Test 1 Flashcards

1
Q

define etiology

A

origins and causes of disorders

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1
Q

what are the 4 types of etiology

A

supernatural, biological, psychological, and social

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2
Q

what is the supernatural view for mental health problems

A

the perceived cause was spirits, demons, divine punishment, and witchcraft

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3
Q

what are the supernatural treatment methods

A

exorcisms (expel spirits), trephination (drilling holes in the skill to release evil spirits), prayer and religious rituals (appealing to divine intervention)

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4
Q

history of clinical psychology - ancient greece

(important person, predominant view, treatments (if applicable))

A

hippocrates (father of western medicine), and the origin of biopsychological view OVER supernatural views

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5
Q

history of clinical psychology - middle ages (~1500)

(important person, predominant view, treatments (if applicable))

A

st vincent de paul. rise of biological view and the asylum model. treatments included blood letting, squalid conditions, chaining to walls.

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6
Q

history of clinical psychology - enlightenmend period (~1700)

(important person, predominant view, treatments (if applicable))

A

psychogenic and rise of the hospital model and scientific approach. continuous advocation for humane treatment of patients

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7
Q

what are willhelm wundt’s contributions

A

experimental psychologist who founded the first lab in germany. first to research psychological constructs and processes such as sensation and perception, laying the groundwork for more applied clinical intervention

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8
Q

what are lightner witmer’s contributions

A

student of wundt who founded the first psychology clinic. emphasized the application of psychological principles for treatment purposes

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9
Q

what effect did the world war I and II have on psychology

A

emergence of psychological testing and intervention (and psychotherapy). due to the need to treat veterans with trauma after the war, there was an increased consideration of diagnostic/therapeutic methods.

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10
Q

how did the US Army benefit from the emergence of psychological testing and intervention

A

screen recruits showing the practical relevance of clinical assessments, and completed personality and intelligence tests to see if they were suited for a certain career

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11
Q

who was francis summer

A

father of black psychology, first black man to earn a PhD in psych. advocated for addressing racial bias in psychological research and promoting diversity in education

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12
Q

what does black psychology focus on

A

understanding/addressing the mental health and experiences of black people through a culturally relevant and empowering lens

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13
Q

who who was inez beverly prosser

A

first black woman to earn a PhD in psychology and studied the impact of segregated/integrated schools on black children’s self esteem/academics. advocated for safe and nurturing educational spaces to support black students

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14
Q

who was albert sidney beckham

A

one of the first black clinical psychologists and pioneer of school psychology, established first psychological clinic in a public school and emphasized the impact of social, emotional and environmental factors on academic performance

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15
Q

which 2 psychologists focused on school psychology

A

albert sidney beckham and inez beverly prosser

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16
Q

who was braulino alonso

A

pioneered work in counseling psychology for latinx students, advocated for educational equity and increased mental health access for marginalized communities. emphasized the need for competent counseling

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17
Q

what treatment did freud and breuer use

A

hypnosis (free association) and catharsis (release repressed memories, therapeutic benefit)

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18
Q

according to freud, what are the 3 levels of unconsciousness

A
  1. conscious with thoughts and perceptions
  2. subconscious with memories and stored knowledge
  3. unconscious with fears and socially unacceptable desires
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19
Q

according to freud, what are the 3 components of the mind

A

id, ego, superego

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20
Q

what disorder does too much id lead to

A

impulsive disorders

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21
Q

what disorder does too much superego lead to

A

compulsive disorders

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22
Q

what theory comes from carl rogers

A

humanistic theory

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23
Q

what is the humanistic theory

A

draws from maslow’s hierarchy of needs and values human connection, thwarted needs (particularly from parental figures)

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24
Q

what is unconditional regard and which theory uses it

A

where the patient is loved regardless of mistakes/achievements, used by humanistic theorists

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25
Q

what treatment is used by humanistic theorists

A

person-centered therapy (unconditional positive regard, empathy, genuineness, self-actualization)

