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Paeds > Surgery > Flashcards

Surgery Flashcards

1
Q

Causes of acute abdo pain in children - medical? (15)

A 
Non-specific abdo pain
Gastroenteritis
Urinary tract
- UTI, pyelonephritis, hydronephosis, renal calculus
HSP
DKA
Sickle cell disease
Hepatitis
IBD
Constipation
Functional/recurrent abdo pain of childhood
Gynae
Psychological
Lead poisoning
Acute porphyria
Unknown
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2
Q

Causes of acute abdo pain in children - surgical? (7)

A 
Acute appendicitis
Intestinal obstruction
Inguinal hernia
Peritonitis
Inflamed Meckel diverticulum
Pancreatitis
Trauma
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3
Q

Causes of acute abdo pain in children - extra-abdominal?

A

URTI
Lower lobe pneumonia
Torsion of testis
Hip/spine

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4
Q

Ix for acute abdo pain?

A
  • Urine dip → UTI/DKA
  • Pregnancy test (maybe)
  • Bloods – U&Es, FBC, LFTs, glucose, calcium
  • Group and save
  • Urinalysis
  • ECG
  • O2 saturation
  • Radiology 

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5
Q

S+S of appendicitis?

A

Sx:

  • Anorexia
  • Vomiting (usually only few times) or diarrhoea (not sig amounts)
  • Abdo pain – initially central and colicky but then localising to 
RIF 


Signs:

  • Flushed face
  • Low-grade fever 37.2-38 degrees
  • Abdo pain aggravated by movement
  • Persistent tenderness with guarding in RIF (McBurney’s point) – pos absent in retrocaecal appendix
  • Rebound tenderness
  • Obturator sign – internal rotation of a flexed right thigh will give 
pain if there is an inflammatory mass overlying obturator space (pelvic appendicitis) 

  • Rovsing sign – pain in RLQ when palpate LLQ → indicates peritoneal irritation
  • May be few abdo signs in pelvic appendix
  • Appendicitis progressive therefore clinical review every few hours essential to diagnosis
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6
Q

Ddx of appendicitis? (9)

A
  • Gastroenteritis – N&V and diarrhoea
  • Constipation
  • PID
  • Volvulus
  • Hirschprung’s disease
  • Intussusception
  • Ovarian cysts
  • Pregnancy
  • Mesenteric adenitis
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7
Q

Ix for appendicitis? (3)

A
  • No lab test/ imaging consistently helpful in making diagnosis
  • Bloods: FBC, CRP, LFTs, amylase and lipase – useful if aetiology unclear
  • USS – may support clinical diagnosis
  • Laparoscopy if available
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8
Q

What is an appendicular mass?

A
  • Complication of appendicitis
  • Omentum and small bowel adhere to appendix
  • Usually presents with fever and palpable mass
  • Initial treatment = fluids, analgesia and abx but urgent surgical intervention may be required if mass enlarges or pt’s condition deteriorates
  • Recovery following conservative treatment usually by appendectomy
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9
Q

Diagnosis and treatment of appendicular abscess?

A
  • Can be shown by USS or CT
  • Initial treatment is by percutaneous or open drainage but open drainage also enables appendectomy
  • A worsening CRP with a good history is a sure signal of rupture and abscess formation
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10
Q

S+S of intestinal obstruction? (6)

A
  • Persistent, bile-stained vomiting – non bilious if before ½ duo
  • Delayed or absent passage of meconium
  • Abdo distention - depends on location
  • Abdo pain usually colicky then constant – child can’t sit still
  • Tenderness - minimal and diffuse or localised and severe
  • Auscultation - High pitched hyperactivity = mechanical obstruction - Over time waves and bowel sounds disappear
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11
Q

Causes of intestinal obstruction? (10)

A
  • NEC
  • Small bowel atresia
  • Volvulus and malrotation
  • Imperforate anus
  • Gastroschisis / exomphalus
  • Duodenal atresia/stenosis
  • Meconium ileus (consider CF)
  • Prenatal perforation (rare)
  • Hirschprungs disease (common)
  • Functional obstruction - defect in nn growth into myenteric plexi of bowel
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12
Q

Ix for intestinal obstruction?

