Surgery 2 Flashcards

1
Q

Hoof bandage

A

Can be wet or dry
After surgical procedures-softening
Extends under fetlock
3-5 days

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2
Q

Distal limb bandage

A

Surgical sites or intraarticular injections
From coronary band to carpus or tarsus
Fixed to hoof capsule by impermeable tape

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3
Q

Robert-Jones bandage

A

Immobilization of the limb and joints!
Standard bandage with additional sheet cotton
1.5x the circumference of the limb
Split can be applied to strengthen

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4
Q

Carpal bandaging

A

From coronary/fetlock to above the carpus
Hole/pressure releasing pads over acc carpal bones to prevent pressure sores or pressure necrosis of skin
Normal bandage plus strengthening additional layers
Splints can be applied for more restriction of movements

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5
Q

Carpal splint

A

When need stabilization of the limb- luxation, fracture, tendon rupture
On top of Robert-Jones
Proper protection of acc!!!
Split from coronary band/fetlock to under the elbow

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6
Q

Tarsal bandaging

A

Same as carpal but this time pressure releasing pads over the common calcaneal tendon

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7
Q

Common problems of bandages

A
Slipping/rotation of bandage and splint 
Too tight or too loose 
Pressure necrosis of skin
Uneven tension of bandage 
Contamination
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8
Q

What is neuroleptanalgesia

A

sedatives and analgesic
for standing position procedures and diagnosis
head comes down to carpal level- head support may be needed
Partial unconsciousness and muscle relax– can add analgesia for surgical procedures

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9
Q

Combos for neuroleptanalgesia

A

ACP and Butorphanol
Xylazine and Butorphanol
Detomidin and Butorphanol
ACP and Xylazine and Butorphanol

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10
Q

Cardiopulmonary effects of alpha2 agonists

A
  1. Vagal tone incr– bradycard– decr CO
  2. Hypertension at beginning.. then hypotension
  3. if give IV– temp grade I and II AV block
  4. Dysrhythmia or arrhythmia
  5. Central resp depression
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11
Q

GI effects of alpha2 agonists

A

Block the swallow reflex
Reduced visceral motility and perfusion to the organs
Hyperglycaemia
Are good visceral analgesics– therefore good for colic

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12
Q

Cardiopulmonary effect of opioids

A

Resp depression
Hypotension
Bradycardia

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13
Q

Opioid drugs used: agonists

A

Methadone: 3-4x more potent than morphine
Morphine
Morphinum hydrochlorium
Fentanyl- lipophilic so use a patch

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14
Q

Opioid drugs used: agonists-antagonist

A

Butorphanol

Pentazocin

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15
Q

Opioid drugs used: antagonist

A

Naloxon

Nalorphin

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16
Q

Local anaestheisa: physical methods

A

Ties and tourniquets: nerve press and anaemia

Cool: at 4degrees- stops the potency of nerve stim

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17
Q

Local anaesthesia: chemical

A

Esters: cocaine, procaine and tetracaine.. are hydrolysed by plasma pseudo-cholinesterase

Amides: lidocaine, mepivacaine and bupivacaine- are metab by the liver and so are better

Cannot be absorbed through intact skin

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18
Q

Local anaesthetic used in optho

A

Oxibuprocaine and proparacaine
These are 10-15x more effective than procaine
Can be toxic for the corneal epithelium

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19
Q

Local anaesthetics for mucus membranes and skin

A

Lidocaine: most stable, good penetration: 1.5-2hrs
Bupivacaine: 4-6 hrs
Mepivacaine: fast effect! only lasts 1-2hrs

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20
Q

Methods of local anesthesia

A
Topical
Infiltration
Regional IV
Intrasynovial
Perineural
Paravertebral
Epidural
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21
Q

Local anaesthesia: Infiltration

A

Safest
2% lidocaine
SE: hematoma

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22
Q

Local anaesthesia: Regional IV

A

IV catheter and Esmarch tourniquet

2% lidocaine (same as for infiltration)

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23
Q

Local anaesthesia: Intrasynovial

A

Intraarticular
Intrathecal bursa
Tendon sheath: Mepivacaine, bupivacaine, lidocaine

