TBI Flashcards
Causes of acquire brain injuries-7
- strokes
- Tumors
- anoxia
- hypoxia
- toxins
- Degenerative disease
- near drowing
Definition of TBI
NOT DEGENERATIVE OR CONGENITAL
- caused by external physical force
- alteration in brain function caused by an external force
Outcomes of TBI depend on 5
immediate damage cumulative effects pre-morbid substance abuse interpersonal relationships and work history
Open Vs Close brain injuries
OPEN:
- penetrating types of wounds
- gunshot, knife
- skull is either fractured or displaced
- brain injury follows path of object entry and exit
- risk of infection increased from open wound and hair, fragments
Closed:
- impact to the head but the skull does not fracture or displace
- brain tissue damaged and dura remains intact
Contusion
- bruising on the surface of the brain
- small blood vessels on the surface hemorrhage
Coup vs CounterCoup
Coup:
- same side of brain where impact is
Counter:
- opp side of trauma
- acceleration can cause further vessel occlusion and edema formation
Epidural Hematomas
- between dura mater and skull
- seen with MVA blow to head and side of skull fracture
- Arterial Hematoma
Signs:
- unconsciousness and then alert&lucid
- immediate surgical intervention needed
Subdural Hematoma
- venous hematoma
- rupture of cortical bridging of veins
- between dura and arachnoid
- common in older adults who fall
- slow to build up and detect
signs:
- resemble CVA and flucuate
Small clots can be reabsorbed vs. large need to be removed
Subarachnoid hematoma
space below arachnoid and above the pia mater
- associated with cerebral aneurysm
- -but can be caused by skull fx
Diffuse Anoxal injury
- common
- usually from shaking brain back and forth
Symptoms of Diffuse Anoxal Injury
disorientation or confusion headache nausea or vomit drowsiness or fatigue trouble sleeping sleeping longer than normal LOB or dizziness
Concussion
most common
can be caused by open or closed injury
momentary loss of consciousness and reflexes
repeated concussion leads to CTE
Symptoms of concussion
dizziness disorientation blurred vision difficulty concentrating altered sleep patterns nausea headache LOB amnesia
3 types of secondary problems
*cerebral damage occurs in result to initial injury
1. Increased ICP
2 anoxic injuries
3. postraumatic epilepsy
Increased ICP
- skull is rigid and does not expand to accommodate edema
- leads to compression of brain tissue, decreased perfusion of blood and possible herniation
Treatment: monitor, drugs, shunt
S&S of increased ICP
decreased responsiveness impaired consciousness severe headache vomiting irritability papilledema changes in vital signs--increased BP and decreased HR
normal ICP range
5-10mmHg—> above 20 is abnormal
anoxic injuries
brain tissue demands 20% of bodys O2 intake to maintain proper o2 sats and metabolic functions
- cardiac arrest most frequent cause
- causes diffuse damage within brain tissue
What areas of the brain are vulnerable from anoxic injuries
hippocampus
cerebellum
basal ganglia
Post traumatic Epilepsy
- at increased risk after TBI
- open injury: subdural hematoma->older adults
- vestibular stimulation is contraindicated
what triggers a seizure
stress poor nutrition electrolyte imbalance missed meds flickering lights infection lack of sleep fever anger worry fear
what do you do if a pt. has a seizure
bring them to lowest safe level
do not put anything in mouth
protect them from hitting head but do not restrain
flip on side if they are starting to vomit
medications for seizures
Phenytoin
phenobarbital
carbamazepine
**also used to control behavior
Glasgow Coma Scale
- used in ER to assess the individuals level of arousal and function of cerebral cortex
- TYPICALLY: 3-4 scores do not make it
- most powerful predictor of prognosis and outcome of TBI
Mild TBI
- GCS of 13 or higher
- LOC less than 20 minutes
- normal CT
- awake when arriving to hospital
- may present dazed, confused
- c/o headache or fatigue
Moderate TBI
GCS 9-12
- confused and unable to answer questions
- most have permanent physical, cognitive and behavioral deficits
Severe TBI
GCS of 3-8
individual is in coma
permanent functional and cognitive impairments
7 manifestations of TBI
decreased level of consciousness cognitive deficits motor deficits sensory deficits communication deficits behavioral deficits associated problems
Definition of Coma
decreased level of awareness
state of unconsciousness
presents with:
eyes remain closed
unable to initiate voluntary activity
sleep/wake cycle not present
vegetative state
person who demonstrates a return of brain stem functions
- respiration
- digestion
- BP control
no sleep/wake cycle
may experience arousal and spontaneous eye opening
pain responses may be evident
unaware of external and internal needs
persistent vegetative
been in this state for a year or longer with no improvements in neurological status
arousal
regulated by RAS