A 
AXR
CXR - pos for perforation
FBC, U&E’s, Creatinine
Glucose
Urinalysis
ABG
Stool for occult blood
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13
Q

Peak age of intussusception?

A

3m-2y

–> Commonest cause of obstruction in infants after neonatal period

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14
Q

Presentation of intussusception? (6)

A 
Pain - severe, colicky
Vomiting
Mass
Redcurrant jelly stool
Abdo distension
Shock
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15
Q

Ix for intussusception? (2)

A

AXR
- Distended small bowel and absence of gas in distal colon/rectum

USS
- Helpful to diagnose/check response to treatment

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16
Q

Management of intussusception?

A
  • IV fluid rescus likely to be needed immediately (often pooling in gut)
  • Unless peritonitis, reduction of intussusception by rectal air insufflation
  • → Only carried out once child has been resuscitated and under supervision of paediatric surgeon in case unsuccessful or bowel perforation occurs
  • Sucess rate = 75%
  • Remaining 25% require operative reduction
  • Recurrence occurs in <5% but more frequent after hydrostatic reduction
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17
Q

Complications of intussusception? (3)

A
  • Shock and dehydration
  • Stretching and constriction of mesentery → venous obstruction → engorgement and bleeding from bowel mucosa, fluid loss and subsequently bowel perforation, peritonitis and gut necrosis
  • Metabolic acidosis
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18
Q

What is volvulus?

A

Loop of intestine twists around self and mesentery → bowel obstruction

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19
Q

Clinical features of volvulus? (7)

A
  • Bilious vomiting
  • Bloody/ dark red stools
  • Constipation + cramps (crying, pulling legs up)
  • Distended abdo
  • Tender abdo
  • Dehydration – dry nappies, sunken fontanelle
  • Ischaemia → acute abdo + peritonitis → raised HR, shock, hypovolaemia
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20
Q

Management of volvulus? (3)

A

Surgery

  • Rotate volvulus (Ladds procedure, not laparoscopic)
  • Bowel straightened, infarcted bowel removed

Fluid resuscitation

NG tube to drain stomach and bowel contents pre-op

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21
Q

RFs for NEC? (4)

A
  • Preterm
  • Low birthweight
  • Cow’s milk formula
  • Patent ductus arteriosus
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22
Q

Most common location of NEC? (2)

A

Terminal ileum

Proximal ascending colon

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23
Q

S+S of NEC? (7)

A
  • Usually in first 2w
  • Distension + tenderness
  • Vomiting – bilious
  • Bloody mucoid stool
  • ↓ Bowel sounds, palpable mass
  • May rapidly become shocked and require artificial ventilation
  • Stop tolerating feeds
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24
Q

Xray findings in NEC? (4)

A
  • Distended loops of bowel
  • Thickened bowel wall
  • Intramural gas and gas in portal tract
  • If perforation may see free air in abdo
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25
Q

Management of NEC?

A
  • Triple Abx therapy (broad spec)
  • Nil by mouth
  • Parental nutrition needed
  • If complications (perforation or stenosis > surgery may be required to remove non-functioning bowel)
  • Artificial ventilation and circulatory support often needed
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26
Q

Clinical features of bowel atresia?

A
  • Bilious vomiting
  • Prematurity
  • Polyhydramnios
  • Low birth weight
  • Additional early signs = jaundice, abdo distension and failure to pass meconium
  • Signs of continuous fluid loss such as dehydration, poor urine output, tachycardia and neurological involvement
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27
Q

What is Meckel’s diverticulum?

A

Ileal remnant of vitello-intestinal duct

  • Contains ectopic gastric mucosa or pancreatic tissue
  • 2cm long, 2% population, 2 feet from ileocaecal valve
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28
Q

What is a

a) Indirect inguinal hernia?
b) Hydrocele?

A

Both from failure of processes vaginalis (tongue of peritoneum) to obliterate after testicular descent

a) Processus vaginalus intact, bowel can enter inguinal canal
b) Processus vaginalus incompletely obliterated, leaving thin tract - peritoneal fluid can tract down into scrotum around testis

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29
Q

Which side is inguinal hernia more common?