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24
Q

Local anaesthesia: Perineural uses and types

A

Lameness diagnosing
Palliative- laminitis hoof cast
Surgery of the head

Periorbital
Dental and muzzle
Corneal

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25
Q

Local anaesthesia: Paravertebral anaesthesia

A

Laparoscopy and flank laparotomy

If successful block– vasodilation, sweating– Horner’s syndrome like

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26
Q

Local anaesthesia: Epidural

A
Sedation 
Btw Cc1 and Cc2
Drugs used:
2%lidocaine
Xylazine (and saline)
Detomidne and morphine
Morphine
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27
Q

ASA classification of risk categories for surgery

A
  1. Healthy horse
  2. Mild systemic disease– mild anemia, RAO
  3. Severe systemic disease– severe RAO
  4. Severe systemic that is life-threatening– colic, polytrauma
  5. Moribound horse, not expected to survive for more than 24hrs– foal with uroperitoneum

E. Emergency

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28
Q

Preoperative evaluation

A

Goal: to define the risk for the owner
To select the best strategy to minimize the risks

  1. Free airway- intubation
  2. O2 supply
  3. IPPV= intermittent Postive Pressure Ventilation
  4. Venous Pressure Catheter
  5. CPR= cardio pulmonary resuscitation
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29
Q

Patient prep for surgery

A
  1. History- prev anaesthesia
  2. Physical exam- focus on resp, CV, musculoskeletal and CNS
  3. For emergency cases- first treat shock and stabilize
  4. Lab tests: elective selection: PCV, TPP sometimes hematology
  5. Fasting– no water for 6 hrs prev–lung function, decr chance of stomach rupture and decr risk of postop ileus
  6. Body weight- drug dosages
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30
Q

Surgical Complications and emergencies

A
Cardiopulmonary resuscitation
Anaphylaxis
Intraoperative hypotension
During maintenance 
Hypoxemia and Hypoxia
Hypercapnia 
Postop myopathy
Postop neuropathy
Postop laryngeal edema
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31
Q

Cardiopulmonary resusitation

A

Intraop mortality 30% due to cardiac arrest
Caused by deep hypotension and the anesthesia

Signs: 
EtCO2 decreases
Weak pulse 
Cyanotic mm
Dilated pupils 
Kussmaul type breathing 
Tx: discontinue anesthetic admin
IPPV
Chest compression 60x/min
O2 supply 
IV drugs
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32
Q

Anaphylaxis

A

Causes:
vasoD and incr vessel permeability
AB’s- penicillin and aminoglycosides

Just after drug admin there is: spO2 decr, weak pulse, bronchospasm, pulmonary edema

Tx: 
Stop giving the drug 
IPPV
O2
Give epinephrine, AH's etc
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33
Q

Intraoperative hypotension: what it is and causes:

A

Happens with inhalational more so than TIVA or PIVA
Make sure ABP is over 70mmHg, foals should be lower

Myocardial depression- endotoxaemia
Bradycard
Hypovolemia, acidosis and electrolyte imbalance–shock

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34
Q

Intraoperative hypotension: consequences

A
Poor tissue perfusion
Postop myopathy
SC ischaemia
Cerebreal necrosis
Myocardial dysfunction
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35
Q

Intraoperative hypotension: treatment

A

Infusion– electrolyte, colloid, hypertonic
(+)inotrop– dobutamin
calcium

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36
Q

During maintenance

A

hypovent– V/Q mismatch– hypoxemia

decr CO

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37
Q

Hypoxemia and hypoxia: what it is and causes

A

Hypoxemia: paCO2 is less than 60mmHg
Hypoxia: inadequate tissue oxygenation

Causes:
Failure in O2 supply 
Hypoventilation
Problems with endotracheal tube 
Distended abdomen putting pressure on thorax
RAO
Acute pulmonary edema
Shunt
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38
Q

