study material Flashcards

already have this info - theses are just worded differently to keep brain thinking

1
Q
A
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2
Q

What are the stages of tissue healing?

A
  1. Acute stay (3-5 days post onset): Inflammatory response phase
  2. Subacute stage (day 4-21 post onset - 6 weeks): Fibroblastic repair phase

These stages represent the timeline and physiological changes occurring during the healing process.

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3
Q

How do we minimize energy loss through tendons?

A

By utilizing resistance to deformation strain underload (recoil)

This method helps maintain tendon function and efficiency.

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4
Q

What different types of manipulation can be performed to progress exercises?

A
  • Dosage
  • Equipment used
  • Speed
  • Contraction type
  • Patient position

These factors can be adjusted to tailor rehabilitation exercises effectively.

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5
Q

What is the goal of Phase 1 in soft tissue injury management?

A
  • Control inflammation
  • Prevent deleterious effects of rest (deconditioning, loss of strength)

This phase focuses on protecting the injured area while promoting healing.

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6
Q

What precautions should be taken during Phase 1 of soft tissue injury management?

A

Proper dosage of rest and movement

This balance is crucial to facilitate healing without exacerbating the injury.

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7
Q

What are contraindications during Phase 1 of soft tissue injury management?

A
  • Stretching to injured site
  • Resistance exercises to injured site

These actions can worsen the injury and hinder recovery.

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8
Q

How does reactive tendinopathy occur?

A

Due to starting up a new level of activity after relative inactivity or as a result of an acute bout of overload to tendon

Understanding this mechanism helps in preventing tendinopathy.

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9
Q

What changes occur at the cellular matrix and collagen level during reactive tendinopathy?

A

Change only to cellular matrix (potential for reversibility due to intact collagen)

This indicates that early intervention can be effective.

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10
Q

What causes disrepair tendinopathy?

A

Chronically overloaded tendon

This condition reflects ongoing stress and insufficient recovery.

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11
Q

What changes occur at the cellular matrix and collagen level during disrepair tendinopathy?

A

Changes to both cellular matrix and collagen (same as degenerative tendinopathy)

Indicates a progression in the severity of the condition.

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12
Q

What causes degenerative tendinopathy?

A

Chronic overloading of tendon

This condition often results from repetitive stress without adequate recovery.

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13
Q

What occurs at the cellular matrix and collagen level during degenerative tendinopathy?

A
  • Changes to both cellular matrix and collagen
  • Progressive disorganization and breakdown results in lower collagen and low capacity for reversibility

This highlights the long-term impact of tendon overload.

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14
Q

What are the common cellular changes in tendinopathy?

A
  • Rounded fibroblasts unevenly distributed throughout tissue
  • Increased ground substance
  • Capillary ingrowth
  • Disorganized type I and III collagen fibrils

These changes reflect the structural and functional impairment of the tendon.

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15
Q

What management considerations are essential for tendinopathy?

A
  • Does not improve with absolute rest (relative rest is better)
  • Modifying load results in reduced tendon pain
  • Exercise is the most evidence-based treatment

These considerations guide effective rehabilitation strategies.

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