A

RHS

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30
Q

RF for inguinal hernia? (2)

A

Boy

Premature

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31
Q

Clinical features of inguinal hernia?

A
  • Intermittent swelling in groin/scrotom on crying/straining
  • May become visible on raising IAP (press on abdo/cough)
  • Diagnosis relies on hx and identification of thickening of spermatic cord (round lig in girls)
  • May present as irreducible lump → firm & tender
  • → Infant may be unwell with irritability/ vomiting
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32
Q

Management of inguinal hernia?

A
  • Incarceration/strangulation in 12-16% → surgery ASAP
  • Most ‘irreducible’ can be reduced following opioids and sustained gentle compression
  • Surgery delayed 24-48h to allow reduction of odema
  • If not- risk of strangulation → operate immediately
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33
Q

Clinical features of hydrocele?

A
  • Asymptomatic scrotal swellings
  • Often bilateral
  • Sometimes blueish discolouration
  • May be tense or lax
  • Transilluminate
  • Some not evident at birth but present early childhood after viral/GI illness
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34
Q

Management of hydrocele?

A
  • Majority resolve spontaneously as processus continues to obliterate
  • Surgery considered if persists >18-24m old
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35
Q

Ddx of acute scrotum? (3)

A
  • Testicular torsion
  • Hyatid torsion
  • Epididymo-orchitis
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36
Q

How quickly must testicular torsion be operated on to prevent testicular necrosis?

A

6-12h after onset of sx

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37
Q

Clinical features of testicular torsion?

A
  • Pain in scrotum, groin or lower abdo
  • May be hx of previous self-limiting episodes
  • More common in L testis
  • Red, swollen testis, retracted upwards, decreased cremasteric reflex
  • In newborn → red, firm hard scrotal mass + doesn’t transilluminate
38
Q

Management of testicular torsion?

A
  • Surgical exploration mandatory unless torsion can be excluded
  • Doppler USS can allow differentiation from epididymitis (yet not used to diagnose)
  • Surgery within 6-12h
  • If torsion confirmed, must fix both testis (anatomical predisposition)
39
Q

Causes of epididymo-orchitis? (5)

A
  • UTI
  • STI
  • Mumps
  • Operation
  • Medication (esp amiodarone)
40
Q

What is balanitis?

A

Inflammation of the glans penis

  • Due to irritation by environmental substances, physical trauma, and infection
  • Usually affects boys <4y
  • Uncommon in circumcised men
41
Q

Clinical features of balanitis? (4)

A
  • Small red erosions on glans (can be small patch or whole glans)
  • Pain + redness of foreskin + penis
  • Other rashes + foul smelling discharge
  • Pain passing urine
42
Q

Causes of balanitis? (4)

A
  • Poor hygine + tight foreskin → most common
  • Infection e.g. candida, STI
  • Allergy or irritants
  • Skin conditions
43
Q

Management of balanitis?

A
  • Washed daily in warm water no soap
  • Swab to culture, check for DM
  • Tropic corticosteroids
  • Infection → antifungals/ abx
44
Q

What is BXO?

A

Balanitis xerotica obliterans

  • Skin condition affecting foreskin and penis → causes pathological phimosis
  • Atrophic white patches on affected area + white ring hardened tissue stops retraction
  • Can also cause urethral meatal stenosis
  • Sx → burning, pruritis, dysuria, painful erection
  • Circumcision only treatment
45
Q

What % have non-retractable foreskin at

a) 1y
b) 4y
c) 16y?

A

a) 50%
b) 10%
c) 1%

46
Q

Indications for circumcision? (3)

A
  • Pathological phimosis
  • Recurrent balanoposthitis – red/inflamed foreskin +/- discharge – usually responds to abx
  • Recurrent UTIs and upper UT abnormality
47
Q

What % have undescended testicle at birth and 3m?

A

4% at birth
- More common if preterm

1.5% by 3m (little change thereafter)

48
Q

3 types of undescended testicle?