Hypoxemia and hypoxia: Methods of improving

A

Early vent–IPPV
Increase FiO2
Albuterol bronchoD
Pulsed delivered NO

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39
Q

Hypercapnia: What it is, causes, effects and treatment

A

When paCO2 is greater than 45mmHg

Causes:
Resp centre depression
Hypovent
Incr CO2 prod

Effects:
Symp stim
Arrhythmia, resp acidosis
Intracranial P incr

Tx:
IPPV– get to anesthesia depth

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40
Q

Postop myopathy: causes and treatment

A
Causes: 
Large body
Long anesthesia time
Inadequate padding 
Intraop hypotension and hypoxemia 
Tx:
Adequate padding 
Assistance in standing 
Mild cases- exercise and walking 
Mannitol infusion
Vit E and selenium
Massage
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41
Q

Postop neuropathy

A

Caused by inadequate padding and conditioning, overextension of limbs

Radial, femoral and facial nerve injury

Treatment similar to myopathy

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42
Q

Postop laryngeal edema: what it is, causes and treatment

A

Spinal cord myelomalacia– very severe
in young- hypotension, embolus
“dog-sit position- loss of deep pain- poor prognosis

Cerebral cortical necrosis- severe and fatal

Causes:
Bilateral nasal/ laryngeal edema
Bilateral laryngeal neuropathy- hemiplagia
Negative pressure pulmonary edema

Tx:
Temp tracheostomy

43
Q

Anesthesia of risk patients: foals– considerations

A

Pulm changes within the first few hours
Circ changes within 3 days
PaO2 is less in lateral recumbency (10-15mmHg) than it is in standing– provide O2 if in lateral recumbency
Lung and chest compliance is less than in adult

44
Q

Anesthesia of risk patients: foals- preoperative evaluation

A

Congenital heart defect very high risk because the ductus arteriosus can reopen- hypoxemia or acidosis
Colostrum intake- measure IgG- if less than 800mg/dl- give plasma infusion or colloids
Blood glucose- hypoglycaemia- not sucking, bacteremic

45
Q

Anesthesia of risk patients: foals– fasting

A

Neonatal- allowed to suck
If tube fed- withold milk- sick foals have delayed gastric emptying
Foals over 3 months- max 4-6 hrs fasting

46
Q

Anesthesia of risk patients: foals- Newborns thermoregulation

A

CO2 production is HR dependent so avoid bradycardia
Small body mass, low fat, large surface area leads to significant heat loss
High metabolic rate- leads to hypoxia and hypoglycaemia faster than it would in adult horses

Effects of hypothermia: 
decr MAC
bradycard and decr CO
decr perfusion
decr metab
incr bleeding time 
delayed recovery with incr O2 consumption
47
Q

Anesthesia of risk patients: foals- sedation

A

T=Young easily become recumbent
Have to prevent hypotension and hypoglycaemia
Always provide O2!!!

Drugs:
Butorphanol
BZD: diazepam midazolam give exact dose IV
Xylazine and detomidine- only to not critically sick and older- because of the CV SE’s
Medetomidine and romifidine IV

All low doses to effect

Avoid giving in the sick neonatal foal

48
Q

Drugs to neonatal foals

A
Injectable 
Considerations:
High sensitivity of receptors 
High TBW and plasma fluid 
Permeable BBB
Low albumin therefore low plasma protein binding 
.. but weaker drug metabolism-- longer lasting effect
Inhal can have a lower MAC
49
Q

Anesthesia of risk patients: foals– Induction

A

INHALATIONAL ANESTHETICS ARE NOT RECOMMENDED
In neonates after minimal sedation
Facemask/ nsotracheal tube
Very quick uptake of these drugs

With IV Drugs:
Light plane: ketamine and xylazine
Deep plane: ketamine and xylazine and alpha2 agonist
Propofol on its own

50
Q

Anesthesia of risk patients: foals– maintenance

A

Inhal or TIVA or PIVA

PIVA: ketamine, isoflurane and lidocaine

51
Q

Anesthesia of risk patients: foals– Monitoring

A

They have a lower blood pressure
bradycardia <50 beats
High RR- this is why the inhal an are very rapidle acting
Endotracheal intubation difficult– therefore easier to get hypercapnia
PaCO2 is less than 50-60mmHg- small volume breathing circle needs to be used
Active heating
Check blood glucose– give IV dextrose if very low
During recovery get to sternal recumbency ASAP because of the compliance of thorax

