survival & response, to stimuli & nervous system Flashcards

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1
Q

Describe the effect of IAA on root and shoot growth.

A

• IAA synthesised in shoots tips.
• IAA diffuses into growing (cell elongating) region.
• Light causes movement of IAA from light side to shaded side.
• Proportionally more IAA on shaded side.
• Causes directional growth as there is greater cell elongation on shaded side.
• Shaded side grows faster and causes shoot to bend towards the light.

• IAA synthesised in root tips.
• IAA diffuses into growing (cell elongating) region.
• IAA moves towards underside/shaded side.
• Inhibits cell elongation.
• Uneven growth (more elongation on side with less IAA).
• Causes directional growth away from light and downwards towards gravity.

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2
Q

Describe what is meant by the term taxis

A

• Moves towards or away from directional stimulus

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3
Q

Describe what is meant by the term kinesis

A

• Movement is random/non-directional response
OR
• Insect is not moving towards a particular stimulus;

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4
Q

Compare & Contrast taxis and tropism

A

• Both are directional responses to stimuli
• Taxis involves the movement of the entire organism whereas tropism is the movement of part of an organism

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5
Q

Explain the advantages of simple reflex arcs

A

• Rapid;
• Protect against damage to body tissues;
• Do not have to be learnt;
• Help escape from predators;
• Enable homeostatic control;

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6
Q

As a neurone transmits an impulse, its rate of oxygen consumption increases.
Explain why.

A

• ATP required for active transport;
• Na+ (actively) moved out only at nodes in myelinated / Na+ (actively) moved out along whole length of axon in non-myelinated;

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7
Q

Describe how a resting potential is maintained in a neurone.

A

• active transport/pumping of sodium (ions across membrane);
• out of neurone/higher concentration outside;
• differential permeability to K+ and Na+;
• Membrane more permeable to K+ ions;

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8
Q

The potential across the membrane is reversed when an action potential is produced.

Describe how.

A

• Sodium ion gates / channel (proteins) open;
• Na+ (rapidly) diffuse in;

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9
Q

Describe the sequence of events leading to the release of acetylcholine and its binding to the postsynaptic membrane.

A

• Depolarisation of presynaptic membrane;
• Ca2+ channels open and calcium ions enter (synaptic knob);
• (Calcium ions cause) synaptic vesicles move to/fuse with presynaptic membrane and release acetylcholine / neurotransmitter;
• Acetylcholine/neurotransmitter diffuses across (synaptic cleft);
• (Acetylcholine attaches) binds to receptors on the postsynaptic membrane;
• Sodium ions enter (postsynaptic neurone) leading to depolarisation;

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10
Q

When a nerve impulse arrives at a synapse, it causes the release of neurotransmitter from vesicles in the presynaptic knob.

Describe how.

A

• Sodium ions enter (postsynaptic neurone) leading to depolarisation;
• (Nerve impulse/depolarisation of membrane) causes Ca2+ channel (proteins) to open;
• Ca2+ enter by (facilitated) diffusion;
• Causes (synaptic) vesicles to fuse with (presynaptic) membrane;

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11
Q

The binding of GABA to receptors on postsynaptic membranes causes negatively charged chloride ions to enter postsynaptic neurones.

Explain how this will inhibit transmission of nerve impulses by postsynaptic neurones.

A

• (Inside of postsynaptic) neurone becomes more negative/hyperpolarised;
• More sodium ions required (to reach threshold) OR Not enough sodium ions enter (to reach threshold);
• For depolarisation/action potential;

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12
Q

Describe how the speed of the conduction could be increased in a neurone.

A

• Axon is myelinated;
• So shows saltatory conduction/impulses jump from node of Ranvier to node of Ranvier;
OR
• Axon has a larger diameter;
• So less resistance to flow of ions;

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13
Q

Myelination affects the rate of conduction of a nerve impulse. Explain how.

A

• Impulse jumps from node of Ranvier to node of Ranvier / depolarisation only at node of Ranvier;
• Fewer jumps / depolarisations to travel length of axon;

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14
Q

Describe how the inhibition of acetylcholinesterase affects the action of synapses.

A

• Acetylcholine not broken down / stays bound to receptor;
• Na+ ions (continue to) enter / (continued) depolarisation / Na+ channels (kept) open / action potentials;

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15
Q

Describe what is meant by the term refractory period

A

• (Refractory period) limits number of impulses per second/frequency of nerve impulses;
• Maximum frequency of impulse transmission
• Period of time between threshold and resting membrane potential.
• When maximum frequency reached/exceeded, no further increase in information/ all (higher) stimuli seem the same;

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16
Q

Describe the effect of IAA on the shoots (6)

A
  1. IAA synthesised in shoot tips
  2. IAA diffuses into growing region
  3. Light causes movement of IAA from light side to shaded side
  4. Proportionally more IAA on shaded side
  5. Causes directional growth as there is greater cell elongation on shaded side
  6. Shaded side grows faster and causes shoot to bend towards light
17
Q

Describe the effect of lAA on the roots (6)

A
  1. IAA synthesised in root tips
  2. IAA diffuses into growing region
  3. IAA moves towards shaded side
  4. Inhibits cell elongation
  5. Uneven growth
  6. Causes directional growth away from light and towards gravity
18
Q

Give two reasons why transmission across a cholinergic synapse is unidirectional (2)

A
  1. Presynaptic neurone releases acetylcholine
  2. Only post synaptic neurone has receptors
19
Q

Describe spatial summation (3)

A
  1. Different neurones converge at a single synapse
  2. Causes release of enough neurotransmitter to reach threshold
  3. Causes action potential in post synaptic membrane
20
Q

Describe temporal summation (3)

A
  1. Impulse arrive in rapid succession
  2. Giving cumulative effect
  3. Sufficient to depolarise post synaptic membrane
21
Q

Explain how a resting potential is maintained across the axon membrane in a neurone (3)

A
  1. Na+ diffuse in and K+ diffuse out
  2. Membrane more permeable to K+ leaving than Nat entering
  3. Na+ actively transported out and K+
22
Q

Explain why the speed of transmission of impulses is faster along a myelinated axon than along a non-myelinated axon (3)

A
  1. Myelination provides insulation
  2. In myelinated, saltatory conduction
  3. In non-myelinated depolarisation occurs along length of axon
23
Q

A scientist investigated the effect of inhibitors on neurones. She added a respiratory inhibitor to a neurone. The resting potential of the neurone changed from -70 mV to 0 mV.

Explain why (3)

A
  1. Less ATP produced
  2. Less active transport
  3. No net movement of Nat and Kt