A
  1. Retractile (with age will go into scrotum - yet follow up)
  2. Palpable
  3. Impalpable
49
Q

Ix for undescended testis? (3)

A

USS
- Performed with bilateral impalpable testes to verify internal pelvic organs

Hormonal
– For bilateral impalpable testes, presence of testicular tissue confirmed by rise in serum testosterone in response to IM HCG

Laparoscopy
- Ix of choice for impalpable testis

50
Q

How does undescended testes affect fertility?

A

Fertility for unilat undescended testis nearly normal

Fertility only 50% for bilateral palpable undescended testis

Usually sterile if bilateral impalpable testis

51
Q

What is pyloric stenosis and RFs?

A

Hypertrophy of pyloric muscle → gastric outlet obstruction

RFs:

  • M> F x4
  • More common in first borns
  • May be FH – esp maternal side
52
Q

Most common age of pyloric stenosis presentation?

A

2-7w (irrespective of gestational age)

53
Q

Clinical features of pyloric stenosis?

A
  • Vomiting – increases in freq and forcefulness over time
  • → Eventually projectile
  • Hunger after vomiting until dehydration → loss of appetite
  • Wt loss if presentation delayed
  • Hypochloraemic metabolic alkalosis with low Na and K due to vomiting
54
Q

Ix for pyloric stenosis?

A
  • Test feed (unless immediate fluid resus needed)
  • Baby given milk – may see peristalsis from L→R across abdo
  • Pyloric mass (feels like olive) usually palpable in RUQ
  • If stomach overdistended with air → must be emptied by NG tube to allow palpation
  • USS if diagnosis in doubt
55
Q

Treatment of pyloric stenosis?

A
  • Initial priority = correct fluid + electrolyte imbalance with IV fluids (0.45% saline and 5%dextrose with potassium supplements
  • Once hydration, acid-base + electrolytes normal → pyloromyotomy
  • → Division of hypertrophies muscle down to but not inc mucosa
  • Open or peri-umbilical procedure or laparascopically
  • Recovery fast – fed 6h later, discharged within 2d
56
Q

Ddx of lymphadenopathy? (3 categories)

A
  1. Infection
    - Viral URTI, Infectious mononucleosis (EBV, CMV), group A strep pharyngitis, acute bacterial lymphadenitis, Kawasaki disease, rubella, mumps, cat scratch disease, toxoplasmosis, TB
  2. Malignancy
    - Acute leukaemia, lymphoma, neuroblastoma, rhabdomyosarcoma
  3. Other
    - Thyroglossal cyst or brachial cleft cyst, cervical ribs, cystic hydroma, haemangioma, laryngocele, deroid cyst
57
Q

What is lymphadenitis?

A

Lymphadenitis = inflammation and/or enlargement of lymph nodes

  • Most due to benign, local or general infection
  • Onset can be acute, sub-acute or chronic
  • Small palpable cervical, axillary and inguinal nodes
58
Q

Ix for lymphadenopathy?

A
  • FBC and film
  • Rule out infectious causes: monospot, CMV, EBV, toxoplasma, bartonella titres, TB skin test, anti-HIV test, CRP, ESR
  • ECH if kawasaka suspected (autoimmune of blood vessels)
  • LFTs, U&Es and urinalysis (systemic disorders)
  • LDH, uric acid, ca, phosphate, Mg if malignancy suspected
  • BM biopsy if malig suspected
  • CT or USS guided lymph node biopsy
59
Q

Most common cause of lymphadenopathy?

A

Transient response to benign local or generalised infection

60
Q

What proportion of cleft lip and palate is each?

A

Half cleft palate
Quarter cleft lip
Quarter both

61
Q

Problems from cleft lip and palate? (5)

A

May be detected antenatally

  • Feeding problems – inadequate suck
  • Ear infections and hearing impairment
  • Speech and language problems (repaired before speech starts to 
develop)
  • Dental health – change in structure
  • Psychological issues
62
Q

When is cleft lip/palate surgery undertaken?