52
Q

Anaesthesia of Geriatric Horse: general considerations

A
Over 20 years of age 
Loss of functional reserve 
Lower ABP
Lower ventricular filling 
Lower TBW
Lower metab, liver, kidney and heart function
53
Q

Anaesthesia of Geriatric Horse: sedation

A

ACP IM and xylazine and butorphanol IV

54
Q

Anaesthesia of Geriatric Horse: anesthesia

A

Not a good option
Inhal, TIVA, PIVA– IPPV is essential

Preferred: Neurolept- analgesia and local anesthetics– in standing position

55
Q

Anaesthesia of Geriatric Horse: monitoring

A
positioning 
body temp thermoreg decr
ABP supporting- hypovol
ECG dysrrhythmias
Blood gas analysis
56
Q

Anaesthesia of Geriatric Horse: some age associated diseases

A

RAO
Cushings
Aortic valve insufficiency
Hypothyroidism

Recovery with assistance

57
Q

Anaesthesia of donkeys and mules: general considerations

A

Narrower, deeper larynx and trachea– smaller ET
Jugular catheterization is more difficult
Faster elim of drugs- therefore more freq dosing needed– around 30% higher than for a horse
Plasma GGT is 3x greater than horses

Sedation: IM first then give IV alpha2

58
Q

Anaesthesia of donkeys and mules: maintenance

A

TIVA: triple drip: alpha2 and ketamine and GGE- small donkeys can be easily OD, GGE sensitivity- hemolysis
PIVA
Inhal an
NSAIDS- shorter elim half life than horse
Opioids

Prone to hypoxemia

59
Q

Anaesthesia of horses with intestinal emergencies (colic): general considerations

A

10x greater risk–which is multifactorial- CV compromise and endotoxemia

60
Q

Anaesthesia of horses with intestinal emergencies (colic): Preop Evaluation

A

Hypovolaemic shock: HR, Pulse and CRT all incr
cyanotic and pale mm
Abdominal pain, shock- stomach and intestinal distension
Endotoxaemic shock

61
Q

Anaesthesia of horses with intestinal emergencies (colic): Preparation

A
  1. Stomach tube- for better resp and CV function
  2. Fluid therapy:
    Isotonic fluids– maybe intestinal edema and reflux
    Hypertonic saline
    Colloids
    Acid-base correction
62
Q

Anaesthesia of horses with intestinal emergencies (colic): Sedation and analgesia

A
Xylazine 
NSAIDS
Opioids can combo with alpha2 agonist
Antiendotoxin like polymixin B
ABx- penicillin or gentamicin before induction 

PHENOTHIAZINES ARE CONTRA!!

63
Q

Anaesthesia of horses with intestinal emergencies (colic): Maintenance and recovery

A

IPPV- can actually further compromise CV
give low dose Isoflurane, make sure there is adequate oxygen
PIVA: Lidocain CRI
If theres hypocalcaemia give Dobutamine in an infusion slowly with calcium

Recovery is usually slow

64
Q

Anaesthesia of pregnant mare: general considerations

A

Paramount to maintain BP and normal O2
Minimal surgery time– minimize the fetal exposure
Big belly presses against thoracic cavity- intraop hypovent and hypoxemia– IPPV is therefore NB!!!

65
Q

Anaesthesia of pregnant mare: sedation

A

alpha2
opioids– but they can cross the placental barrier
Flunixin can block PGF2alpha release- ensures fetus is not lost after uterine manipulation

66
Q

Anaesthesia of pregnant mare: Maintenance

A

Inhal:
isoflurane
half-lateral recumbency to avoid v.cava compression
TIVA can cause fetal bradycard if continuously given
PIVA lidocaine can be toxic

67
Q

Anaesthesia of pregnant mare: Monitoring

A

ABP
Blood gas analysis
Fetal HR
If doing C-section- give oxytocin to prevent blood loss

Trendelenberg position- worst position for pregnant mare becuase put more P on the thorax

68
Q

Anaesthesia of pregnant mare: Recovery

A

Myopathy: because large body, hypotonia and hypoxemia
Mare is exhausted so needs assistance
Fracture risks: so calcium decreases

69
Q

Hyperkalemic periodic paralysis (HYPP)