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26
Q

who came up with the behaviorist model

A

skinner and watson

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27
Q

what is the behaviorist model

A

operational and classical conditioning

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28
Q

define classical conditioning

A

a NS is paired with an UR. NS becomes CS. now, CS will elicit a CR without the need for a NS

behaviour is learned through repeated associations between stimuli

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29
Q

what are the 2 treatments in the behaviorist model

e.g., used for classical conditioning

A

extinguishing and systematic desensitization

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30
Q

define cognitive model

A

disorders are a result of biased and distorted thoughts

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31
Q

what treatment is presented by the cognitive model

A

identify biased thought and challenge them with evidence

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32
Q

what are the 3 ds in abnormality

A
  1. dysfunction, breakdown in cognitive, emotional, or behavioural functioning
  2. distress or impairment, feeling upset about the problem or interfering with function
  3. deviant, atypical or not culturally expected from socio-cultural norms
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33
Q

give some examples of unidimensional explanations

A
  • genes cause schizophrenia
  • video games cause violent baheviours
  • coddling children cause narcissism
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34
Q

what is interconnectedness and feedback loops in the multidimensional approach?

A

when one action/event leads to a domino effect

low self-esteem -> not socializing -> isolation -> depression

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35
Q

what treatments would be used in the biological aspect of the biopsychosocial model?

A
  • medical evaluation for contributors to depression
  • explore the use of antidepressants
  • encourage regular physical activity
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36
Q

what treatments would be used in the psychological aspect of the biopsychosocial model?

A
  • increase daily pleasurable activities to improve mood
  • challenge negative thinking
  • process emotions in a healthy way
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37
Q

what treatments would be used in the social aspect of the biopsychosocial model?

A
  • gradually reconnect with friends
  • engage with community to find others with common interests
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38
Q

what does the multidimensional model explain?

A

mental illness is a system of reciprocal influences with complex interactions.

treating one aspect will likely influence other components

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39
Q

how do genes contribute to psychological disorders?

A

disorders are mostly polygenetic, meaning no individual gene have been identified to any major psychological disorder (except alzheimer’s)

if one identical twin has a disorder, 50% chance the other will too

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40
Q

how does the environment contribute to psychological disorders?

A

environmental factors influence the expression of different genes

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41
Q

what is the diathesis-stress model?

A
  • Genetic Vulnerability (aka Diathesis) x Stress (environmental factors) = Psychological Disorder
  • Environmental stress interacts with genetics to predict whether a perosn will exprience a psychological disorder
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42
Q

identify and define the 2 types of stress in the diathesis-stress model

A
  • percipitating stress: specific events that trigger an issue
  • predisposing stress: genetic
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43
Q

what is the reciprocal gene-environment model?

A
  • people with certain genetic traits are more likely to experience stress that can lead to psychopathology
  • genetic trait -> stress -> psychological disorder

it is a bit of victim blaming

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44
Q

what is epigenetics?

A
  • genes are turned on/off by cellular material outside of the genome
  • environmental factors influence epigenome
  • biological evidence of the effects of generational trauma
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45
Q

how does the frontal lobe contribute to psychological disorders?

A
  • involved in decision making, stm, impulse control
  • dysfunctions like depression, anxiety, ADHD, other mood disorders

not fully developed until 25 years old

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46
Q

how does the limbic system contribute to psychological disorders?

A
  • emotion regulation, fear response, memory formation
  • imbalances associated with anxiety disorder, PTSD, and depression

includes structures like the amygdala, hippocampus, and the hypothalamus

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47
Q

what is serotonin? what are the consequences when it is imbalanced?

A
  • regulates mood, appetite, and sleep
  • low levels associated with depression
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48
Q

what is dopamine? what are the consequences when it is imbalanced?

A
  • involved in reward, motivation, and pleasure systems
  • dysregulation linked to schizophrenia, addiction, and Parkinson’s disease
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49
Q

what is norepinephrine? what are the consequences when it is imbalanced?

A
  • influences alertness, arousal, and the stress response
  • imbalances can contribute to anxiety disorders and depression
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50
Q

what is neuroplasticity?

A

brain’s wiring is plastic and can be reconstructed

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51
Q

what can psychological intervention do to the brain inference to rewiring?

A
  • alter neural pathways associated with negative thinking
  • increase activity in the prefrontal cortex, enhancing control over limbic system
  • reduce activity in the amygdala, reducing stress/anxiety
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52
Q

how does pharmacotherapy work?