A
  • Cleft lip can be done in 1st week

- Cleft palate usually done at several months of age

63
Q

Associated conditions/drugs with cleft lip and palate? (5)

A
  • Anticonvulsant therapy
  • Isotretinoin (acne treatment)
  • Patau syndrome and other chromosomal disorders
  • Maternal factors - smoking, alcohol, obesity, lack of folate & HTN
  • 
Pierre Robin syndrome → rare condition where baby born with abnormally small lower jaw that causes their tongue to fall backwards in throat, causing breathing difficulties
64
Q

3 types of diaphragmatic hernia?

A

Bochdalek

  • > 90%
  • Posterolateral, usually on the left
  • In L → small and large bowel and solid intra-abdo viscera

Morgagni

  • Less common
  • Anterior – behind sternum, most slightly to R
  • In R → liver and portion of large bowel

Hiatus

Nb if bilateral usually fatal

65
Q

Signs of diaphragmatic hernia?

A
  • Cyanosis shortly after birth
  • Tachypnoea
  • Tachycardia
  • Asymmetry of chest wall
  • Absent breath sounds on 1 side of chest (usually left)
  • Bowel sounds audible over the chest wall
  • Abdo feels ‘less full’ on palpation
  • Shift of cardiac sounds
  • Signs of pneumothorax
66
Q

Management of diaphragmatic hernia?

A

Immediately after birth:

  • Optomise oxygenation whilst avoiding barotrauma
  • Stabilize baby before surgery
  • Resuscitate if needed
  • Intubate and artificially ventilate
  • Place NG tube and suction to remove air from stomach to prevent further lung compression
  • Continuous monitoring of O2, BP, perfusion – use drugs to maintain if needed
  • Maintain glucose and Ca levels

Once stable:
- Surgical repair

67
Q

2 pulmonary effects from diaphragmatic herniation?

A 
Pulmonary hypoplasia
Pulmonary HTN (decreased vasculature)
68
Q

Types of trachea-oesophageal atresia?

A

Trachea-oesophageal atresia w/o fistula (8%)

Trachea-oesophageal atresia with fistula

  • Proximal fistula 1%
  • Distal fistula (84%)
  • Prox + distal fistula (3%)

Trachea-oesophageal fistula w/o atresia (4%)

69
Q

What abnormality throughout pregnancy is trachea-oesophageal atresia associated with?

A

Polyhydramnios

70
Q

Clinical features of trachea-oesophageal atresia?

A
  • Persistant salivation + drooling
  • May → coughing and choking when fed and have cyanotic episodes
  • May be aspiration into lungs of saliva (or milk) from upper airways and acid secretions from stomach
  • Almost ½ have other congenital malformations eg VACTERL association
71
Q

Diagnosis of trachea-oesophageal atresia? (3)

A
  • If suspected – pass wide-calibre feeding tube and check by XRay to see if it reaches stomach
  • CT scan – clear picture of type of malformation
  • Oesophagoscopy and bronchoscopy
72
Q

Management of trachea-oesophageal atresia? (2)

A
  • Continuous suction applied to tube passed into oesophageal pouch to reduce aspiration
  • Transfer to neonatal surgical unit for repair
73
Q

Features of gastroschisis?

A
  • Bowel protrudes through defect in anterior abdo wall, adjacent to umbilicus
  • No covering sac
  • Not associated with other congenital abnormalities
  • High risk of dehydration and protein loss
  • → Abdo of affected infants wrapped in several layers of cling film to minimize fluid/ heat loss
  • NG tube passed and aspirated often
  • IV infusion of dextrose
  • Colloid sipper often required to replace protein loss
  • Repaired by surgery
  • If not pos to do in 1 surgery, intestine enclosed in silastic sac sutured to edges of abdo wall and contents gradually returned to peritoneal cavity
74
Q

Features of exompholos?

A
  • Also called omphalocele
  • Abdo contents protrude through umbilical ring into umbilical cord, covered with transparent sac formed by amniotic membrane and peritoneum
  • Often associated with other major congenital abnormalities eg trisomy 13, 18, 21, Beckwith-Weidemann syndrome and others
  • Minor = <4cm opening, only intestines
  • Major >4cm, intestines + liver
75
Q

Name 3 umbilical abnormalities?