Signs and treatment

A
GA can act as a trigger 
Signs: 
Hyperkalemia
Tachy/ bradycardia 
ECG changes 
Hypotension
Muscle tremor
Hypercapnia
Normothermia
Treatment:
Ca-gluconate infusion
Dextrose inf 
insulin 
all act to move K out of blood into the IC space
70
Q

Anaesthetic and equine malignant hyperthermia

A

Freq: QH’s, thoroughbred, appaloosa, arabian, pony
When an. for more than 3hrs

ETCO2 incr
PaCO2 incr 
Muscle rigidity
Tachpnea 
Body temp incr 
2nd metab acidosis- arrhythmia, CK and K incr 

Mechanism: the volatile anesthetic stim Ca release from the SR into the myoplasm– muscle contration– heat and acid prod

Tx:
Pure O2 IPPV
TIVA and good monitoring is preferred
Dantrolene to block the calcium release

71
Q

Anaesthesia of horse with RAO:

A
Prior to surgery: 
Secretolytic and bronchodilator 
Inhal an:
IPPV and O2 supplementation is essential
Longer insp and exp phase- defective elastic recoil- airway closure 
Albuterol into ET- Ventolin spray
72
Q

Anaesthesia of horse with CV problem

A

Rare!! is usually secondary

73
Q

Anaesthesia Practice- principles

A
Physical exam 
Catheter site 
Lavage of oral cavity
Premed
Induction
Intubation
Reflexes in surgery-plane 
Recovery
74
Q

Anaesthesia Practice: physical exam

A
CV and resp systems!!!
Heart beat auscultation at least 5 mins on both sides ]Auscultate lungs both sides RR and quality of breath 
Intestinal- colic 
Conjunctiva and mm
CRT in the mouth
Core temp
75
Q

Anaesthesia Practice: Catheter site

A

Clipping
Clean with soap
Disinfection with alcohol containing solutions
No LA or sedation to be used through it
Secure with one-stitch suture- short term

76
Q

Anaesthesia Practice: premedication drugs

A

IM in semitendinous muscle

IV for sedative e.g xylazine

77
Q

Anaesthesia Practice: Reflexes in surgery plane

A

Remains:
Corneal reflex
Ventromedial rotated eyeball
Pupillary light reflex

Gone:
Palpebral reflex
Nystagmus
Tears

78
Q

Anaesthesia Practice: recovery

A

Stop inhal an and disconnect the breathing circuit
Move to recovery box
ET remains with the inflated cuff- give O2 and then remove it
Cover shoes with tape
Keep in lat recumbency for as long as possible

79
Q

Anaesthesia practice: IPPV

A

Volume icr– injury to lung
Pressure incr– less injury to lung

small: 10 H2Ocm
large: 20 H2Ocm

80
Q

Anaesthesia practice: TV

A

In generak 10ml/kg bw, for a thin horse it is more

81
Q

Anaesthesia practice: O2 consumption

A

Minimum 2-3ml/kg

82
Q

Types of narcosis systems

A

Open
Semi-open
Semi-closed
Closed

83
Q

Narcosis system- open

A

No

  • rebreathing balloon
  • CO2 absorption
  • re-breathing
84
Q

Narcosis system- semi-open

A

Yes- rebreathing balloon
No- CO2 absorption
Partial- rebreathing -pendulum like cyst

85
Q

Narcosis system- semi-closed

A

Yes
- rebreathing balloon
-CO2 absorption
Partial- rebreathing

86
Q

Narcosis system- closed

A

Yes

  • rebreathing ballon
  • CO2 absorption
  • rebreathing
87
Q

Anaesthesia machine: components

A
Mixing part
Breathing circle 
Endotracheal tube 
IPPV (when is it indicated)
Monitoring
88
Q

Anaesthesia machine: Mixing part

A
Gas source
Regulator and reductor valves, manometer 
Flow meter: l/min- max is 10l/min
O2 flush valve/bypass
Vaporiser= out of circuit
89
Q

Anaesthesia machine: Breathing circuit

A

Y-shape rebreathing hoses
Rebreathing balloon/bag- needs to be at least 2x thhe TV
CO2 absorber- sodalime or sodium and Calcium hydroxide
Pop-off valve
Central scaveneger apparatus and tube
One- way inhal and exhal valves