A

influences the chemical processes in the brain

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53
Q

what are the 4 main types of psychopharmaceuticals?

A
  • SSRIs
  • antipsychotic drugs
  • stimulants
  • benzodiazepines
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54
Q

how do SSRIs work?

A

increase serotonin levels, affecting mood and anxiety

selective serotonin reuptake inhibitors

55
Q

how do antipsychotic drugs work?

A

regulate dopamine activity, essential for schizophrenia treatment

56
Q

how do stimulants work?

A

increases dopamine and norepinephrine, used for ADHD

57
Q

how do benzodiazepines work?

A

fast-acting anxiety medications that enhance the effect of GABA (NT that inhibits brain activity)

58
Q

what is the classification of psychological disorders?

A

it is a central element of all branches of science, allowing us to organize, describe, and relate

59
Q

what are the pros of classification or labelling?

A
  • taxonomy: organizes presentations into meaningful groups
  • nomenclature: helps with communication, and helps people access certain accessibility services
  • treatment and insurance
  • social understanding of the severity of the disorder
60
Q

what are the cons of classification or labelling?

A
  • labelling and stereotyping
  • stigma-based discrimination
61
Q

what are the pros to categorical approaches?

A
  • simplifies communication
  • helps with treatment planning
  • easy research participant classification
  • ease of administration in hospital settings
62
Q

what are the cons to categorical approaches?

A
  • heterogeneity issues: depression may look very different for two people
  • boundary and threshold disputes/arbitrary timelines and thresholds
63
Q

what is the dimensional approach to psychological disorders?

A

views psychopathology as existing on a continuum rather than in discrete categories (scales, dimensions), recognizing overlapping and variability in symptoms, experience, and intensity

64
Q

what is cloninger’s seven factor model?

A
  • temperament: novelty seeking, harm avoidance, reward dependence, persistence
  • character: self-directedness, cooperativeness, self-transcendence
65
Q

what are millon’s three polarities?

A
  • pleasure - pain
  • active - passive
  • self - other
66
Q

what are the pros to the dimensional approach?

A
  • diagnostic reliability and stability
  • eliminate boundary disputes
  • gather more patient information
  • may benefit therapy
67
Q

what are the cons to the dimensional approach?

A
  • still requires thresholds
  • lack of agreement on dimensions (which ones, and how many?)
68
Q

what type of approach does the DSM-V follow? explain.

A

prototypical approach: essential characteristics and non-essential variations

69
Q

what is the DSM-V?

A

main classification system of adult and child mental illnesses with broad categories, symptom descriptions and differential diagnosis

70
Q

what is the prolonged grief disorder?

A

intense and persistent longing or preoccupation with the deceased, causing significant distress and impairment in daily functioning which last for an extended period post-loss

71
Q

what are the pros/strengths of DSM-V?

A
  • common language and criteria for diagnosis
  • consistency acorss clinicians, researchers, and institutions
  • not biased towards one theory
72
Q

what are the cons/criticisms of the DSM-V?

A
  • not consistent with scientific advances
  • over-influenced by the drug industry
  • does not take whole person into account
  • limites use for psychotherapy
73
Q

what are some DSM’s unresolved issues?

A
  • appropriateness of medical model (too symptom-focuses, superficial)
  • dimensions vs categories
  • validity and reliability of categories; no natural boundaries
74
Q

why is clincial assessment important?

A
  • formulate diagnosis
  • understand individuals
  • predict behaviours
  • plan and narrow treatment
  • evaluate treatment outcomes
  • help clients understand, t/f making them feel less ashamed

this is the clinical treatment reasons (other is research reasons)

75
Q

what are the 3 characteristics of strong assessment tools?

A
  • reliability: measure consistently
  • validity: measurement accuracy
  • standardization: guidelines to ensure consistency
76
Q

what are the 2 components of reliability in assessment tools?

A
  • inter-rater: different observers should agree that a person’s behaviour fit a certain diagnosis
  • test-retest: taking a test multiple times should produce the same results
77
Q

give some examples of assessment tools

A
  • physical exams
  • psychology tests
  • behavioural assessment
  • psychophsyiological assessment
  • neurophysiological assessment
  • neuroimaging
  • clinical interviews
78
Q

what is a diagnosis?

