens to stage 4 if you go to bed late?

A
  • disappears after the first few cycles, while REM dominates in the morning
  • if you go to bed late, your body prioritizes REM and you won’t get deep sleep which is important for physical recovery
85
Q

why is REM sleep important?

A

crucial for memory consolidation and emotional processing

86
Q

what is the restoration theory in relation to theories on why we sleep?

A
  • suggests sleep is essential for repairing and rejuvenating the body and brain
  • helps in muscle growth, tissue repair, and protein synthesis
87
Q

what is the evolutionary theory in relation to theories on why we sleep?

A
  • sleep evolved as a survival mechanism
  • conserves energy and protects individuals from predators during parts of the day where there’s little value or danger in being awake
  • preserves energy so we don’t need to eat as much as well
88
Q

what is the information consolidation theory in relation to theories on why we sleep?

A
  • sleep plays a role in processing and cosolidating memories from the day
  • strengthens neural connections and supports learning and memory formation
89
Q

what is the waste clearance theory (glymphatic theory) in relation to theories on why we sleep?

A
  • sleep facilitates the clearance of waste products from the brain, including toxins associated with neurodegenerative diseases (e.g., dementia), through the glymphatic system
90
Q

how many hours a night do you need to be considered a ‘deprevation’?

91
Q

what are the cognitive effects of sleep deprivation?

A
  • irritability
  • cognitive/moral judgement impairment
  • memory lapses or loss
  • severe yawning
  • hallucinations
  • symptoms related to ADHD
92
Q

what are some health effects of sleep deprivation?

A
  • risk of type 2 diabetes, heart disease, obesity
  • impaired immune system
  • decreased reaction time, accuracy, and temperature
  • tremors and aches
  • growth supression
93
Q

what are the psychological effects of sleep deprivation?

A

losing sleep for one night can lead to deficits equivalent to having a blood alcohol concentration of 0.1% which is above the legal limit for driving in many jurisdictions

has historically been used as a form of torture

94
Q

according to the dsm-5-tr, what are the two major types of sleep disorders?

A
  • dyssomnias
  • parasomnias
95
Q

what is dyssomnia?

A
  • difficulties getting enough sleep or quality of sleep
  • insomnia disorder, hypersomnolence disorder, narcolepsy, breathing-related sleep disorders, circadian rhythm disorders
96
Q

what is parasomnias? what are its type?

A
  • abnormal events that occur during sleep
  • nightmare disorder
  • sleep terrors
  • sleep-walking
97
Q

what is the ‘equation’ for sleep disturbance?

A

biological vulnerability <–> sleep stress (e.g., poor sleep hygiene, cultural demands on sleep) –> sleep disturbance <–> maladaptive reactions (e.g., naps, sleep schedule changes, parental reactions, self-medication)

98
Q

what is insomnia disorder’s symptoms according to dsm-5?

A
  • dissatisfaction with sleep quality/quantity associated with at least one of the following difficulties:
  • initiating sleep or;
  • maintaining sleep (frequent awakenings); and/or
  • early morning awakening with inability to return to sleep
  • clinically significant distress/impairment
  • occurs at least 3 nights a week for at least 3 months
  • occurs despite adequate opportunity for sleep
99
Q

what are the medications for sleep disorders?

A
  • e.g., benzodiazepines and sedatives
  • prolonged use of meds cause dependence
  • rebound insomnia
  • CBT is more effective
  • melatonin: hormone that regulates sleep cycle. helps with sleep onset but no sustained sleep
100
Q

relative to good sleepers, those with insomnia are more likely to:

A
  • distorted perception of sleep length and quality
  • increased instrusive worries when trying to sleep
  • endorse unhelpful, negative beliefs about sleep (unrealistic expectations, catastophizing)
  • endorse positive meta-beliefs about benefits of worrying in bed
  • conserve energy during waking hourse (e.g., cancel appointments, avoid exercise, avoid social events, etc.)
101
Q

what are some cbts for insomnia?

A
  • individualized treatment, outlining behaviours for night-time and day-time symptoms
  • improve sleep hygiene
  • correct misperceptions of sleep and daytime deficits; reduce worring, rumination, and unhelpful beliefs
102
Q

what are some healthy sleep tips?

A
  • same bed/wake time, even on weekends
  • avoid naps
  • exercise daily
  • use bright light to help manage circadian rhythms
  • avoid alcohol, cigarettes and heavy meals in the evening
  • wind down
  • if you can’t sleep, go into another room and do something relaxing until you feel tired