A

Patent vitello-intestinal duct

Urachus
- leakage of urine out of umbilicus

Umbilical granuloma
- XS granulation tissue

76
Q

Management of low and high anorectal malformations?

A

Low
- Anoplasty

High

  • Initial colostomy
  • Appropriate surgery to form anus
  • Further surgery to remove colostomy
77
Q

4 types of congenital neck cysts?

A

Thyroglossal duct cyst
Branchial cleft cyst
Dermoid cyst
Enlarged lymph nodes

78
Q

What primary damage can occur from a head injury? (3)

A

Cerebral contusions/lacerations
Dural tears
Diffuse axonal damage

79
Q

What secondary damage can occur from a head injury? (5)

A

Hypoxia from airway obstruction/inadequate ventilation
Hypoglycaemia/ hyperglycaemia
Reduced cerebral perfusion
- Hypotension from bleeding
- Raised ICP
Haematoma (extradural, subdural, intracranial)
Infection (from open wound or CSF leak)

80
Q

What factors indicate a potentially serious head injury? (11)

A

LOC >5min
Amnesia >5min
Abnormal drowsiness
3 or more discrete episodes of vomiting
Clinical suspicion of NAI
Post traumatic seizure (no hx of epilepsy)
GCS <15 (<14 if <1y)
Suspicion of open/depressed skull injury/tense fontanelle
Sign of basal skull fracture (e.g. CSF leak from nose/ears)
<1y + bruise/swelling >5cm on head
Dangerous mechanism eg RTA, >3m fall

81
Q

General management of head injury?

A

Mild → discharge home with written advice

Potentially severe → monitor to avoid secondary damage

Severe → resuscitate, CT scan + neurosurgical referral

82
Q

What is a neuroblastoma?

A

Solid tumour arising from neural crest tissue in the adrenal medulla and sympathetic NS

  • Biologically unusual as occasionally regresses
  • Most common <5yo
83
Q

Common clinical features of neuroblastoma?

A 
Pallor
Wt loss
Abdo mass
Hepatomegaly
Bone pain
Limp

–> If >2yo, most sx from metastatic disease

84
Q

Diagnosis of neuroblastoma?

A

Characteristic clinical and radiological features with raised urinary catecholamine levels suggest neuroblastoma

  • Confirmatory biopsy usually obtained
85
Q

Management of neuroblastoma?

A
  • Localised primaries w/o metastatic disease → often cured with surgery alone
  • Metastatic disease → chemo, incl high­dose therapy with autologous stem cell rescue, surgery and radiotherapy
  • Risk of relapse high and prospect of cure for metastatic disease is little better than 30%
  • Immunotherapy and use of long­term ‘maintenance’ treatment with differentiating agents (retinoic acid) are establishing role in those with high-­risk disease
86
Q

What is a saccrococcygeal teratoma?

A
  • Teratoma located at base of coccyx thought to be derived from primitive streak
  • Benign
  • Most common tumour in newborns
  • Treatment = surgical removal
87
Q

What is Wilm’s tumour?

A

= Nephroblastoma

  • Originates from embryonal renal tissue
  • Commonest renal tumour of childhood
  • > 80% of patients present <5yo
  • Very rarely seen after 10yo
88
Q

Common and uncommon presenting features of Wilm’s tumour?

A

Common
- Abdo mass

Uncommon

  • Abdo pain
  • Anorexia
  • Anaemia (haemorrhage into mass)
  • Haematuria
  • HTN
89
Q

Ix for Wilm’s tumour?

A

USS +/or CT/MRI usually characteristic

90
Q

Management of Wilm’s tumour?

A
  • Initial chemo followed by delayed nephrectomy, after which tumour is staged histologically and subsequent treatment planned
  • Radiotherapy restricted to those with more advanced disease
  • Prognosis good
  • > 80% of all patients cured
  • Cure rate for metastatic disease at presentation (~15%) is >60%, but relapse carries a poor prognosis

Paeds (15 decks)

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