A

a clinical judgement about an individual’s mental health condition based on observed symptoms, behaviurs, and history

identifies/labels psych disorders using established criteria like DSM

79
Q

what is the controlled act of a diagnosis?

A

a task/procedure that is legally restricted to certain regulated professionals due to potential harm if performed incorrectly. E.g., diagnosing is considered a controlled act because it requires specialized knowledge, training, and judgement

80
Q

who can diagnose in ontario?

A
  • psychiatrists
  • clinical psychologists
  • nurse practitioners
  • physicians

clinical psychologists can only diagnose psychological disorders

81
Q

how can psychologists determine a diagnosis?

A
  • compile collected data from assessment
  • identify patterns and clustering symptoms that may fit a mental health condition
  • may refer to the DSM for specific criteria
  • symptoms must meet the required criteria
82
Q

what is a differential diagnosis?

A

when clinicians evaluate overlapping symptoms to rule out disorders that may present similarly

e.g., distinguishing between depression and bipolarity

83
Q

what are cultural considerations in diagnosis?

A

clinicians assess how cultural context may influence symptoms or behaviorus

84
Q

why is feedback important in diagnosis?

A

clinicians discuss their findings with the client for clarity and to ensure accuracy

85
Q

what is an important part of the diagnosis?

communciation

A

clinicians share the diagnosis with the client, explaining it in a clear and supportive manner

86
Q

what are the 4 components when designing a study?

A
  • ethics
  • research questions and hypotheses
  • samples
  • validity issues
87
Q

what is important in research studies?

there are 4

A
  • competence: must ensure participants have capacity to provide consent
  • voluntarism
  • full disclosure
  • comprehension: participants understand all possible risks and benefits
88
Q

what is the independent variable?

A

(IV or X) a variable that stands alone and isn’t changed by the other variables you are trying to measure

89
Q

what is a dependent variable?

A

(DV or Y) something that depends on other factors

90
Q

what is an example of a research question with IV and DV?

A

what is the studying (IV) on test grades (DV)

91
Q

what is a hypothesis?

A

researcher’s supposition about probable links between variables.

starting point for further investigation

92
Q

what are the types of sampling bias?

A
  • experimenter bias
  • self-selection bias
  • attrition bias
93
Q

what is the experimeneter bias?

A

occurs when scientists’ hypotheses influence their results, even if involuntarily

94
Q

what is the self-selection bias?

A

occurs when participants voluntarily choose to participate in a study or program group rather than being randomly selected into one

95
Q

what is attrition bias?

A

the selective dropout of some participants who systematically differ from those who remain in the study

96
Q

the ____ the line on a correlation graph the ____ the correspondance

A

steeper, higher

97
Q

what is the third variable problem?

A

type of confounding in which a third variable leads to a mistaken causal relationship between two others

98
Q

what does the manipulation of IV cause?

A

ehtical issues – cannot assign participants to harmful conditions (e.g., abuse, neglect, war)

99
Q

what are internal/external validity issues?

A
  • internal validity is the degree of confidence that the causal relationship you are testing is not influenced by other factors or variables.
  • external validity is the extent to which your results can be generalized to other contexts
100
Q

what are some experimental research strengths and weaknesses?

A
  • allows conclusions about causality
  • caution still required (interal validity achieved? limited external validity?)
  • sometimes impossible or unethical
101
Q

what is the nature of studying inidividual cases?

A
  • extensive observation
  • rich source of info about a case
  • foundation of early historic developments in psychopathology
102
Q

what are some limitations of the case study?

A
  • lacks scientific rigor and suitable contorls
  • often entails numerous factors that make results uninterpretable
103
Q

what are cross sectional trends over time?

A
  • snapshots in time
  • different age groups
104
Q

what are some disadvantages of cross sectional trends over time?

A

risk of cohort effects (something that affected the results unique to that snapshot in time)

105
Q

what are longitudinal trends over time?

A

same individuals over time

106
Q

what are some challenges in longitudinal trends?

A
  • cross-generational effect
  • resources
  • patience
  • study drop-outs
107
Q

what are some helpful natures/functions of anxiety?

A
  • alerts us to real/potential danger
  • prepares us to act
  • helps us escape danger
  • exists in moderate amounts
108
Q

what are some harmful natures/functions of anxiety?