90
Q

Anaesthesia machine: Endotracheal tubes

A

Cuffed silicone

Bifurcation at shoulder joint

91
Q

Anaesthesia machine: IPPV and when is it indicated

A
When RR <2-6/min
Large abd volume causes dyspnoea
Open thorax surgery 
Resp acidosis
Hypoxemia nd hypercapnia

It is a pressure limited ventilator- insp pressure should reach 20 H2Ocm at the end of inhalation, at expiration should be >0 H2Ocm
Volume/time limited

To return to spontaneous at the end of IPPV: decr iso and incr O2– and watch capnogram

92
Q

Anaesthesia machine: Monitoring

A
Eye 
Pulse 
MM
Breathing frequency and quality
ECG
RR
Pulsoximetry <90%--- IPPV
Capnography: ETCO2> 45Hgmm-- hypercapnia 
Blood gas analysis- use arterial sample:
pH <7.35-- acidosis (ABE < 2.5)
PaCO2 > 45mmHg-- resp acidosis-- hypercapnia 
HCO3 < 20mmol/l-- metabolic acidosis

If ABP > 70mmHg- give Dobutamine

93
Q

Stages and planes of GA

A
I stadium analgesiae 
II stadium excitationis 
III stadium tolerantiae 
III/1 superf III/2 surgical- this is the one that has to be reached III/3 deep
III/4 stadium paralyticum
94
Q

General anaesthesia: CV system- ECG

A

Rate and rhythm
Leads: Left- caud to olecranon and electrode
Right: caud to olecranon
Neutral: on loose skin on chest

95
Q

General anaesthesia: CV system– heart rate

A

Normal: 35-45/min
brady < 25/min
tachy > 55/min

Frequency largely determines CO

96
Q

General anaesthesia: CV system– causes of bradycardia

A

Dtugs: alpha2, opioids, OD any anaesthetic
Increased vagal tone
Metabolic: hypotherm, end stage hypoxaemia, hyperkalemia
Heart diseases

97
Q

General anaesthesia: CV system– causes of tachycardia

A

Light level of anaesthesia- slight nociception during surgery
Drugs- ketamine
Metabolic- hypovolemia, hypoxemia, hypercapnia
Endocrine- pheochromocytoma, hyperthyreoidosis
Heart diseases

98
Q

General anaesthesia: CV system– Blood pressure

A

Indirect: not useful in horses but maybe in foals, on forelimb and tail
Direct:
Facial, transv facial and metatarsalis

Normotension

systolic: 90-130 mmHg
diastolic: 60-90 mmHg
mean: 70-110 mmHg

Hypotension
When systolic is under 80mmHg and /or mean is under 60 mmHg– compromised cerebral and coronary perfusion

Hypertension
When systolic pressure is above 180mmHg and/or mean pressure above 140 mmHg– edema and bleedings in the brain and lungs

99
Q

General anaesthesia: Resp system– changes

A

Apnea- can even occur during light anaesthesia
Bradypnea– deep anesthesia hypothermia
Tachypnea– occurs during light due to hypoxemia, hypercapnia, atelectasis airway obstruction

100
Q

General anaesthesia: Resp system– pulse oximetry

A

Measures HB oxygen saturation, emits red and UV light
Normal: 98-99%
Hypoxaemia: < 95%
Severe hypoxaemia: < 90%

101
Q

General anaesthesia: Resp system– partial pressure of oxygen in the arterial blood (PaCO2)

A
Determined by blood-gas analysis
Normal: 80-110 mmHg
Hypoxaemia- anything under 80
Severe life-threatening hypoxaemia under 60 mmHg
Hyperoxaemia- above 110mmHg
102
Q

General anaesthesia: Resp system– Capnography

A
Uses infrared light absorption technology- can be side-stream or mainstream
CO2 of exhaled air 
End tidal CO2 partial pressure/conc 
Resp status 
Ventilation 
Perfusion 
Metabolism
103
Q

General anaesthesia: Resp system– Temperature

A

Hypothermia is not a big issue because of the large size, but may be an issue for foals