A
  • excessive levels relative to “threat”
  • interferes with functioning
109
Q

what is the yerkes-dodson law?

A
  • low: increasing attention and interest
  • medium: optimal arousal/performance
  • high: impaired performance due to strong anxiety

describes how anciety affects performance

110
Q

where does anxiety come from?

A
  • evolved threat system
  • activated in response to dangers in the external environment
  • in humans, activated by danger that is not physical (real or imagined, external or internal)
111
Q

people with anxiety disorders have a ________ “threat sysetm”

A

highly sensitive

112
Q

what is generalized anxiety disorder?

A

anxiety that causes excessive wory and difficulty controlling these worries.

must last for 6 months or more

113
Q

what are some symptoms of GAD?

A
  • restlessness or feeling on edge
  • being easily fatigued
  • difficulty concentrating or mind going blank
  • irritability
  • muscle tension
  • sleep disturbance
114
Q

what are some biological factors associated with GAD?

A
  • inherited tendency to be tense and reactive
  • highly sensitive nervous system
115
Q

what are some learning factors associated with GAD?

A
  • early stressful experiences
  • modelling from others (learn that the world is dangerous and you cannot cope)
116
Q

what are some cognitive factors associated with GAD?

A
  • belief that worrying is helpful
  • difficulty tolerating uncertainty
  • cognitive avoidance: lost in mid, avoid engaging with reality
  • attention: biased towards threatening information
117
Q

what are the 2 types of medication used to treat GAD?

A
  • benzodiazepines
  • antidepressants
118
Q

how do benzodiazepines work as a treatment of GAD?

A
  • short term relief
  • significant side effects: impairment and poor concentration
  • dependence
119
Q

how do antidepressants work as a treatment of GAD?

A
  • safer long term
  • fewer significant side effects
  • not habit-forming
120
Q

how does psychotherapy work as a treatment of GAD? give examples.

A
  • as effective as medication short-term
  • more beneficial long-term
  • cognitive behaviour therapy: relaxation techniques, challenge thoughts, mindfulness and worry time
121
Q

what are obsessions?

A

intrusive, nonsensical thoughts, images, or urges that one tries to resist or eliminate, causing anxiety

122
Q

what are compulsions?

A

repetitive, ritualistic actions meant to provide relief and supress obsessions

123
Q

the obsessions and/or compulsions are ____ and cause ____.

A

time consuming, significant distress or impairment in social, occupational and other important areas of functioning

124
Q

what is OCD not caused by?

A

substance, medical conditions, or other psychological disorders

125
Q

how does OCD cause impairment?

A
  • disrupts daily life (work, relationships, self-care)
  • causes intense anxiety if rituals aren’t performed
  • triggers avoidance
  • lack of rational control over compulsions
126
Q

what are some DSM-5 TR specifiers for OCD?

A
  • with good or fair insight: knows what they are doing and why
  • with poor insight
  • with absent insight/delusional beliefs

clincially meaningful information on how the disorder presents itself

127
Q

what are the 3 most common obsessions in OCD?

A
  1. agression: unrealistic thoughts of hurting a loved one
  2. contamination
  3. symmetry and exactness
128
Q

what are the 3 most common compulsions in OCD?

A
  1. checking: to see if the stove is off, doors are locked, etc.
  2. washing/cleaning
  3. ordering/arranging
129
Q

how does cognitive function cause OCD?

A
  • structural abnormalities in the caudate nucleus
  • low serotonin strongly implied
  • attention drawn to disturbing materal relevent to obsessive concerns
130
Q

what are 3 causes of OCD?

A
  • genes: moderately heritable
  • brain and cognitive function
  • early learning: taught that some thoughts are dangerous and unacceptable
131
Q

what are some maintenance factors in OCD?

A
  • thought-action fusions
  • attempts at suppression
  • inflated sense of responsibility and guilt
132
Q

what are the medical treatments for OCD?

A
  • antidepressants (SSRIs)
  • in severe cases, psychosurgery (cingulotomy - incision to cingulate gyrus)
133
Q

what is a psychological treatment for OCD?

A

exposure and reponse prevention (ERP)

134
Q

what is used to measure correlation?

A

pearson correlation coefficient (r)